ライフサイエンスコーパス: endometrial adenocarcinoma

コーパス検索結果 (１語後でソート)

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1 y medications (NSAID), and acetaminophen and endometrial adenocarcinoma.

2 ease states such as renal cell carcinoma and endometrial adenocarcinoma.

3 ls, derived from a well-differentiated human endometrial adenocarcinoma.

4 Five had thymomas and one had an endometrial adenocarcinoma.

5 with E2 developed invasive endometrioid-type endometrial adenocarcinoma.

6 allele spontaneously develop highly invasive endometrial adenocarcinomas.

7 hich are premalignant precursors of invasive endometrial adenocarcinomas.

8 One patient with endometrial adenocarcinoma achieved a complete response

9 hysterectomies with both well-differentiated endometrial adenocarcinoma and adjacent (normal or abnor

10 However, these mice did not develop invasive endometrial adenocarcinoma, and instead succumbed premat

11 -menopausal endometrium, well-differentiated endometrial adenocarcinoma, and poorly differentiated ma

12 gresses to endometrial carcinoma in situ and endometrial adenocarcinoma as evidenced by myometrial in

13 These lesions progressed to invasive endometrial adenocarcinomas as early as 9 months of age

14 by RNA interference in a well differentiated endometrial adenocarcinoma cell line (Ishikawa) stimulat

15 ne responsiveness, and differentiation of an endometrial adenocarcinoma cell line (Ishikawa).

16 cell line HCT-116 and sublines of the human endometrial adenocarcinoma cell line HEC59.

17 ter expression in the Ishikawa human uterine endometrial adenocarcinoma cell line.

18 tor-dependent p38 activation by estrogens in endometrial adenocarcinoma cells and in vitro and in viv

19 ell proliferation and invasive phenotypes in endometrial adenocarcinoma cells, where it helps maintai

20 ked highly invasive and sometimes metastatic endometrial adenocarcinomas closely resembling those obs

21 Committee on Cancer stage IA-C node-negative endometrial adenocarcinoma comprised the study populatio

22 tumors are hypothesized to develop from the endometrial adenocarcinoma (EAC) through epithelial-mese

23 Normal proliferative, EIN, and malignant (endometrial adenocarcinoma) endometrial tissues were imm

24 Endometrial adenocarcinomas exhibited changes in the lev

25 ters early uterine morphogenesis and induces endometrial adenocarcinomas in adults.

26 Endometrial adenocarcinoma is the most common gynecologi

27 currently shown, treatment of cultured human endometrial adenocarcinoma (Ishikawa) cells with resvera

28 alysis to date of adjuvant RT in early stage endometrial adenocarcinoma, our study reveals a statisti

29 In clinical specimens of endometrial adenocarcinoma, PME-1 levels were increased

30 f adjuvant radiation therapy (RT) in stage I endometrial adenocarcinoma remains controversial despite

31 l endometrial proliferation and, ultimately, endometrial adenocarcinoma, the fourth most common cance

32 ncer cell lines from multiple origins and in endometrial adenocarcinoma tumors, there appears to be n

33 These tumors were poorly differentiated endometrial adenocarcinomas with prominent nuclear atypi

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