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1 nths of age, the absence of Mig-6 results in endometrial hyperplasia.
2 ma, prostatic intraepithelial neoplasia, and endometrial hyperplasia.
3 ortant, since unexpected bleeding may signal endometrial hyperplasia.
4 the mechanism by which tamoxifen results in endometrial hyperplasia.
5 accompanied by an unacceptably high rate of endometrial hyperplasia.
6 ocrine therapy and reduces tamoxifen-induced endometrial hyperplasia, a strategy with potential clini
7 and was restricted primarily to the atypical endometrial hyperplasia (AEH) type with coexisting endom
9 lost in a high percentage of cases of human endometrial hyperplasia and adenocarcinoma, which are li
10 and phosphorylated ER-alpha was increased in endometrial hyperplasia and atypical hyperplasia compare
12 cated as a mediator of the increased risk of endometrial hyperplasia and cancer resulting from the us
13 Cables mutant mice are viable, but develop endometrial hyperplasia and carcinoma in situ at a young
15 osity in stromal APC is sufficient to induce endometrial hyperplasia and endometrial carcinogenesis b
16 e cellular proliferation in vivo in atypical endometrial hyperplasia and endometrial endometrioid ade
18 he murine uterus leads to the development of endometrial hyperplasia and estrogen-induced endometrial
20 oxifen include vaginal endometrial bleeding, endometrial hyperplasia, and cancer, conditions associat
22 ession on a TMA, including a large series of endometrial hyperplasia, atypical hyperplasia, and adeno
25 nt in endometrial tumorigenesis we evaluated endometrial hyperplasia (EH) characterized as simple, co
26 en and progesterone bear on the incidence of endometrial hyperplasia (EH), a noninvasive proliferatio
32 ogy clinics in Manchester, UK, with atypical endometrial hyperplasia or endometrial endometrioid aden
33 may present with vaginal bleeding caused by endometrial hyperplasia or uterine cancer as a result of
34 R+ putative endometrial precancers (atypical endometrial hyperplasias) progress to RER+ carcinomas, w
35 ovarian and endometrial tumors as well as in endometrial hyperplasia, signifying the importance of PR
39 tumors of uncertain malignant potential, or endometrial hyperplasia) were 0.67% (n = 257) (95% CI, 0
40 carcinoma, endometrial polyps, and atypical endometrial hyperplasias, whereas normal and anovulatory
41 e whether PTEN mutations also are present in endometrial hyperplasias, which are premalignant precurs
42 ved human specimens of normal endometrium; 7 endometrial hyperplasia with or without atypia; 32 endom
43 was no higher frequency of PTEN mutations in endometrial hyperplasias with atypia (6 of 32; 19%) rela
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