ライフサイエンスコーパス: endodontic

1 scaffolds and/or stem cells in regenerative endodontics.

2 mately one-third of the RAG-2 mice developed endodontic abscesses, while no immunocompetent controls

3 in clinical practice and requires effective endodontic and regenerative periodontal therapy.

4 membrane in association with teeth that had endodontic and/or periodontic involvement, and a nearly

5 the impact of technical advances for modern endodontics and endodontic microsurgery on the long-term

6 The outcomes of traditional periodontal, endodontic, and prosthodontic treatment approaches are c

7 e for detecting vital bacteria within mature endodontic biofilms, with an improved sensitivity over c

8 As there was a periapical radiolucency, an endodontic consultation was obtained.

9 ur results demonstrated that ODN can inhibit endodontic disease development, bone erosion, and immune

10 knockdown in periapical tissues can inhibit endodontic disease development, bone resorption, and inf

11 Dental caries may lead to endodontic disease, where the bacterial infection progre

12 bone resorption by 80% in the mouse model of endodontic disease.

13 improve the health of those who suffer from endodontic disease.

14 ts regarding systemic health consequences of endodontic disease.

15 Most common etiology of apical RPI is endodontic infection from neighboring teeth, which was d

16 tic lesion (EL) is a common manifestation of endodontic infection where Porphyromonas endodontalis is

17 Bacterial DNA typical for endodontic infection, mainly oral viridans streptococci,

18 and can be used to identify a wider range of endodontic-infection-related bacteria including the pres

19 results can help explain the persistence of endodontic infections and demonstrate a new virulence me

20 Dental caries, endodontic infections and periodontal diseases are bacte

21 protects the host from the dissemination of endodontic infections and that RAG-2 mice are more susce

22 sults indicate that bacterial communities in endodontic infections are more diverse than previously d

23 Bacterial diversity in endodontic infections has not been sufficiently studied.

24 re cultivable; hence, bacterial diversity in endodontic infections is underestimated.

25 fect of TLR4 deficiency on host responses to endodontic infections is unknown.

26 ed a more diverse microflora associated with endodontic infections than that revealed by cultural met

27 is study investigated bacterial diversity in endodontic infections through taxonomic classification b

28 s from 15 de novo and 26 refractory cases of endodontic infections were assessed.

29 iques can detect the presence of bacteria in endodontic infections when culture techniques yield a ne

30 has been identified in peri-implantitis, in endodontic infections, and in patients with localized ag

31 Endodontic infections, in which oral bacteria access the

32 bic polymicrobial consortium responsible for endodontic infections.

33 quencings were performed on 7 specimens from endodontic infections.

34 A hybridization to examine the microbiota of endodontic infections.

35 y-six samples were collected from teeth with endodontic infections.

36 et of the oral microflora is responsible for endodontic infections.

37 The goal of regenerative endodontics is to reinstate normal pulp function in necr

38 An endodontic lesion (EL) is a common manifestation of endo

39 infections, such as periodontal diseases or endodontic lesion formation.

40 radiographically as a common periodontal or endodontic lesion.

41 Periodontitis associated with endodontic lesions are among the most daunting diagnosti

42 Chronic periodontic-endodontic lesions are not uncommon in clinical practice

43 the diagnosis and management of periodontal-endodontic lesions is also presented.

44 dity in chronic otitis media, periodontitis, endodontic lesions, and loosening of orthopedic implants

45 The endodontic microbiota was far more complex than previous

46 echnical advances for modern endodontics and endodontic microsurgery on the long-term prognosis of to

47 the relationship between incident lesions of endodontic origin and time to CHD diagnosis.

48 incident radiographically evident lesions of endodontic origin were related to development of coronar

49 ose < or = 40 years old, incident lesions of endodontic origin were significantly associated with tim

50 to periodontal disease as well as lesions of endodontic origin.

51 s commonly associated with these infections (endodontic pathogens [EP]), i.e., Fusobacterium nucleatu

52 d and infected with a mixture of four common endodontic pathogens, and bone destruction was determine

53 We conclude that the endodontic pathogens, particularly P. intermedia, can ef

54 hogen that has recently been associated with endodontic pathology.

55 Treatment of concomitant endodontic-periodontal lesions remains a challenge in cl

56 promoting periodontal healing in concomitant endodontic-periodontal lesions.

57 viable modality in the treatment of chronic endodontic/periodontal lesions.

58 us cell carcinoma that presented as a benign endodontic-periodontic lesion with a 7-mm periodontal po

59 Regenerative endodontic procedures (REPs) are a treatment alternative

60 Regenerative endodontic procedures are stem cell-based treatments for

61 The translation of regenerative endodontic procedures into treating mature teeth depends

62 teeth with apical rarefactions had received endodontic procedures.

63 gative effect on the outcome of regenerative endodontic procedures.

64 prosthetic (risk difference [RD], 0.21) and endodontic (RD, 0.11) treatment was higher among histori

65 More recently, cell-based approaches in endodontic regeneration based on pulpal mesenchymal stem

66 Species commonly detected in endodontic samples included P. tannerae, Prevotella oris

67 al pathogen Porphyromonas gingivalis and the endodontic species Enterococcus faecalis were grown to e

68 depth, PI, and BOP), and background factors (endodontic status, smoking status, and surgeon's experie

69 n had been present for 42 years since having endodontic surgery at teeth #8 and #9 after a traumatic

70 regeneration techniques in combination with endodontic surgery using an induced perio-endo defect mo

71 illing materials are commonly used following endodontic surgical procedures; however, their effect on

72 h conditions were assessed, including decay, endodontic therapy (root canal therapy [RCT]), and/or ex

73 estore the lesion, and facilitate subsequent endodontic therapy and restoration.

74 e present findings demonstrate that adequate endodontic therapy performed >/=6 months before surgical

75 canal system and are therefore promising for endodontic therapy.

76 The impact of the endodontic treatment (ET) of the tooth on the healing po

77 OR, 1.52), restorative treatment (OR, 1.35), endodontic treatment (OR, 1.63), and more than 10 oral c

78 , with minimum 4 weeks between completion of endodontic treatment and actual implant placement.

79 A major cause of apical periodontitis after endodontic treatment is the bacterial infiltration which

80 ant and adjacent tooth and/or with time from endodontic treatment of adjacent tooth to implant placem

81 ded in this study (N = 20) were referred for endodontic treatment of mature teeth with apical lesions

82 n of human dental pulp stem cells (DPSCs) in endodontic treatment.

83 special attention and careful assessment for endodontic treatment.

84 for dentin/pulp tissue engineering in future endodontic treatment.

85 for dentin/pulp tissue engineering in future endodontic treatment.