ライフサイエンスコーパス: embryonic death

1 c coronary transmural patterning, leading to embryonic death.

2 ptosis, growth retardation, and, ultimately, embryonic death.

3 that for fecundity reduction, manifested as embryonic death.

4 causes arrest before gastrulation and early embryonic death.

5 entiation of skeletal muscle, culminating in embryonic death.

6 the genome for which PatDp results in early embryonic death.

7 synthetic lethality and, in the mouse, early embryonic death.

8 al in the umbilical artery and ultimately to embryonic death.

9 severe placental and heart defects preceded embryonic death.

10 normal, whereas TFEB deficiency causes early embryonic death.

11 hic placentas shared by multiple embryos and embryonic death.

12 4 expression at the epiblast stage and early embryonic death.

13 rganized, resulting in their stenosis and in embryonic death.

14 ditis causes defective cell arrangements and embryonic death.

15 ansgenes may be particularly likely to cause embryonic death.

16 ligand, complete loss of Ron leads to early embryonic death.

17 used failure of primitive erythropoiesis and embryonic death.

18 p300 and cbp was invariably associated with embryonic death.

19 ent is severely impaired, resulting in early embryonic death.

20 long with intrauterine growth restriction or embryonic death.

21 identifying a recessive mutation underlying embryonic death and a dominant mutation underlying letha

22 Mice with this mutation exhibited increased embryonic death and alpha-thalassemia intermedia.

23 The knock-down of Ce-imp-2 leads to embryonic death and an abnormal molting phenotype in Cae

24 c cells at frequencies high enough to induce embryonic death and cancer in wild-type mice.

25 s1 leads to severe developmental impairment, embryonic death and chromosomal instability.

26 Finally, embryonic death and phenotypes of Adar1(E861A/E861A) wer

27 We show here that Cyr61-null mice suffer embryonic death: approximately 30% succumbed to a failur

28 ns by which this modification leads to early embryonic death are currently unresolved.

29 rap220(-)(/)(-) fibroblasts (isolated before embryonic death) are impaired in specific nuclear recept

30 se severely compromised cardiac function and embryonic death around embryonic day 11.5.

31 tion of beta1 integrin expression results in embryonic death at ca. embryonic day 5 (E5), a developme

32 These defects lead to embryonic death at E14.5 and are similar to those observ

33 erning, severe fetal growth retardation, and embryonic death at E9.5 to 10, although there were no ov

34 anched fetal vessels entering the LA causing embryonic death at embryonic day 11.5.

35 geted deletion of FIP200 in the mouse led to embryonic death at mid/late gestation associated with he

36 Germ line Aurora-A deficiency causes embryonic death at the blastocyst stage with pronounced

37 omplete deletion of EPCR function results in embryonic death, at least in part due to placental throm

38 d deletion of the mouse Dlx3 gene results in embryonic death between day 9.5 and day 10 because of pl

39 in aortic arch artery patterning defects or embryonic death, but does result in ventricular septal d

40 tion results in segment polarity defects and embryonic death, but how nkd affects Wnt signaling is un

41 pes lead to a dramatic development delay and embryonic death by 8 to 9 days of gestation, which are i

42 mutation of the murine Arnt locus results in embryonic death by day 10.5 associated with placental, v

43 widespread developmental defects, leading to embryonic death by day 14.5.

44 ythrocytes were produced in vivo, leading to embryonic death by E13.5.

45 of NFATC1 expression in EPDCs in mice causes embryonic death by E18.5 with reduced coronary vessel an

46 tumour suppressor gene in the mouse leads to embryonic death caused by failure of erythroblasts to en

47 Embryonic death coincided with the period of more widely

48 ernal factors interact to prevent Tgfb1(-/-) embryonic death due to defective yolk sac angiogenesis.

49 corneal implants, and Cyr61-null mice suffer embryonic death due to vascular defects, thus establishi

50 Those succumbing to early embryonic death had markedly deformed vasculature of the

51 ING) is responsible for inflammation-related embryonic death in DNase II defective mice initiated by

52 RagA deficiency leads to E10.5 embryonic death, loss of mTORC1 activity, and severe gro

53 ains the pericardial bleeding and subsequent embryonic death observed in Wt1 null embryos.

54 actor VIII null (F8(-/-)) did not rescue the embryonic death of TFPI null (Tfpi(-/-)) mice.

55 of cellular differentiation in vivo, the mid-embryonic death of these mutants precludes an analysis o

56 erations, and this decrease seems to reflect embryonic death rather than impaired fertilization.

57 aracterized them with respect to the time of embryonic death, revealing that most act at midgestation

58 before the 16-cell stage and causes eventual embryonic death, suggesting that activation of cyclin D/

59 In the mouse, RAD51 deletions cause early embryonic death, suggesting that in higher eukaryotes Ra

60 results in failed lumen formation and early embryonic death through an endothelial cell autonomous m

61 of phenotypes ranging in severity from early embryonic death to viable dysmyelination.

62 (PA14 or PAO1) by microinjection results in embryonic death, unlike infection with Escherichia coli

63 on of the mdm2 gene in mice results in early embryonic death while concomitant mdm2 and p53 deletion