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1 the setting of spotty skin pigmentation and endocrinopathy.
2 ted with spotty pigmentation of the skin and endocrinopathy.
3 isfolding of exportable proteins can trigger endocrinopathies.
4 ated by bilateral involvement and associated endocrinopathies.
5 zed by medullary thyroid carcinoma and other endocrinopathies.
6 , but at higher incidences in particular for endocrinopathies.
7 age, gender, and the occurrence of systemic endocrinopathies also influence sulfotransferase gene ex
9 pes, which also increase risk for autoimmune endocrinopathies and other autoimmune disorders, encode
11 gery, the medical treatment specifically for endocrinopathies, and finally, radiation treatments incl
13 on irAEs including rash, colitis, hepatitis, endocrinopathies, and pneumonitis have been established.
17 only treatment that can prevent or cure the endocrinopathies associated with multiple endocrine neop
19 the most commonly encountered perioperative endocrinopathy, continues to increase dramatically in pr
20 ome characterized by autoimmune enteropathy, endocrinopathy, dermatitis, and other autoimmune phenome
21 disease, organomegaly (or lymphadenopathy), endocrinopathy, edema (peripheral edema, ascites, or eff
23 sprue in asymptomatic patients, and multiple endocrinopathies in individuals with type 2 disease may
27 ullary thyroid carcinoma (MTC), the dominant endocrinopathy in patients with these hereditary syndrom
28 nic kidney disease (CKD), colorectal cancer, endocrinopathies (including type 2 diabetes mellitus [T2
30 ed to refer to polyneuropathy, organomegaly, endocrinopathy, M protein, and skin changes) remains poo
31 lyradiculoneuropathy, organomegaly, multiple endocrinopathies, monoclonal protein, skin changes) is a
32 ally including coexisting Castleman disease, endocrinopathy, monoclonal plasma cell neoplasm, and ski
33 mmon features: Polyneuropathy, Organomegaly, Endocrinopathy, Monoclonal protein and Skin changes) is
34 aracterized by polyneuropathy, organomegaly, endocrinopathy, monoclonal protein, and skin changes.
36 s undergoing CSI were more likely to develop endocrinopathies (P < .0001) and to require special educ
37 c findings of MAS including hyperfunctioning endocrinopathies, polyostotic fibrous dysplasia, and caf
38 al plasma cell disorder (PCD), organomegaly, endocrinopathy, skin changes, edema, sclerotic bone lesi
39 nimal operative morbidity and eradication of endocrinopathy support the minimally invasive approach f
40 neonatal diabetes mellitus, enteropathy and endocrinopathy syndrome (IPEX; MIM 304930) is the geneti
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