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1 pared with another intervention or a vehicle/emollient.
2 ents in the control arm were asked to use no emollients.
3 example, fragrances and other ingredients in emollients.
4 or tacrolimus ointment than for steroids and emollients.
5 without the requirement to constantly apply emollients.
6 ty of various topical agents, and the use of emollients.
10 ally higher in children with frequent use of emollients compared to uncommon users, reaching statisti
11 show that applying a thin layer of glycerine emollient containing nanoparticles of either calcium car
14 stamines, topical corticosteroids, and thick emollient creams, rendering their eruption tolerable for
15 measures such as the application of topical emollients have shown mixed results in the prevention of
17 sise the evidence for topical application of emollients in the prevention of invasive infection and m
19 ffect of prophylactic application of topical emollient (ointments, creams, or oils) on the incidence
20 ctive effect was found with the use of daily emollient on the cumulative incidence of atopic dermatit
21 conceivable to design resveratrol-containing emollient or patch, as well as sunscreen and skin-care p
22 ial data do not provide strong evidence that emollients prevent invasive infection or death in preter
24 cidence of infection in infants treated with emollient (relative risk 1.20, 95% CI 1.01-1.42), but no
25 s explained by increased use of the specific emollients that are used to treat pruritic and inflamed
26 ntion arm were instructed to apply full-body emollient therapy at least once per day starting within
27 The results of this trial demonstrate that emollient therapy from birth represents a feasible, safe
29 to ascertain whether topical application of emollients to enhance skin barrier function would preven
32 a vapor-permeable membrane (Gore-Tex) or an emollient (Vaseline), applied after acute barrier disrup
33 formation about atopic dermatitis and use of emollients was obtained from questionnaires completed by
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