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1 total glucose, generating UDP-GlcNAc as the end product.
2 alase system, which produces d-lactate as an end product.
3 ribute to the synthesis of at least this one end product.
4 and 10,11-dihydroxy carbamazepine as a dead-end product.
5 also limited the sensorial complexity of the end product.
6 get, the {Co(NO)2}(10) dinitrosyl (3) is the end product.
7 ather than promote the formation of the dead-end product.
8 oholic fermentation with ethanol as the main end product.
9 d its chemical activation as a useful carbon end product.
10 t this is a true intermediate and not a dead-end product.
11 oduction of simple nitriles as the bioactive end product.
12 l expertise for performance and detection of end product.
13 cessing and reduces the quality of the sugar end-product.
14 ygen by producing ethanol as their metabolic end-product.
15 iochemical pathway without alteration of the end products.
16 ssion of the receptor for advanced glycation end products.
17 reducing the formation of advanced glycation end products.
18 clusters to any of 1172 clusters with known end products.
19 s (HAAs) as major reaction intermediates and end products.
20 fications that are called advanced glycation end products.
21 fied as both intermediates and apparent dead-end products.
22 genes and subsequent accumulation of pathway end products.
23 , and rerouting carbon flux toward desirable end products.
24 TLR4 or the receptor for advanced glycation end products.
25 4 and/or the receptor for advanced glycation end products.
26 ructurally and morphologically heterogeneous end products.
27 umulation of irreversible advanced glycation end products.
28 sform specific central metabolites into many end products.
29 ith an aerobic on site treatment to harmless end products.
30 ) enzymes convert proproteins into bioactive end products.
31 ough the production of different biochemical end products.
32 long-chain base precursors into sphingolipid end-products.
33 evels of the receptor for advanced glycation end-products.
34 the quantities contained in intermediate and end-products.
35 challenges and assess the quality of traded end-products.
36 noic acid, results in the accumulation of an end product, 2-(omega-carboxyethyl)pyrrole (CEP), which
37 1]), soluble receptor for advanced glycation end products (3.5 [1.7-7.2]), and von Willebrand Factor
38 d thiols can undergo isomerization to a dead-end product (a 4-methylcoumarin-3-yl thioether) upon pho
39 nsferases to gain insight into the extent of end product accumulation and possible feedback inhibitio
40 he dinitrosyl complex is converted to a dead-end product after the dissociation of the proximal histi
42 preferentially recognize advanced glycation end product (AGE)-modified proteins, upregulate expressi
45 o determine the effect of advanced glycation end products (AGE) in the presence and absence of Porphy
46 howed reduced fluorescence advanced glycated end-products (AGE) and a reduced amount of alpha-dicarbo
51 digestibility, release of advanced glycation end products (AGEs) and antioxidant capacity of puffed c
52 osis include formation of advanced glycation end products (AGEs) and reactive oxygen species (ROS), b
53 increased accumulation of advanced glycation end products (AGEs) and receptor for AGE (RAGE) expressi
56 on cascade, the so-called advanced glycation end products (AGEs) are formed, including proteins with
60 predominant precursor for advanced glycation end products (AGEs) due to its protein glycation reactio
62 fication by oxidation and advanced glycation end products (AGEs) had been shown to give rise to color
64 l data suggest a role for advanced glycation end products (AGEs) in cardiovascular disease (CVD), par
66 F) noninvasively measures advanced glycation end products (AGEs) in the skin and is a risk indicator
68 nsumption of diets low in advanced glycation end products (AGEs) on cardiometabolic parameters are co
70 s in the oxidative stress-advanced glycation end products (AGEs) receptor for AGEs (RAGE) pathway, an
71 alpha-dicarbonyls) yield advanced glycation end products (AGEs) that can alter the structures and fu
73 examined associations of advanced glycation end products (AGEs) with renal function loss (RFL) and i
75 n results in formation of advanced glycation end products (AGEs), also originating from alpha-dicarbo
76 -derived macrophages with advanced glycation end products (AGEs), which accumulate in diabetic nerve
86 For investigating the advanced glycation end-product-albumin (AGE-albumin) from activated macroph
87 cted between days 16 and 21 for fermentative end-product analysis and 16S ribosomal RNA bacterial gen
88 ssion of the receptor for advanced glycation end products and could be reversed by inhibiting p38 MAP
90 e of soluble receptor for advanced glycation end products and receptor for advanced glycation end pro
91 ex) inhibits formation of advanced glycation end products and scavenges reactive oxygen species and t
92 lial injury (receptor for advanced glycation end products and surfactant protein D) and endothelial i
93 ed by plasma receptor for advanced glycation end products and systemic endothelial injury by the urin
95 eventing the formation of advanced glycation end products and therefore potentially mitigate the diab
96 c; only two showed accumulation of anaerobic end products and three exhibited the classical heat shoc
98 ation of the receptor for advanced glycation end-products and its ligand S100A8 in lung samples from
99 hydroxymethylfurfural and advanced glycation end products) and microbiological safety and sensory tes
100 ntent (i.e. less elastin, advanced glycation end-products) and increase in conduit artery diameter ar
103 (EcoRV and HincII) that also generate blunt-ended products, and to a structurally distinct enzyme (t
105 y, as secondary metabolites and fermentation end products are absorbed either by the epithelial layer
108 tion of terpenoid phytoalexin precursors and end products as potential signals mediating plant physio
109 multiligand receptor for advanced glycation end products as receptors during MRP8-mediated effects o
110 Ib and RAGE (receptor for advanced glycation end products) as model type I receptor systems, we demon
112 ite synthesis has a remarkable effect on the end product, because PbCl2 nanocrystals are present duri
113 y identified receptor for advanced glycation end products-binding protein levels only showed a signif
115 eptor that interacts with advanced glycation end products, but also with C3a, CpG DNA oligonucleotide
116 quire several months to identify a metabolic end product by biosynthetic pathway screens, but this ti
117 lites in blood and is a precursor as well as end product central to numerous important metabolic path
119 osed reaction scheme, and the calculation of end product concentrations are in general good agreement
120 es between subunits, followed by disassembly end-products consisting of highly curved oligomers and 1
124 ate (MEP) pathway of isoprenoid synthesis by end products dimethylallyl diphosphate (DMADP) and isope
127 hesized that receptor for advanced glycation end products expression might be deleterious, whereas so
128 Ph-NH-CN 8), which are therefore the thermal end products for nitrile imines of the types RCNNH and H
130 non-fermentable pentoses inhibit growth and end-product formation during fermentation of cellulose-d
131 in a variety of organic compounds, including end products from the degradation of influent substrates
132 cumulating intermediates but not the pathway end product fumigaclavine C also were less virulent than
133 zymes studied caused significantly different end product functionality, presumably due to the differe
136 ors 2 and 4, receptor for advanced glycation end-products, high-mobility group box 1, uric acid, IL-3
137 hese NRPS and PKS gene clusters have unknown end products highlighting the power of genome mining in
140 ient consumption and production of metabolic end products in ATL as measured by gas chromatography-ti
141 lowing accumulation of aromatic aldehydes as end products in E. coli, the RARE strain expands the cla
142 products and receptor for advanced glycation end products in humans with cardiogenic shock complicati
145 erglycemia and exposure to advanced glycated end products inactivated MFG-E8, recognizing a key mecha
146 ment cellulosic biomass to formate and other end products, including CO2 This organism lacks formate
148 chains of proteins to form Maillard reaction end products, inducing a negative impact on the digestib
149 lIPMS3 (Solyc08g014230) encodes a functional end product inhibition-insensitive version of the commit
150 by consolidating process steps and reducing end-product inhibition of enzymes compared with separate
151 t fibroblasts from affected individuals, and end-product inhibition of PSS1 by phosphatidylserine was
153 t a drug load of 40%, the solid state of the end product is reached further down the barrel when the
157 epressed by inhibitors of advanced glycation end products, L-type calcium channels, protein kinase C,
158 ress, and accumulation of advanced glycation end products, leading to altered bone metabolism, struct
159 nables precise control of pyrimidine pathway end-product levels, even in the presence of dysregulated
160 form of the receptor for advanced glycation end-products levels in the sevoflurane group, compared w
162 cemia causes receptor for advanced glycation end products-mediated epigenetic modification of naive T
163 reas soluble receptor for advanced glycation end products might be beneficial in cardiogenic shock.
165 on of label into metabolic intermediates and end products observed when cells were labelled with [1-(
171 with a model where spontaneous SCEs are the end product of endogenous recombination events and impli
175 nic nitrate was once considered an oxidation end product of nitric oxide metabolism with little biolo
179 ques were used to affirm formaldehyde as the end product of the dicamba monooxygenase-catalyzed O-dem
180 eas the liver and brain content of the major end product of the enzymatic cascade, NAD(+), did not di
181 y involving the direct quantification of the end product of the enzymatic reaction, 1-deoxy-D-xylulos
182 ified as a target of S-glutathionylation, an end product of the nitric-oxide-signaling cascade that i
183 ation to intracellular proteins utilizes the end product of the nutrient sensing hexosamine biosynthe
186 e that these immature particles are not dead-end products of assembly, but progress into mature 30S s
187 ed for capturing methanol or formaldehyde as end products of biochemical reactions or in environmenta
188 few seconds to the common intermediates and end products of both glycolysis and gluconeogenesis unde
193 Polymorphism observed within the aggregation end products of fibrils are known to arise due to micros
194 e, histidine, and methionine; (iv) catabolic end products of lignin (pyruvate and oxaloacetate) must
195 expression of proinflammatory cytokines, and end products of lipid oxidation had a synergistic effect
199 calorimetric studies to demonstrate that the end products of Xyn10A hydrolysis of xylan are recognize
201 ) phase uraninite, or UO2, is the preferable end-product of bioremediation due to its relatively high
211 ation of the stabilities of various proposed end-products of sulfamate-induced sulfatase inactivation
212 htly regulated by feedback inhibition of the end product on the first enzyme of the lysine-specific b
215 t-chain fatty acids (SCFAs) are fermentation end products produced by the intestinal microbiota and h
217 engaging the receptor for advanced glycation end product (RAGE) that may sustain inflammation and tis
218 DNA through receptor for advanced glycation end products (RAGE) and induces production of type I int
219 ytes through receptor for advanced glycation end products (RAGE) and Toll-like receptor 2, leading to
220 tion of both receptor for advanced glycation end products (RAGE) and Toll-like receptor-9 (TLR9).
222 role of the receptor for advanced glycation end products (RAGE) in neuroinflammation, neurodegenerat
224 ction of the receptor for advanced glycation end products (RAGE) in the development of phenotypes ass
232 However, in Receptors for Advanced Glycation End Products (RAGE) knockout mice after postnatal day 3,
235 egulation of receptor for advanced glycation end products (RAGE) messenger RNA, but not toll-like rec
236 act with the receptor for advanced glycation end products (RAGE) on hepatic Kupffer cells, resulting
237 binds to the receptor for advanced glycation end products (RAGE) on Kupffer cells, ultimately leading
239 through the receptor for advanced glycation end products (RAGE) to reverse apoptosis-induced toleran
240 suppress the receptor for advanced glycation end products (RAGE) via nuclear factor erythroid-2-relat
241 ors, such as receptor for advanced glycation end products (RAGE), allows for a complex regulatory net
242 ation of the receptor for advanced glycation end products (RAGE), as deletion of RAGE was able to red
243 antibody to receptor for advanced glycation end products (RAGE), inhibited responses to cell debris
244 (TLR)2, the receptor for advanced glycation end products (RAGE), myeloid differentiation primary res
245 or 4 (TLR4), receptor for advanced glycation end products (RAGE), p-ERK1/2, nuclear NF-kappaB p65, an
247 ckade of the receptor for advanced glycation end products (RAGE)/nuclear factor-kappaB (NF-kappaB) ax
248 gene for the receptor for advanced glycation end-products (RAGE) are associated with an increased inc
250 xpression of receptor for advanced glycation end-products (RAGE) is suggested to play a crucial role
251 how that the receptor for advanced glycation end-products (RAGE) promoted DNA uptake into endosomes a
252 rts that the receptor for advanced glycation end-products (RAGE) promotes uptake of DNA into endosome
253 LR4) and the receptor for advanced glycation end-products (RAGE) revealed the involvement of alarmins
254 TLRs) and the receptor for advanced glycated end-products (RAGE), are upregulated within inflamed gin
256 through the receptor for advanced glycation end-products (RAGE; ie, its receptor), are involved in f
257 ed receptor (receptor for advanced glycation end products [RAGE]) has no significant effect on inflam
258 s [TLRs] and receptor for advanced glycation end products [RAGE]) present on vascular and innate immu
260 idely depending, among other factors, on the end product's location in the pathway, which determines
262 heir receptors, including advanced glycation end product-specific receptor (RAGE), trigger various in
265 tive soluble receptor for advanced glycation end-product (sRAGE) levels, a marker of type I alveolar
266 form of the receptor for advanced glycation end-products (sRAGE) are elevated during acute respirato
268 PPARgamma is activated by lipid peroxidation end products, such as 4-hydroxynonenal, whose levels are
269 -lysine, one of the major advanced glycation end products, suggesting the prominent role of carbamyla
270 observed in receptor for advanced glycation end products, surfactant protein D, angiopoietin-2, inte
271 xpression of receptor for advanced glycation end products, surfactant protein-B, type III procollagen
272 crobial fermentation and, in turn, result in end products that may be used by the host (eg, short-cha
273 oreover, the receptor for advanced glycation end products that recognizes MG and GO adducts and glyox
275 d as shelf-stable precursors to a variety of end products through simple functional group transformat
278 he increased formation of advanced glycation end products under certain pathological conditions.
280 F-alpha, the receptor for advanced glycation end products, vascular endothelial growth factor, perios
281 athways converge to the desired biosynthetic end product via the (SAM-dependent) retro-Claisen rearra
285 exquisite biological sensor, the fibrin clot end-product was replaced with a synthetic material under
287 Under humid conditions (50% RH), the major end products were 4-oxopentanal, 4-oxobutanoic acid, and
288 n m/z 300 and 350, the major condensed-phase end products were levulinic acid (LLA) and succinic acid
289 duct soluble receptor for advanced glycation end products were significantly attenuated in fat-fed mi
290 on as well as growth on the surface, but the end products were variable proportions of nantokite, cup
293 lites, the precursors for advanced glycation end products, were significantly elevated in plasma and
294 n of (13) C into metabolic intermediates and end products when cells were labelled with [1-(13) C], [
295 a, TLR4, and receptor for advanced glycation end products, whereas mRNA levels of anti-inflammatory T
296 anipulation of autologous cells, ideally the end product will be the equivalent of an autograft such
299 tools towards the production of high quality end-product with increased bioactive properties without
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