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1  that blood and lymphatic vessels within the endomysial and perimysial spaces undergo significant cha
2               Mild inflammatory exudates, in endomysial and perivascular spaces, consisted of lymphoc
3      Group 2: the sensitivities of Simtomax, endomysial and tissue-transglutaminase antibodies were c
4    BACKGROUND & AIMS: Tests to measure serum endomysial antibodies (EMA) and antibodies to tissue tra
5 cted and apparently asymptomatic adults with endomysial antibodies (EmA) benefit from a gluten-free d
6 psy; in 5 countries, HLA, and in one country endomysial antibodies (EMA) were not available.
7 awn for later evaluations of serum IgA-class endomysial antibodies (EMA).
8 noglobulins, IgA/IgG anti-gliadin (AGA), and endomysial antibodies (EMA).
9  serum IgA antigliadin, IgG antigliadin, and endomysial antibodies (EMA).
10                                              Endomysial antibodies and HLA typing were routinely avai
11                                              Endomysial antibodies indicate celiac sprue in asymptoma
12                                          IgA endomysial antibodies tests were associated with high sp
13 of IgA, IgA antitissue transglutaminase, and endomysial antibodies were measured, and a duodenal biop
14 d on analysis of tissue transglutaminase and endomysial antibodies.
15 issue transglutaminase and, if abnormal, for endomysial antibodies.
16 d for IgA anti-tTG, and if positive, for IgA endomysial antibodies.
17 sis of tissue transglutaminase (tTg) levels; endomysial antibody (EMA) levels were analyzed in sample
18  tTG) and, if positive, testing for IgA anti-endomysial antibody (IgA EMA) as a confirmatory test.
19 and deposition of complement causes lysis of endomysial capillaries and muscle ischaemia.
20 delta T cells are cytotoxic against adherent endomysial cardiac cells, mostly macrophages, but not ag
21 re evaluated by measuring serum CK activity, endomysial CD45 immunoreactive inflammation area, endomy
22 with muscle fiber invasion by leukocytes and endomysial collagen deposition, was associated with elev
23 ysial CD45 immunoreactive inflammation area, endomysial collagen III deposition, and hind limb grip s
24 a fibrogenic cytokine immunolocalized in the endomysial connective tissue of pretreatment DM muscles,
25 ents of complement were present diffusely in endomysial connective tissue.
26 often with a prominent B cell component, and endomysial dendritic cells.
27                      Although studies on the endomysial expression of cytokines and cell adhesion mol
28                                     However, endomysial fibrosis (distance between myocytes within bu
29 a severe muscular dystrophy in DKO mice with endomysial fibrosis and ectopic calcification.
30                                       Atrial endomysial fibrosis content was lower and myocyte diamet
31 ontinuity of the epicardial layer because of endomysial fibrosis may reduce its synchronizing effect,
32 confirmed the potential impact of epicardial endomysial fibrosis on AF complexity.
33 cence, obvious foci of severe capillary loss/endomysial fibrosis, and muscle infarcts predicted chron
34 myofiber size, centrally located nuclei, and endomysial fibrosis, in the absence of intracytoplasmic
35 uclei, elevated creatine kinase activity and endomysial fibrosis.
36 muscle fibers, increased central nuclei, and endomysial fibrosis.
37 iac disease was confirmed using an assay for endomysial IgA.
38 found that CD8+ T lymphocytes persist in the endomysial infiltrates in all biopsies during a 19-22 mo
39 ve the strength of some patients, and reduce endomysial inflammation and stressor molecules.
40  with mRNA of ICOS, ICOS-L and the degree of endomysial inflammation as assessed in coded haematoxyli
41 at least two members from each family showed endomysial inflammation, red-rimmed vacuoles, intracellu
42 n expression was compared with the degree of endomysial inflammation.
43   The HIV gag antigen was present on several endomysial macrophages but not within the muscle fibers.
44 omyositis specimens contained C5b-9 reactive endomysial microvessels but none of these or other vesse
45  particle antibodies; immune myopathies with endomysial pathology (IM-EP), illustrated by brachio-cer
46  model predicts that the blood volume in the endomysial space increases 24% and decreases 22% for a 2
47 t only peripheral blood lymphocytes but also endomysial T cells and alters the natural course of the
48 n of certain Vb families was noted among the endomysial T cells with evidence of in situ expansion.
49                    The clonal restriction of endomysial T cells, compared with peripheral blood, was
50 r understanding of TCR gene rearrangement in endomysial T cells, regulation of MHC expression, activi
51 istent, clonally expanded and antigen-driven endomysial T cells, the disease is resistant to immunoth
52 ysed the T-cell receptor (TCR) gene usage by endomysial T lymphocytes in three sequential muscle biop
53  putative antibody may not be limited to the endomysial vessels but may also involve the blood vessel

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