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1 that were distinct from archetypical physeal endochondral ossification.
2 actor-I (IGF-I) is an important regulator of endochondral ossification.
3 for the genesis of normal cartilage and thus endochondral ossification.
4 ic secretion of HMGB1 in cartilage regulates endochondral ossification.
5 vertebral segmentation, joint formation and endochondral ossification.
6 equently fails in growth, chondrogenesis and endochondral ossification.
7 e destined for a chondrogenic lineage during endochondral ossification.
8 protein kinase (MAPK) pathway is involved in endochondral ossification.
9 ned role for Nell1 in signal transduction in endochondral ossification.
10 physes, also suggest an underlying defect in endochondral ossification.
11 Type I Collagen and Osteocalcin), suggesting endochondral ossification.
12 nization, cartilage boundary definition, and endochondral ossification.
13 and proper closure of the PF suture through endochondral ossification.
14 ferentiation during both intramembranous and endochondral ossification.
15 y distinct mechanisms in intramembranous and endochondral ossification.
16 ion and specification of intramembranous and endochondral ossification.
17 dgehog and collagen X, and failed to undergo endochondral ossification.
18 roduced by hypertrophic cartilage undergoing endochondral ossification.
19 peared to be required for the progression of endochondral ossification.
20 sterior cranium and other bones derived from endochondral ossification.
21 now determined the role of EGFR signaling in endochondral ossification.
22 and the cranial base are both formed through endochondral ossification.
23 ndrium and the vasculature are essential for endochondral ossification.
24 ormed within a soft connective tissue, or by endochondral ossification.
25 n, as well as chondrocyte maturation, during endochondral ossification.
26 th overlapping and distinct functions during endochondral ossification.
27 ocyte hypertrophy is a mandatory step during endochondral ossification.
28 a change of matrilin oligomeric forms during endochondral ossification.
29 ly as embryonic day 14, during initiation of endochondral ossification.
30 on to its co-assembly with matrilin-1 during endochondral ossification.
31 both chondrogenesis and osteogenesis during endochondral ossification.
32 owed by mineralizing chondrocytes undergoing endochondral ossification.
33 or, a signaling molecule that also regulates endochondral ossification.
34 fgfr3 gene expression during the process of endochondral ossification.
35 nar structures of chondrocytes and defective endochondral ossification.
36 ot express RARgamma, and were not undergoing endochondral ossification.
37 oduces a delay in chondrocyte maturation and endochondral ossification.
38 with other signaling pathways that regulate endochondral ossification.
39 enchyme, but not in cartilages formed during endochondral ossification.
40 ized by short-limbed dwarfism and a delay in endochondral ossification.
41 development, indicating a role for Acp 5 in endochondral ossification.
42 equired for chondrogenic differentiation and endochondral ossification.
43 th FGFR1 in hypertrophic chondrocytes during endochondral ossification.
44 bar vertebrae revealed delayed or incomplete endochondral ossification.
45 nal-induced chondrogenic differentiation and endochondral ossification.
46 hat the protein phosphatase Phlpp1 regulates endochondral ossification.
47 cal role in chondrogenic differentiation and endochondral ossification.
48 nitors and proliferating chondrocytes during endochondral ossification.
49 nvolving Foxp1/2/4 may regulate Runx2 during endochondral ossification.
50 rast, the isotropic control groups underwent endochondral ossification.
51 ification, vascular invasion, and subsequent endochondral ossification.
52 mbrane-anchored metalloproteinase ADAM17, in endochondral ossification.
53 erein, DeltaNP63alpha and TAP63alpha, during endochondral ossification.
54 ypertrophic chondrocytes during growth plate endochondral ossification.
55 differentiation to osteoblasts and impaired endochondral ossification.
56 f primary ossification centers and disrupted endochondral ossification.
57 s, likely by reducing hedgehog signaling and endochondral ossification.
58 es heal predominantly through the process of endochondral ossification.
59 t is replaced by bone through the process of endochondral ossification.
60 were impaired in digit/limb development and endochondral ossification.
61 mous function of Atf4 in chondrocytes during endochondral ossification.
62 he proximal region of the mandible undergoes endochondral ossification.
63 osure of growth plates reflecting defects in endochondral ossification.
64 rmation of skeletal elements derived through endochondral ossification.
68 terminal phalanx forms late in gestation by endochondral ossification and continues to elongate unti
69 e for pRB and p107 in cartilage development, endochondral ossification and enchondroma formation that
70 e for pRB and p107 in cartilage development, endochondral ossification and enchondroma formation that
71 vo system to unlink the processes initiating endochondral ossification and establish more precisely t
72 sed hypertrophic domains as well as delay in endochondral ossification and formation and vascularizat
73 ificial tissue undergoes intramembranous and endochondral ossification and forms a trabecular-like bo
74 monstrate that Fgfr3 is essential for normal endochondral ossification and inner ear development.
77 ides in vivo evidence for the role of p38 in endochondral ossification and suggests that Sox9 is a li
79 that syndecan 4 is functionally involved in endochondral ossification and that its loss impairs frac
80 rview of the processes of chondrogenesis and endochondral ossification and their control at the molec
81 els of active canonical Wnt signaling enable endochondral ossification and therefore PF-suture closur
82 ical Wnt signaling in the PF suture inhibits endochondral ossification and therefore, suture closure,
84 FR3) plays a critical role in the control of endochondral ossification, and bone growth and mutations
85 oreseen link between hypertrophic cartilage, endochondral ossification, and establishment of the marr
86 tional partner of Ihh-Gli2 signalling during endochondral ossification, and that disruption of the Fo
87 ority of skeletal elements that form through endochondral ossification are absent, and the ones that
90 n and Has2 expression to control the rate of endochondral ossification as a negative feedback mechani
91 (P < 0.05), and had enhanced early and late endochondral ossification as demonstrated by Safranin O,
92 dentified a previously unrecognized delay in endochondral ossification associated with the loss of Gp
93 ion in hypertrophic chondrocytes accelerates endochondral ossification at both E17.5 and P1 stages.
94 natal viability and growth, with a defect in endochondral ossification at epiphyseal plates similar t
95 that Phd2 expressed in chondrocytes inhibits endochondral ossification at the epiphysis by suppressin
98 e regeneration via either intramembranous or endochondral ossification, both within and outside of th
99 how that redifferentiation does not occur by endochondral ossification but by the direct ossification
101 Shn3 impairs growth plate maturation during endochondral ossification but simultaneously results in
102 bone marrow (BM) is tightly associated with endochondral ossification, but little is known about the
103 minal differentiation of chondrocytes during endochondral ossification by activating the TGFalpha/EGF
105 rmone (PTH) and its related peptide regulate endochondral ossification by inhibiting chondrocyte diff
106 , which drive the early steps of heterotopic endochondral ossification by lowering oxygen tension in
107 F signaling pathway plays essential roles in endochondral ossification by regulating osteoblast proli
109 re we hypothesized that hMSCs pushed through endochondral ossification can engineer a scaled-up ossic
110 the oc/oc mouse, a mouse model with impaired endochondral ossification caused by a loss of osteoclast
111 ort limbs, a defect attributable to abnormal endochondral ossification caused by delayed cell cycle e
112 This is followed by the formation of a new endochondral ossification center at the distal end of th
114 mmalian limb structures by stimulating a new endochondral ossification center that utilizes an existi
118 eoblasts and periosteal cells during primary endochondral ossification, consistent with a role in bon
119 hat Axin2(-/-) PF-sutures lack physiological endochondral ossification, contain ectopic cartilage and
122 data show that humans with CCD have altered endochondral ossification due to altered RUNX2 regulatio
124 ibit chondrodysplasia and a complete lack of endochondral ossification even though Runx2 expression,
125 ion from mesenchymal stem cells in vitro and endochondral ossification ex vivo, and GEP-knockdown mic
126 vertebral segmentation, joint formation and endochondral ossification for this ubiquitously expresse
127 tal mandible or calvaria that do not undergo endochondral ossification formed only bone without marro
131 formed that grew in mass and cellularity by endochondral ossification in a manner similar to normal
134 drocytes induced chondrocyte hypertrophy and endochondral ossification in locations where it normally
135 ng of terminal cartilage differentiation and endochondral ossification in mandibular condylar cartila
138 fferentiation of chondrocytes that initiates endochondral ossification in the midsection of endochond
141 , which contribute to suture closure through endochondral ossification, in a process regulated in par
142 ssion of Sox9, a major negative regulator of endochondral ossification, in Col2a1-TAP63alpha transgen
145 loss of Foxc1 function mouse (Foxc1(ch/ch)), endochondral ossification is delayed and the expression
147 erefore, gene expression of ccn2 mRNA during endochondral ossification is properly regulated, at leas
148 /-) mice during embryogenesis and found that endochondral ossification is significantly impaired due
149 containing endothelial cell masses, abnormal endochondral ossification, leading to stunted long bone
151 thening, and repair of most bones proceed by endochondral ossification, namely through formation of a
153 eleton arises from the continuous process of endochondral ossification occurring at the ends of growi
154 any inflammatory reaction, and they form by endochondral ossification of enthesis fibrocartilage.
155 analyze the functional role of syndecan 4 in endochondral ossification of mouse embryos and in adult
159 on embryonic days 10.5-12.5 and to sites of endochondral ossification on embryonic days 12.5 and 13.
161 s play essential roles in crucial aspects of endochondral ossification: osteoblast differentiation, c
162 B-catalyzed proteoglycans regulate postnatal endochondral ossification partially through the mediatio
163 vidence for the association of these two key endochondral ossification pathway genes with BMD and ost
167 ively differentiate and the tissue undergoes endochondral ossification, recapitulating the developmen
170 spatiotemporal regulation in vitro resisted endochondral ossification, retained the expression of ca
175 uppressing expression of factors involved in endochondral ossification, such as osterix and vascular
176 he replacement of cartilage by bones through endochondral ossification, the growth of long bones thro
177 s to defects in cartilage development during endochondral ossification, the process by which long bon
178 e observed in the calvarium, indicating that endochondral ossification, the process needed for the fo
181 ide, negatively regulates chondrogenesis and endochondral ossification via associating with progranul
185 To investigate the role of this pathway in endochondral ossification, we generated transgenic mice
186 - and PTHrP-receptor-mediated signals during endochondral ossification were examined with embryonic m
187 in the growth plates and a general delay in endochondral ossification, whereas chondrocyte prolifera
188 ouse posterior frontal (PF) suture closes by endochondral ossification, whereas sagittal (SAG) remain
189 elements blocks chondrocyte hypertrophy and endochondral ossification, whereas signaling starting at
190 ure (PF) of the cranial vault closes through endochondral ossification, while other sutures remain pa
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