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1 inflammatory complications (e.g., sepsis and endocarditis).
2 independently associated with acute Q fever endocarditis.
3 mportant in the pathogenesis of experimental endocarditis.
4 Mycobacterium spp. are a rare cause of endocarditis.
5 patients with suspected PHV obstruction and endocarditis.
6 apparently culture-negative prosthetic valve endocarditis.
7 ny bacterial species implicated in infective endocarditis.
8 ce the understanding of left-sided infective endocarditis.
9 ation of laboratory testing for diagnosis of endocarditis.
10 f Mycobacterium mageritense prosthetic valve endocarditis.
11 s and can result in chronic life-threatening endocarditis.
12 at can progress to chronic, life-threatening endocarditis.
13 netii can cause a life-threatening infective endocarditis.
14 se urinary tract infections, bacteremia, and endocarditis.
15 and long-term patient outcomes in left-sided endocarditis.
16 alve replacement is necessary for left-sided endocarditis.
17 tion and low incidence of stent fracture and endocarditis.
18 al pathogen that is rarely reported to cause endocarditis.
19 nfection, and no clinical signs of infective endocarditis.
20 nfirm the role of Acm in the pathogenesis of endocarditis.
21 We excluded patients presenting with endocarditis.
22 teeth and is an important cause of infective endocarditis.
23 nhancement in imaging of S. aureus infective endocarditis.
24 sanguinis is a causative agent of infective endocarditis.
25 the embolism risk in patients with infective endocarditis.
26 ases such as skin infections, pneumonia, and endocarditis.
27 tic complications, and nonseptic complicated endocarditis.
28 olic events (EEs) in patients with infective endocarditis.
29 transesophageal echocardiography findings of endocarditis.
30 nced disease severity as tested by infective endocarditis.
31 17 patients (1.9-fold increase) had definite endocarditis.
32 ut the Ross procedure is of limited value in endocarditis.
33 negative impact on the outcome of infective endocarditis.
34 implant, and 2.7% of patients had prosthetic endocarditis.
35 (ER) at admission of patients with infective endocarditis.
36 y biofilm formation and the establishment of endocarditis.
37 Infective endocarditis.
38 priate management and treatment of infective endocarditis.
39 cifically increased in patients with Q fever endocarditis.
40 vasive manifestations like osteomyelitis and endocarditis.
41 without antibiotic prophylaxis progressed to endocarditis.
42 262,658 beneficiaries were hospitalized with endocarditis.
43 n the subgroup with a principal diagnosis of endocarditis.
44 ations of Streptococcus agalactiae infective endocarditis.
45 e clinical syndromes, such as meningitis and endocarditis.
46 ot meet the CSTE case definition for Q fever endocarditis.
47 were reviewed to identify reports describing endocarditis.
48 lis is a leading cause of subacute infective endocarditis.
49 he contemporary epidemiology and outcomes of endocarditis.
50 terial disease, rheumatic heart disease, and endocarditis.
51 ure perfusion model and a new mouse model of endocarditis.
52 lococcus lugdunensis is an emerging cause of endocarditis.
53 is and in-hospital mortality after infective endocarditis.
54 sociated with an increased risk of infective endocarditis.
55 ike illness that can also present as chronic endocarditis.
56 infections (UTIs), bacteremia, and infective endocarditis.
57 ptococcus mutans and virulence for infective endocarditis.
58 ococcus aureus, a leading cause of bacterial endocarditis.
59 ermease A [BepA]), as important in infective endocarditis.
60 current acute rheumatic fever, and infective endocarditis.
62 sociated with higher rates of a diagnosis of endocarditis (14%-28%) compared with transthoracic echoc
63 ination was administered in 12 patients with endocarditis, 2 with vascular graft infection, and 2 wit
68 cus sanguinisis a leading cause of infective endocarditis, a life-threatening infection of the cardio
70 hospitalization or mortality associated with endocarditis after publication of the 2007 guidelines.
71 ry included patients with definite infective endocarditis after TAVR from 47 centers from Europe, Nor
73 with higher risk of progressing to infective endocarditis after TAVR was younger age (78.9 years vs 8
74 s and outcomes of patients who had infective endocarditis after undergoing transcatheter aortic valve
76 aureus infection with cardiac involvement (2 endocarditis and 1 pericarditis), secondary to intraveno
77 sis patients were hospitalized for bacterial endocarditis and 1267 (11.4%) underwent valvular replace
82 B is critical for S. sanguinis virulence for endocarditis and belongs to the LraI family of conserved
83 is of many E. faecalis infections, including endocarditis and catheter-associated urinary tract infec
85 s of Mycobacterium chimaera prosthetic valve endocarditis and disseminated disease were notified in E
86 lass Ib RNR in an animal model for infective endocarditis and establishing whether the manganese requ
87 intigraphy was classified as having possible endocarditis and had positive (18)F-FDG PET results and
88 eptococcus sanguinis is a cause of infective endocarditis and has been shown to require a manganese t
91 valve replacement for incidence of infective endocarditis and infective endocarditis for in-hospital
94 le reviews the challenges posed by infective endocarditis and outlines current and future strategies
95 t a case of R. mucilaginosa prosthetic valve endocarditis and review the literature of prosthetic dev
96 an important factor involved in E. faecalis endocarditis and that rEfbA immunization is effective in
98 mes were crude and standardized incidence of endocarditis and trends in patient characteristics and d
99 us faecalis), encoding a virulence factor in endocarditis and urinary tract infection models, has bee
101 ortic aneurysm, peripheral arterial disease, endocarditis, and all other cardiovascular diseases comb
102 rly implanted leads risk vascular injury and endocarditis, and can be difficult to locate in desired
103 ding meningitis, septicaemia, gonorrhoea and endocarditis, and extracts haem from haemoglobin as an i
104 isease in the Young and its Rheumatic Fever, Endocarditis, and Kawasaki Disease Committee organized a
105 uspices of the Committee on Rheumatic Fever, Endocarditis, and Kawasaki Disease, Council on Cardiovas
107 Patients with congenital heart disease, with endocarditis, and undergoing concomitant cardiac operati
111 Prospective analyses of culture-negative endocarditis are needed to better assess the clinical sp
113 atients with MRSA bacteremia and right-sided endocarditis as well as in complicated SSSIs, but should
114 ansferase system (PTS) permease (biofilm and endocarditis-associated permease A [BepA]), as important
115 middle-aged patients, and for patients with endocarditis, bacterial meningitis, lymphoma/leukemia, H
116 Studies comparing outcomes in left-sided endocarditis based on the prosthesis selected for implan
119 ed as a diagnostic tool for prosthetic valve endocarditis, but its specificity is limited by uptake o
120 virulent than wild-type in a rabbit model of endocarditis, but significantly more virulent than the s
121 dence of embolism in high-risk patients with endocarditis, but the quantification of ER remains chall
123 after left ventricular assist device (LVAD) endocarditis caused by methicillin-resistant Staphylococ
124 had received a diagnosis of tricuspid valve endocarditis caused by MRSA was evaluated for replacemen
126 ical and epidemiological features of Q fever endocarditis collected through passive surveillance in t
127 causes more-aggressive infections, including endocarditis, compared with other coagulase-negative sta
129 ients with health care-associated nosocomial endocarditis decreased (from 17.7% to 15.3%; APC, -1.0%;
130 the proportion of patients with native-valve endocarditis decreased (from 74.5% to 68.4%; APC, -0.7%;
131 mendations for valve selection in left-sided endocarditis depend on patient age and whether the infec
136 icin plus cyclophosphamide group), infective endocarditis (docetaxel plus bevacizumab followed by dox
137 ibe a case involving a patient who developed endocarditis due to Rhodotorula mucilaginosa and Staphyl
138 of mortality were arrhythmia, heart failure, endocarditis, during valvular surgery, pulmonary hyperte
139 l-associated lineages were predominant among endocarditis E. faecalis isolates recovered during this
140 mbined with antibiotics against experimental endocarditis (EE) due to Pseudomonas aeruginosa, an arch
144 thrombus), bioprosthesis calcifications, and endocarditis extent (valve dehiscence and pseudoaneurysm
145 prophylaxis for the prevention of infective endocarditis fell substantially after introduction of th
148 Dialysis patients hospitalized for bacterial endocarditis from 2004 to 2007 were studied retrospectiv
150 racteristics of patients with meningitis and endocarditis from a nationwide cohort study of adults wi
158 in patients at risk of developing infective endocarditis has historically been the focus of infectiv
159 substantially and the incidence of infective endocarditis has increased significantly in England sinc
161 laxis and treatment guidelines for infective endocarditis have changed substantially over the past de
162 .55; 95% confidence interval, 2.14-3.03) and endocarditis (hazard ratio, 1.60; 95% confidence interva
163 erity from bite wounds and rhinosinusitis to endocarditis; historically, these infections were though
165 ical variability and complexity in infective endocarditis, however, dictate that these recommendation
166 atients hospitalized with a first episode of endocarditis identified from mandatory state databases i
167 dental procedures in patients with infective endocarditis (IE) according to whether the IE-causing mi
169 stics, management, and outcomes of infective endocarditis (IE) after transcatheter aortic valve impla
170 fy patients with increased risk of infective endocarditis (IE) among patients with Enterococcus faeca
171 se of surgery for the treatment of infective endocarditis (IE) as related to surgical indications and
172 ays a key role in the diagnosis of infective endocarditis (IE) but can be inconclusive in patients in
173 ogy and American Heart Association infective endocarditis (IE) guideline update, antibiotic prophylax
174 ctious embolisms in patients with infectious endocarditis (IE) in comparison with a historic cohort o
180 ating the portal of entry (POE) of infective endocarditis (IE) is important, but published research o
184 phylaxis (AP) for those at risk of infective endocarditis (IE) undergoing dental procedures in the Un
187 normalities have increased risk of infective endocarditis (IE), it is unknown whether these abnormali
188 rgical mortality for patients with infective endocarditis (IE), presumably because of improved diagno
196 c tamponade (1B), valvular dysfunction (1C), endocarditis in native (2C) or mechanical valves (1B), g
197 initial pathogenic step of S aureus-induced endocarditis in patients with an apparently intact endot
202 In univariate analysis, male sex, previous endocarditis, in situ stents in the right ventricular ou
203 haracteristics associated with a low risk of endocarditis include absence of a permanent intracardiac
205 ts with health care-associated nonnosocomial endocarditis increased (from 32.1% to 35.9%; APC, 0.8%;
206 arch, 2008, the number of cases of infective endocarditis increased significantly above the projected
207 t of large caseous lesions, and in infective endocarditis, increases the size of pathognomonic vegeta
209 onic Q fever develops, mostly manifesting as endocarditis, infected aneurysms, or infected vascular p
214 of nosocomial infections, of which infective endocarditis is associated with substantial mortality.
215 interaction in the pathogenesis of infective endocarditis is attachment of the organisms to host plat
220 stinct surface carbohydrates from E. faecium endocarditis isolate Tx16, shown previously to be resist
222 ased approach for the treatment of infective endocarditis, leading to a strong reduction of mortality
223 cus aureus left-sided native valve infective endocarditis (LNVIE) has higher complication and mortali
225 Among 75829 patients with first episodes of endocarditis (mean [SD] age, 62.3 [18.9] years; 59.1% ma
227 ixed-inoculum (TX82 plus TX82 DeltaccpA) rat endocarditis model and also in an in vitro competitive g
228 mapping (M-TraM) approach to evaluate a rat endocarditis model and identified a gene, originally ann
231 istance both in vitro and in an experimental endocarditis model, using prototypic healthcare- and com
242 struction (n=27, with stent fracture in 22), endocarditis (n=3, 2 with stenosis and 1 with pulmonary
244 characteristics associated with low risk of endocarditis (negative predictive values from 93% to 100
245 (1.2%) were considered to have acute Q fever endocarditis, none of whom had a previously known VHD.
251 n age of the 5 male patients, diagnosed with endocarditis or aortic graft infection, was 57.8 years.
254 etween 49 and 64 years with prosthetic valve endocarditis or vascular graft infection due to M. chima
255 erculosis (OR = 8.5; CI, 1.2-61.5; P = .03), endocarditis (OR = 8.3; CI, 4.9-13.9; P < .0001), bacter
259 tonella quintana; it was also found to cause endocarditis, peliosis hepatis, and bacillary angiomatos
260 Brucella spp, and most commonly manifests as endocarditis, peripheral and cerebrovascular aneurysms,
262 ger than 1 year; Maori or Pacific ethnicity; endocarditis, pneumonia, or sepsis; and receiving no tre
265 ined from the International Collaboration on Endocarditis-Prospective Cohort Study of 4794 patients w
267 ations raised at the horizon as arrhythmias, endocarditis, pulmonary hypertension, and heart failure,
272 children with E. faecalis bacteremia without endocarditis receiving ampicillin monotherapy with those
273 patients with complicated MRSA bacteremia or endocarditis requiring rescue therapy were eligible for
277 c prophylaxis and the incidence of infective endocarditis since the introduction of these guidelines.
279 RHD, RHD complication rates (heart failure, endocarditis, stroke, and atrial fibrillation), and mort
280 cology (n=26), suspicion of prosthetic valve endocarditis subsequently excluded (n=17), and history o
281 emphasize the need for the functioning of an endocarditis team, including cardiac surgeons, cardiolog
282 r of anatomic lesions associated with active endocarditis than PET/nonenhanced CT (P=0.006) or echoca
283 not a random sample of cases of E. faecalis endocarditis, these results indicate that nonencapsulate
284 atients with Enterococcus faecalis infective endocarditis treated in the years before and after endor
285 ring a second TPV, and 2 developed bacterial endocarditis treated with antibiotics, 1 of whom then un
286 monary disease, neurological disease, active endocarditis, unstable angina, recent myocardial infarct
290 This increase in the incidence of infective endocarditis was significant for both individuals at hig
291 overall standardized incidence of infective endocarditis was stable from 1998 through 2013, with cha
299 fin-embedded heart valves from subjects with endocarditis who had positive valve and/or blood culture
300 ing FDG PET/CT in suspected prosthetic valve endocarditis, with specific attention to uptake pattern.
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