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1 trix proteins (amelogenin, ameloblastin, and enamelin).
2 ecies show evidence of positive selection on enamelin.
3 re correlated with the adaptive evolution of enamelin.
4 monstrated ameloblast-specific expression of enamelin.
5 es that encode ameloblastin, amelogenin, and enamelin.
6 on of mRNA encoding a fourth enamel protein: enamelin.
7 bservations strongly suggest that the 32-kDa enamelin and amelogenins cooperate to promote nucleation
9 emonstrate ameloblast-specific expression of enamelin and reveal that enamelin is essential for prope
10 tations to the secreted proteins amelogenin, enamelin, and enamelysin result in visibly, structurally
11 atrix proteins amelogenin, ameloblastin, and enamelin are also expressed during this same approximate
12 rter assays revealed increased expression of enamelin but decreased expression of kallikrein 4 (prote
13 e density was not affected in the absence of enamelin, but its volume was, which is likely a conseque
14 rified antipeptide antibody specific for the enamelin carboxyl terminus demonstrate that enamelin is
15 cloned and characterized a full-length human enamelin cDNA and determined by radiation hybrid mapping
17 Polymerase chain-reaction phenotyping of enamelin cDNA suggests that porcine enamelin transcripts
19 of the developing enamel matrix, while other enamelin cleavage products are observed in deeper enamel
20 on/exon junctions within the mouse and human enamelin coding regions are between codons, so there are
22 e a knock-in mouse carrying a null allele of enamelin (Enam) that has a lacZ reporter gene replacing
23 matrix proteins (EMPs), amelogenin (AMELX), enamelin (ENAM), ameloblastin (AMBN), amelotin (AMTN), t
30 ttern of enamelin expression makes the human enamelin gene a prime candidate in the etiology of amelo
31 To gain information on the structure of the enamelin gene and to facilitate the future assessment of
32 l enamel formation, and defects in the human enamelin gene cause autosomal dominant amelogenesis impe
35 ate the search for specific mutations in the enamelin gene in kindreds suffering from amelogenesis im
37 , addition of 18 and 80 micro g/mL of 32-kDa enamelin in gels containing 1.5% amelogenin accelerated
38 ilitate the future assessment of the role of enamelin in normal and diseased enamel formation, we hav
39 To improve our understanding of the roles of enamelin in normal enamel formation, and to gain informa
43 cific expression of enamelin and reveal that enamelin is essential for proper enamel matrix organizat
44 y 1 mouse developing incisor that shows that enamelin is expressed by ameloblasts, but not by odontob
45 ing the secretory stage of enamel formation, enamelin is found among the crystallites in the rod and
49 enamelin carboxyl terminus demonstrate that enamelin is synthesized and secreted by secretory-phase
53 ing AI in mice carrying a p.S55I mutation in enamelin; one of the most commonly mutated proteins unde
55 evelop a deeper understanding of the role of enamelin protein during formation with regard to crystal
57 The effects of amelogenin and the 32-kDa enamelin proteins on apatite nucleation were investigate
58 Ser(216) is one of two serines on the 32-kDa enamelin that is phosphorylated by Golgi casein kinase a
60 yping of enamelin cDNA suggests that porcine enamelin transcripts are not alternatively spliced and u
61 he gene encoding the enamel-specific protein enamelin underlies the phenotype observed in this family
62 oblast markers amelogenin, ameloblastin, and enamelin was down-regulated, as was expression of Msx2 a
63 We present analyses of one such candidate, enamelin, whose protein product operates in tooth enamel
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