ライフサイエンスコーパス: endoplasmic reticulum

1 n of cellular protein homeostasis beyond the endoplasmic reticulum.

2 idylcholine (PC) and causes expansion of the endoplasmic reticulum.

3 he Golgi apparatus and from the Golgi to the endoplasmic reticulum.

4 esized as a proprotein that dimerizes in the endoplasmic reticulum.

5 ac1 when unfolded proteins accumulate in the endoplasmic reticulum.

6 al N-linked glycosylation of proteins in the endoplasmic reticulum.

7 serine palmitoyltransferase localized to the endoplasmic reticulum.

8 lding of nascent proteins synthesized in the endoplasmic reticulum.

9 cular chaperones that normally reside in the endoplasmic reticulum.

10 ses, and mutant FUS accumulates at the rough endoplasmic reticulum.

11 rane and clearing clogged translocons on the endoplasmic reticulum.

12 the C terminus of precursor proteins in the endoplasmic reticulum.

13 mitochondria and their interaction with the endoplasmic reticulum.

14 olgi apparatus and between the Golgi and the endoplasmic reticulum.

15 Here we discover that BAP1 localizes at the endoplasmic reticulum.

16 donor located in the cytoplasmic side of the endoplasmic reticulum.

17 volved in collagen biosynthesis in the rough endoplasmic reticulum.

18 he lumen of both the Golgi apparatus and the endoplasmic reticulum.

19 type and mutant ANO5 protein localize to the endoplasmic reticulum.

20 a time-dependent manner, mainly in the cell endoplasmic reticulum.

21 lected from a large diversity present in the endoplasmic reticulum.

22 ol via a reaction-diffusion process from the endoplasmic reticulum.

23 urprisingly only partially surrounded by the endoplasmic reticulum, a key mediator of mitochondrial C

24 ration, IP3 receptors (IP3Rs) located on the endoplasmic reticulum allow the 'quasisynaptical' feedin

25 rocyclic trypanosomes, (ii) localizes to the endoplasmic reticulum and (iii) represents the unique ro

26 lacenta specific miRNAs from STBEVs into the endoplasmic reticulum and mitochondria of these recipien

27 ellular membranes, specifically those of the endoplasmic reticulum and mitochondria, are crucial fact

28 atched the chemical composition of the human endoplasmic reticulum and served as an ER biomimetic.

29 fraction of dendrimers, however, localize to endoplasmic reticulum and the Golgi apparatus, presumabl

30 ence microscopy localized TbRFT1 to both the endoplasmic reticulum and the Golgi, consistent with the

31 the Sec61 complex and the Get complex in the endoplasmic reticulum and the SecYEG complex and YidC in

32 astin (ATL) catalyzes membrane fusion of the endoplasmic reticulum and thus establishes a network of

33 mutant, because it mostly failed to exit the endoplasmic reticulum and was degraded.

34 n molecule 1 (STIM1), a Ca(2+) sensor in the endoplasmic reticulum, and the Ca(2+) ion channel Orai i

35 Herpud1, a component of the endoplasmic reticulum-associated degradation (ERAD) comp

36 sterol-induced ubiquitination and subsequent endoplasmic reticulum-associated degradation of the rate

37 nase ataxin3, which collaborates with p97 in endoplasmic reticulum-associated degradation.

38 ons and, soon after starvation, nucleates in endoplasmic reticulum-associated foci that colocalize wi

39 degraded by the proteasome-a pathway termed endoplasmic reticulum-associated protein degradation (ER

40 Although endoplasmic reticulum association was occasionally noted

41 en fluorescent protein was detectable in the endoplasmic reticulum but that it also could be recogniz

42 The sarco/endoplasmic reticulum Ca ATPase (SERCA) pump then refill

43 he L-type Ca(2+) channel (LCC) and the sarco/endoplasmic reticulum Ca(2+) -ATPase (SERCA) as the prin

44 oregulin and DWORF, which regulate the sarco/endoplasmic reticulum Ca(2+) -ATPase (SERCA).

45 Sarcoplasmic/endoplasmic reticulum Ca(2+) adenosine triphosphatase (S

46 upon stimulation was due to increased sarco/endoplasmic reticulum Ca(2+) ATPase (SERCA)-mediated reu

47 ropyridine receptor (DHPR), and sarcoplasmic/endoplasmic reticulum Ca(2+) ATPase (SERCA).

48 ntifying the L-type Ca(2+) channel and sarco/endoplasmic reticulum Ca(2+) ATPase as the principal reg

49 in MNCs from RVH rats, partly due to blunted endoplasmic reticulum Ca(2+) buffering capacity.

50 gh TRPM7 is essential for the maintenance of endoplasmic reticulum Ca(2+) concentration in resting ce

51 of resistant parasites identifies the sarco/endoplasmic reticulum Ca(2+) transporting PfATP6 as a pu

52 Sarco/endoplasmic reticulum Ca(2+)-ATPase (SERCA), a member of

53 hanced ATP-dependent Ca(2+) cycling by sarco/endoplasmic reticulum Ca(2+)-ATPase 2b (SERCA2b) and rya

54 nd is a potent inhibitor of the sarcoplasmic-endoplasmic reticulum Ca(2+)-ATPase calcium pump in mamm

55 holamban (PLN), inhibiting the cardiac sarco/endoplasmic reticulum calcium ATPase 2a (SERCA2a) in the

56 ted in reduced expression of Serca2, reduced endoplasmic reticulum calcium levels, and induction of t

57 a disrupted CD19 membrane export in the post-endoplasmic reticulum compartment as molecular basis for

58 nction is compromised, the morphology of the endoplasmic reticulum deteriorates, and these defects ca

59 eloid-expressed transmembrane protein in the endoplasmic reticulum, develop spontaneous neurological

60 over, Pet10p functionally interacts with the endoplasmic reticulum droplet assembly factors seipin an

61 s the cell's protein folding capacity in the endoplasmic reticulum (ER) according to need.

62 Insufficient folding capacity of the endoplasmic reticulum (ER) activates the unfolded protei

63 acidification disturb protein folding in the endoplasmic reticulum (ER) and activate the Unfolded Pro

64 ins with folding problems are trapped in the endoplasmic reticulum (ER) and are eventually degraded i

65 Although alterations in both endoplasmic reticulum (ER) and cytosolic free calcium le

66 l is accompanied by reduced juxtaposition of endoplasmic reticulum (ER) and mitochondria as well as e

67 Lipid exchange between the endoplasmic reticulum (ER) and peroxisomes is necessary

68 BiP is the only Hsp70 chaperone in the endoplasmic reticulum (ER) and similar to other Hsp70s,

69 in Gn colocalizes and accumulates within the endoplasmic reticulum (ER) and the transport of Gn from

70 antibodies, biosynthetic substrates to fuel endoplasmic reticulum (ER) biogenesis, and additional ca

71 tes two Brassicaceae-specific traits, namely endoplasmic reticulum (ER) body formation and induction

72 Thapsigargin (Tg) blocks the sarco/endoplasmic reticulum (ER) Ca(2+)-ATPase (SERCA), disrup

73 Unfavorable redox conditions in the endoplasmic reticulum (ER) can decrease the capacity for

74 The endoplasmic reticulum (ER) consists of the nuclear envel

75 Peroxisomes (POs) and the endoplasmic reticulum (ER) cooperate in cellular lipid m

76 Electron microscopic observation revealed endoplasmic reticulum (ER) dilatation, suggestive of ER

77 l nutrient requiring tight constraint in the endoplasmic reticulum (ER) due to its uniquely challengi

78 The turnover of endoplasmic reticulum (ER) ensures the correct biologica

79 Although the endoplasmic reticulum (ER) extends throughout axons and

80 Disturbances in endoplasmic reticulum (ER) homeostasis create a conditio

81 Native cargo proteins exit the endoplasmic reticulum (ER) in COPII-coated vesicles, whe

82 ce mechanism that ensures homeostasis of the endoplasmic reticulum (ER) in eukaryotes.

83 ficking by assembling onto subdomains of the endoplasmic reticulum (ER) in two layers to generate car

84 The endoplasmic reticulum (ER) integral membrane protein VAP

85 The endoplasmic reticulum (ER) is a network of tubules and s

86 The endoplasmic reticulum (ER) is a single organelle in euka

87 Here we report that the endoplasmic reticulum (ER) is asymmetrically partitioned

88 y, alteration of the folding capacity of the endoplasmic reticulum (ER) is becoming a common patholog

89 Glycosylation in the endoplasmic reticulum (ER) is closely associated with pr

90 Homotypic membrane fusion of the endoplasmic reticulum (ER) is mediated by a class of dyn

91 folding, and disulfide formation within the endoplasmic reticulum (ER) is poorly understood.

92 The endoplasmic reticulum (ER) is the largest cellular membr

93 Cholesterol biosynthesis in the endoplasmic reticulum (ER) is tightly controlled by mult

94 The endoplasmic reticulum (ER) mediates the folding, maturat

95 , is degraded when cholesterol levels in the endoplasmic reticulum (ER) membrane are high, but the si

96 t be transported across or inserted into the endoplasmic reticulum (ER) membrane by the ER protein tr

97 nchoring and the compartmentalization of the endoplasmic reticulum (ER) membrane confine protein depo

98 n-enveloped polyomavirus SV40 penetrates the endoplasmic reticulum (ER) membrane to reach the cytosol

99 tor tyrosine kinases are mislocalized in the endoplasmic reticulum (ER) of AML and play an important

100 Contacts between endosomes and the endoplasmic reticulum (ER) promote endosomal tubule fiss

101 The extended synaptotagmins (E-Syts) are endoplasmic reticulum (ER) proteins that bind the plasma

102 ween plasma membrane P/Q Ca(2+) channels and endoplasmic reticulum (ER) ryanodine receptors and anoth

103 n sensing the depletion of (Ca(2+)) from the endoplasmic reticulum (ER) store, organizes as puncta th

104 It induced endoplasmic reticulum (ER) stress and activated the prot

105 gulation of pre-rRNA level led to attenuated endoplasmic reticulum (ER) stress and cell death.

106 study, we focused on a relationship between endoplasmic reticulum (ER) stress and cGVHD, and aimed t

107 s studies have suggested that ORMDL3 induces endoplasmic reticulum (ER) stress and production of the

108 Endoplasmic reticulum (ER) stress arises from accumulati

109 gated the unfolded protein response (UPR) to endoplasmic reticulum (ER) stress by Mvarphis in a longi

110 R) is a cytoprotective pathway that relieves endoplasmic reticulum (ER) stress by promoting ER-associ

111 RATIONALE: Endoplasmic reticulum (ER) stress causes the accumulatio

112 We found that the PERK axis of endoplasmic reticulum (ER) stress elicited prominent nuc

113 Endoplasmic reticulum (ER) stress elicits EC dysregulati

114 mpaired glucose tolerance to overt diabetes; endoplasmic reticulum (ER) stress expedites beta cell fa

115 Endoplasmic reticulum (ER) stress has been recognized to

116 ession was stimulated by tunicamycin-induced endoplasmic reticulum (ER) stress in both KRAS wild-type

117 ical role of reticulon (RTN) 1A in mediating endoplasmic reticulum (ER) stress in kidney tubular cell

118 how that increased ECM accumulation leads to endoplasmic reticulum (ER) stress in the TM.

119 ons result in protein misfolding, leading to endoplasmic reticulum (ER) stress in the trabecular mesh

120 hysiological growth as well as management of endoplasmic reticulum (ER) stress in unfavorable growth

121 Endoplasmic reticulum (ER) stress is a local factor that

122 Programmed cell death (PCD) induced by endoplasmic reticulum (ER) stress is implicated in vario

123 The regulatory control of cardiac endoplasmic reticulum (ER) stress is incompletely charac

124 The endoplasmic reticulum (ER) stress occurs frequently in c

125 Endoplasmic reticulum (ER) stress occurs in the early st

126 ent of cardiac hypertrophy and heart failure.Endoplasmic reticulum (ER) stress promotes cardiac dysfu

127 e we present evidence that ECD regulates the endoplasmic reticulum (ER) stress response.

128 nished stemness, in part due to induction of endoplasmic reticulum (ER) stress that resulted in apopt

129 was also potently upregulated by triggering endoplasmic reticulum (ER) stress with thapsigargin and

130 In response to endoplasmic reticulum (ER) stress, ATF6 migrates from th

131 s associated with metabolic inflammation and endoplasmic reticulum (ER) stress, both of which promote

132 We demonstrate that metformin induces endoplasmic reticulum (ER) stress, calcium release from

133 riggered in Paneth cells by bacteria-induced endoplasmic reticulum (ER) stress, required extrinsic si

134 Endoplasmic reticulum (ER) stress, triggered by unfolded

135 ghly sensitive to differentiation induced by endoplasmic reticulum (ER) stress.

136 acrophages under growth-factor limitation or endoplasmic reticulum (ER) stress.

137 verexpression, which sensitizes cells to the endoplasmic reticulum (ER) stress.

138 ondrial dysfunction, impaired autophagy, and endoplasmic reticulum (ER) stress.

139 unction by regulating miR-204 expression and endoplasmic reticulum (ER) stress.

140 lator of interferon genes (STING) to mediate endoplasmic reticulum (ER) stress.

141 ctive oxygen species, and in the response to endoplasmic reticulum (ER) stress.

142 nd one of the cellular manifestations is the endoplasmic reticulum (ER) stress.

143 oduce IFN by 5-azacytidine (5-aza) underwent endoplasmic reticulum (ER) stress.

144 Oxidative and endoplasmic reticulum (ER) stresses are hallmarks of the

145 and resulted in its accumulation within the endoplasmic reticulum (ER) suggesting impaired ER-to-Gol

146 While the endoplasmic reticulum (ER) supports dendritic translatio

147 many secretory protein mRNAs is to encode an endoplasmic reticulum (ER) targeting sequence.

148 that MCTP localizes to the membranes of the endoplasmic reticulum (ER) that elaborate throughout the

149 ia form close physical associations with the endoplasmic reticulum (ER) that regulate a number of phy

150 trafficking from the Golgi apparatus to the endoplasmic reticulum (ER) through an interaction with Z

151 Here we demonstrate that upregulation of an endoplasmic reticulum (ER) to Golgi trafficking gene sig

152 cilitate the transport of DP(84Gly) from the endoplasmic reticulum (ER) to the endosomal/lysosomal pa

153 tate nonvesicular ceramide transfer from the endoplasmic reticulum (ER) to the Golgi complex, where c

154 0-nm procollagen I (PC1) molecules, from the endoplasmic reticulum (ER) to the Golgi.

155 eurons induces trafficking of GluA2 from the endoplasmic reticulum (ER) to the synapse by enhancing G

156 ClC-4 is retained in the endoplasmic reticulum (ER) upon overexpression in HEK293

157 occurs with TAG-synthesizing enzymes on the endoplasmic reticulum (ER), and nascent TAGs are sequest

158 rf is generated as a membrane protein in the endoplasmic reticulum (ER), and that it undergoes auto-p

159 a membrane (PM), contain a strand of tubular endoplasmic reticulum (ER), and the space between these

160 s signaling has so far mostly focused on the endoplasmic reticulum (ER), emerging data suggest that t

161 e biogenesis of VLDL particles occurs in the endoplasmic reticulum (ER), followed by subsequent lipid

162 Y141C-Prph2 showed signs of retention in the endoplasmic reticulum (ER), however co-expression with R

163 scription of procollagen I, which enters the endoplasmic reticulum (ER), is trafficked through the se

164 the persistent metabolic overloading of the endoplasmic reticulum (ER), leading to its functional im

165 sed zymogen granules, and alterations in the endoplasmic reticulum (ER), ranging from vesicular ER to

166 at INF2 mediates actin polymerization at the endoplasmic reticulum (ER), resulting in increased ER-mi

167 When unfolded proteins accumulate in the endoplasmic reticulum (ER), the unfolded protein respons

168 disturb the protein-folding capacity of the endoplasmic reticulum (ER), thereby provoking a cellular

169 A fraction of RIP2 localizes to the endoplasmic reticulum (ER), where it interacts with ZNRF

170 karyotic secretory pathway begin life in the endoplasmic reticulum (ER), where their folding is surve

171 s wild-type (WT) proinsulin from exiting the endoplasmic reticulum (ER), which is essential for insul

172 mitochondrial matrix, nucleus, cytosol, and endoplasmic reticulum (ER), with specificity and sensiti

173 We showed that TMEM24 is an endoplasmic reticulum (ER)-anchored membrane protein who

174 and show that CLas induces the formation of endoplasmic reticulum (ER)-associated bodies.

175 DNAJB12 (JB12) is an endoplasmic reticulum (ER)-associated Hsp40 family prote

176 rium translocates proteins that establish an endoplasmic reticulum (ER)-associated replication compar

177 eric complexes associated with intracellular endoplasmic reticulum (ER)-derived membranes.

178 se structural changes dramatically decreased endoplasmic reticulum (ER)-exit and plasma membrane loca

179 tiple proteostatic mechanisms, including the endoplasmic reticulum (ER)-induced unfolded protein resp

180 The endoplasmic reticulum (ER)-localized Hsp70 chaperone BiP

181 hese activities are mediated largely through endoplasmic reticulum (ER)-localized vIL-6, which can in

182 In yeast, the ERMES complex is an endoplasmic reticulum (ER)-mitochondria tether composed

183 (HDAC6) increase alpha-tubulin acetylation, endoplasmic reticulum (ER)-mitochondrial overlay, and re

184 DAcT colocalizes to the same fractions as an endoplasmic reticulum (ER)-specific marker.

185 ERdj3/DNAJB11 is an endoplasmic reticulum (ER)-targeted HSP40 co-chaperone t

186 re mitochondria are closely apposed with the endoplasmic reticulum (ER).

187 nhibitor of cholesterol transport from PM to endoplasmic reticulum (ER).

188 connections between viral membranes and the endoplasmic reticulum (ER).

189 adjusts the protein folding capacity of the endoplasmic reticulum (ER).

190 ocalization with the Golgi apparatus and the endoplasmic reticulum (ER).

191 class of tail-anchored proteins (TAs) to the endoplasmic reticulum (ER).

192 ted proteins, exported through the classical endoplasmic reticulum (ER)/Golgi-dependent pathway, but

193 amine the assembly of the MECA (mitochondria-endoplasmic reticulum [ER]-cortex anchor), which tethers

194 70 (HSP70) inhibitor pifithrin-mu such that endoplasmic reticulum export of and radioligand binding

195 ong six different membrane-bound organelles (endoplasmic reticulum, Golgi, lysosome, peroxisome, mito

196 chaperone protein that localizes within the endoplasmic reticulum-Golgi intermediate compartment.

197 ause exosome secretion without affecting the endoplasmic reticulum/Golgi pathway.

198 gulation of phospholipid synthesis maintains endoplasmic reticulum homeostasis and is critical for tr

199 ed protein (GRP78/HSPA5), a key regulator of endoplasmic reticulum homeostasis and PI3K/AKT signaling

200 (i) ion fluxes, (ii) Ca(2+) release from the endoplasmic reticulum, (iii) intercellular coupling, and

201 phate (IP3)-mediated Ca(2+) release from the endoplasmic reticulum in several rare monogenic syndrome

202 filopodia, and the 2D and 3D dynamics of the endoplasmic reticulum, in living cells.

203 ndent fashion, and retention of GPR31 on the endoplasmic reticulum inhibited delivery of KRAS4B to th

204 Ca(2+) tunnelling through the lumen of the endoplasmic reticulum is a mechanism for delivering Ca(2

205 ng of most integral membrane proteins to the endoplasmic reticulum is controlled by the signal recogn

206 The endoplasmic reticulum is particularly intriguing, as it

207 s to proteins as they enter the lumen of the endoplasmic reticulum, is a membrane-bound hetero-pentam

208 the potential division spots contacting the endoplasmic reticulum, it appears on IMM independently o

209 at local reduced protein kinase R (PKR)-like endoplasmic reticulum kinase (PERK) expression or activi

210 The endoplasmic reticulum kinase inositol-requiring enzyme 1

211 Hilpda localized to the endoplasmic reticulum-LD interface, the site of LD forma

212 Among these, depleting the endoplasmic reticulum-localized protein FAM134A impairs

213 Proteins with misfolded domains in the endoplasmic reticulum lumen or membrane are discarded th

214 ity extending from the cytosol almost to the endoplasmic reticulum lumen, while a segment of the neig

215 re of S. cerevisiae Hrd1 in complex with its endoplasmic reticulum luminal binding partner, Hrd3.

216 repeat (TPR) domain that is anchored to the endoplasmic reticulum membrane by Sec71.

217 ah1Delta effects on lipid synthesis, nuclear/endoplasmic reticulum membrane morphology, and lipid dro

218 oorly characterized secreted protein, EMC10 (endoplasmic reticulum membrane protein complex subunit 1

219 Insig-2 is one of two endoplasmic reticulum membrane proteins that inhibit cho

220 is the core structural component of a large endoplasmic reticulum membrane-embedded protein complex

221 vement of misfolded polypeptides through the endoplasmic reticulum membrane.

222 te targeting to and translocation across the endoplasmic reticulum membrane.

223 n to cause extensive remodeling of Golgi and endoplasmic reticulum membranes, and a number of the hos

224 r cytosolic localization associated with the endoplasmic reticulum, not co-localizing with endosomal

225 ERthermAC accumulated in the endoplasmic reticulum of BAs and displayed a marked chan

226 of secretory proteins into the lumen of the endoplasmic reticulum or the periplasm of bacteria is me

227 in the sec22b gene, a critical regulator of endoplasmic reticulum-phagosome traffic required for cro

228 , RIDalpha did not reconstitute transport to endoplasmic reticulum pools that regulate SREBP transcri

229 receptor and caused retention of Fz8 in the endoplasmic reticulum possibly by preventing N-linked gl

230 racellular Toll-like receptors (TLRs) in the endoplasmic reticulum prevents their activation under ba

231 Misfolded endoplasmic reticulum proteins are retro-translocated th

232 namic tubular membrane network with purified endoplasmic reticulum proteins.

233 Protein disulfide isomerases (PDIs) support endoplasmic reticulum redox protein folding and cell-sur

234 identified as the ortholog of the mammalian endoplasmic reticulum-resident chaperone gp96.

235 hares key structural elements with MEC-6, an endoplasmic reticulum-resident molecular chaperone in Ca

236 tissue, revealing its identity as TMEM97, an endoplasmic reticulum-resident transmembrane protein tha

237 s known to form highly dynamic contacts with endoplasmic reticulum-resident VAP proteins that regulat

238 Retention in the endoplasmic reticulum seems to be the cause of the lower

239 arcolipin (SLN) is an inhibitor of the sarco/endoplasmic reticulum (SR) Ca(2+) ATPase (SERCA) and is

240 odine receptor-mediated calcium release from endoplasmic reticulum stores, leading to calcineurin-med

241 egenerating livers, XBP1 deficiency leads to endoplasmic reticulum stress and DNA damage.

242 Endoplasmic reticulum stress causes unfolded proteins to

243 Cytokine-induced endoplasmic reticulum stress enhanced exosome secretion

244 ndrogen receptor, induces AR aggregation and endoplasmic reticulum stress in the prostate glands of E

245 Endoplasmic reticulum stress is an evolutionarily conser

246 rther validated by demonstrating increase in endoplasmic reticulum stress of MDA-MB-468 cells with ti

247 lpha/ATF4 signaling branch of the integrated endoplasmic reticulum stress response (IERSR) is activat

248 stimulate protein synthesis, resulting in an endoplasmic reticulum stress response mediated by Perk.

249 d activation of the proapoptotic arms of the endoplasmic reticulum stress response that is probably s

250 ls was TNFalpha independent but involved the endoplasmic reticulum stress response.

251 ggesting that specific TG6 mutants elicit an endoplasmic reticulum stress response.

252 pathway causing cardiac hypertrophy involves endoplasmic reticulum stress sensor PERK (protein kinase

253 GGF1 protein therapy and miR-183-5p regulate endoplasmic reticulum stress signaling and block endopla

254 onical, AGGF1-mediated regulatory system for endoplasmic reticulum stress signaling associated with i

255 Mechanistically, AGGF1 regulates endoplasmic reticulum stress signaling by inhibiting ERK

256 The major endoplasmic reticulum stress signaling pathway causing c

257 Accumulation of misfolded proteins triggers endoplasmic reticulum stress that leads to unfolded prot

258 veolar epithelial metaplasia, and epithelial endoplasmic reticulum stress that were evident after the

259 iptional and translational analyses revealed endoplasmic reticulum stress was not the etiology of our

260 e genes impaired protein translation, caused endoplasmic reticulum stress, activated DNA-damage-respo

261 except for genes associated with apoptosis, endoplasmic reticulum stress, and autophagy (P < 0.05).

262 -kappaB activation, proinflammatory markers, endoplasmic reticulum stress, and insoluble phosphorylat

263 dverse effects on mitochondrial function and endoplasmic reticulum stress, could have contributed to

264 XBP1, we observed of liver tissue persistent endoplasmic reticulum stress, defects in acute-phase res

265 tic proteins BIM and BAX, JNK signaling, and endoplasmic reticulum stress, explaining why SRp55 deple

266 es consistent with facets of T2DM, including endoplasmic reticulum stress, inflammation, and hyperpro

267 Endocrine disrupting chemicals induce endoplasmic reticulum stress, perturb NF-kappaB, and p53

268 100 only) exhibited only subtle increases in endoplasmic reticulum stress, suggesting that an altered

269 tokine and microbial stimulation to suppress endoplasmic reticulum stress, thereby assuring antiinfla

270 binding and inhibiting p53, but its role in endoplasmic reticulum stress-induced apoptosis remains u

271 plasmic reticulum stress signaling and block endoplasmic reticulum stress-induced apoptosis, cardiac

272 In addition, acrolein induced endoplasmic reticulum stress-mediated death of epithelia

273 edistribution of tight junction proteins and endoplasmic reticulum stress-mediated epithelial cell de

274 as they tend to aggregate and induce robust endoplasmic reticulum stress.

275 tal to the adaptation to AKI associated with endoplasmic reticulum stress.

276 rols and had decreased lung inflammation and endoplasmic reticulum stress.

277 tion, disordered autophagy, and pathological endoplasmic reticulum stress.

278 rotein metabolism and adaptive activation of endoplasmic-reticulum-stress-induced survival pathways.

279 machinery and the IRE1-alpha-MKK4 arm of the endoplasmic-reticulum-stress-response pathway.

280 ocyte specific (CREBH), is a liver-enriched, endoplasmic reticulum-tethered transcription factor know

281 promote contacts between FYCO1 lysosomes and endoplasmic reticulum that contain the PtdIns3P effector

282 lar trafficking, lipid metabolism and in the endoplasmic reticulum that could impact viral entry and

283 The endoplasmic reticulum, the cytoplasmic organelle that ma

284 Like the endoplasmic reticulum, these vesicles are a distributed

285 cycling of extracellular calcium through the endoplasmic reticulum to activate SK channels.

286 ere incubated with MKC-3946, an inhibitor of endoplasmic reticulum to nucleus signaling 1 (ERN1, also

287 on by the ubiquitin-proteasome system at the endoplasmic reticulum to regulate hERG levels and channe

288 is required for transport of UNC5A from the endoplasmic reticulum to the cell surface.

289 the sigma1-receptor to translocate from the endoplasmic reticulum to the cytosol and nucleus.

290 non-vesicular transport of ceramide from the endoplasmic reticulum to the Golgi by the multidomain pr

291 g with sfCherry211 and GFP11, revealing that endoplasmic reticulum translocon complex Sec61B has redu

292 such as the unfolded protein response of the endoplasmic reticulum (UPR(ER)).

293 hieved by primary release of Ca(2+) from the endoplasmic reticulum via Ca(2+) release channels placed

294 ma membrane, as well as mitochondria and the endoplasmic reticulum, we demonstrate local RI measureme

295 ausing DAT mutants that were retained in the endoplasmic reticulum when heterologously expressed in H

296 t delivers cholesterol from endosomes to the endoplasmic reticulum, where it is esterified and stored

297 DL-derived cholesterol from endosomes to the endoplasmic reticulum, where it was converted to cholest

298 restingly, PtNTT5 is probably located in the endoplasmic reticulum, which in diatoms also represents

299 molecules that link calcium depletion of the endoplasmic reticulum with opening of plasma membrane ca

300 d to localize to the lumen of the epiplastid endoplasmic reticulum, with its expression regulated by