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1  the setting of spotty skin pigmentation and endocrinopathy.
2 ted with spotty pigmentation of the skin and endocrinopathy.
3 isfolding of exportable proteins can trigger endocrinopathies.
4 ated by bilateral involvement and associated endocrinopathies.
5 zed by medullary thyroid carcinoma and other endocrinopathies.
6 , but at higher incidences in particular for endocrinopathies.
7  age, gender, and the occurrence of systemic endocrinopathies also influence sulfotransferase gene ex
8 e of the sulfotransferases in the genesis of endocrinopathies and cancer in humans.
9 pes, which also increase risk for autoimmune endocrinopathies and other autoimmune disorders, encode
10 ontribute to the most common human pituitary endocrinopathies and tumors.
11 gery, the medical treatment specifically for endocrinopathies, and finally, radiation treatments incl
12 malignant neoplasms, cardiovascular disease, endocrinopathies, and neurocognitive impairment.
13 on irAEs including rash, colitis, hepatitis, endocrinopathies, and pneumonitis have been established.
14 nt reduction in IQ and cognitive impairment, endocrinopathy, and risk of second malignancy.
15                                          The endocrinopathies are a group of illnesses that either pr
16                                              Endocrinopathies are significant consequences of childho
17  only treatment that can prevent or cure the endocrinopathies associated with multiple endocrine neop
18                     Clustering of autoimmune endocrinopathies can result from polygenic predispositio
19  the most commonly encountered perioperative endocrinopathy, continues to increase dramatically in pr
20 ome characterized by autoimmune enteropathy, endocrinopathy, dermatitis, and other autoimmune phenome
21  disease, organomegaly (or lymphadenopathy), endocrinopathy, edema (peripheral edema, ascites, or eff
22                             No recurrence of endocrinopathy has been documented at a mean follow-up o
23 sprue in asymptomatic patients, and multiple endocrinopathies in individuals with type 2 disease may
24 e pathogenesis of the more common autoimmune endocrinopathies in our population.
25                                              Endocrinopathies in survivors increased substantially ov
26                           The development of endocrinopathies in survivors of childhood cancer as the
27 ullary thyroid carcinoma (MTC), the dominant endocrinopathy in patients with these hereditary syndrom
28 nic kidney disease (CKD), colorectal cancer, endocrinopathies (including type 2 diabetes mellitus [T2
29                            PHPT is a complex endocrinopathy involving calcium metabolism and a potent
30 ed to refer to polyneuropathy, organomegaly, endocrinopathy, M protein, and skin changes) remains poo
31 lyradiculoneuropathy, organomegaly, multiple endocrinopathies, monoclonal protein, skin changes) is a
32 ally including coexisting Castleman disease, endocrinopathy, monoclonal plasma cell neoplasm, and ski
33 mmon features: Polyneuropathy, Organomegaly, Endocrinopathy, Monoclonal protein and Skin changes) is
34 aracterized by polyneuropathy, organomegaly, endocrinopathy, monoclonal protein, and skin changes.
35 including non-convulsive status epilepticus, endocrinopathy, or thiamine deficiency.
36 s undergoing CSI were more likely to develop endocrinopathies (P < .0001) and to require special educ
37 c findings of MAS including hyperfunctioning endocrinopathies, polyostotic fibrous dysplasia, and caf
38 al plasma cell disorder (PCD), organomegaly, endocrinopathy, skin changes, edema, sclerotic bone lesi
39 nimal operative morbidity and eradication of endocrinopathy support the minimally invasive approach f
40  neonatal diabetes mellitus, enteropathy and endocrinopathy syndrome (IPEX; MIM 304930) is the geneti
41                   We identified a pattern of endocrinopathies that develop in an age-dependent manner

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