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1  ears often lack a distinct saccule, and the endolymphatic duct and common crus are invariably fused.
2 , the most common phenotype being failure of endolymphatic duct and common crus formation, accompanie
3  central vestibule, a coiled cochlea, and an endolymphatic duct and sac allowing fluid balance with t
4 e loss of dorsal otic structures such as the endolymphatic duct and sac is attributable to the downre
5 cular canals were the most sensitive and the endolymphatic duct and sac most resistant to exogenous N
6 cells in the thyroid gland as well as in the endolymphatic duct and sac of the inner ear, whereas pre
7 ed effects of VHL deficiency on "tumor-free" endolymphatic duct and sac of VHL patients.
8 alysis of clinically uninvolved "tumor-free" endolymphatic duct and sac tissues of VHL patients, we d
9 lar canals, utricle, saccule, cochlear duct, endolymphatic duct and sac, and neurons of the eighth cr
10   Pds expression was detected throughout the endolymphatic duct and sac, in distinct areas of the utr
11 2 normally promotes dorsal fates such as the endolymphatic duct and semicircular canals by positively
12                               Absence of the endolymphatic duct and swelling of the membranous labyri
13 e ventromedial otic vesicle, the base of the endolymphatic duct and the fusion plates of the semicirc
14 ivatives, including the semicircular canals, endolymphatic duct and utricle, are malformed or absent.
15 5 revealed that mtl and bsd homozygotes lack endolymphatic ducts and semicircular canals and have sho
16 inner ears lack a non-sensory structure, the endolymphatic duct, and the membranous labyrinth is poor
17 r work has shown that sensory organs and the endolymphatic duct each arise near the boundaries of bro
18 ntire dorsal rim of the otic cup becomes the endolymphatic duct (ED), while the posteroventral rim be
19 (facial nerve exiting through jugular canal, endolymphatic ducts exiting posterior to the skull roof)
20 ing that leads to dorsal otic patterning and endolymphatic duct formation.
21 in the hindbrain from otic induction through endolymphatic duct outgrowth, and in the prospective neu
22 hat may be critical for the specification of endolymphatic duct outgrowth.
23 selected topographical sites within the CNS, endolymphatic duct/sac epithelia are preferentially and
24 he primary effect of VHL deficiency on human endolymphatic duct/sac epithelium seems to be the genera
25 arise within the intraosseous portion of the endolymphatic duct/sac, the vestibular aqueduct.
26 be placed in the intraosseous portion of the endolymphatic duct/sac.

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