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1 spread of the branch points directly with T4 endonuclease VII.
2 in the phosphodiester cleavage mechanism of endonuclease VII.
3 xyl radicals, KMnO4, the junction resolvases endonuclease VII and RuvC, and RuvC activation of KMNO4
4 he rate of resolution of the Hol75 DNA by T4 endonuclease VII and T7 endonuclease I, two enzymes know
6 rly and middle genes, but two late proteins, endonuclease VII and terminase, are uniquely important i
8 tic lysyl-tRNA synthetases, bacteriophage T4 endonuclease VII, and several uncharacterized proteins.
12 e accumulation of blocked forks increased in endonuclease VII-deficient infections, suggesting that e
14 aracter is observed between Hjc and T4-phage endonuclease VII despite a complete lack of structural h
15 uding nondenaturing PAGE, Ferguson analysis, endonuclease VII digestion, and hydroxyl radical autofoo
19 ication reaction is only partly dependent on endonuclease VII (gp49), suggesting that either another
22 f the cruciform by the junction resolvase T4 endonuclease VII is independent of PARP-1, which indicat
23 imer of active enzyme and an inactive mutant endonuclease VII leads to the formation of nicked circul
24 ber of other DNA junction-resolving enzymes, endonuclease VII of bacteriophage T4 binds to a four-way
26 n low-GC Gram-positive bacteria and AQUIFEX: Endonuclease VII of phage T4 is shown to serve as a stru
27 rrelation spectroscopy that purified T4 gp49 endonuclease VII resolvase can release DNA compression i
29 Fourth, mutations that inactivate UvsW and endonuclease VII (which cleaves DNA branches) synergisti
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