ライフサイエンスコーパス: end-diastolic

1 remodeling at 6 months included decreases in end-diastolic (161 +/- 36 ml to 122 +/- 30 ml; p < 0.001

2 modeled with increase in LV left ventricular end-diastolic (175 mL +/- 35 to 201 mL +/- 40) and end-s

3 Left ventricular end-diastolic (2.2 +/- 0.7 versus 1.59 +/- 0.6 mL; P<0.0

4 ic (51 +/- 13 vs 50 +/- 14 mm, p = 0.05) and end-diastolic (55 +/- 13 vs 52 +/- 14 mm, p = 0.003) dim

5 d hearts revealed that all LV parameters (LV end-diastolic and -systolic dimensions, ejection fractio

6 ith PA:A>1 had higher right ventricular (RV) end-diastolic and end-systolic volume indices accompanie

7 LV end-diastolic and end-systolic volumes and diastolic fun

8 The LV end-diastolic and end-systolic volumes and LV ejection f

9 LV end-diastolic and end-systolic volumes decreased as earl

10 In controls, end-diastolic and end-systolic volumes increased and eje

11 LV end-diastolic and end-systolic volumes, ejection fractio

12 Left ventricular end-diastolic and left atrial end-systolic volumes incre

13 ciliary (TPCA) arteries, the systolic (PSV), end-diastolic and mean blood flow velocities as well as

14 Maximum end-diastolic aortic root diameters were measured in the

15 s area (r=0.651; P<0.001), right ventricular end-diastolic area (r=0.347; P=0.003), fractional area c

16 as associated with greater right ventricular end-diastolic area and worse right ventricular fractiona

17 In contrast, median RV end-diastolic area increased (P=0.001 versus baseline).

18 eft atrial area <26 cm(2), right ventricular end-diastolic area index <10.0 cm(2)/m(2), right atrial

19 t atrial volume index, 44 +/- 19 mL/m(2); RV end-diastolic area, 34 +/- 9 cm(2)), and reduced RV func

20 V function was assessed by echocardiographic end-diastolic area, end-systolic area, fractional area c

21 rsus 172+/-28 mL, P<0.001), right ventricle (end-diastolic area=27.0+/-4.8 versus 28.6+/-4.3 cm(2); P

22 with the ratio of the right/left ventricular end-diastolic areas suggesting a decrease of the right v

23 elow tenth percentile and absent or reversed end-diastolic blood flow in the umbilical artery on Dopp

24 .0001]); and a reversal of LV remodeling (LV end diastolic diameter -2.49 mm [95% CI: -4.09 to -0.90;

25 py before implant (OR 2.2), left ventricular end-diastolic diameter <6.5 cm (OR 1.7), pulmonary systo

26 d reverse LV remodeling with reduction of LV end-diastolic diameter (-0.20 +/- 0.4 mm) and volume (-2

27 ARNi versus MI-vehicle demonstrated lower LV end-diastolic diameter (by echocardiography; 9.7+/-0.2 v

28 mary endpoint was change in left ventricular end-diastolic diameter (LVEDD) after 5 years, measured a

29 m (-2.9 to -0.6, p=0.0027), left ventricular end-diastolic diameter -1.3 mm, (-2.3 to 0.3, p=0.0128),

30 ion (LVEF), and remodeling (left ventricular end-diastolic diameter [LVEDD]) at presentation, were as

31 lts for cardiac (2 SNPs for left ventricular end-diastolic diameter and 5 SNPs for aortic root diamet

32 ion were a low LVEF, a high left ventricular end-diastolic diameter and a low heart rate after beta-b

33 ween HIIT and MCT (P=0.45); left ventricular end-diastolic diameter changes compared with RRE were -2

34 Change in left ventricular end-diastolic diameter from baseline to 12 weeks was not

35 oup comparison of change in left ventricular end-diastolic diameter from baseline to 12 weeks.

36 tion (beta=0.42, P<0.0001), left ventricular end-diastolic diameter index (beta=-0.22, P=0.002), and

37 atrial diameter, increased left ventricular end-diastolic diameter, additional linear ablation lesio

38 l diastolic thickness, left ventricular (LV) end-diastolic diameter, LV posterior wall thickness, LV

39 Women with preeclampsia had smaller mean LV end-diastolic diameters (5.2 versus 6.0 cm; P=0.001), gr

40 Left-ventricular end diastolic dimension was 179+/-65 mL at rest and incr

41 ccompanied by a decrease in left ventricular end-diastolic dimension >/=10% at 12 months of follow-up

42 (LV) function (LV ejection fraction >50%, LV end-diastolic dimension </=70 mm, LV end-systolic dimens

43 =0.005) and a decrease of 0.7+/-0.2 cm in RV end-diastolic dimension (P<0.001) after intervention.

44 ction fraction z-score <-2) and LV dilation (end-diastolic dimension [LVEDD] z-score >2) at diagnosis

45 Baseline LV size was assessed by LV end-diastolic dimension and LV end-systolic dimension (L

46 (n=161; 48%) was associated with pre-CRT LV end-diastolic dimension index <3.1 cm/m(2), global longi

47 n fraction was 0.50+/-0.16, left ventricular end-diastolic dimension was 5.0+/-0.9 cm, and left atria

48 nsistently, the increase in left ventricular end-diastolic dimension was of lesser magnitude (+0.47 v

49 nd regression tree analysis identified an LV end-diastolic dimension z score less than -1.85 or the c

50 less than -1.85 or the combination of an LV end-diastolic dimension z score of -1.85 or higher and a

51 3.98 versus -9.06+/-3.89, P<0.001) and lower end-diastolic dimension z scores (4.12+/-2.61 versus 4.9

52 wall thickness of 22+/-4 mm and nondilated (end-diastolic dimension, 39+/-7 mm).

53 assessed on measurements of left ventricular end-diastolic dimension, area, and volume.

54 ce, heart rate variability, left ventricular end-diastolic dimension, left ventricular ejection fract

55 parameters, including diastolic function, LV end-diastolic dimension, LV mass, and right ventricular

56 riables associated with reduction in MR were end-diastolic dimension, MR severity, clip location, and

57 lculated as the ratio of LV to ring size (LV end-diastolic dimension/ring size and LVESd/ring size).

58 o determine LV end-systolic elastance (Ees), end-diastolic elastance (Eed), and effective arterial el

59 IUGR placentas displaying absent or reversed end-diastolic flow contained reduced myosin heavy chain,

60 Elevated diabetic end-diastolic intensity ratios were correlated with the

61 95 near MTSS1 and rs10774625 in ATXN2 for LV end-diastolic internal dimension; rs806322 near KCNRG, r

62 On the basis of end-diastolic left ventricular pressure, 34 MI rats were

63 that adjusted for age, sex, height, weight, end-diastolic LV volume, augmentation index, end-systoli

64 sensitivity and 94% specificity based on LV end-diastolic M-mode dimension z-score >/=7.7, LV ejecti

65 year; P<0.001), larger left ventricular (LV) end-diastolic M-mode dimension z-score (hazard ratio=1.4

66 volume index (B=0.6; P=0.0001), and lower LV end-diastolic mass index (B=0.4; P=0.0001).

67 reduced LV end-diastolic volume index and LV end-diastolic mass index in a large multiethnic populati

68 Irrespective of breath holding, LV end-diastolic mass was overestimated with SSIR (standard

69 ters (median: 41.2 versus 31.5; P=0.004) and end-diastolic mitral annular diameters (median: 35.5 ver

70 rd cine and CP-BOLD images, end-systolic and end-diastolic myocardium was segmented.

71 Elevated LV end-diastolic or pulmonary capillary wedge pressure, con

72 on LV end-diastolic pressure (LVEDP) or the end-diastolic P-V relationship (EDPVR) in sham rats.

73 4 different ways: the first was 1 gated bin (end-diastolic phase with 25% of the counts), the second

74 ional shortening, or higher left ventricular end-diastolic posterior wall thickness or end-diastolic

75 5 vs. 28+/-10 mmHg, P<0.001), and lowered LV end diastolic pressure (10+/-1 vs. 86+/-13 mmHg, P<0.001

76 ased left ventricular developed pressure and end diastolic pressure.

77 onary artery pressure (25+/-10 mm Hg) and LV end-diastolic pressure (11+/-5 mm Hg; P<0.001 for both c

78 ) mass (70 +/- 2 vs. 63 +/- 1%), enhanced LV end-diastolic pressure (14 +/- 2 vs. 8 +/- 1 mmHg) and t

79 (mean pressure, 39+/-12 mm Hg), elevated LV end-diastolic pressure (19+/-5 mm Hg), and reduced strok

80 5; P=0.02) and more likely to have higher RV end-diastolic pressure (HR, 1.07; 95% CI, 1.00-1.15; P=0

81 ine paradoxically decreased left ventricular end-diastolic pressure (LVEDP) and left ventricular end-

82 d diastolic volumes with little effect on LV end-diastolic pressure (LVEDP) or the end-diastolic P-V

83 tion was seen for stiffness with ventricular end-diastolic pressure (P = 0.001) and pulmonary artery

84 roved ejection fraction and left ventricular end-diastolic pressure (P<0.05).

85 ased fractional shortening, and increased LV end-diastolic pressure and fibrosis (P<0.05 versus contr

86 ary cardiospheres decreased left ventricular end-diastolic pressure and increased cardiac output.

87 F as evidenced by increased left ventricular end-diastolic pressure and left ventricular volume index

88 ved between the plasma DPPIV activity and LV end-diastolic pressure and lung congestion.

89 nduced HF, CXL-1020 reduced left ventricular end-diastolic pressure and myocardial oxygen consumption

90 ncluding heart rate, peak-systolic pressure, end-diastolic pressure and volume, end-systolic pressure

91 End-diastolic pressure correlated significantly with tis

92 Although left ventricular end-diastolic pressure decreased in 45/10, it increased

93 fluid protocol based on the left ventricular end-diastolic pressure for the prevention of contrast-in

94 ere further dichotomized by left ventricular end-diastolic pressure into postcapillary (left ventricu

95 d LV end-diastolic volume at an idealized LV end-diastolic pressure of 20 mm Hg (EDV20), and RV remod

96 l resection again blunted the increase in LV end-diastolic pressure secondary to volume expansion (+4

97 l approach would mitigate the increase in LV end-diastolic pressure that develops during volume loadi

98 ction fraction, end-systolic volume, and the end-diastolic pressure volume relationship by Ang-(1-9)

99 s, pericardiotomy blunted the increase in LV end-diastolic pressure with saline infusion, while enhan

100 (ejection fraction [EF] and left ventricular end-diastolic pressure) was assessed at days 28 and 56.

101 c pulmonary artery pressure-left ventricular end-diastolic pressure) was normal (<7 mm Hg) or elevate

102 fibrosis and left atrium diameter (marker of end-diastolic pressure), suggesting an improvement in di

103 ressure into postcapillary (left ventricular end-diastolic pressure, >15 mm Hg; n=269) and precapilla

104 9) and precapillary groups (left ventricular end-diastolic pressure, </=15 mm Hg; n=56).

105 us saturation, elevated systemic ventricular end-diastolic pressure, and elevated main pulmonary arte

106 orrelated with PH severity, left ventricular end-diastolic pressure, and left ventricular dilatation.

107 h HTN(+)HFpEF had increased left ventricular end-diastolic pressure, left atrial volume, N-terminal p

108 tion fraction) and hemodynamic variables (LV end-diastolic pressure, LV dP/dtmax, preload adjusted ma

109 TN(-)HFpEF had no change in left ventricular end-diastolic pressure, myocardial passive stiffness, co

110 HF-related cardiac dysfunction, including LV end-diastolic pressure, systolic performance, and chambe

111 Left ventricular end-diastolic pressure-guided fluid administration seems

112 equently in patients in the left ventricular end-diastolic pressure-guided group (6.7% [12/178]) than

113 allocated in a 1:1 ratio to left ventricular end-diastolic pressure-guided volume expansion (n=196) o

114 action (p = 0.014) and improvement of the RV end-diastolic pressure-volume relationship in PH pigs tr

115 ced fibrosis, and decreased left ventricular end-diastolic pressure.

116 l, and maximum LV dP/dt, as well as lower LV end-diastolic pressure.

117 ad in Fontan, manifested by high ventricular end-diastolic pressures and pulmonary arterial wedge pre

118 ction, which subsequently led to elevated LV end-diastolic pressures and pulmonary hypertension.

119 n develop increases in left ventricular (LV) end-diastolic pressures during exercise that contribute

120 levant parameters, including RV systolic and end-diastolic pressures, cardiac output, RV size, and mo

121 s, had higher indexed right ventricular (RV) end-diastolic (range 85-326 mL/m(2), mean 148 mL/m(2)) v

122 Both end-systolic and end-diastolic RV volumes increased in SSc-PAH patients t

123 , in the HAART-exposed group, LV mass and LV end-diastolic septal thickness were lower whereas LV con

124 cant differences (P<0.01) in preoperative RV end-diastolic short-/long-axis and long-axis/length-area

125 for postoperative RV failure were found for end-diastolic short-/long-axis ratio >/= 0.6, tricuspid

126 End-diastolic short-/long-axis ratio <0.6, tricuspid ann

127 tio (S/D), quotient of mean end-systolic and end-diastolic signal intensities (on CP-BOLD and standar

128 s caused septal shift as demonstrated by the end-diastolic transseptal pressure gradient (4.1 +/- 3.3

129 ameters, including mean flow velocity (MFV), end diastolic velocity (EDV), peak systolic velocity (PS

130 ar end-diastolic posterior wall thickness or end-diastolic ventricular septal thickness at the time o

131 T intensive versus conventional treatment in end diastolic volume (EDV), end systolic volume, stroke

132 jection fraction (LVEF) and left ventricular end diastolic volume (LVEDV) on cardiac magnetic resonan

133 en by associations with baseline and DeltaLV end diastolic volume (P<0.0001 for each) and not wall th

134 ed as the change in indexed left ventricular end diastolic volume and LVEF.

135 ype and G6PDX mice but significantly greater end diastolic volume and wall thinning in G6PDX mice.

136 Right ventricular end diastolic volume increased by 49.4% after transannul

137 les of trastuzumab, indexed left ventricular end diastolic volume increased in patients treated with

138 nance to assess LV remodeling (LV mass-to-LV end diastolic volume ratio), function, tissue characteri

139 ciations of RV mass and, to a lesser extent, end diastolic volume with PM10-2.5 mass among susceptibl

140 ed percentiles of LV end systolic volume, LV end diastolic volume, relative wall and septal thickness

141 n a 57% increase in LV mass (no change in LV end diastolic volume, resulting in an increase in the LV

142 tion >/=10 U, combined with a decrease in LV end-diastolic volume >/=10% at follow-up.

143 s defined as an increase in left ventricular end-diastolic volume >/=20% at 6 months.

144 tein-losing enteropathy, ventricular indexed end-diastolic volume >125 mL/body surface area raised to

145 were predicted by a preoperative indexed RV end-diastolic volume </=158 mL/m(2) and RVESVi </=82 mL/

146 enhancing the saline-mediated increase in LV end-diastolic volume (+17+/-1 versus +10+/-2 mL; P=0.016

147 , a significant decrease in left ventricular end-diastolic volume (-18 mL; P=0.009) and end-systolic

148 o -3.80; P<0.00001), and tended to reduce LV end-diastolic volume (-2.26 mL; 95% confidence interval,

149 Changes in end-diastolic volume (-6+/-14 versus -4+/-16 mL; P=0.67)

150 /-4 and 65+/-4%; P<0.05), greater indexed LV end-diastolic volume (102+/-34 versus 84+/-14 and 85+/-1

151 grade was mild with significant decreases in end-diastolic volume (139 to 107 mL; P=0.03) and left at

152 A significant reduction of LV end-diastolic volume (149.7 +/- 41.4-140.1 +/- 43.9 mL;

153 e was a larger increase in right ventricular end-diastolic volume (15 mL; 95% confidence interval, 3-

154 ignificant attenuation in the increase in LV end-diastolic volume (64.8 mL [60.7-71.3] to 83.5 mL [74

155 had more than mild FMR despite increased LV end-diastolic volume (82 +/- 22, 86 +/- 23, and 51 +/- 1

156 bcutaneous fat was associated with higher LV end-diastolic volume (beta=0.48), reduced concentricity

157 4.02 to -0.23; P=0.028) and left ventricular end-diastolic volume (coefficient, 7.85; 95% confidence

158 Remodeling was defined as an increase in LV end-diastolic volume (DeltaEDV>0) between discharge and

159 global function was assessed by measuring LV end-diastolic volume (EDV), end-systolic volume (ESV), a

160 analyzed to provide the reference limits for end-diastolic volume (EDV), end-systolic volume (ESV), e

161 End-diastolic volume (EDV), end-systolic volume (ESV), s

162 Filling was assessed by changes in LV end-diastolic volume (EDV; conductance catheter techniqu

163 follow-up documented a stepwise reduction in end-diastolic volume (from 147 mL [IQR, 95-191 mL] to 12

164 g LV strain (HR, 1.63; P=0.005), exercise LV end-diastolic volume (HR, 1.38; P=0.048), and resting RV

165 r end-diastolic volume and right ventricular end-diastolic volume (left ventricular end-diastolic vol

166 CMR showed higher left ventricle end-diastolic volume (mean difference: 43+/-22.5 mL), hi

167 resonance (1.5 T) to measure RV mass (g), RV end-diastolic volume (mL), RV mass/volume ratio, and LV

168 F23 on left ventricular (LV) mass (P=0.016), end-diastolic volume (P=0.029), and end-systolic volumes

169 on, and IMH correlated with the change in LV end-diastolic volume (Pearson's rho of 0.64, 0.59, and 0

170 stolic pressure (LVEDP) and left ventricular end-diastolic volume (preload) in CHF rats, which was no

171 cardial lidocaine paradoxically decreased LV end-diastolic volume (preload) in CHF rats, which was no

172 = 0.95), end-systolic volume (r = 0.93), and end-diastolic volume (r = 0.90), and slightly lower corr

173 d mechanical dyssynchrony correlated with RV end-diastolic volume (r=0.39; P=0.03).

174 ed on LV mass index and the ratio of LV mass:end-diastolic volume (relative wall mass).

175 Right ventricular end-diastolic volume (RVEDV) was reduced in COPD compare

176 compared with patients with GLS</=-15.0% (LV end-diastolic volume 123+/-44 versus 106+/-36 mL and 121

177 assification based on LV dilatation (high LV end-diastolic volume [EDV] index) and concentricity (mas

178 scular events and a dilated LV (increased LV end-diastolic volume [EDV] indexed to body surface area)

179 volume [EDV] index) and concentricity (mass/end-diastolic volume [M/EDV](2/3)) in hypertensive patie

180 increased from pre-Norwood to pre-stage II (end-diastolic volume [milliliters]/body surface area [BS

181 of the right ventricle were used to measure end-diastolic volume and ejection fraction.

182 MRI results showed a decrease of indexed RV end-diastolic volume and end-systolic volume (98 ml/m(2)

183 RV volumes (end-diastolic volume and end-systolic volume), stroke vo

184 of women were classified as having abnormal end-diastolic volume and internal diameter by ASE 2015 c

185 with placebo did not change left ventricular end-diastolic volume and left ventricular mass nor did i

186 iminated the progressive deterioration in LV end-diastolic volume and LV end-systolic volume.

187 Indexed left ventricular end-diastolic volume and mass correlated with O2max (r=0

188 /m(2); P<0.001) and indexed left ventricular end-diastolic volume and right ventricular end-diastolic

189 During PEI, LV end-diastolic volume and stroke volume were increased in

190 ndent relationship between GLS groups and LV end-diastolic volume at 3 and 6 months (adjusted for cli

191 reased LV compliance, measured as reduced LV end-diastolic volume at an idealized LV end-diastolic pr

192 ty indices ([RA+aRV]/[fRV+LA+LV]) and fRV/LV end-diastolic volume corresponded only to some parameter

193 In both men and women, LV end-diastolic volume decreased (-9.8 and -13.3 mL per de

194 Left ventricular end-diastolic volume decreased (from 125.1 +/- 40.1 ml t

195 l adjustment for city attenuated the RV mass/end-diastolic volume findings.

196 ncrease and dilatation, but left ventricular end-diastolic volume improved because of reduced blood r

197 baseline to 12 months, left ventricular (LV) end-diastolic volume improved from 161 +/- 56 ml to 143

198 se; P<0.001), BMI was not correlated with RV end-diastolic volume in men (R=0.04; P=0.51).

199 (n = 167), and after replacing LV mass by LV end-diastolic volume in the regression models.

200 remental value of LVGLS for prediction of LV end-diastolic volume increase (0.14 [95% confidence inte

201 After intervention, LV mass and end-diastolic volume increased and exercise capacity imp

202 control animals, LV end-systolic volume and end-diastolic volume increased from 6 to 30 weeks (media

203 surgery at 5 years, 90% for left ventricular end-diastolic volume index <100 mL/m(2) versus 48% for >

204 tial shortening (B=-0.1; P=0.0001), lower LV end-diastolic volume index (B=0.6; P=0.0001), and lower

205 0.0001), obesity (beta=1.3 mL/m(2), P<0.01), end-diastolic volume index (beta=0.4 mL/m(2), P<0.0001),

206 outcome was mean change from baseline in LV end-diastolic volume index (LVEDVI) at 6 months.

207 small, stable reduction in left ventricular end-diastolic volume index (P<0.001), with a concomitant

208 all stable increase in the right ventricular end-diastolic volume index (P<0.001).

209 Stroke volume index (P=0.015) and end-diastolic volume index (P=0.001) decreased during ex

210 olume index (P<0.0001), and left ventricular end-diastolic volume index (P=0.0015).

211 Patients with a right ventricular end-diastolic volume index above 150 ml/m(2) and peak ex

212 Patients with a right ventricular end-diastolic volume index above 150 ml/m(2) and peak ex

213 se in change from baseline right ventricular end-diastolic volume index and a 429 ml (P < 0.001) redu

214 Left ventricle end-diastolic volume index and end-systolic volume index

215 Greater ECE is associated with reduced LV end-diastolic volume index and LV end-diastolic mass ind

216 The left ventricular end-diastolic volume index decreased (90.1 +/- 28.2 ml/m

217 Resting left ventricular end-diastolic volume index increased (P=0.001) within th

218 Right ventricular end-diastolic volume index measured by means of cardiova

219 CMR-derived end-diastolic volume index showed a weaker association w

220 had a greater extent of LGE and a higher LV end-diastolic volume index than other groups, but levels

221 as change from baseline in right ventricular end-diastolic volume index versus placebo.

222 All patients were hypovolemic (global end-diastolic volume index<680 mL/m) on enrollment.

223 ET sympathetic denervation, left ventricular end-diastolic volume index, creatinine, and no angiotens

224 edictors (age, body mass index, diabetes, LV end-diastolic volume index, LGE, EF) (hazard ratio = 2.0

225 and 87.3+/-18.7 mL/m(2) at 3-year follow-up (end-diastolic volume index, P=0.0056; end-systolic volum

226 roke volume variation monitoring, and global end-diastolic volume index.

227 iuretic peptide, and larger left ventricular end-diastolic volume index.

228 al volume index, and larger left ventricular end-diastolic volume index.

229 maximal NC/C ratio and preceding changes in end-diastolic volume indexed (EDVi) to body surface area

230 CMR-derived ventricular indexed end-diastolic volume is an independent predictor of tran

231 nly decreased the temporal variability of LV end-diastolic volume measurements by the 2D biplane meth

232 tal right/left-volume index was defined from end-diastolic volume measurements in CMR: total right/le

233 Indexed RV end-diastolic volume plotted against neoaortic stroke vo

234 RV end-systolic volumes (RVESVi) (indexed RV end-diastolic volume pPVR versus immediately after PVR v

235 dditional decline in the RV/left ventricular end-diastolic volume ratio (P=0.05) and trended toward i

236 (95% CI: 0.04, 0.36)] and a greater RV mass/end-diastolic volume ratio conditional on LV parameters.

237 associated with greater RV mass and RV mass/end-diastolic volume ratio conditional on the LV; howeve

238 associated with greater RV mass and RV mass/end-diastolic volume ratio relative to the LV.

239 measures of PM2.5, whereas those for RV mass/end-diastolic volume ratio were attenuated.

240 were directly associated with LV mass to LV end-diastolic volume ratio, a marker of cardiac remodell

241 2 models showed that the addition of indexed end-diastolic volume resulted in a significantly improve

242 fraction was 34+/-17%, and right ventricular end-diastolic volume was 114+/-39 cc/m(2).

243 LV end-diastolic volume was 2-fold (P=0.005) higher in MR p

244 LA end-diastolic volume was increased in patients with mitr

245 In CAP-treated dogs, end-diastolic volume was unchanged; ejection fraction in

246 infarct size, LV end-systolic volume, and LV end-diastolic volume were analyzed with random-effects m

247 ; stroke volume (SV), ejection fraction, and end-diastolic volume were assessed by echocardiography.

248 Pulmonary capillary wedge pressure and LV end-diastolic volume were measured at baseline, during d

249 ssociated with greater RV mass and larger RV end-diastolic volume with or without further adjustment

250 echocardiogram, the median left ventricular end-diastolic volume z score was +1.7 (range, -1.3 to +8

251 ed normal values, left and right ventricular end-diastolic volume z scores were mildly enlarged (0.48

252 eas women exhibited RV cavity dilatation (RV end-diastolic volume, +1.0 mL per BMI point increase; P<

253 004) with stable LV chamber volumes (DeltaLV end-diastolic volume, 1+/-1 mL/m(2); P=0.48).

254 4), and increased echocardiographic volumes (end-diastolic volume, 126+/-39 versus 112+/-33 mL/BSA(1.

255 acterized by dilation of the left ventricle (end-diastolic volume, 156+/-26 versus 172+/-28 mL, P<0.0

256 creased E-wave velocity and left ventricular end-diastolic volume, 2) exhibit a higher plasma volume,

257 28.6+/-4.3 cm(2); P=0.02), and left atrium (end-diastolic volume, 65+/-19 versus 72+/-19; P=0.02).

258 pertrophy was driven by LV dilation (DeltaLV end-diastolic volume, 9+/-3 mL/m(2); P=0.004) with stabl

259 in RV ejection fraction and left ventricular end-diastolic volume, although correlation coefficients

260 LV end-diastolic volume, an index of LV remodeling, increas

261 We quantified associations with RV mass, end-diastolic volume, and ejection fraction after contro

262 54 +/- 13 years of age), noncontrast 3D EF, end-diastolic volume, and end-systolic volume had signif

263 demographic data, risk factors for scar, LV end-diastolic volume, and LV mass.

264 rdiovascular risk factors, calcium score, LV end-diastolic volume, and mass in addition to resting he

265 associated with preserved stroke volume, LV end-diastolic volume, and mass/volume ratio as measured

266 al effective regurgitant orifice, indexed LV end-diastolic volume, and right ventricular systolic pre

267 dary efficacy end points included changes in end-diastolic volume, end-systolic volume, and ejection

268 ng tachycardia, low blood pressure, enlarged end-diastolic volume, high ejection fraction, and high c

269 periencing MACE showed higher left ventricle end-diastolic volume, higher left ventricle end-systolic

270 ts underwent TTE and CMR, and left ventricle end-diastolic volume, left ventricle end-systolic volume

271 d improvements in LV end-systolic volume, LV end-diastolic volume, left ventricular ejection fraction

272 as a reduction in LV wall stress, reduced LV end-diastolic volume, LV end-systolic volume and increas

273 ined percent change in left ventricular (LV) end-diastolic volume, LV end-systolic volume, LV ejectio

274 line LV function, who showed reduction in LV end-diastolic volume, LV wall stress, no change in LV en

275 .96%; 95% confidence interval, -0.2 to 2.1), end-diastolic volume, or systolic volume were observed c

276 oved overall explained variance for volumes (end-diastolic volume, R(2)=0.43; end-systolic volume, R(

277 ter adjustment for baseline left ventricular end-diastolic volume.

278 rease of 15% or more in the left ventricular end-diastolic volume.

279 elated with the postoperative decrease in LV end-diastolic volume.

280 ssociated with greater RV mass and larger RV end-diastolic volume.

281 enhancing the saline-mediated increase in LV end-diastolic volume.

282 cular end-diastolic volume (left ventricular end-diastolic volume/body surface area, 104+/-13 and 69+

283 69+/-18 mL/m(2); P<0.001; right ventricular end-diastolic volume/body surface area, 110+/-22 and 66+

284 7.9 [7.3-28.5], then decreased by 14 months (end-diastolic volume/BSA(1.3), end-systolic volume/BSA(1

285 lume: -4.3 [11.3] versus 7.4 [11.8], P=0.02; end-diastolic volume: -9.1 [14.9] versus 7.4 [15.8], P=0

286 size (Delta ejection fraction: P<0.04, Delta end-diastolic volume: P<0.02, Delta end-systolic volume:

287 remodeling (>20% change in left ventricular end-diastolic volume; 21.91 [2.75-174.29]; P=0.004).

288 ge of 15 to 30 mL in 3DTTE right ventricular end-diastolic volume; sample sizes were 2x to 2.5x those

289 dilated right ventricles (right ventricular end-diastolic volume=101+/-26 mL/m(2)) with good systoli

290 jects in the highest quintile had smaller LV end-diastolic volumes (68+/-13 versus 58+/-12 mL/m(2); P

291 ection fraction (P<0.01), reduced stroke and end-diastolic volumes (both P<0.001), decreased peak E'

292 mediate and midterm reductions in indexed RV end-diastolic volumes and RV end-systolic volumes (RVESV

293 By CMR, right ventricular end-diastolic volumes were 118 +/- 31 cc/m(2), 108 +/- 2

294 End-systolic and end-diastolic volumes were calculated using blood pool i

295 Both the LV and RV end-systolic and LV end-diastolic volumes were increased compared with refer

296 ing demonstrated increasing left ventricular end-diastolic volumes, end-systolic volumes, stroke volu

297 (LV) ejection fraction, LV end-systolic and end-diastolic volumes, infarct size, and major adverse c

298 all thickening fraction, LV end-systolic and end-diastolic volumes, LV ejection fraction) and hemodyn

299 Systolic function (EF, end-systolic and end-diastolic volumes, stroke volumes) was not different

300 an age (1.6 vs. 1.7 years), left ventricular end-diastolic z-scores (+4.2 vs. +4.2), and left ventric