ライフサイエンスコーパス: end systole

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1 erformed separately at both end diastole and end systole.

2 as the dicrotic notch and retrograde flow at end systole.

3 ains occurring from control to ring state at end-systole.

4 postoperative MR was LV sphericity index at end-systole.

5 nulus and leaflets were computed at mid- and end-systole.

6 astole, isovolumic systole, peak-systole and end-systole.

7 re calculated from 3-D marker coordinates at end-systole.

8 LV systole with minimum MA area occurring at end-systole.

9 trol, Duran, and Physio; ANOVA=0.005) and at end systole (14.5+/-6.2, 10.5+/-5.5, and 5.8+/-2.5 mm, r

10 .5 +/- 2.7 to 13.7 +/- 2.4 mm, p = 0.03) and end systole (16.1 +/- 2.9 to 18.5 +/- 1.8 mm, p = 0.03),

11 er at the endocardium than the epicardium at end systole (24+/-5% versus 16+/-3%; P<0.05, n=8), consi

12 se-apex length increased at end-diastole and end-systole (all +1 mm, P<0.05).

13 ains occurring from control to ring state at end-systole along the annulus were calculated.

14 the origins of the mitral valve leaflets at end systole and end diastole.

15 1.5 T, with images aquired simultaneously at end systole and middiastole.

16 periods of cardiac motion identified during end systole and middiastole.

17 reases in LV volumes and sphericity index at end-systole and end-diastole.

18 Volume and mass were calculated at end-systole and end-diastole.

19 tip (10.3 versus 6.4 mm, MR versus no-MR, at end-systole) and increased r of the anterior papillary m

20 MR severity, LV volumes at end-diastole and end-systole, and LA volumes were measured at baseline, d

21 illary blood volume between end diastole and end systole at baseline.

22 y and serial changes in regional geometry at end systole.Beginning as a narrow band of fully perfused

23 inded, independent readers on cine images in end systole by using a freely available software package

24 tance (left ventricular maximum elastance at end systole), cardiac output, circumflex artery blood fl

25 real time (32-64 Hertz) or triggered 1:1 at end systole during a 20% C3 or C3C4 droplet infusion.

26 marker coordinates at end diastole (ED) and end systole (ES) were computed.

27 of the annulus increased only 9.2+/-6.3% at end systole (ES).

28 ted from 3-dimensional marker coordinates at end-systole (ES).

29 ange in the cavity area from end diastole to end systole (fractional area change [FAC]), was related

30 Ejection after end systole has a positive effect on ventricular perform

31 astole and LA volumes, but not LV volumes at end-systole in degenerative MR, is consistent with corre

32 both continuously and intermittently (every end systole) in the fundamental (2 MHz) and harmonic (tr

33 tment, left ventricular maximum elastance at end systole increased and was unchanged in controls (30

34 levated border zone fiber stresses from mean end-systole levels of 28.2 kPa (control) to 23.3 kPa (tr

35 ic contours were automatically propagated to end systole, mean differences were 2.0 g +/- 3.6 (P =.05

36 tion between diameter and volume was good at end-systole (r = 0.91, p < 0.0001) and end-diastole (r =

37 tole and 42+/-17%, 37+/-17%, and 21+/-10% at end systole, respectively, for Control, Duran, and Physi

38 ring acute ischemic mitral regurgitation, at end systole, the anterolateral edge of the central scall

39 d 2.5+/-0.12 mm toward the mitral annulus at end systole; the posterior papillary muscle geometry was

40 PV data measured at normalized time (tN) and end systole (tmax) to predict intercept: Vo(SB) = [EN(tN

41 jected onto the MA plane at end diastole and end systole to assess PM dynamics.

42 nd a correspondence between end diastole and end systole was computed with a novel algorithm.

43 LV volumes at end-systole was significantly reduced in functional MR b

44 ular free wall curvature (C(FW)) measured at end systole were used to derive the curvature ratio (C(I

45 te left ventricular volume reduction (VR) at end systole, with EDV kept constant.

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