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1 ancomycin for definitive therapy but not for empiric treatment.
2 of guideline adherence on appropriateness of empiric treatment.
3 e often lacking, leading to the necessity of empiric treatment.
4 effectiveness of assay-guided treatment and empiric treatment.
5 signed patients to assay-guided treatment or empiric treatment.
6 o monitor antimicrobial resistance and guide empiric treatment.
7 ive microbiology resulted in less changes of empiric treatment (33 [40%] vs 112 [60%]; p=0.005) and s
8 nosed with a stepwise approach incorporating empiric treatment and antisecretory therapy, combined MI
9 s among all isolates with the following: (1) empiric treatment (B and C), and treatment guided by POC
10 instructed versus induced patients initiated empiric treatment based on clinical and radiography find
11 se) to guide treatment changes compared with empiric treatment changes, and the benefits and risks of
12 tential clinical and public health impact on empiric treatment, contact investigation, and housing in
13 duce the frequency and impact of unnecessary empiric treatment, contact investigation, and housing, p
17 Therapeutic plasma exchange is an effective empiric treatment for thrombotic thrombocytopenic purpur
18 ame-day diagnoses, and-because of widespread empiric treatment-may not result in more patients starti
20 idime-avibactam was highly effective for the empiric treatment of cUTI (including acute pyelonephriti
22 e to beta-lactam agents commonly used in the empiric treatment of febrile neutropenia was observed on
23 f ceftazidime and tobramycin for the initial empiric treatment of hospital-acquired bacterial pneumon
25 lness with no laboratory-confirmed etiology, empiric treatment of iNTS disease is a major challenge i
29 nerally is the recommendation as the initial empiric treatment of suspected bacterial endophthalmitis
30 d change of antibiotic classes, used for the empiric treatment of suspected gram-negative bacterial i
31 neration cephalosporin (ceftazidime) for the empiric treatment of suspected gram-negative bacterial i
32 to adapt therapy in patients unresponsive to empiric treatment options, which occurs in 10% of all ca
33 prophylaxis regimens, programs should devise empiric treatment regimens that are directed against the
36 erratically and, instead, clinicians rely on empiric treatment strategies and ignore public health im
38 h to determine the appropriate threshold for empiric treatment when PE is suspected before diagnostic
39 platelet count remained at 2000/mm3 despite empiric treatment with intravenous immune globulin and m
41 patients with BSI due to ESBL-E who received empiric treatment with OADs or carbapenems was performed
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