ライフサイエンスコーパス: encephalitis

1 effect for the development of MAV-1-induced encephalitis.

2 patients admitted to an ICU with anti-NMDAR encephalitis.

3 cally suspected antibody-mediated autoimmune encephalitis.

4 role of IL-1 signaling during MAV-1-induced encephalitis.

5 ected in brain tissue of five cases of human encephalitis.

6 h anti-N-methyl-D-aspartate receptor (NMDAR) encephalitis.

7 permeability, and neuroimaging damage during encephalitis.

8 rove aetiological diagnosis in children with encephalitis.

9 nst neuronal antigens, leading to autoimmune encephalitis.

10 and increases susceptibility to lethal viral encephalitis.

11 otizing stromal keratitis and herpes simplex encephalitis.

12 on when animals are no longer suffering from encephalitis.

13 an also cause blindness and life-threatening encephalitis.

14 ctivated Bak is bound to p53 during reovirus encephalitis.

15 similar to those for human survivors of NiV encephalitis.

16 ith the virus spreading to the brain causing encephalitis.

17 rmed in brain samples from patients with HIV encephalitis.

18 nosed as stroke or as noninfectious forms of encephalitis.

19 ed and SIV-infected macaques with or without encephalitis.

20 lex virus 1 (HSV-1) infections, usually from encephalitis.

21 d be investigated as adjunctive treatment in encephalitis.

22 ithout classic presentation of toxoplasmosis encephalitis.

23 of the leading etiologies of sporadic viral encephalitis.

24 serious human diseases such as arthritis and encephalitis.

25 iscuss the concept of a specific Ebola virus encephalitis.

26 increased in infected brains during reovirus encephalitis.

27 cits are important long-term sequelae of the encephalitis.

28 nia (NMT), Morvan syndrome (MoS), and limbic encephalitis.

29 the host cellular factors leading to lethal encephalitis.

30 se of a 62-year-old female with seizures and encephalitis.

31 itions, including corneal blindness or fatal encephalitis.

32 distinguishing HSE from non-HSE causes of TL encephalitis.

33 el system to study the pathogenesis of virus encephalitis.

34 a superimposed antibody-mediated autoimmune encephalitis.

35 es upon induction of experimental autoimmune encephalitis.

36 ase, neonatal infection, and, in rare cases, encephalitis.

37 en with signs and symptoms of meningitis and encephalitis.

38 dies is the leading cause of immune-mediated encephalitis.

39 itted flavivirus that can cause debilitating encephalitis.

40 everity from periodic "cold sores" to lethal encephalitis.

41 most prevalent astrovirus in cases of human encephalitis.

42 proven to have antibody-positive autoimmune encephalitis.

43 most universally fatal granulomatous amoebic encephalitis.

44 (MRI) alterations in patients with anti-LGI1 encephalitis.

45 V infection (6%) died (1 with GBS and 1 with encephalitis), 18 (51%) had chronic pain, and the median

46 (1 patient), and a severe form of fulminant encephalitis (2 patients).

47 axonal, and 1 Miller Fisher syndrome), 5 had encephalitis (3 with concomitant acute neuromuscular dis

48 0% Brighton level 1 certainty), 7 (18%) with encephalitis, 3 (8%) with transverse myelitis, and 1 (3%

49 The most common clinical syndromes were encephalitis (52%), acute disseminated encephalomyelitis

50 itis (4) , meningoencephalitis (5) and fatal encephalitis (6) .

51 coids in attenuating experimental autoimmune encephalitis, a model of multiple sclerosis.

52 The virus causes tick-borne encephalitis, a severe infection of the CNS with a high

53 uninfected and SIV-infected animals without encephalitis, a trend that was also confirmed in brain s

54 Serum samples were tested for autoimmune encephalitis Abs as well as thyroperoxidase (TPO) and gl

55 (FluCAN) study and the Australian Childhood Encephalitis (ACE) study between 2013 and 2015.

56 (FluCAN) study and the Australian Childhood Encephalitis (ACE) study between 2013 and 2015.

57 The numbers of encephalitis admissions without a specific diagnosis are

58 Forty patients with anti-NMDAR encephalitis after the acute disease stage and 25 health

59 lular infiltrate found in the CNS during WNV encephalitis, although the molecular cues involved in th

60 ith symptoms suggestive of autoimmune limbic encephalitis, although they can be paucisymptomatic or m

61 mice suffering from experimental autoimmune encephalitis ameliorates the severity of the clinical sy

62 Data on 30 patients with anti-LGI1 encephalitis and 27 healthy control individuals matched

63 and genital sores as well as rare yet severe encephalitis and a panoply of ocular ailments.

64 Balamuthia infection in donors with possible encephalitis and also assess donors carefully for signs

65 tribute to tissue-specific diseases, such as encephalitis and dementia in brain and pneumonia in lung

66 n the progression and outcome of VSV-induced encephalitis and demonstrated a significant decrease in

67 ney-pancreas recipients developed Balamuthia encephalitis and died.

68 are associated with immunotherapy-responsive encephalitis and epilepsies.

69 ll populations from two patients with limbic encephalitis and faciobrachial dystonic seizures associa

70 morphologically distinct from prototypic SIV encephalitis and human immunodeficiency virus encephalit

71 severe human neurological diseases including encephalitis and meningitis.

72 hese include the relevance of herpes simplex encephalitis and of epilepsy to AD, the action of IFN, a

73 rome-based diagnostic approach to autoimmune encephalitis and providing guidelines to navigate throug

74 an emergent paramyxovirus that causes deadly encephalitis and respiratory infections in humans.

75 ntly emerged as a causative agent of febrile encephalitis and severe respiratory disease in humans.

76 The findings highlight the complex nature of encephalitis and suggests that IL-1 has a protective eff

77 of TLR3 mutations to varicella-zoster virus encephalitis and support the role of TLR3 genetic defect

78 clinical picture in keeping with autoimmune encephalitis and very high VGKC-complex/LGI1 antibodies.

79 to-borne flavivirus that causes epidemics of encephalitis and viscerotropic disease worldwide.

80 orms" in ruminants and by hemorrhagic fever, encephalitis, and blindness in humans.

81 as those that cause Zika, dengue, West Nile encephalitis, and chikungunya, have become endemic or ha

82 reduced severity of experimental autoimmune encephalitis, and defective immune responses to lymphocy

83 ighly debilitating and lethal herpes simplex encephalitis, and generalized infections that can lead t

84 to more severe symptoms, such as hepatitis, encephalitis, and hemorrhagic fever.

85 transmission and disease, such as keratitis, encephalitis, and neurodegeneration, have been linked to

86 to develop pulmonary hypertension, seizures, encephalitis, and pneumonia.

87 ith Japanese encephalitis, Venezuelan equine encephalitis, and Rift Valley fever viruses; and urbaniz

88 antibodies described in patients with limbic encephalitis, and the subsequent seminal paper describin

89 e mice is a tractable small-animal model for encephalitis, and the virus causes disruption of the BBB

90 revents the pathogenic effects of anti-NMDAR encephalitis antibodies on memory and behavior, levels o

91 s and treatment of infectious meningitis and encephalitis are critical to minimize morbidity and mort

92 Meningitis and encephalitis are potentially life-threatening diseases w

93 However, existing criteria for autoimmune encephalitis are too reliant on antibody testing and res

94 ive adult patients diagnosed with anti-NMDAR encephalitis at the French National Reference Centre, ad

95 Bickerstaff's brainstem encephalitis (BBE) is a very rare disease of the central

96 consecutively diagnosed as having anti-NMDAR encephalitis between May 1, 2008, and January 31, 2013.

97 lt patients (>/=18 years) with meningitis or encephalitis by International Classification of Diseases

98 re is a risk of the appearance of LCMV acute encephalitis cases.

99 RATIONALE: Encephalitis caused by anti-N-methyl-d-aspartate recepto

100 in preventing neuroinvasion and the ensuing encephalitis caused by OROV and LACV.

101 c anaplasmosis, an emerging disease, and the encephalitis-causing Langat virus, and a population stru

102 al analysis revealed signs of meningitis and encephalitis, characteristic of severe human disease.

103 n the CNS and decreased survival during LGTV encephalitis compared with wild-type controls.

104 The donor had Balamuthia encephalitis confirmed on autopsy.

105 from 0.32 to 0.009) and admissions for mumps encephalitis decreased by 98% (from 0.60 to 0.01) after

106 t of 11 adult Italian patients in whom viral encephalitis developed.

107 good model for studying factors involved in encephalitis development.

108 iological information for patients with POWV encephalitis diagnosed at 2 hospitals in Massachusetts f

109 neurosciences center, studied patients with encephalitis diagnosed by brain biopsy from January 1, 1

110 ENOS is the most common initial type of encephalitis diagnosed by brain biopsy.

111 Eastern equine encephalitis diagnostic serum antibody can appear 6 days

112 ts, including one death (treatment-unrelated encephalitis due to influenza B infection), one life-thr

113 One patient died from influenza B encephalitis during an endemic outbreak 10 months after

114 The fourth had viral encephalitis during infancy.

115 In experimental autoimmune encephalitis (EAE), autoimmune T cells are activated in

116 nuates Th17-mediated experimental autoimmune encephalitis (EAE).

117 we show that during La Crosse Virus-induced encephalitis, egress of iMOs was surprisingly independen

118 in children, including influenza-associated encephalitis/encephalopathy (IAE).

119 in children, including influenza associated encephalitis/encephalopathy (IAE).

120 (CASPR2), are found in patients with limbic encephalitis, faciobrachial dystonic seizures, Morvan's

121 We report a fatal case of DTV encephalitis following a documented bite from an Ixodes

122 nalysis of hospital admission statistics for encephalitis for individuals aged 0-19 years using natio

123 mmon presenting feature in antibody-mediated encephalitis, for which prompt recognition and treatment

124 We present 8 cases of POWV encephalitis from Massachusetts and New Hampshire in 201

125 Patients with anti-LGI1 encephalitis had incomplete recovery with significant an

126 Although herpes simplex encephalitis has been extensively studied, HSV-1 general

127 Admissions for measles and mumps encephalitis have decreased substantially.

128 t common causes of infectious meningitis and encephalitis have the potential for high clinical impact

129 We found 16 571 encephalitis hospital admissions in the period 1979-2011

130 y of acyclovir (ACV) therapy, herpes simplex encephalitis (HSE) continues to cause substantial morbid

131 Herpes simplex encephalitis (HSE) is the most common form of acute vira

132 The herpes simplex encephalitis (HSE) model was used for induction of neuro

133 usceptibility to herpes simplex virus type 1 encephalitis (HSE).

134 to this, there had been only 2 cases of POWV encephalitis identified in Massachusetts.

135 ded encephalomyelitis in 23 (40%), brainstem encephalitis in 20 (35%), encephalitis in 6 (11%), acute

136 13 patients with VGKC antibodies and limbic encephalitis in 2004.

137 3 (40%), brainstem encephalitis in 20 (35%), encephalitis in 6 (11%), acute flaccid paralysis in 4 (7

138 ose an unusual and fatal case of progressive encephalitis in an immunocompromised adult presenting at

139 including one recovered from a case of fatal encephalitis in an immunosuppressed child.

140 irus responsible for causing acute and fatal encephalitis in animal and human hosts.

141 vere hand, foot, and mouth disease and viral encephalitis in children across the Asia-Pacific region,

142 is (JE) virus (JEV) is an important cause of encephalitis in children of South and Southeast Asia.

143 trated pathological findings consistent with encephalitis in control and knockout mice; however, intr

144 s, and current best tests for meningitis and encephalitis in developed countries.

145 ital admission rates for all-cause childhood encephalitis in England are increasing.

146 a on admission rates for all-cause childhood encephalitis in England are scarce.

147 ed to estimate admission rates for childhood encephalitis in England over 33 years (1979-2011), to de

148 (VEEV), which elicits flu-like symptoms and encephalitis in humans, with an estimated 14% of cases r

149 can also cause lethal hemorrhagic fever and encephalitis in humans.

150 tor-activated virus entry.Nipah virus causes encephalitis in humans.

151 se severe and acute respiratory diseases and encephalitis in humans.

152 that can cause fatal respiratory illness or encephalitis in humans.

153 (HSE) is the most common form of acute viral encephalitis in industrialized countries.

154 when injected into the brain leads to lethal encephalitis in Irf2(-/-) mice after peripheral inoculat

155 MAV-1 produces viral encephalitis in its natural host, providing a good model

156 tive-strand RNA virus causes acute and fatal encephalitis in many mammals, including humans.

157 that are capable of causing acute and fatal encephalitis in many mammals, including humans.

158 We describe 8 cases of POWV encephalitis in Massachusetts and New Hampshire in 2013-

159 sed to study the pathogenesis of acute viral encephalitis in mice for many years.

160 euroinvasive properties (SVNI) causes lethal encephalitis in mice, and its replication in cultured ce

161 d the development of experimental autoimmune encephalitis in mice.

162 ge II) of Powassan virus, is a rare cause of encephalitis in North America.

163 WNV) is the most important cause of epidemic encephalitis in North America.

164 arly recognition and treatment of autoimmune encephalitis in patients receiving immune checkpoint blo

165 on between TRIF mutations and herpes simplex encephalitis in patients.

166 nsmitted by insects and cause meningitis and encephalitis in subsets of individuals in the Americas.

167 in turn may contribute to BBB disruption and encephalitis in susceptible mice.IMPORTANCE RNA and DNA

168 e adenovirus type 1 (MAV-1) infection causes encephalitis in susceptible strains of mice and alters t

169 a neurotropic coronavirus that causes acute encephalitis in susceptible strains of mice.

170 osuppressive therapy in suspected autoimmune encephalitis in the setting of immune checkpoint inhibit

171 esting for POWV in patients who present with encephalitis in the spring to fall in New England.

172 s, and outcomes of adults with meningitis or encephalitis in the United States (US) are lacking.

173 are the most common cause of meningitis and encephalitis in the United States and are treated with a

174 most important cause of mosquito-transmitted encephalitis in the United States.

175 virus (POWV) is a rarely diagnosed cause of encephalitis in the United States.

176 (WNV) is an emerging cause of meningitis and encephalitis in the United States.

177 ry of HAstV-VA1/HMO-C-UK1(a) as the cause of encephalitis in this case provides further evidence that

178 ice.IMPORTANCE RNA and DNA viruses can cause encephalitis; in some cases, this is accompanied by MMP-

179 fluid (n = 37) specimens from patients with encephalitis, including 17 with disease due to HSV infec

180 urologic complications associated with viral encephalitis, including seizures and cognitive impairmen

181 0 per month to 5.6 per month, admissions for encephalitis increased from 0.4 per month to 1.4 per mon

182 Encephalitis is a serious neurological disorder, yet dat

183 Encephalitis is a severe inflammatory disorder of the br

184 Anti-N-methyl D-aspartate receptor (NMDAR) encephalitis is a severe neuropsychiatric disorder that

185 Japanese encephalitis is an acute zoonotic, mosquito-borne diseas

186 Leucine-rich glioma-inactivated1 (LGI1) encephalitis is an antibody-associated inflammation of t

187 Anti-LGI1 encephalitis is associated with cognitive deficits and d

188 Anti-NMDAR encephalitis is associated with extensive superficial wh

189 Japanese encephalitis is characterized by extensive inflammation

190 though the role of acyclovir in treating HSV encephalitis is clear, the role of antiviral therapy in

191 The structural and molecular basis of this encephalitis is not fully understood.

192 Encephalitis is parenchymal brain inflammation, commonly

193 The diagnosis of encephalitis is particularly challenging in immunocompro

194 ugh several different flaviviruses may cause encephalitis, Japanese encephalitis virus is the most si

195 The live-attenuated Japanese encephalitis (JE) vaccine (JE-CV; IMOJEV) induces a prot

196 Japanese encephalitis (JE) virus (JEV) is an important cause of e

197 fluenza viruses, as well as agents of lethal encephalitis like Nipah virus.

198 tion of CCL2 expression during viral-induced encephalitis may have a protective effect.

199 The FilmArray Meningitis/Encephalitis (ME) Panel is a multiplexed in vitro diagno

200 neurotropic ssRNA flavivirus that can cause encephalitis, meningitis, and death in humans and mice.

201 appears broader as cases of encephalopathy, encephalitis, meningitis, myelitis, and seizures have al

202 the vesicular stomatitis virus (VSV)-induced encephalitis model, the replication, caudal penetration,

203 n = 1,433) meningitis or with herpes simplex encephalitis (n = 115), who were alive 1 year after diag

204 studies, none of the deferred cases had HSV encephalitis (n = 3120).

205 are generally referred to pathologically as encephalitis, not otherwise specified (ENOS).

206 Selectivity was validated using Japanese Encephalitis NS1, a homologous and potentially cross-rea

207 ed whether IL-1 signaling contributes to the encephalitis observed in mouse adenovirus type 1 (MAV-1)

208 Hospital admission rates for encephalitis of unknown aetiology have increased by 37%

209 Four main modes of encephalitis onset were observed, including confusion (3

210 0 +/- 2.55 years, median 4 years post-limbic encephalitis onset; n = 18 controls].

211 al fluid (CSF) from patients with anti-NMDAR encephalitis or controls, with or without ephrin-B2, wer

212 by a febrile disease that sometimes leads to encephalitis or hemorrhagic fever.

213 N-methyl-d-aspartate-encephalitis or inborn errors of metabolism may present

214 t's immune status and site of reactivation - encephalitis or myositis can develop.

215 abortion in ruminants and hemorrhagic fever, encephalitis, or blindness in humans.

216 d 1 in a patient with varicella-zoster virus encephalitis (p.Leu199Phe).

217 the performance of the FilmArray meningitis/encephalitis panel compared to conventional methods.

218 The investigation of encephalitis pathogenesis produced by different viruses

219 Data from this largest cohort of anti-NMDAR encephalitis patients that underwent extensive multimoda

220 is crucial to the survival of herpes simplex encephalitis patients; however, many survivors suffer fr

221 diagnosis, levels of evidence for autoimmune encephalitis (possible, probable, or definite) are achie

222 Diagnosis of autoimmune encephalitis presents some challenges in the clinical se

223 oint inhibition may contribute to autoimmune encephalitis require further study.

224 prognosis of adult patients with anti-NMDAR encephalitis requiring intensive care is good, especiall

225 Advances in autoimmune encephalitis research in the past 10 years have led to t

226 measles and rubella, yellow fever, Japanese encephalitis, rotavirus, and invasive bacterial diseases

227 hat antibodies from patients with anti-AMPAR encephalitis selectively eliminate surface and synaptic

228 se in CD68+Ki-67+ cells in macaques with SIV encephalitis (SIVE) compared to uninfected and SIV-infec

229 mation and in the perivascular space and SIV encephalitis (SIVE) lesions late.

230 to simian AIDS and potential SIV-associated encephalitis (SIVE).

231 h anti-N-methyl-D-aspartate receptor (NMDAR) encephalitis suffer from persistent memory impairment de

232 s are not readily regenerated, recovery from encephalitis suggests that mature neurons utilize unique

233 Tick-borne encephalitis (TBE) is caused by a virus that belongs to

234 n has been previously reported in Toxoplasma encephalitis (TE)-susceptible model, our current work de

235 ecipients from donors with signs of possible encephalitis to facilitate early diagnosis and targeted

236 ody titres from a prior inactivated Japanese encephalitis vaccination enhanced yellow fever (YF) immu

237 s of CpG oligodeoxynucleotides or tick-borne encephalitis vaccine, which occurred in an S1PR4-depende

238 domesticated animals, as seen with Japanese encephalitis, Venezuelan equine encephalitis, and Rift V

239 Eastern equine encephalitis virus (EEEV) is a representative member of

240 thogenic phenotype.IMPORTANCE Eastern equine encephalitis virus (EEEV) is one of the most pathogenic

241 ne encephalitis virus (VEEV), eastern equine encephalitis virus (EEEV), and western equine encephalit

242 encephalitis virus (VEEV) and Eastern equine encephalitis virus (EEEV), which have demonstrated poten

243 presumptive serodiagnosis of acute Japanese encephalitis virus (JEV) and West Nile virus (WNV) infec

244 Using Japanese encephalitis virus (JEV) as a model, we performed a syst

245 ns against neurotropic flaviviruses.Japanese encephalitis virus (JEV) is a Flavivirus responsible for

246 and efficacy of the live-attenuated Japanese encephalitis virus (JEV) SA14-14-2 vaccine are attribute

247 g EIIIs from Koutango virus (KOUV), Japanese encephalitis virus (JEV), St. Louis encephalitis virus (

248 three flaviviruses, DENV, WNV, and Japanese encephalitis virus (JEV), using a high-content immunoflu

249 c, mosquito-borne disease caused by Japanese encephalitis virus (JEV).

250 Japanese encephalitis virus (JEV), St. Louis encephalitis virus (SLEV), and Bagaza virus (BAGV) were

251 Tick-borne encephalitis virus (TBEV) is a flavivirus that is transf

252 Tick-borne encephalitis virus (TBEV) is a vector-transmitted flaviv

253 encephalitic alphaviruses Venezuelan equine encephalitis virus (VEEV) and Eastern equine encephaliti

254 Venezuelan equine encephalitis virus (VEEV) is a mosquito-borne RNA virus

255 Venezuelan equine encephalitis virus (VEEV) is a previously weaponized art

256 Venezuelan equine encephalitis virus (VEEV) is a select agent that has bee

257 Venezuelan equine encephalitis virus (VEEV) is an alphavirus that is preva

258 nfectious titer of WNV and Venezuelan equine encephalitis virus (VEEV) TC83 in the brains of Asyn-kno

259 ve the assembly of vRCs of Venezuelan equine encephalitis virus (VEEV), and G3BPs were shown to funct

260 Valley fever virus (RVFV), Venezuelan equine encephalitis virus (VEEV), and herpes simplex virus 1 (H

261 The alphaviruses Venezuelan equine encephalitis virus (VEEV), eastern equine encephalitis v

262 currently unavailable for Venezuelan equine encephalitis virus (VEEV), which elicits flu-like sympto

263 ate that Tc bovine-derived Venezuelan equine encephalitis virus (VEEV)-specific TcPAbs are highly eff

264 ncephalitis virus (EEEV), and western equine encephalitis virus (WEEV) are arthropod-borne positive-s

265 phalitic arboviruses, such as eastern equine encephalitis virus and West Nile virus, underscore the n

266 tron microscopy structure of mature Japanese encephalitis virus at near-atomic resolution, which reve

267 mino-terminal subdomain of Venezuelan equine encephalitis virus capsid protein, SD1, plays a critical

268 sed plasmid VRC5288 (Zika virus and Japanese encephalitis virus chimera), and the VRC 320, done in on

269 an enzootic member of the Venezuelan Equine Encephalitis Virus complex and belongs to the New World

270 ovirulence and neuroinvasiveness of Japanese encephalitis virus in mice.

271 laviviruses may cause encephalitis, Japanese encephalitis virus is the most significant, being respon

272 with the corresponding region from Japanese encephalitis virus NS1 to create chimeric DJ NS1 protein

273 presence of dengue virus (DENV) and Japanese encephalitis virus NS1s in the blood of infected interfe

274 -vectored vaccine (Kp47/47-Venezuelan equine encephalitis virus replicon particle) for safety, immuno

275 avirus 229E), Togaviridae (Venezuelan equine encephalitis virus), and Hepeviridae (HEV), indicating t

276 other NT human arbovirus (Venezuelan equine encephalitis virus), which is also poorly understood.

277 flavivirus family: West Nile virus, Japanese encephalitis virus, and dengue virus 2.

278 alphavirus infection with Venezuelan equine encephalitis virus, and this was associated with greater

279 chikungunya, dengue, yellow fever, Japanese encephalitis virus, GBS, and control datasets.

280 de viral (for example, HIV, rabies, Japanese encephalitis virus, herpes simplex virus, varicella zost

281 of neurovirulence and stability in Japanese encephalitis virus, opening up new avenues for therapeut

282 bunyaviruses La Crosse virus and California encephalitis virus, suggesting a conserved role for Wnt

283 susceptible virus, the flavivirus tick-borne encephalitis virus.

284 s to the NS5 protein of the related Japanese encephalitis virus.

285 Venezuelan and western equine encephalitis viruses (VEEV and WEEV; Alphavirus; Togavir

286 uses include dengue, West Nile, and Japanese encephalitis viruses, and the nonpathogenic flaviviruses

287 n with neuroinvasive West Nile and La Crosse encephalitis viruses.

288 cluding yellow fever, West Nile and Japanese encephalitis viruses.

289 Using a mouse model of West Nile virus (WNV) encephalitis, we show that RIPK3 restricts WNV pathogene

290 ncephalitis and human immunodeficiency virus encephalitis were identified.

291 A total of 26429 patients with meningitis or encephalitis were identified.

292 Forty-six patients with anti-NMDAR encephalitis were included.

293 d LGI1 VGKC-complex antibody-mediated limbic encephalitis were investigated using in vivo ultra-high

294 Symptoms of limbic encephalitis were observed in all patients, including te

295 of the most frequent variants of autoimmune encephalitis with antibodies targeting neuronal surface

296 nnel (VGKC) complex antibody-mediated limbic encephalitis with generalized hippocampal atrophy.

297 should be aware of Balamuthia as a cause of encephalitis with high rate of fatality, and should noti

298 Limbic encephalitis with leucine-rich, glioma-inactivated 1 (LG

299 are associated with a subtype of autoimmune encephalitis with prominent limbic involvement and seizu

300 egated squirrels (Sciurus variegatoides) had encephalitis with similar clinical signs and died 2 to 4