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1 apillary mucinous neoplasms and 3 pancreatic endocrine tumors).
2 by a gastric carcinoid and not a pancreatic endocrine tumor.
3 sion in both human and mouse MEN1 pancreatic endocrine tumors.
4 ce are more susceptible to hematopoietic and endocrine tumors.
5 which dysregulation of cAMP signaling causes endocrine tumors.
6 7 of 8 (88%) cases of multifocal pancreatic endocrine tumors.
7 , which is mutated in heritable and sporadic endocrine tumors.
8 ntified as a promising marker for pancreatic endocrine tumors.
9 d by multiple endocrine and occasionally non-endocrine tumors.
10 wn of its expression in malignant pancreatic endocrine tumors.
11 carcimomas but are never found in pancreatic endocrine tumors.
12 astases in patients with sporadic pancreatic endocrine tumors.
13 h diabetes and for targeting menin-sensitive endocrine tumors.
14 Pheochromocytomas are rare endocrine tumors.
15 le in the development of sporadic pancreatic endocrine tumors.
16 , are thus successfully used in PET to image endocrine tumors.
17 advances have been made in the management of endocrine tumors.
18 se (AADC), the level of which is enhanced in endocrine tumors.
19 as, 13 other pancreatic cysts, 27 pancreatic endocrine tumors, 16 chronic pancreatitis, 9 ductal aden
20 c response rate of 25% (45% among pancreatic endocrine tumors, 33% among pheochromocytomas, and 7% am
22 sion patterns in normal pancreas, pancreatic endocrine tumors and acinar carcinomas to evaluate their
23 in approximately 40% of sporadic pancreatic endocrine tumors and hypothesize that MENIN is involved
24 cal carcinoma (ACC) is a rare but aggressive endocrine tumor, and the prognostic factors associated w
26 n pancreatic tumors including cystic tumors, endocrine tumors, and intraductal pancreatic mucinous ne
27 a number of nonendocrine tumors and in a few endocrine tumors, and its presence can correlate with tu
34 l carcinoid tumors and multifocal pancreatic endocrine tumors arise independently or whether they ori
35 Medullary thyroid carcinoma (MTC) is a rare endocrine tumor arising from the C-cells of the thyroid
36 tions in the MEN1 gene on chromosome 11, and endocrine tumors arising in these patients usually show
37 erior pituitary, and pancreatic and duodenal endocrine tumors as well as foregut carcinoids in affect
39 lated adult pancreatic islets and pancreatic endocrine tumor cell lines was determined to compile lis
49 ur understanding of the hereditary nature of endocrine tumors increases, the role of genetic counseli
50 nsus region they may play a role in sporadic endocrine tumors independent of the MEN1 gene or in othe
52 unregulated secretion often associated with endocrine tumors may result, at least in part, from inef
57 LOH rates of parathyroid lesions (100%) and endocrine tumors of the pancreas (83%) were strikingly d
61 alterations that override these controls in endocrine tumors often are not associated with oncogenes
62 Forty-two patients with advanced pancreatic endocrine tumors or carcinoid tumors were treated with r
63 objective response rate (ORR) in pancreatic endocrine tumor patients was 16.7% (11 of 66 patients),
66 nomas as well as other functional pancreatic endocrine tumors (PETs) in whom the excess-hormone state
69 ine neoplasia type 1 (MEN1) develop multiple endocrine tumors, primarily affecting the parathyroid, p
71 l molecule Gbetagamma inhibition in treating endocrine tumors such as pheochromocytoma, in addition t
73 sent in both benign and malignant pancreatic endocrine tumors, suggesting that a MENIN gene mutation
74 in, a protein previously characterized as an endocrine tumor suppressor and transcriptional regulator
75 hromosome 3p25 may harbor a novel pancreatic endocrine tumor suppressor gene, and allelic loss of thi
76 NETs occur either sporadically or as part of endocrine tumor susceptibility syndromes such as multipl
82 Twenty-nine sporadic and MEN1 pancreatic endocrine tumors were studied for loss of heterozygosity
84 ine tumors include carcinoids and pancreatic endocrine tumors, which share a number of common feature
86 activation of the Men1 in pancreas developed endocrine tumors with no exocrine tumor manifestation, r
87 ntinued inclusion and analysis of pancreatic endocrine tumors with small intestinal neuroendocrine tu
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