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1 that blood and lymphatic vessels within the endomysial and perimysial spaces undergo significant cha
4 BACKGROUND & AIMS: Tests to measure serum endomysial antibodies (EMA) and antibodies to tissue tra
5 cted and apparently asymptomatic adults with endomysial antibodies (EmA) benefit from a gluten-free d
13 of IgA, IgA antitissue transglutaminase, and endomysial antibodies were measured, and a duodenal biop
17 sis of tissue transglutaminase (tTg) levels; endomysial antibody (EMA) levels were analyzed in sample
18 tTG) and, if positive, testing for IgA anti-endomysial antibody (IgA EMA) as a confirmatory test.
20 delta T cells are cytotoxic against adherent endomysial cardiac cells, mostly macrophages, but not ag
21 re evaluated by measuring serum CK activity, endomysial CD45 immunoreactive inflammation area, endomy
22 with muscle fiber invasion by leukocytes and endomysial collagen deposition, was associated with elev
23 ysial CD45 immunoreactive inflammation area, endomysial collagen III deposition, and hind limb grip s
24 a fibrogenic cytokine immunolocalized in the endomysial connective tissue of pretreatment DM muscles,
31 ontinuity of the epicardial layer because of endomysial fibrosis may reduce its synchronizing effect,
33 cence, obvious foci of severe capillary loss/endomysial fibrosis, and muscle infarcts predicted chron
34 myofiber size, centrally located nuclei, and endomysial fibrosis, in the absence of intracytoplasmic
38 found that CD8+ T lymphocytes persist in the endomysial infiltrates in all biopsies during a 19-22 mo
40 with mRNA of ICOS, ICOS-L and the degree of endomysial inflammation as assessed in coded haematoxyli
41 at least two members from each family showed endomysial inflammation, red-rimmed vacuoles, intracellu
43 The HIV gag antigen was present on several endomysial macrophages but not within the muscle fibers.
44 omyositis specimens contained C5b-9 reactive endomysial microvessels but none of these or other vesse
45 particle antibodies; immune myopathies with endomysial pathology (IM-EP), illustrated by brachio-cer
46 model predicts that the blood volume in the endomysial space increases 24% and decreases 22% for a 2
47 t only peripheral blood lymphocytes but also endomysial T cells and alters the natural course of the
48 n of certain Vb families was noted among the endomysial T cells with evidence of in situ expansion.
50 r understanding of TCR gene rearrangement in endomysial T cells, regulation of MHC expression, activi
51 istent, clonally expanded and antigen-driven endomysial T cells, the disease is resistant to immunoth
52 ysed the T-cell receptor (TCR) gene usage by endomysial T lymphocytes in three sequential muscle biop
53 putative antibody may not be limited to the endomysial vessels but may also involve the blood vessel
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