ライフサイエンスコーパス: end-diastolic volume

1 to pump failure revealed a rapid decrease in end diastolic volume.

2 ssociated with greater RV mass and larger RV end-diastolic volume.

3 significantly reduced because of diminished end-diastolic volume.

4 ) which relaxes quickly and fills to a large end-diastolic volume.

5 ter adjustment for baseline left ventricular end-diastolic volume.

6 rease of 15% or more in the left ventricular end-diastolic volume.

7 enhancing the saline-mediated increase in LV end-diastolic volume.

8 elated with the postoperative decrease in LV end-diastolic volume.

9 Allogeneic MSCs reduced LV end-diastolic volumes.

10 T was associated with lower left ventricular end-diastolic volume (0.01 mm/mL; P<0.01), a lower left

11 e ratio of elevated left ventricular mass to end-diastolic volume (0.02 g/mL per unit).

12 ned as percent reduction in left ventricular end-diastolic volume 1 year after CRT-D implantation) an

13 004) with stable LV chamber volumes (DeltaLV end-diastolic volume, 1+/-1 mL/m(2); P=0.48).

14 eas women exhibited RV cavity dilatation (RV end-diastolic volume, +1.0 mL per BMI point increase; P<

15 n (45% vs. 56%; p < 0.001), increased median end-diastolic volume (100 ml/body surface area [BSA](1.3

16 dilated right ventricles (right ventricular end-diastolic volume=101+/-26 mL/m(2)) with good systoli

17 /-4 and 65+/-4%; P<0.05), greater indexed LV end-diastolic volume (102+/-34 versus 84+/-14 and 85+/-1

18 s not associated with changes in LV volumes (end-diastolic volume 106 +/- 15 versus 110 +/- 22 mL; en

19 compared with patients with GLS</=-15.0% (LV end-diastolic volume 123+/-44 versus 106+/-36 mL and 121

20 4), and increased echocardiographic volumes (end-diastolic volume, 126+/-39 versus 112+/-33 mL/BSA(1.

21 grade was mild with significant decreases in end-diastolic volume (139 to 107 mL; P=0.03) and left at

22 cant reduction in right ventricular volumes (end diastolic volume 142+/-43 to 91+/-18, end systolic v

23 A significant reduction of LV end-diastolic volume (149.7 +/- 41.4-140.1 +/- 43.9 mL;

24 e was a larger increase in right ventricular end-diastolic volume (15 mL; 95% confidence interval, 3-

25 acterized by dilation of the left ventricle (end-diastolic volume, 156+/-26 versus 172+/-28 mL, P<0.0

26 crease of 15 mL at >500 ng/mL, p=0.0026) and end-diastolic volumes (16 mL, p=0.0096) that might have

27 enhancing the saline-mediated increase in LV end-diastolic volume (+17+/-1 versus +10+/-2 mL; P=0.016

28 , a significant decrease in left ventricular end-diastolic volume (-18 mL; P=0.009) and end-systolic

29 o -3.80; P<0.00001), and tended to reduce LV end-diastolic volume (-2.26 mL; 95% confidence interval,

30 creased E-wave velocity and left ventricular end-diastolic volume, 2) exhibit a higher plasma volume,

31 CMR at 1 year demonstrated a decrease in end diastolic volume (208.7+/-20.4 versus 167.4+/-7.32 m

32 remodeling (>20% change in left ventricular end-diastolic volume; 21.91 [2.75-174.29]; P=0.004).

33 action (30+/-10% versus 29+/-7%, P=0.79), LV end-diastolic volume (220+/-70 versus 228+/-57 mL, P=0.6

34 (178 +/- 72 to 145 +/- 23 ml, p < 0.001), LV end-diastolic volume (242 +/- 85 ml to 212 +/- 63 ml, p

35 ricular [LV] ejection fraction 23 +/- 9%, LV end-diastolic volume 275 +/- 127 ml) underwent evaluatio

36 ding 22 years were inversely associated with end-diastolic volume (-3.0 mL per unit mean hemoglobin A

37 nts (mean LV ejection fraction 22 +/- 7%, LV end-diastolic volume 323 +/- 140 ml, 40% ischemic) had e

38 ly related to reductions in left ventricular end-diastolic volume (-4.1 ml; 95% confidence interval [

39 2.7]; p = 0.002), and a trend toward reduced end-diastolic volume (-4.6 ml [95% CI -10.4 to 1.1]; p =

40 val (94% versus 57%) and less LV dilatation (end-diastolic volume, 46+/-8 versus 85+/-32 microL), mec

41 <0.05) greater reduction in left ventricular end-diastolic volume (-49+/-16% versus -35+/-20%), left

42 nterval, -11.57 to -6.25; P<0.00001), and LV end-diastolic volume (-5.23 mL; 95% confidence interval,

43 Older age was associated with lower volumes (end-diastolic volume, -5 mLdecade; end-systolic volume,

44 Changes in end-diastolic volume (-6+/-14 versus -4+/-16 mL; P=0.67)

45 ignificant attenuation in the increase in LV end-diastolic volume (64.8 mL [60.7-71.3] to 83.5 mL [74

46 28.6+/-4.3 cm(2); P=0.02), and left atrium (end-diastolic volume, 65+/-19 versus 72+/-19; P=0.02).

47 jects in the highest quintile had smaller LV end-diastolic volumes (68+/-13 versus 58+/-12 mL/m(2); P

48 ts/min; p = 0.03), but no difference in peak end-diastolic volume (77 +/- 18 ml vs. 77 +/- 17 ml; p =

49 was associated with a small right ventricle (end diastolic volume, 79.8+/-13.2 versus 88.5+/-11.8 mL/

50 had more than mild FMR despite increased LV end-diastolic volume (82 +/- 22, 86 +/- 23, and 51 +/- 1

51 pertrophy was driven by LV dilation (DeltaLV end-diastolic volume, 9+/-3 mL/m(2); P=0.004) with stabl

52 lume: -4.3 [11.3] versus 7.4 [11.8], P=0.02; end-diastolic volume: -9.1 [14.9] versus 7.4 [15.8], P=0

53 in RV ejection fraction and left ventricular end-diastolic volume, although correlation coefficients

54 LV end-diastolic volume, an index of LV remodeling, increas

55 On echocardiography, left ventricular end diastolic volume and deceleration time improved with

56 ed as the change in indexed left ventricular end diastolic volume and LVEF.

57 ype and G6PDX mice but significantly greater end diastolic volume and wall thinning in G6PDX mice.

58 of the right ventricle were used to measure end-diastolic volume and ejection fraction.

59 MRI results showed a decrease of indexed RV end-diastolic volume and end-systolic volume (98 ml/m(2)

60 RV volumes (end-diastolic volume and end-systolic volume), stroke vo

61 of women were classified as having abnormal end-diastolic volume and internal diameter by ASE 2015 c

62 , and was highly reflective of changes in LV end-diastolic volume and LA pressure.

63 ints, including structural (left ventricular end-diastolic volume and left ventricular ejection fract

64 with placebo did not change left ventricular end-diastolic volume and left ventricular mass nor did i

65 iminated the progressive deterioration in LV end-diastolic volume and LV end-systolic volume.

66 Indexed left ventricular end-diastolic volume and mass correlated with O2max (r=0

67 /m(2); P<0.001) and indexed left ventricular end-diastolic volume and right ventricular end-diastolic

68 During PEI, LV end-diastolic volume and stroke volume were increased in

69 mediate and midterm reductions in indexed RV end-diastolic volumes and RV end-systolic volumes (RVESV

70 d mean values for the left ventricular mass, end diastolic volume, and stroke volume, but not with ej

71 reases in left and right ventricular masses, end-diastolic volume, and diastolic dysfunction, and an

72 We quantified associations with RV mass, end-diastolic volume, and ejection fraction after contro

73 54 +/- 13 years of age), noncontrast 3D EF, end-diastolic volume, and end-systolic volume had signif

74 1) were directly correlated with LV mass, LV end-diastolic volume, and left atrial dimension.

75 e measures of left ventricular (LV) mass, LV end-diastolic volume, and left atrial dimension.

76 re, 6-minute walk distance, left ventricular end-diastolic volume, and left ventricular ejection frac

77 demographic data, risk factors for scar, LV end-diastolic volume, and LV mass.

78 rdiovascular risk factors, calcium score, LV end-diastolic volume, and mass in addition to resting he

79 associated with preserved stroke volume, LV end-diastolic volume, and mass/volume ratio as measured

80 al effective regurgitant orifice, indexed LV end-diastolic volume, and right ventricular systolic pre

81 Before training, Vo(2)max, LV mass, LV end-diastolic volume, and stroke volume were significant

82 Improvement in end-diastolic volume at 1 year was predictive of subsequ

83 ndent relationship between GLS groups and LV end-diastolic volume at 3 and 6 months (adjusted for cli

84 independently of load/extrinsic forces, the end-diastolic volume at a common end-diastolic pressure

85 reased LV compliance, measured as reduced LV end-diastolic volume at an idealized LV end-diastolic pr

86 centric remodeling characterized by lower LV end-diastolic volume (beta=-0.21), higher concentricity

87 on (beta=-0.02/%; P=0.015), left ventricular end-diastolic volume (beta=0.01/mL; P<0.0001), and left

88 bcutaneous fat was associated with higher LV end-diastolic volume (beta=0.48), reduced concentricity

89 cular end-diastolic volume (left ventricular end-diastolic volume/body surface area, 104+/-13 and 69+

90 69+/-18 mL/m(2); P<0.001; right ventricular end-diastolic volume/body surface area, 110+/-22 and 66+

91 endpoint variables for primary outcomes: LV end-diastolic volume/body surface area, LV ejection frac

92 ection fraction (P<0.01), reduced stroke and end-diastolic volumes (both P<0.001), decreased peak E'

93 7.9 [7.3-28.5], then decreased by 14 months (end-diastolic volume/BSA(1.3), end-systolic volume/BSA(1

94 tly higher ratio of left ventricular mass to end-diastolic volume (by 8%).

95 4.02 to -0.23; P=0.028) and left ventricular end-diastolic volume (coefficient, 7.85; 95% confidence

96 The 3DE end-diastolic volume correlated well (r=0.96) but was sm

97 ty indices ([RA+aRV]/[fRV+LA+LV]) and fRV/LV end-diastolic volume corresponded only to some parameter

98 In both men and women, LV end-diastolic volume decreased (-9.8 and -13.3 mL per de

99 Left ventricular end-diastolic volume decreased (from 125.1 +/- 40.1 ml t

100 Cardiac index increased and left ventricular end-diastolic volume decreased significantly only with 1

101 Remodeling was defined as an increase in LV end-diastolic volume (DeltaEDV>0) between discharge and

102 o a significant increase in left ventricular end-diastolic volume, demonstrating increased blood flow

103 The mean left ventricular end systolic and end diastolic volumes did not differ between the groups.

104 ction (LVEF), end systolic volume (ESV), and end diastolic volume (EDV) using 4 different commercial

105 T intensive versus conventional treatment in end diastolic volume (EDV), end systolic volume, stroke

106 tolic (ePAD) pressures and an increase in LV end diastolic volume (EDV).

107 End-diastolic volume (EDV) and Doppler-derived end-diast

108 re- to post-therapy (treatment effect) in LV end-diastolic volume (EDV) and end-systolic volume (ESV)

109 losure reversed remodeling at 3 months, with end-diastolic volume (EDV) and ESV 135% and 128% of base

110 2.6 mL of CHAM (n=5) or saline (n=13) and LV end-diastolic volume (EDV) and MMP/TIMP profiles in the

111 a small difference in left ventricular (LV) end-diastolic volume (EDV) and thus, LV stroke volume an

112 +/- 13.0 mL; P < 0.0001), difference between end-diastolic volume (EDV) at rest and stress (DeltaEDV[

113 n left ventricular ejection fraction (LVEF), end-diastolic volume (EDV), and end-systolic volume (ESV

114 gated PET images, LV ejection fraction (EF), end-diastolic volume (EDV), and end-systolic volume (ESV

115 global function was assessed by measuring LV end-diastolic volume (EDV), end-systolic volume (ESV), a

116 e overall bias and limits of agreement of LV end-diastolic volume (EDV), end-systolic volume (ESV), a

117 analyzed to provide the reference limits for end-diastolic volume (EDV), end-systolic volume (ESV), e

118 arameters, including ejection fraction (EF), end-diastolic volume (EDV), end-systolic volume (ESV), s

119 End-diastolic volume (EDV), end-systolic volume (ESV), s

120 eft ventricular ejection fraction (LVEF) and end-diastolic volume (EDV).

121 Filling was assessed by changes in LV end-diastolic volume (EDV; conductance catheter techniqu

122 LV end-diastolic volumes (EDV) were measured by echocardiog

123 assification based on LV dilatation (high LV end-diastolic volume [EDV] index) and concentricity (mas

124 scular events and a dilated LV (increased LV end-diastolic volume [EDV] indexed to body surface area)

125 ocardiographic measures of EF: cardiac size (end-diastolic volume [EDV]); contractile function (the e

126 End-systolic and end-diastolic volumes, ejection fraction, and left ventr

127 The mean LV end-diastolic volume, end-systolic volume, and ejection

128 dary efficacy end points included changes in end-diastolic volume, end-systolic volume, and ejection

129 However, changes in end-diastolic volume, end-systolic volume, and LVEF did

130 Biventricular end-diastolic volume, end-systolic volume, stroke volume

131 ing demonstrated increasing left ventricular end-diastolic volumes, end-systolic volumes, stroke volu

132 l adjustment for city attenuated the RV mass/end-diastolic volume findings.

133 rformed to compare stroke, end-systolic, and end-diastolic volumes for the left ventricle (LV) and th

134 greater than 20% relative increase in the LV end-diastolic volume from 1 to 12 wk (percentage injecte

135 - 5.8 mm Hg (p = 0.016) and a decrease in LV end-diastolic volume from 172 +/- 37 ml to 158 +/- 38 ml

136 follow-up documented a stepwise reduction in end-diastolic volume (from 147 mL [IQR, 95-191 mL] to 12

137 tion >/=10 U, combined with a decrease in LV end-diastolic volume >/=10% at follow-up.

138 lization for heart failure, left ventricular end-diastolic volume >/=125 mL/m(2), and left atrial vol

139 s defined as an increase in left ventricular end-diastolic volume >/=20% at 6 months.

140 tein-losing enteropathy, ventricular indexed end-diastolic volume >125 mL/body surface area raised to

141 CMR-derived left ventricular end-diastolic volume >246 mL had good, although lower, d

142 ng tachycardia, low blood pressure, enlarged end-diastolic volume, high ejection fraction, and high c

143 periencing MACE showed higher left ventricle end-diastolic volume, higher left ventricle end-systolic

144 g LV strain (HR, 1.63; P=0.005), exercise LV end-diastolic volume (HR, 1.38; P=0.048), and resting RV

145 ncrease and dilatation, but left ventricular end-diastolic volume improved because of reduced blood r

146 baseline to 12 months, left ventricular (LV) end-diastolic volume improved from 161 +/- 56 ml to 143

147 Left ventricular end-diastolic volume improved from 172 ml to 140 ml and

148 Cine MRI demonstrated a >40% increase in LV end-diastolic volume in both groups, consistent with a f

149 clusion initially induced an expansion of LV end-diastolic volume in IPAH (+7%; P < 0.05), whereas en

150 se; P<0.001), BMI was not correlated with RV end-diastolic volume in men (R=0.04; P=0.51).

151 (n = 167), and after replacing LV mass by LV end-diastolic volume in the regression models.

152 were recognized for stroke, end-systolic, or end-diastolic volumes in either the LV or the RV.

153 this shear strain perturbation, minimized LV end diastolic volume increase, and augmented the LV sphe

154 remental value of LVGLS for prediction of LV end-diastolic volume increase (0.14 [95% confidence inte

155 mL/m(2), P<0.0001), whereas left ventricular end diastolic volume increased (66+/-12 to 73+/-13 mL/m(

156 Right ventricular end diastolic volume increased by 49.4% after transannul

157 LV end diastolic volume increased in all groups but was att

158 les of trastuzumab, indexed left ventricular end diastolic volume increased in patients treated with

159 Cine-magnetic resonance-derived LV end-diastolic volume increased 2-fold (P=0.005), and LV

160 After intervention, LV mass and end-diastolic volume increased and exercise capacity imp

161 control animals, LV end-systolic volume and end-diastolic volume increased from 6 to 30 weeks (media

162 End-diastolic volume increased in placebo, but not in CD

163 Subsequently, RV end-systolic and end-diastolic volume increased more in both groups, resu

164 to the Frank-Starling relationship, greater end-diastolic volume increases ventricular output.

165 surgery at 5 years, 90% for left ventricular end-diastolic volume index <100 mL/m(2) versus 48% for >

166 ravascular lung water (<10 mL/kg) and global end-diastolic volume index (<850 mL/m) in the transpulmo

167 had greater improvement in left ventricular end-diastolic volume index (-26.2 versus -7.4 mL/m(2)),

168 (66 +/- 5% to 54 +/- 9%, p < 0.0001) and LV end-diastolic volume index (108 +/- 28 ml/m(2) to 78 +/-

169 iuretic peptide (79 pg/mL), left ventricular end-diastolic volume index (110 mL/m2), and left ventric

170 The decrease in end-diastolic volume index (14+/-3) was different from t

171 tial shortening (B=-0.1; P=0.0001), lower LV end-diastolic volume index (B=0.6; P=0.0001), and lower

172 0.0001), obesity (beta=1.3 mL/m(2), P<0.01), end-diastolic volume index (beta=0.4 mL/m(2), P<0.0001),

173 3.4 vs 3.8 L/min/m(2); P = .001), and global end-diastolic volume index (GEDVI) (726 vs 775 mL/m(2);

174 outcome was mean change from baseline in LV end-diastolic volume index (LVEDVI) at 6 months.

175 small, stable reduction in left ventricular end-diastolic volume index (P<0.001), with a concomitant

176 all stable increase in the right ventricular end-diastolic volume index (P<0.001).

177 Stroke volume index (P=0.015) and end-diastolic volume index (P=0.001) decreased during ex

178 olume index (P<0.0001), and left ventricular end-diastolic volume index (P=0.0015).

179 Patients with a right ventricular end-diastolic volume index above 150 ml/m(2) and peak ex

180 Patients with a right ventricular end-diastolic volume index above 150 ml/m(2) and peak ex

181 se in change from baseline right ventricular end-diastolic volume index and a 429 ml (P < 0.001) redu

182 d more severe renal dysfunction, yet smaller end-diastolic volume index and cardiac output and increa

183 Left ventricle end-diastolic volume index and end-systolic volume index

184 Greater ECE is associated with reduced LV end-diastolic volume index and LV end-diastolic mass ind

185 The left ventricular end-diastolic volume index decreased (90.1 +/- 28.2 ml/m

186 Resting left ventricular end-diastolic volume index increased (P=0.001) within th

187 Right ventricular end-diastolic volume index measured by means of cardiova

188 CMR-derived end-diastolic volume index showed a weaker association w

189 had a greater extent of LGE and a higher LV end-diastolic volume index than other groups, but levels

190 as change from baseline in right ventricular end-diastolic volume index versus placebo.

191 End-diastolic volume index was determined by echo Dopple

192 All patients were hypovolemic (global end-diastolic volume index<680 mL/m) on enrollment.

193 central venous pressure, 0.56 for the global end-diastolic volume index, and 0.64 for the left ventri

194 istic of the central venous pressure, global end-diastolic volume index, and left ventricular end-dia

195 ith infarct volume, LV ejection fraction, LV end-diastolic volume index, and LV end-systolic volume i

196 ET sympathetic denervation, left ventricular end-diastolic volume index, creatinine, and no angiotens

197 e were no significant differences in mean LV end-diastolic volume index, end-systolic volume index an

198 edictors (age, body mass index, diabetes, LV end-diastolic volume index, LGE, EF) (hazard ratio = 2.0

199 and 87.3+/-18.7 mL/m(2) at 3-year follow-up (end-diastolic volume index, P=0.0056; end-systolic volum

200 roke volume variation monitoring, and global end-diastolic volume index.

201 iuretic peptide, and larger left ventricular end-diastolic volume index.

202 al volume index, and larger left ventricular end-diastolic volume index.

203 ure (LV mass index: r = 0.35, p < 0.0001; LV end-diastolic volume index: r = 0.43, p < 0.0001) and LV

204 maximal NC/C ratio and preceding changes in end-diastolic volume indexed (EDVi) to body surface area

205 In the 50-mg group, end-systolic and end-diastolic volume indexes decreased relative to basel

206 (LV) ejection fraction, LV end-systolic and end-diastolic volumes, infarct size, and major adverse c

207 CMR-derived ventricular indexed end-diastolic volume is an independent predictor of tran

208 n was defined as (LA end-systolic volume--LA end-diastolic volume) / LA end-systolic volume.

209 r end-diastolic volume and right ventricular end-diastolic volume (left ventricular end-diastolic vol

210 ts underwent TTE and CMR, and left ventricle end-diastolic volume, left ventricle end-systolic volume

211 d improvements in LV end-systolic volume, LV end-diastolic volume, left ventricular ejection fraction

212 s percentage of myocardium, left ventricular end-diastolic volume, left ventricular end-systolic volu

213 olume by CMR is superior to left ventricular end-diastolic volume, left ventricular end-systolic volu

214 ments in ejection fraction, left ventricular end-diastolic volume, left ventricular end-systolic volu

215 A relatively aggressive PVR policy (end diastolic volume <150 mL/m(2)) leads to normalizatio

216 were predicted by a preoperative indexed RV end-diastolic volume </=158 mL/m(2) and RVESVi </=82 mL/

217 as a reduction in LV wall stress, reduced LV end-diastolic volume, LV end-systolic volume and increas

218 ined percent change in left ventricular (LV) end-diastolic volume, LV end-systolic volume, LV ejectio

219 line LV function, who showed reduction in LV end-diastolic volume, LV wall stress, no change in LV en

220 all thickening fraction, LV end-systolic and end-diastolic volumes, LV ejection fraction) and hemodyn

221 of the Fick equation: (left ventricular (LV) end-diastolic volume--LV end-systolic volume) x heart ra

222 jection fraction (LVEF) and left ventricular end diastolic volume (LVEDV) on cardiac magnetic resonan

223 tan (30 mg/day) on reducing left ventricular end-diastolic volume (LVEDV) compared with placebo in pa

224 efined as percent change in left ventricular end-diastolic volume (LVEDV), was analyzed in 3 prespeci

225 volume [EDV] index) and concentricity (mass/end-diastolic volume [M/EDV](2/3)) in hypertensive patie

226 nts of left ventricular end-systolic volume, end-diastolic volume, mass, and ejection fraction were p

227 ite normalization of LV stroke volume and LV end-diastolic volume/mass ratio, there was a persistent

228 CMR showed higher left ventricle end-diastolic volume (mean difference: 43+/-22.5 mL), hi

229 nly decreased the temporal variability of LV end-diastolic volume measurements by the 2D biplane meth

230 tal right/left-volume index was defined from end-diastolic volume measurements in CMR: total right/le

231 increased from pre-Norwood to pre-stage II (end-diastolic volume [milliliters]/body surface area [BS

232 resonance (1.5 T) to measure RV mass (g), RV end-diastolic volume (mL), RV mass/volume ratio, and LV

233 P=0.002), exercise indexed left ventricular end-diastolic volume (odds ratio, 1.04; P=0.026), exerci

234 tolic distensibility index (ratio of ePAD to end-diastolic volume) of 0.11+/-0.06 mm Hg/mL.

235 left ventricular (LV) ejection fraction, LV end-diastolic volume, or New York Heart Association (NYH

236 .96%; 95% confidence interval, -0.2 to 2.1), end-diastolic volume, or systolic volume were observed c

237 en by associations with baseline and DeltaLV end diastolic volume (P<0.0001 for each) and not wall th

238 wer ejection fraction (p = 0.002), increased end-diastolic volume (p < 0.001), increased mass(i) (p <

239 G-but not CT-significantly overestimated the end-diastolic volume (p < 0.001), whereas 2D Echo and 3D

240 ly predictor of increase in left ventricular end-diastolic volume (p < 0.001).

241 ts were observed for the ratio of LV mass to end-diastolic volume (P=0.02) and with hyperinflation me

242 F23 on left ventricular (LV) mass (P=0.016), end-diastolic volume (P=0.029), and end-systolic volumes

243 0.0002), and increased ventricular dilation (end-diastolic volume, P=0.01).

244 size (Delta ejection fraction: P<0.04, Delta end-diastolic volume: P<0.02, Delta end-systolic volume:

245 on, and IMH correlated with the change in LV end-diastolic volume (Pearson's rho of 0.64, 0.59, and 0

246 Indexed RV end-diastolic volume plotted against neoaortic stroke vo

247 RV end-systolic volumes (RVESVi) (indexed RV end-diastolic volume pPVR versus immediately after PVR v

248 stolic pressure (LVEDP) and left ventricular end-diastolic volume (preload) in CHF rats, which was no

249 cardial lidocaine paradoxically decreased LV end-diastolic volume (preload) in CHF rats, which was no

250 ntric geometry, defined by higher LV mass to end-diastolic volume quartiles, were associated with hig

251 t zone strongly correlated with reduction of end diastolic volume (r(2)=0.69, P=0.04) and end systoli

252 e was a significant predictor of increase in end-diastolic volume (r = 0.51, P < 0.05).

253 fraction correlated closely with indexed RV end-diastolic volume (R = 0.79, p < 0.001) and modestly

254 = 0.95), end-systolic volume (r = 0.93), and end-diastolic volume (r = 0.90), and slightly lower corr

255 d mechanical dyssynchrony correlated with RV end-diastolic volume (r=0.39; P=0.03).

256 ed earlier and accounted for the increase in end-diastolic volume (r=0.65), and VO2max (r=0.74, both

257 oved overall explained variance for volumes (end-diastolic volume, R(2)=0.43; end-systolic volume, R(

258 nance to assess LV remodeling (LV mass-to-LV end diastolic volume ratio), function, tissue characteri

259 dditional decline in the RV/left ventricular end-diastolic volume ratio (P=0.05) and trended toward i

260 (95% CI: 0.04, 0.36)] and a greater RV mass/end-diastolic volume ratio conditional on LV parameters.

261 associated with greater RV mass and RV mass/end-diastolic volume ratio conditional on the LV; howeve

262 associated with greater RV mass and RV mass/end-diastolic volume ratio relative to the LV.

263 measures of PM2.5, whereas those for RV mass/end-diastolic volume ratio were attenuated.

264 were directly associated with LV mass to LV end-diastolic volume ratio, a marker of cardiac remodell

265 nce of LVH and abnormal/normal LV mass to LV end-diastolic volume ratio.

266 tion, preload-recruitable stroke work, dP/dt-end-diastolic volume relation) were significantly depres

267 ed on LV mass index and the ratio of LV mass:end-diastolic volume (relative wall mass).

268 ed percentiles of LV end systolic volume, LV end diastolic volume, relative wall and septal thickness

269 2 models showed that the addition of indexed end-diastolic volume resulted in a significantly improve

270 n a 57% increase in LV mass (no change in LV end diastolic volume, resulting in an increase in the LV

271 Right ventricular end-diastolic volume (RVEDV) was reduced in COPD compare

272 ge of 15 to 30 mL in 3DTTE right ventricular end-diastolic volume; sample sizes were 2x to 2.5x those

273 on are inversely related to left ventricular end-diastolic volume, stroke volume, and cardiac output

274 Systolic function (EF, end-systolic and end-diastolic volumes, stroke volumes) was not different

275 sed relaxation velocity and left ventricular end diastolic volume to produce higher left ventricle ma

276 fraction was 34+/-17%, and right ventricular end-diastolic volume was 114+/-39 cc/m(2).

277 LV end-diastolic volume was 2-fold (P=0.005) higher in MR p

278 es and treatment group; each 10% decrease in end-diastolic volume was associated with a 40% reduction

279 LA end-diastolic volume was increased in patients with mitr

280 The RV end-diastolic volume was measured at 70.97 +/- 15.0 ml w

281 th LV mass and left atrial dimension, but LV end-diastolic volume was not consistently associated wit

282 In CAP-treated dogs, end-diastolic volume was unchanged; ejection fraction in

283 mean left ventricular ejection fraction and end-diastolic volume were 63.8% +/- 9.8% and 82.0 +/- 53

284 infarct size, LV end-systolic volume, and LV end-diastolic volume were analyzed with random-effects m

285 ; stroke volume (SV), ejection fraction, and end-diastolic volume were assessed by echocardiography.

286 infarct size, LV end-systolic volume, and LV end-diastolic volume were estimated with random-effects

287 Pulmonary capillary wedge pressure and LV end-diastolic volume were measured at baseline, during d

288 By CMR, right ventricular end-diastolic volumes were 118 +/- 31 cc/m(2), 108 +/- 2

289 End-systolic and end-diastolic volumes were calculated using blood pool i

290 LV end-systolic and end-diastolic volumes were defined with a phase analysis

291 Both the LV and RV end-systolic and LV end-diastolic volumes were increased compared with refer

292 Pulmonary capillary wedge pressures and LV end-diastolic volumes were measured at baseline, during

293 follow-up, left atrial and left ventricular end-diastolic volumes were significantly more reduced in

294 id not significantly change left ventricular end-diastolic volume, while ventricular end-systolic vol

295 ciations of RV mass and, to a lesser extent, end diastolic volume with PM10-2.5 mass among susceptibl

296 ssociated with greater RV mass and larger RV end-diastolic volume with or without further adjustment

297 echocardiogram, the median left ventricular end-diastolic volume z score was +1.7 (range, -1.3 to +8

298 ed normal values, left and right ventricular end-diastolic volume z scores were mildly enlarged (0.48

299 LV end-diastolic volume z-score increased (P=0.02), LV mass

300 The mean right ventricular (RV) end-diastolic volume z-score was 1.8 +/- 1.9; RV dilatio