ライフサイエンスコーパス: endoplasmic

1 mplex signaling sequence involving reticulum endoplasmic destalilization which leads to Ca(2+) rise,

2 signaling in many cell types by sensing low endoplasmic reticular Ca(2+) levels and then coupling to

3 Down regulation of dStim, the endoplasmic reticular calcium sensor and a principal com

4 injury marker), local caspase-3/7, and local endoplasmic reticular stress-related genes.

5 s the cell's protein folding capacity in the endoplasmic reticulum (ER) according to need.

6 Insufficient folding capacity of the endoplasmic reticulum (ER) activates the unfolded protei

7 acidification disturb protein folding in the endoplasmic reticulum (ER) and activate the Unfolded Pro

8 ins with folding problems are trapped in the endoplasmic reticulum (ER) and are eventually degraded i

9 Although alterations in both endoplasmic reticulum (ER) and cytosolic free calcium le

10 l is accompanied by reduced juxtaposition of endoplasmic reticulum (ER) and mitochondria as well as e

11 Lipid exchange between the endoplasmic reticulum (ER) and peroxisomes is necessary

12 BiP is the only Hsp70 chaperone in the endoplasmic reticulum (ER) and similar to other Hsp70s,

13 in Gn colocalizes and accumulates within the endoplasmic reticulum (ER) and the transport of Gn from

14 antibodies, biosynthetic substrates to fuel endoplasmic reticulum (ER) biogenesis, and additional ca

15 tes two Brassicaceae-specific traits, namely endoplasmic reticulum (ER) body formation and induction

16 Thapsigargin (Tg) blocks the sarco/endoplasmic reticulum (ER) Ca(2+)-ATPase (SERCA), disrup

17 Unfavorable redox conditions in the endoplasmic reticulum (ER) can decrease the capacity for

18 The endoplasmic reticulum (ER) consists of the nuclear envel

19 Peroxisomes (POs) and the endoplasmic reticulum (ER) cooperate in cellular lipid m

20 Electron microscopic observation revealed endoplasmic reticulum (ER) dilatation, suggestive of ER

21 l nutrient requiring tight constraint in the endoplasmic reticulum (ER) due to its uniquely challengi

22 The turnover of endoplasmic reticulum (ER) ensures the correct biologica

23 Although the endoplasmic reticulum (ER) extends throughout axons and

24 Disturbances in endoplasmic reticulum (ER) homeostasis create a conditio

25 Native cargo proteins exit the endoplasmic reticulum (ER) in COPII-coated vesicles, whe

26 ce mechanism that ensures homeostasis of the endoplasmic reticulum (ER) in eukaryotes.

27 ficking by assembling onto subdomains of the endoplasmic reticulum (ER) in two layers to generate car

28 The endoplasmic reticulum (ER) integral membrane protein VAP

29 The endoplasmic reticulum (ER) is a network of tubules and s

30 The endoplasmic reticulum (ER) is a single organelle in euka

31 Here we report that the endoplasmic reticulum (ER) is asymmetrically partitioned

32 y, alteration of the folding capacity of the endoplasmic reticulum (ER) is becoming a common patholog

33 Glycosylation in the endoplasmic reticulum (ER) is closely associated with pr

34 Homotypic membrane fusion of the endoplasmic reticulum (ER) is mediated by a class of dyn

35 folding, and disulfide formation within the endoplasmic reticulum (ER) is poorly understood.

36 The endoplasmic reticulum (ER) is the largest cellular membr

37 Cholesterol biosynthesis in the endoplasmic reticulum (ER) is tightly controlled by mult

38 The endoplasmic reticulum (ER) mediates the folding, maturat

39 , is degraded when cholesterol levels in the endoplasmic reticulum (ER) membrane are high, but the si

40 t be transported across or inserted into the endoplasmic reticulum (ER) membrane by the ER protein tr

41 nchoring and the compartmentalization of the endoplasmic reticulum (ER) membrane confine protein depo

42 n-enveloped polyomavirus SV40 penetrates the endoplasmic reticulum (ER) membrane to reach the cytosol

43 tor tyrosine kinases are mislocalized in the endoplasmic reticulum (ER) of AML and play an important

44 Contacts between endosomes and the endoplasmic reticulum (ER) promote endosomal tubule fiss

45 The extended synaptotagmins (E-Syts) are endoplasmic reticulum (ER) proteins that bind the plasma

46 ween plasma membrane P/Q Ca(2+) channels and endoplasmic reticulum (ER) ryanodine receptors and anoth

47 n sensing the depletion of (Ca(2+)) from the endoplasmic reticulum (ER) store, organizes as puncta th

48 It induced endoplasmic reticulum (ER) stress and activated the prot

49 gulation of pre-rRNA level led to attenuated endoplasmic reticulum (ER) stress and cell death.

50 study, we focused on a relationship between endoplasmic reticulum (ER) stress and cGVHD, and aimed t

51 s studies have suggested that ORMDL3 induces endoplasmic reticulum (ER) stress and production of the

52 Endoplasmic reticulum (ER) stress arises from accumulati

53 gated the unfolded protein response (UPR) to endoplasmic reticulum (ER) stress by Mvarphis in a longi

54 R) is a cytoprotective pathway that relieves endoplasmic reticulum (ER) stress by promoting ER-associ

55 RATIONALE: Endoplasmic reticulum (ER) stress causes the accumulatio

56 We found that the PERK axis of endoplasmic reticulum (ER) stress elicited prominent nuc

57 Endoplasmic reticulum (ER) stress elicits EC dysregulati

58 mpaired glucose tolerance to overt diabetes; endoplasmic reticulum (ER) stress expedites beta cell fa

59 ession was stimulated by tunicamycin-induced endoplasmic reticulum (ER) stress in both KRAS wild-type

60 ical role of reticulon (RTN) 1A in mediating endoplasmic reticulum (ER) stress in kidney tubular cell

61 how that increased ECM accumulation leads to endoplasmic reticulum (ER) stress in the TM.

62 ons result in protein misfolding, leading to endoplasmic reticulum (ER) stress in the trabecular mesh

63 hysiological growth as well as management of endoplasmic reticulum (ER) stress in unfavorable growth

64 Endoplasmic reticulum (ER) stress is a local factor that

65 Programmed cell death (PCD) induced by endoplasmic reticulum (ER) stress is implicated in vario

66 The regulatory control of cardiac endoplasmic reticulum (ER) stress is incompletely charac

67 The endoplasmic reticulum (ER) stress occurs frequently in c

68 Endoplasmic reticulum (ER) stress occurs in the early st

69 ent of cardiac hypertrophy and heart failure.Endoplasmic reticulum (ER) stress promotes cardiac dysfu

70 e we present evidence that ECD regulates the endoplasmic reticulum (ER) stress response.

71 nished stemness, in part due to induction of endoplasmic reticulum (ER) stress that resulted in apopt

72 was also potently upregulated by triggering endoplasmic reticulum (ER) stress with thapsigargin and

73 In response to endoplasmic reticulum (ER) stress, ATF6 migrates from th

74 s associated with metabolic inflammation and endoplasmic reticulum (ER) stress, both of which promote

75 We demonstrate that metformin induces endoplasmic reticulum (ER) stress, calcium release from

76 riggered in Paneth cells by bacteria-induced endoplasmic reticulum (ER) stress, required extrinsic si

77 Endoplasmic reticulum (ER) stress, triggered by unfolded

78 ghly sensitive to differentiation induced by endoplasmic reticulum (ER) stress.

79 acrophages under growth-factor limitation or endoplasmic reticulum (ER) stress.

80 verexpression, which sensitizes cells to the endoplasmic reticulum (ER) stress.

81 ondrial dysfunction, impaired autophagy, and endoplasmic reticulum (ER) stress.

82 unction by regulating miR-204 expression and endoplasmic reticulum (ER) stress.

83 lator of interferon genes (STING) to mediate endoplasmic reticulum (ER) stress.

84 ctive oxygen species, and in the response to endoplasmic reticulum (ER) stress.

85 nd one of the cellular manifestations is the endoplasmic reticulum (ER) stress.

86 oduce IFN by 5-azacytidine (5-aza) underwent endoplasmic reticulum (ER) stress.

87 and resulted in its accumulation within the endoplasmic reticulum (ER) suggesting impaired ER-to-Gol

88 While the endoplasmic reticulum (ER) supports dendritic translatio

89 many secretory protein mRNAs is to encode an endoplasmic reticulum (ER) targeting sequence.

90 that MCTP localizes to the membranes of the endoplasmic reticulum (ER) that elaborate throughout the

91 ia form close physical associations with the endoplasmic reticulum (ER) that regulate a number of phy

92 trafficking from the Golgi apparatus to the endoplasmic reticulum (ER) through an interaction with Z

93 Here we demonstrate that upregulation of an endoplasmic reticulum (ER) to Golgi trafficking gene sig

94 cilitate the transport of DP(84Gly) from the endoplasmic reticulum (ER) to the endosomal/lysosomal pa

95 tate nonvesicular ceramide transfer from the endoplasmic reticulum (ER) to the Golgi complex, where c

96 0-nm procollagen I (PC1) molecules, from the endoplasmic reticulum (ER) to the Golgi.

97 eurons induces trafficking of GluA2 from the endoplasmic reticulum (ER) to the synapse by enhancing G

98 ClC-4 is retained in the endoplasmic reticulum (ER) upon overexpression in HEK293

99 occurs with TAG-synthesizing enzymes on the endoplasmic reticulum (ER), and nascent TAGs are sequest

100 rf is generated as a membrane protein in the endoplasmic reticulum (ER), and that it undergoes auto-p

101 a membrane (PM), contain a strand of tubular endoplasmic reticulum (ER), and the space between these

102 s signaling has so far mostly focused on the endoplasmic reticulum (ER), emerging data suggest that t

103 e biogenesis of VLDL particles occurs in the endoplasmic reticulum (ER), followed by subsequent lipid

104 Y141C-Prph2 showed signs of retention in the endoplasmic reticulum (ER), however co-expression with R

105 scription of procollagen I, which enters the endoplasmic reticulum (ER), is trafficked through the se

106 the persistent metabolic overloading of the endoplasmic reticulum (ER), leading to its functional im

107 sed zymogen granules, and alterations in the endoplasmic reticulum (ER), ranging from vesicular ER to

108 at INF2 mediates actin polymerization at the endoplasmic reticulum (ER), resulting in increased ER-mi

109 When unfolded proteins accumulate in the endoplasmic reticulum (ER), the unfolded protein respons

110 disturb the protein-folding capacity of the endoplasmic reticulum (ER), thereby provoking a cellular

111 A fraction of RIP2 localizes to the endoplasmic reticulum (ER), where it interacts with ZNRF

112 karyotic secretory pathway begin life in the endoplasmic reticulum (ER), where their folding is surve

113 s wild-type (WT) proinsulin from exiting the endoplasmic reticulum (ER), which is essential for insul

114 mitochondrial matrix, nucleus, cytosol, and endoplasmic reticulum (ER), with specificity and sensiti

115 We showed that TMEM24 is an endoplasmic reticulum (ER)-anchored membrane protein who

116 and show that CLas induces the formation of endoplasmic reticulum (ER)-associated bodies.

117 DNAJB12 (JB12) is an endoplasmic reticulum (ER)-associated Hsp40 family prote

118 eric complexes associated with intracellular endoplasmic reticulum (ER)-derived membranes.

119 se structural changes dramatically decreased endoplasmic reticulum (ER)-exit and plasma membrane loca

120 tiple proteostatic mechanisms, including the endoplasmic reticulum (ER)-induced unfolded protein resp

121 The endoplasmic reticulum (ER)-localized Hsp70 chaperone BiP

122 hese activities are mediated largely through endoplasmic reticulum (ER)-localized vIL-6, which can in

123 In yeast, the ERMES complex is an endoplasmic reticulum (ER)-mitochondria tether composed

124 (HDAC6) increase alpha-tubulin acetylation, endoplasmic reticulum (ER)-mitochondrial overlay, and re

125 DAcT colocalizes to the same fractions as an endoplasmic reticulum (ER)-specific marker.

126 ERdj3/DNAJB11 is an endoplasmic reticulum (ER)-targeted HSP40 co-chaperone t

127 re mitochondria are closely apposed with the endoplasmic reticulum (ER).

128 nhibitor of cholesterol transport from PM to endoplasmic reticulum (ER).

129 connections between viral membranes and the endoplasmic reticulum (ER).

130 adjusts the protein folding capacity of the endoplasmic reticulum (ER).

131 ocalization with the Golgi apparatus and the endoplasmic reticulum (ER).

132 class of tail-anchored proteins (TAs) to the endoplasmic reticulum (ER).

133 ted proteins, exported through the classical endoplasmic reticulum (ER)/Golgi-dependent pathway, but

134 arcolipin (SLN) is an inhibitor of the sarco/endoplasmic reticulum (SR) Ca(2+) ATPase (SERCA) and is

135 such as the unfolded protein response of the endoplasmic reticulum (UPR(ER)).

136 amine the assembly of the MECA (mitochondria-endoplasmic reticulum [ER]-cortex anchor), which tethers

137 ration, IP3 receptors (IP3Rs) located on the endoplasmic reticulum allow the 'quasisynaptical' feedin

138 rocyclic trypanosomes, (ii) localizes to the endoplasmic reticulum and (iii) represents the unique ro

139 lacenta specific miRNAs from STBEVs into the endoplasmic reticulum and mitochondria of these recipien

140 ellular membranes, specifically those of the endoplasmic reticulum and mitochondria, are crucial fact

141 atched the chemical composition of the human endoplasmic reticulum and served as an ER biomimetic.

142 fraction of dendrimers, however, localize to endoplasmic reticulum and the Golgi apparatus, presumabl

143 ence microscopy localized TbRFT1 to both the endoplasmic reticulum and the Golgi, consistent with the

144 the Sec61 complex and the Get complex in the endoplasmic reticulum and the SecYEG complex and YidC in

145 astin (ATL) catalyzes membrane fusion of the endoplasmic reticulum and thus establishes a network of

146 mutant, because it mostly failed to exit the endoplasmic reticulum and was degraded.

147 Although endoplasmic reticulum association was occasionally noted

148 en fluorescent protein was detectable in the endoplasmic reticulum but that it also could be recogniz

149 The sarco/endoplasmic reticulum Ca ATPase (SERCA) pump then refill

150 he L-type Ca(2+) channel (LCC) and the sarco/endoplasmic reticulum Ca(2+) -ATPase (SERCA) as the prin

151 oregulin and DWORF, which regulate the sarco/endoplasmic reticulum Ca(2+) -ATPase (SERCA).

152 Sarcoplasmic/endoplasmic reticulum Ca(2+) adenosine triphosphatase (S

153 upon stimulation was due to increased sarco/endoplasmic reticulum Ca(2+) ATPase (SERCA)-mediated reu

154 ropyridine receptor (DHPR), and sarcoplasmic/endoplasmic reticulum Ca(2+) ATPase (SERCA).

155 ntifying the L-type Ca(2+) channel and sarco/endoplasmic reticulum Ca(2+) ATPase as the principal reg

156 in MNCs from RVH rats, partly due to blunted endoplasmic reticulum Ca(2+) buffering capacity.

157 gh TRPM7 is essential for the maintenance of endoplasmic reticulum Ca(2+) concentration in resting ce

158 of resistant parasites identifies the sarco/endoplasmic reticulum Ca(2+) transporting PfATP6 as a pu

159 Sarco/endoplasmic reticulum Ca(2+)-ATPase (SERCA), a member of

160 hanced ATP-dependent Ca(2+) cycling by sarco/endoplasmic reticulum Ca(2+)-ATPase 2b (SERCA2b) and rya

161 nd is a potent inhibitor of the sarcoplasmic-endoplasmic reticulum Ca(2+)-ATPase calcium pump in mamm

162 holamban (PLN), inhibiting the cardiac sarco/endoplasmic reticulum calcium ATPase 2a (SERCA2a) in the

163 ted in reduced expression of Serca2, reduced endoplasmic reticulum calcium levels, and induction of t

164 a disrupted CD19 membrane export in the post-endoplasmic reticulum compartment as molecular basis for

165 nction is compromised, the morphology of the endoplasmic reticulum deteriorates, and these defects ca

166 over, Pet10p functionally interacts with the endoplasmic reticulum droplet assembly factors seipin an

167 70 (HSP70) inhibitor pifithrin-mu such that endoplasmic reticulum export of and radioligand binding

168 gulation of phospholipid synthesis maintains endoplasmic reticulum homeostasis and is critical for tr

169 ed protein (GRP78/HSPA5), a key regulator of endoplasmic reticulum homeostasis and PI3K/AKT signaling

170 phate (IP3)-mediated Ca(2+) release from the endoplasmic reticulum in several rare monogenic syndrome

171 ndent fashion, and retention of GPR31 on the endoplasmic reticulum inhibited delivery of KRAS4B to th

172 ng of most integral membrane proteins to the endoplasmic reticulum is controlled by the signal recogn

173 The endoplasmic reticulum is particularly intriguing, as it

174 at local reduced protein kinase R (PKR)-like endoplasmic reticulum kinase (PERK) expression or activi

175 The endoplasmic reticulum kinase inositol-requiring enzyme 1

176 Proteins with misfolded domains in the endoplasmic reticulum lumen or membrane are discarded th

177 ity extending from the cytosol almost to the endoplasmic reticulum lumen, while a segment of the neig

178 re of S. cerevisiae Hrd1 in complex with its endoplasmic reticulum luminal binding partner, Hrd3.

179 repeat (TPR) domain that is anchored to the endoplasmic reticulum membrane by Sec71.

180 ah1Delta effects on lipid synthesis, nuclear/endoplasmic reticulum membrane morphology, and lipid dro

181 oorly characterized secreted protein, EMC10 (endoplasmic reticulum membrane protein complex subunit 1

182 Insig-2 is one of two endoplasmic reticulum membrane proteins that inhibit cho

183 is the core structural component of a large endoplasmic reticulum membrane-embedded protein complex

184 vement of misfolded polypeptides through the endoplasmic reticulum membrane.

185 te targeting to and translocation across the endoplasmic reticulum membrane.

186 n to cause extensive remodeling of Golgi and endoplasmic reticulum membranes, and a number of the hos

187 ERthermAC accumulated in the endoplasmic reticulum of BAs and displayed a marked chan

188 of secretory proteins into the lumen of the endoplasmic reticulum or the periplasm of bacteria is me

189 , RIDalpha did not reconstitute transport to endoplasmic reticulum pools that regulate SREBP transcri

190 receptor and caused retention of Fz8 in the endoplasmic reticulum possibly by preventing N-linked gl

191 racellular Toll-like receptors (TLRs) in the endoplasmic reticulum prevents their activation under ba

192 Misfolded endoplasmic reticulum proteins are retro-translocated th

193 namic tubular membrane network with purified endoplasmic reticulum proteins.

194 Protein disulfide isomerases (PDIs) support endoplasmic reticulum redox protein folding and cell-sur

195 Retention in the endoplasmic reticulum seems to be the cause of the lower

196 odine receptor-mediated calcium release from endoplasmic reticulum stores, leading to calcineurin-med

197 egenerating livers, XBP1 deficiency leads to endoplasmic reticulum stress and DNA damage.

198 Endoplasmic reticulum stress causes unfolded proteins to

199 Cytokine-induced endoplasmic reticulum stress enhanced exosome secretion

200 ndrogen receptor, induces AR aggregation and endoplasmic reticulum stress in the prostate glands of E

201 Endoplasmic reticulum stress is an evolutionarily conser

202 rther validated by demonstrating increase in endoplasmic reticulum stress of MDA-MB-468 cells with ti

203 lpha/ATF4 signaling branch of the integrated endoplasmic reticulum stress response (IERSR) is activat

204 stimulate protein synthesis, resulting in an endoplasmic reticulum stress response mediated by Perk.

205 d activation of the proapoptotic arms of the endoplasmic reticulum stress response that is probably s

206 ls was TNFalpha independent but involved the endoplasmic reticulum stress response.

207 ggesting that specific TG6 mutants elicit an endoplasmic reticulum stress response.

208 pathway causing cardiac hypertrophy involves endoplasmic reticulum stress sensor PERK (protein kinase

209 GGF1 protein therapy and miR-183-5p regulate endoplasmic reticulum stress signaling and block endopla

210 onical, AGGF1-mediated regulatory system for endoplasmic reticulum stress signaling associated with i

211 Mechanistically, AGGF1 regulates endoplasmic reticulum stress signaling by inhibiting ERK

212 The major endoplasmic reticulum stress signaling pathway causing c

213 Accumulation of misfolded proteins triggers endoplasmic reticulum stress that leads to unfolded prot

214 veolar epithelial metaplasia, and epithelial endoplasmic reticulum stress that were evident after the

215 iptional and translational analyses revealed endoplasmic reticulum stress was not the etiology of our

216 e genes impaired protein translation, caused endoplasmic reticulum stress, activated DNA-damage-respo

217 except for genes associated with apoptosis, endoplasmic reticulum stress, and autophagy (P < 0.05).

218 -kappaB activation, proinflammatory markers, endoplasmic reticulum stress, and insoluble phosphorylat

219 dverse effects on mitochondrial function and endoplasmic reticulum stress, could have contributed to

220 XBP1, we observed of liver tissue persistent endoplasmic reticulum stress, defects in acute-phase res

221 tic proteins BIM and BAX, JNK signaling, and endoplasmic reticulum stress, explaining why SRp55 deple

222 es consistent with facets of T2DM, including endoplasmic reticulum stress, inflammation, and hyperpro

223 Endocrine disrupting chemicals induce endoplasmic reticulum stress, perturb NF-kappaB, and p53

224 100 only) exhibited only subtle increases in endoplasmic reticulum stress, suggesting that an altered

225 tokine and microbial stimulation to suppress endoplasmic reticulum stress, thereby assuring antiinfla

226 binding and inhibiting p53, but its role in endoplasmic reticulum stress-induced apoptosis remains u

227 plasmic reticulum stress signaling and block endoplasmic reticulum stress-induced apoptosis, cardiac

228 In addition, acrolein induced endoplasmic reticulum stress-mediated death of epithelia

229 edistribution of tight junction proteins and endoplasmic reticulum stress-mediated epithelial cell de

230 as they tend to aggregate and induce robust endoplasmic reticulum stress.

231 tal to the adaptation to AKI associated with endoplasmic reticulum stress.

232 rols and had decreased lung inflammation and endoplasmic reticulum stress.

233 tion, disordered autophagy, and pathological endoplasmic reticulum stress.

234 promote contacts between FYCO1 lysosomes and endoplasmic reticulum that contain the PtdIns3P effector

235 lar trafficking, lipid metabolism and in the endoplasmic reticulum that could impact viral entry and

236 cycling of extracellular calcium through the endoplasmic reticulum to activate SK channels.

237 ere incubated with MKC-3946, an inhibitor of endoplasmic reticulum to nucleus signaling 1 (ERN1, also

238 on by the ubiquitin-proteasome system at the endoplasmic reticulum to regulate hERG levels and channe

239 is required for transport of UNC5A from the endoplasmic reticulum to the cell surface.

240 the sigma1-receptor to translocate from the endoplasmic reticulum to the cytosol and nucleus.

241 non-vesicular transport of ceramide from the endoplasmic reticulum to the Golgi by the multidomain pr

242 g with sfCherry211 and GFP11, revealing that endoplasmic reticulum translocon complex Sec61B has redu

243 hieved by primary release of Ca(2+) from the endoplasmic reticulum via Ca(2+) release channels placed

244 ausing DAT mutants that were retained in the endoplasmic reticulum when heterologously expressed in H

245 molecules that link calcium depletion of the endoplasmic reticulum with opening of plasma membrane ca

246 (i) ion fluxes, (ii) Ca(2+) release from the endoplasmic reticulum, (iii) intercellular coupling, and

247 urprisingly only partially surrounded by the endoplasmic reticulum, a key mediator of mitochondrial C

248 n molecule 1 (STIM1), a Ca(2+) sensor in the endoplasmic reticulum, and the Ca(2+) ion channel Orai i

249 eloid-expressed transmembrane protein in the endoplasmic reticulum, develop spontaneous neurological

250 ong six different membrane-bound organelles (endoplasmic reticulum, Golgi, lysosome, peroxisome, mito

251 filopodia, and the 2D and 3D dynamics of the endoplasmic reticulum, in living cells.

252 s to proteins as they enter the lumen of the endoplasmic reticulum, is a membrane-bound hetero-pentam

253 the potential division spots contacting the endoplasmic reticulum, it appears on IMM independently o

254 r cytosolic localization associated with the endoplasmic reticulum, not co-localizing with endosomal

255 The endoplasmic reticulum, the cytoplasmic organelle that ma

256 Like the endoplasmic reticulum, these vesicles are a distributed

257 t delivers cholesterol from endosomes to the endoplasmic reticulum, where it is esterified and stored

258 DL-derived cholesterol from endosomes to the endoplasmic reticulum, where it was converted to cholest

259 restingly, PtNTT5 is probably located in the endoplasmic reticulum, which in diatoms also represents

260 d to localize to the lumen of the epiplastid endoplasmic reticulum, with its expression regulated by

261 Herpud1, a component of the endoplasmic reticulum-associated degradation (ERAD) comp

262 sterol-induced ubiquitination and subsequent endoplasmic reticulum-associated degradation of the rate

263 nase ataxin3, which collaborates with p97 in endoplasmic reticulum-associated degradation.

264 ons and, soon after starvation, nucleates in endoplasmic reticulum-associated foci that colocalize wi

265 degraded by the proteasome-a pathway termed endoplasmic reticulum-associated protein degradation (ER

266 chaperone protein that localizes within the endoplasmic reticulum-Golgi intermediate compartment.

267 Hilpda localized to the endoplasmic reticulum-LD interface, the site of LD forma

268 Among these, depleting the endoplasmic reticulum-localized protein FAM134A impairs

269 in the sec22b gene, a critical regulator of endoplasmic reticulum-phagosome traffic required for cro

270 identified as the ortholog of the mammalian endoplasmic reticulum-resident chaperone gp96.

271 hares key structural elements with MEC-6, an endoplasmic reticulum-resident molecular chaperone in Ca

272 tissue, revealing its identity as TMEM97, an endoplasmic reticulum-resident transmembrane protein tha

273 s known to form highly dynamic contacts with endoplasmic reticulum-resident VAP proteins that regulat

274 ocyte specific (CREBH), is a liver-enriched, endoplasmic reticulum-tethered transcription factor know

275 n of cellular protein homeostasis beyond the endoplasmic reticulum.

276 idylcholine (PC) and causes expansion of the endoplasmic reticulum.

277 he Golgi apparatus and from the Golgi to the endoplasmic reticulum.

278 esized as a proprotein that dimerizes in the endoplasmic reticulum.

279 ac1 when unfolded proteins accumulate in the endoplasmic reticulum.

280 al N-linked glycosylation of proteins in the endoplasmic reticulum.

281 serine palmitoyltransferase localized to the endoplasmic reticulum.

282 lding of nascent proteins synthesized in the endoplasmic reticulum.

283 cular chaperones that normally reside in the endoplasmic reticulum.

284 ses, and mutant FUS accumulates at the rough endoplasmic reticulum.

285 rane and clearing clogged translocons on the endoplasmic reticulum.

286 the C terminus of precursor proteins in the endoplasmic reticulum.

287 mitochondria and their interaction with the endoplasmic reticulum.

288 olgi apparatus and between the Golgi and the endoplasmic reticulum.

289 Here we discover that BAP1 localizes at the endoplasmic reticulum.

290 donor located in the cytoplasmic side of the endoplasmic reticulum.

291 type and mutant ANO5 protein localize to the endoplasmic reticulum.

292 volved in collagen biosynthesis in the rough endoplasmic reticulum.

293 he lumen of both the Golgi apparatus and the endoplasmic reticulum.

294 a time-dependent manner, mainly in the cell endoplasmic reticulum.

295 lected from a large diversity present in the endoplasmic reticulum.

296 ol via a reaction-diffusion process from the endoplasmic reticulum.

297 ause exosome secretion without affecting the endoplasmic reticulum/Golgi pathway.

298 rotein metabolism and adaptive activation of endoplasmic-reticulum-stress-induced survival pathways.

299 machinery and the IRE1-alpha-MKK4 arm of the endoplasmic-reticulum-stress-response pathway.

300 on in MM cells activates caspase cascade and endoplasmic stress response signaling.