ライフサイエンスコーパス: endolymph

1 hlea that maintains the ionic composition of endolymph.

2 ar epithelial cells that secretes HCO3- into endolymph.

3 the saccule was merely derived from cochlear endolymph.

4 cium for crystallization in the calcium-poor endolymph.

5 se of the drastic reduction in the volume of endolymph.

6 in maintaining the ionic composition of the endolymph.

7 n the recycling of potassium to the cochlear endolymph.

8 efractive index greater than the surrounding endolymph.

9 ir bundles above the low level found in bulk endolymph.

10 ation of 70 microM similar to that in turtle endolymph.

11 pproximately the concentration found in frog endolymph.

12 at isk mediates potassium secretion into the endolymph.

13 s of the reticular lamina separate K(+)-rich endolymph and Na(+)-rich perilymph.

14 canal using known physical properties of the endolymph and perilymph in three diverse conditions: sur

15 ally distinct extracellular cochlear fluids, endolymph and perilymph, are separated by tight junction

16 ochlear duct are thought to compartmentalize endolymph and provide structural support for the auditor

17 ents involved in generation of the K(+)-rich endolymph and the endocochlear potential.

18 upporting cells in K(+ )recycling within the endolymph, and its apical turn location may explain the

19 a, where it is involved in the generation of endolymph, and on the basolateral membrane in the distal

20 Endolymph collapse in the lte/nkcc1 mutant shows distinc

21 ia lining the scala media, thus defining the endolymph compartment.

22 The results demonstrate that endolymph containing high K+, 50 microns Ca2+ and low Mg

23 The effects of perfusion of 50 microM Ca2+ endolymph depended on the BAPTA concentration of the ele

24 dating zebrafish as a model for the study of endolymph disorders.

25 f indentation and at low frequencies compels endolymph displacement along the canal.

26 , its displacement should be proportional to endolymph displacement.

27 at the midline, while in mutant adults, the endolymph-filled lumen of the semicircular canals is sev

28 ntrast, the PN cupula is probably coupled to endolymph flow by viscous forces, in which case its disp

29 ocation of the PN, which allows it to sample endolymph flow from both vertical semicircular canals.

30 sts EphB2 may regulate ionic homeostasis and endolymph fluid production through macromolecular associ

31 o inner ear sensory organs, dysplasia of the endolymph fluid space, and abnormally formed otoconia (e

32 occurring ionic currents in the labyrinthine endolymph fluid.

33 um in the local regulation of the [Na(+)] of endolymph for maintenance of our sense of balance.

34 epends upon regulation of its luminal fluid, endolymph, for normal transduction of linear acceleratio

35 In mammals, disruptions to endolymph homeostasis can result in either collapse or d

36 phatic sac (ES) and duct (ED), which mediate endolymph homeostasis.

37 behavioral dysfunction and signs of abnormal endolymph homeostasis.

38 We emphasize that the unique, dual role of endolymph in the delivery of both ionic current and flui

39 disorder associated with an accumulation of endolymph in the membranous labyrinth in the inner ear.

40 Previous studies suggested that endolymph in the saccule was merely derived from cochlea

41 temporal bones have found an accumulation of endolymph in the scala media of the cochlea.

42 proteins, as well as tight regulation of the endolymph ionic environment.

43 The endolymph is an extracellular fluid with an atypical com

44 on and volume of the inner ear luminal fluid endolymph is essential for normal hearing and balance.

45 In the mammalian inner ear, endolymph is produced and resorbed by a complex series o

46 Endolymph is the specialised extracellular fluid present

47 (56 +/- 11 microM), a value similar to bulk endolymph levels.

48 cells, which enwrap JO neuronal dendrites in endolymph-like compartments.

49 amenable model to investigate generation of endolymph-like extracellular compartments.

50 the apical surface was bathed in artificial endolymph, many hair bundles exhibited spontaneous oscil

51 lus the unique ionic microenvironment of the endolymph near its epithelium likely contribute to the s

52 tly by external factors such as innervation, endolymph, normal mechanical stimulation, or an intact o

53 enovirus with the Math1 gene insert into the endolymph of the mature guinea pig cochlea results in Ma

54 o mechanisms by infusing substances into the endolymph or perilymph of the chinchilla's cochlea and t

55 ain proper endolymph pH, implicate ATP6B1 in endolymph pH homeostasis and in normal auditory function

56 r active proton secretion to maintain proper endolymph pH, implicate ATP6B1 in endolymph pH homeostas

57 essed in and controls the development of the endolymph-producing cells of the inner ear: the strial m

58 EphB2 is expressed in the endolymph-producing dark cells in the inner ear epitheli

59 a role in the development and/or function of endolymph-producing epithelia.

60 n of several genes thought to play a role in endolymph production is downregulated, including the sod

61 e they are involved in NaCl reabsorption and endolymph production, respectively.

62 y NHE9 in hair bundles, exploits the high-K+ endolymph, responds only to pH imbalance across the bund

63 hasized the prominent role of K+ channels in endolymph secretion and mechanical transduction.

64 the inner ear consistent with a decrease in endolymph secretion.

65 the extracellular Ca2+ concentration in the endolymph surrounding the hair bundles is < 100 microM,

66 Lorentz force mechanism acting on vestibular endolymph that acts to stimulate semicircular canals.

67 ), is associated with abnormal production of endolymph, the fluid bathing sensory hair cells.

68 In artificial endolymph, the stiffness gradient for the transversal co

69 y pathway for K(+) that is secreted into the endolymph, thus playing a critical role in hearing.

70 e its displacement should be proportional to endolymph velocity.

71 lin-deficient mice when generation of normal endolymph was inhibited by a concomitant deletion of the

72 ons Ca2+, a concentration similar to that in endolymph, which bathes the hair bundles in vivo.