ライフサイエンスコーパス: endometrium

1 riate for detecting bacteria adherent to the endometrium.

2 in latent precancers of normal premenopausal endometrium.

3 endometrial cancer, matched blood and normal endometrium.

4 greater degree in the ectocervix than in the endometrium.

5 nce that VEGF has pleiotropic effects in the endometrium.

6 ded upon its first contact with the maternal endometrium.

7 e pro-estrogenic effects of tamoxifen on the endometrium.

8 carcinoma of the breast, prostate, colon and endometrium.

9 trioid endometrial cancer compared to normal endometrium.

10 which may be relevant to pathologies of the endometrium.

11 nd therefore do not implant into the uterine endometrium.

12 ltrastructural hallmark of the postovulation endometrium.

13 ation of epithelial and stromal cells in the endometrium.

14 e not a prominent feature of the nonpregnant endometrium.

15 yonic activities with the developing uterine endometrium.

16 ficient detoxification of tamoxifen in human endometrium.

17 m the recruitment of blood NK cells into the endometrium.

18 d in endometrial tumors compared with normal endometrium.

19 ng W21, C98, and V102 are expressed in sheep endometrium.

20 urce of cells that differentiate to form the endometrium.

21 Incident invasive cancer of the ovary and endometrium.

22 ausative role in malignant transformation of endometrium.

23 l hyperplasia compared with normal secretory endometrium.

24 seven patients with benign neoplasia of the endometrium.

25 eases in stromal cell Ang-1 in LTPOC-exposed endometrium.

26 eus in PE, and only nuclear in the secretory endometrium.

27 ar hyperplasia (n = 25) compared with normal endometrium.

28 ostic marker for patients with cancer of the endometrium.

29 r to evaluate the receptive functions of the endometrium.

30 ssion in proliferative (estrogen-stimulated) endometrium.

31 ogenesis and endothelial repair in the human endometrium.

32 menopausal endometrium than in premenopausal endometrium.

33 distal colon, prostate, pancreas, ovary, and endometrium.

34 motypic guidance of trophoblast cells in the endometrium.

35 he uterus, raloxifene does not stimulate the endometrium.

36 hould prompt an aggressive evaluation of the endometrium.

37 melanoma, and carcinoma of the prostate and endometrium.

38 initiation of decidualization in the baboon endometrium.

39 ied in a search for genes expressed in human endometrium.

40 g cancer promoting effects on the breast and endometrium.

41 ly linked to tumorigenesis in the breast and endometrium.

42 F is confined predominantly to the secretory endometrium.

43 tamoxifen might be a tumor promoter in human endometrium.

44 olving matrix remodeling such as the cycling endometrium.

45 generate a 3D rendering of the cycling human endometrium.

46 d to abrogation of AKT activation within the endometrium.

47 ificant for clear cell adenocarcinoma of the endometrium.

48 ions compared with the peritoneum or eutopic endometrium.

49 d-type females and exhibited a thick uterine endometrium.

50 ult in detectable DNA damage or apoptosis in endometrium.

51 ved in the higher EDA exon inclusion rate in endometrium.

52 B during the cyclic development of the human endometrium.

53 tients with endometriosis compared to normal endometrium.

54 ons in hormonal milieu similar to the native endometrium.

55 (1.32, 1.24-1.40), breast (1.17, 1.15-1.19), endometrium (1.19, 1.13-1.24), ovary (1.17, 1.11-1.23),

56 stologic examination, 16 had a proliferative endometrium, 12 had a secretory endometrium, six had pol

57 tmenopausal (27.3) compared to premenopausal endometrium (4.9) mainly as a result of lower ER-beta ex

58 lpha in 4 archived human specimens of normal endometrium; 7 endometrial hyperplasia with or without a

59 e than 90%; cervix, 40% and 70%-80%; uterus (endometrium), 80% and more than 90%; bladder, 30% and 50

60 , and ultrasonography revealed a 10 mm thick endometrium, a poorly visualised left ovary, and a 2 cm

61 Benign cycling endometrium, a rapidly proliferating tissue, features po

62 tissue-adherent bacteria compared to that in endometrium absent of biofilm.

63 road map connecting the blastocyst with the endometrium across species is diverse (1) and not fully

64 Here, we show that loss of Pten in the mouse endometrium activates Akt and results in increased phosp

65 graphy and histopathologic evaluation of the endometrium after dilation and curettage or after hyster

66 giogenic mediator in the regeneration of the endometrium after menses and as a vasodilator to promote

67 Regeneration of the endometrium after menstruation requires a rapid and high

68 Progesterone protects the endometrium against the hyperplastic effects of estradio

69 rains of fibroblasts from the myometrium and endometrium also demonstrated heterogeneous Thy 1 expres

70 oblast and extracellular matrix (ECM) in the endometrium anchors the embryo to the uterine wall.

71 n requires communication between a receptive endometrium and a healthy blastocyst.

72 BP-1) is a secretory product of decidualized endometrium and a major constituent of amniotic fluid.

73 sets of sex-steroid-related tumors including endometrium and breast carcinomas that are associated wi

74 luid, PrP-Sc was detected only in caruncular endometrium and cotyledonary chorioallantois of pregnant

75 eable population of uILC3s is found in human endometrium and decidua, which are mostly NCR(+) and par

76 cin 1 (PROK1), is expressed in the receptive endometrium and during early pregnancy.

77 opian tube and cervical tissues, followed by endometrium and ectocervix.

78 Here, we show that both normal human endometrium and endometrial cancers express the receptor

79 erone-resistant molecular environment of the endometrium and endometriosis lesions; and (v) restores

80 analysis showed chlamydial inclusions in the endometrium and fetal membranes.

81 r transgene expression specifically in mouse endometrium and found that endometrial-specific VEGF ove

82 ve been noted in cancers of the prostate and endometrium and in glioblastoma multiforme, among many o

83 hows is transcribed in normal and neoplastic endometrium and in MCF-7 cells, forms a stable RNA quadr

84 ntral target for growth regulation of normal endometrium and in the pathogenesis of ECA.

85 ight was accompanied by proliferation of the endometrium and induction of progesterone receptor (PgR)

86 ic cells and luminal epithelial cells of the endometrium and is implicated in the initial attachment

87 rm by all the tissues except estrous uterine endometrium and lactating mammary gland indicates RUSH p

88 ial carcinoma compared with levels in normal endometrium and low-grade tumors.

89 onic day 9 (e9) and in decidualizing uterine endometrium and myometrial smooth muscle at even earlier

90 miological feature of cancers of the breast, endometrium and ovary is the sharp slowing down in their

91 Akt1 deficiency had a profound effect on endometrium and prostate neoplasia, two types of human c

92 rds the mouse embryo by invading the uterine endometrium and remodelling the maternal vasculature.

93 he ratios of RUSH mRNA isoforms from uterine endometrium and testis.

94 e conducive to HIV-1 replication than is the endometrium and that IL-6 enhances HIV-1 transcription a

95 tant role in the malignant transformation of endometrium and that Lkb1 loss promotes a highly invasiv

96 logue between the hormonally primed maternal endometrium and the free-floating blastocyst.

97 eepithelialization in both the postmenstrual endometrium and the mouse uterus after decidual breakdow

98 been shown to be induced by tamoxifen in the endometrium and to be a growth factor for endometrial en

99 The endometrium and uterus also arise from the coelomic epit

100 factor 1 (SRSF1) is more highly expressed in endometrium and, using RNA interference, that it is invo

101 entiated tissue, or during remodeling of the endometrium, and a homologous gene, derailed, is known t

102 pausal endometrium, reduced in premenopausal endometrium, and absent or reduced in a majority of prim

103 tecture and cellular morphology of the mouse endometrium, and allows for the recovery of high-quality

104 blastocyst interacts with and regulates the endometrium, and endometrial fluid secreted by the endom

105 but variably expressed by normal endocervix, endometrium, and fallopian tube (60, 64, and 29% of spec

106 In positive endocervix, endometrium, and fallopian tube specimens, HD-5 was loca

107 n in normal human tissues, including cycling endometrium, and in breast carcinomas, tissues in which

108 ncers in the oesophagus, colorectum, breast, endometrium, and kidney.

109 than one primary cancer of the colorectum or endometrium, and mean age of presentation) and performs

110 metrium, six had polyps, two had an inactive endometrium, and one had carcinoma.

111 dent cancers, including those of the breast, endometrium, and ovary.

112 pig inclusion conjunctivitis in the cervix, endometrium, and oviducts at various times following a p

113 pathway regulates innate immunity within the endometrium, and that isoprenoids are regulatory molecul

114 sed expression of Cyr61 compared with normal endometrium, and this lowered expression may provide the

115 expressed in epithelial cells of the breast, endometrium, and thymus, in tingible body macrophages of

116 entation, but their functions in nonpregnant endometrium are not understood.

117 IL-15 levels in normal endometrium are progesterone-responsive.

118 d into endometriotic lesions but not eutopic endometrium, as revealed by flow cytometry and immunohis

119 etected only in the epithelia of the uterine endometrium, as well as epithelia of the oviduct, cervix

120 mRNA of IL-22R1 was detected in pregnant endometrium at levels similar to other equine epithelia.

121 Of 88 women, 37 had a benign-appearing endometrium at transvaginal hysterosonography; at histol

122 n distinct, changing patterns in the uterine endometrium, at the decidual boundary, in the decidual v

123 osphorylation and activation of Stat3 in the endometrium before implantation.

124 ) in the number of inflammatory cells in the endometrium between areas with and without tissue-adhere

125 d in endometrial cancer compared with normal endometrium but the underlying mechanisms are not well u

126 clic differentiation and apoptosis in normal endometrium, but its role in endometrial carcinogenesis

127 plasmid-free C. muridarum directly into the endometrium by intrauterine inoculation.

128 Infection of the endometrium by Neisseria gonorrhoeae is a pivotal stage

129 Colonization of the endometrium by pathogenic bacteria ascending from the lo

130 t PAPPA is essential to maintain a receptive endometrium by up-regulating N-fucosylation, which is a

131 ages present in the peritoneum and in menses endometrium can contribute to the inflammatory microenvi

132 We compare dermis and endometrium capacities to support trophoblast invasion,

133 uman cancers of the prostate, breast, ovary, endometrium, cervix, and bladder, and a region of deleti

134 o 6 was observed in fallopian tubes, uterine endometrium, cervix, and ectocervix.

135 Interestingly, increased apoptosis in human endometrium coincides with the implantation window.

136 lignancies, including cancers of the breast, endometrium, colon, and prostate.

137 thin its coding region, in MMP tumors of the endometrium, colon, and stomach (28, 62, and 44%, respec

138 ast cells and fibronectin as invasion of the endometrium commences.

139 eveal an increase of SIRT1 expression in the endometrium compared to control mice.

140 tamoxifen can have estrogenic effects on the endometrium; consensus opinion is that tamoxifen increas

141 ioallantois, allantoic fluid, and caruncular endometrium contained higher levels of PrP-C than did in

142 The uterine endometrium coordinates a wide spectrum of physiologic a

143 could affect the angiogenic potential of the endometrium, creating a feed forward loop resulting in m

144 pared to more relevant host human epithelial endometrium-derived HEC-1B and cervix-derived HeLa cells

145 reproductive biology could also be played by endometrium-derived viral particles that influence devel

146 inal hysterosonography was defined as a thin endometrium, diffuse smooth endometrial thickening, or a

147 we report on the composition of uILCs in the endometrium during the luteal phase and in the decidua d

148 account for the increased NK cell numbers in endometrium during the menstrual cycle.

149 The human endometrium (EM) contains macrophages, NK cells, T cells

150 ry epithelial cells and fibroblasts from the endometrium (EM), endocervix (CX) and ectocervix (ECX) s

151 nally within the RT (Fallopian tube, uterine endometrium, endocervix, ectocervix, and vaginal mucosa)

152 Ex vivo organ cultures of endometrium, endometrial cells and peripheral blood mono

153 carcinomas arising from the bladder, cervix, endometrium, esophagus, gallbladder, kidney, liver, and

154 neoplasms, such as those in the thyroid and endometrium, exhibit more than one pattern of differenti

155 ons have shown that while macrophages in the endometrium express adrenomedullin at a low level, endom

156 a of the colon, prostate, ovary, breast, and endometrium, express high levels of fatty acid synthase

157 Our data reveal that endometrium expresses a higher rate of the fibronectin (

158 on in the upper tissues (fallopian tubes and endometrium), followed by cervix and ectocervix.

159 (PR) and its coregulators prepares the human endometrium for receptivity to embryo implantation and m

160 d to matched eutopic patient samples as well endometrium from healthy controls.

161 ed significantly increased staining in human endometrium from late secretory and menstrual phases.

162 eacetylase and gene silencer, in the eutopic endometrium from women with endometriosis throughout the

163 ic endometriotic lesions and matched eutopic endometrium from women with endometriosis.

164 cancer cells, including breast, lung, ovary, endometrium, gastric, and melanoma, which could be rescu

165 In eutopic endometrium, GLI1 expression is reduced in women with en

166 ted effects in the breast, bone, and uterine endometrium has been described, a frequently overlooked

167 Endometrium has its characteristic DNA methylation profi

168 Previous transcriptome studies of the human endometrium have revealed hundreds of simultaneously up-

169 cer showed increased risk for cancers of the endometrium (HR, 1.76; 95% CI, 1.34-2.31), breast (HR, 1

170 ks (AH v SH, CH, or disordered proliferative endometrium [ie, equivocal EH]) from the case-control an

171 ive neurons, the cervix, the vagina, and the endometrium in 5- to 400-fold higher numbers when cultur

172 chemokines CXCL10 and/or CXCL11 within human endometrium in 85% of patient samples tested.

173 e, the first study to compare ectocervix and endometrium in a tissue explant model of HIV-1 infection

174 Regeneration of the endometrium in each menstrual cycle is required for repr

175 ler (uNK) cells are abundant in decidualized endometrium in early pregnancy; they surround spiral art

176 itro, as well as N-fucosylation level of the endometrium in pregnant mice.

177 esignated as SERPINA14, are expressed in the endometrium in response to progesterone.

178 responses and also is upregulated within the endometrium in response to the developing embryo during

179 EMB was used to monitor the endometrium in the majority (95%) of breast cancer patie

180 a physiological effect of CG on the uterine endometrium in vivo and suggest that the primate blastoc

181 ed electrogenic glucose transport across the endometrium in wild type (Slc5a1 (+/+)) but not in SGLT1

182 kidney, pancreas, esophageal adenocarcinoma, endometrium) in CRC survivors, and compared associations

183 es the neoplastic effect of Pten loss in the endometrium, in contrast to complete estrogen depletion.

184 d with increased expression in the quiescent endometrium, indicate that this homeodomain gene is invo

185 ight support a role for enJSRVs in conceptus-endometrium interactions during the peri-implantation pe

186 rtment, deregulated SGK1 activity in cycling endometrium interferes with embryo implantation, leading

187 e C57BL/6 mice by implantation of autologous endometrium into the peritoneal cavity.

188 The endometrium is a complex, steroid-dependent tissue that

189 Human endometrium is a high dynamic tissue that contains endom

190 ing of the mechanisms of angiogenesis in the endometrium is a major limitation for use of antiangioge

191 rate that hCG-mediated LIF expression in the endometrium is dependent on prior induction of PROK1.

192 Human trophoblast invasion of decidualized endometrium is essential for placentation and is tightly

193 lish whether activation of Stat3 in maternal endometrium is essential for successful implantation.

194 Undesirable stimulation of the breast and endometrium is not apparent.

195 In women, much of the endometrium is shed and regenerated each month during th

196 Endometrium is the inner lining of the uterus which is c

197 The inner uterine lining (endometrium) is a unique tissue going through remarkable

198 f the colon, female breast (postmenopausal), endometrium, kidney (renal cell), and esophagus (adenoca

199 as those originating in ovary, lung, breast, endometrium, kidney, and brain.

200 diverse human tumours including breast, CNS, endometrium, kidney, liver, lung, lymphoid, oesophagus,

201 The Laparoscopic Approach to Cancer of the Endometrium (LACE) trial was a multinational, randomized

202 es such as PTEN within histologically normal endometrium (latent precancers) is an early step in endo

203 hanges the regulation of angiogenesis in the endometrium, likely by reducing angiogenic activity.

204 tragonadal tissues including bone, placenta, endometrium, liver, and blood vessels from a number of m

205 d neoplasms in multiple organs including the endometrium, liver, prostate, gastrointestinal tract, th

206 poplasia characterized by loss of the entire endometrium (luminal and glandular epithelium and stroma

207 ancer cells of the prostate, ovary, bladder, endometrium, lung and melanocytes by Western blot to det

208 lso significant in carcinomas of the kidney, endometrium, lung, breast, bladder, and pancreas.

209 from neoplasms of the breast, ovary, cervix, endometrium, lung, colon, placenta, or hematopoietic sys

210 ere also observed for cancers of the breast, endometrium, lung, kidney, upper aerodigestive tract, li

211 found that the epithelial compartment of the endometrium maintains its epithelial identity during the

212 demonstrate that the high FN content of the endometrium matrix, and not specifically the EDA domain,

213 n cells that support the survival of ectopic endometrium may be an effective therapeutic approach in

214 he tamoxifen-DNA adducts detected in patient endometrium may cause mutations and initiate endometrial

215 The Na+-absorptive function of the endometrium may provide an appropriate environment for s

216 her levels of PrP-C than did intercaruncular endometrium, myometrium, oviduct, ovary, fetal bladder,

217 d significant excess risks of cancers of the endometrium (n = 11; observed rate, 16.1/10,000 person-y

218 arker of eosinophil degranulation) in normal endometrium (n = 20) and endometriosis samples (n = 24)

219 device in preventing uterine changes in the endometrium needs to be assessed in the context of decre

220 We have recently detected TAM-DNA adducts in endometrium obtained from patients treated with TAM and

221 e system, and intrinsic abnormalities in the endometrium of affected women and secreted products of e

222 antly higher levels of IL-33 compared to the endometrium of healthy, fertile controls.

223 nstrated phosphorylation of STAT3 in eutopic endometrium of infertile women with this disorder leadin

224 e detected previously TAM-DNA adducts in the endometrium of women receiving TAM (Shibutani et al., Ca

225 luminal epithelium, but downregulated in the endometrium of women suffering from RPL.

226 NA adducts are mutagenic and detected in the endometrium of women treated with TAM, TAM adducts are s

227 of K-ras mutations has been observed in the endometrium of women treated with TAM.

228 ertheless this population is resident in the endometrium of women who have RM, more than three months

229 6 are coordinately over-expressed in eutopic endometrium of women with endometriosis and likely parti

230 aimed to identify the most stable HKG in the endometrium of women with recurrent implantation failure

231 nto layers (myometrium, junctional zone, and endometrium) of uterine remnants.

232 s characterized by the growth of the uterine endometrium on the surface of organs within the pelvic r

233 Uterine amyloid accumulated in the endometrium, only at the site of placental attachment, c

234 efined as either irregular thickening of the endometrium or an inhomogeneous endoluminal mass.

235 A thin endometrium or diffuse smooth endometrial thickening is

236 associated with proliferative changes of the endometrium, or even endometrial cancer.

237 for carcinomas of the anus, bladder, cervix, endometrium, ovary, penis, prostate, rectum, testis, vag

238 iking reduction of uNK in asoprisnil-treated endometrium (p < 0.001).

239 gnaling between the cloned conceptus and the endometrium, particularly the intercaruncular tissue.

240 lial cells derived from normal proliferative endometrium (PE; n = 10) were dose-dependently and maxim

241 w concomitant expression of the two genes in endometrium, placenta, and stimulated KG1 cells (phenoty

242 y on estrogenic stimulation of the breast or endometrium) precludes recommending long-term use.

243 d (stomach, biliary tract, pancreas, cervix, endometrium, prostate, kidney, bladder, and lymphoma) th

244 sylation of integrin alphaVbeta3, a critical endometrium receptivity biomarker, was up-regulated by P

245 itioned medium of trophoblast cells promoted endometrium receptivity in vitro.

246 However, the relationship between PAPPA and endometrium receptivity, as well as the regulation of N-

247 EMX2OS are abundant in normal postmenopausal endometrium, reduced in premenopausal endometrium, and a

248 e the exact localization of Ang in the human endometrium remains a subject of controversy, we have ad

249 gh uterine papillary serous carcinoma of the endometrium represents only 3% to 4% of endometrial canc

250 The uterine endometrium responds to unopposed estrogen stimulation w

251 ced by the chorionic girdle binds IL-22R1 on endometrium, serving as a mechanism of fetal-maternal co

252 Endothelial cell proliferation analysis in endometrium showed a peak during the late menstrual and

253 In situ staining of human myometrium and endometrium showed heterogeneous staining for Thy 1.

254 fen acts as an agonist in the postmenopausal endometrium, similar to estrogen in the breast, we compa

255 roliferative endometrium, 12 had a secretory endometrium, six had polyps, two had an inactive endomet

256 cancers of the liver, pancreas, gallbladder, endometrium, stomach, kidney, brain (benign), brain (mal

257 type II (Lynch II) with tumors found in the endometrium, stomach, ovary, and upper urinary tract in

258 ization of heparan sulfate and TIMP-3 in the endometrium subjacent to the lumen of the uterus.

259 Proliferative diseases of the endometrium such as endometriosis and cancer are common

260 gen-A11 (MAGE-11) in the mid-secretory human endometrium suggested a novel function in human PR signa

261 T1, 1,514 msec +/- 156; T2, 79 msec +/- 10), endometrium (T1, 1,453 msec +/- 123; T2, 59 msec +/- 1),

262 en procedure, transcervical resection of the endometrium (TCRE), for women with heavy menstrual loss.

263 as more abundant in quiescent postmenopausal endometrium than in premenopausal endometrium.

264 uce specific chemokines in nonpregnant human endometrium that can activate NK cell migration, and sug

265 ion of localized VEGF secretion in the human endometrium that may be necessary for the successful est

266 e hormonally regulated, since in the uterine endometrium, the capacity for CD3+ T cell cytolytic acti

267 Approximately 20% of cancers of the uterine endometrium, the fifth most common cancer of women world

268 Within the endometrium, the most prominent change is that expressio

269 Substituting endometrium, the natural trophoblast target, with dermis

270 ugh VEGF expression has been detected in the endometrium, the relationship between VEGF production, r

271 In asoprisnil-treated endometrium, there is a marked downregulation of stromal

272 In normal human endometrium, this gene was transiently expressed before

273 ctivity by receptor phosphorylation in human endometrium throughout the menstrual cycle.

274 Tumors of the endometrium, thyroid, prostate, and liver were not assoc

275 es, 60 endometrial cancer tissues, 10 normal endometrium tissues from normal healthy controls, and 32

276 a critical regulator of the response of the endometrium to E2 in regulating tissue homeostasis.

277 Excessive and prolonged exposure of the endometrium to estrogens unopposed by progesterone and a

278 ells of the equine placenta migrate into the endometrium to form endometrial cups, dense accumulation

279 to regulate the proinflammatory response of endometrium to IL1B2 during conceptus elongation and att

280 nvade and reorganize vessels of the maternal endometrium to initiate blood flow to the intervillous s

281 trial glands, however, the responsiveness of endometrium to physiological concentrations of PAF is co

282 The human endometrium undergoes regular cycles of synchronous tiss

283 ality in patients with adenocarcinoma of the endometrium (ungrouped P = 0.034).

284 y and adrenal glands, and the liver, kidney, endometrium, uterus, and ovary.

285 have shown that estrogen largely acts on the endometrium via estrogen receptor ERalpha, we generated

286 Failure of maturation of the endometrium was also observed.

287 In addition, endometrium was profiled to identify the communication p

288 and PTEN expression in an individual normal endometrium was seen in 21% of patients, but usually inv

289 Expression in endometrium was the highest during the early secretory p

290 xpression in normal pre- and post-menopausal endometrium, well-differentiated endometrial adenocarcin

291 SAA3 mRNA levels in the uterine endometrium were as high as SAA2 in the liver, yet mass

292 Samples from the endometrium were collected for cytology, histopathology,

293 pes of tamoxifen-DNA adducts detected in the endometrium were repaired with moderate to poor efficien

294 ring a Hand2 knock-out specifically in their endometrium were shown to develop precancerous endometri

295 ophoblasts invading the maternal vessels and endometrium, whereas syncytiotrophoblasts covering troph

296 can be relatively large and have functioning endometrium, which can be associated with pain.

297 ms by which the trophoblast cells invade the endometrium while evading maternal immune destruction ar

298 rs within the vascular zone, myometrium, and endometrium, with greater density in the ovarian and cer

299 s markedly in proliferative versus secretory endometrium, with high expression in proliferative (estr

300 Emx2, and Emx2os are abundant in the uterine endometrium, with sense and antisense transcripts exhibi