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1 level was determined by identifying the most emetogenic agent in the combination and then assessing t
2 mbination by one level greater than the most emetogenic agent in the combination; and (3) adding leve
6 ind, parallel-group study, patients naive to emetogenic chemotherapy (N = 1,085) who were scheduled t
7 r, who had not received moderately or highly emetogenic chemotherapy before, with a Karnofsky perform
9 omiting associated with moderately or highly emetogenic chemotherapy in adults, but its efficacy and
11 sk period after administration of moderately emetogenic chemotherapy or regimens containing an anthra
12 th cancer after administration of moderately emetogenic chemotherapy or regimens containing an anthra
15 duled to receive either moderately or highly emetogenic chemotherapy were randomly assigned with an i
17 inical trials with patients receiving highly emetogenic chemotherapy, the neurokinin antagonist aprep
18 ents receiving taxanes as part of moderately emetogenic chemotherapy, who received dexamethasone acco
31 (1) level 1 agents do not contribute to the emetogenic level of a combination; (2) adding > or = one
34 s were challenged intraperitoneally with the emetogenic NK agonist GR73632 at different doses, and vo
35 provides a practical means to determine the emetogenic potential of individual chemotherapy agents a
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