ライフサイエンスコーパス: endoplasmic reticulum stress

1 ated protein kinases activation and elevated endoplasmic reticulum stress.

2 e, deletion of P5-ATPase Spf1p gives rise to endoplasmic reticulum stress.

3 er for autophagy under conditions that cause endoplasmic reticulum stress.

4 al role in determining cell viability during endoplasmic reticulum stress.

5 uction of metabolic stresses, such as AAS or endoplasmic reticulum stress.

6 beit with slower kinetics in response to the endoplasmic reticulum stress.

7 appaBalpha, and JNK, indicating induction of endoplasmic reticulum stress.

8 nally advantageous, conferring resistance to endoplasmic reticulum stress.

9 cts with a unique carbon skeleton that cause endoplasmic reticulum stress.

10 ibitor CFTR(inh)-172, CF BAL, or inducers of endoplasmic reticulum stress.

11 ssociated with mitochondrial dysfunction and endoplasmic reticulum stress.

12 ggestive of impaired protein degradation and endoplasmic reticulum stress.

13 , GAS delivers streptolysin toxins, creating endoplasmic reticulum stress.

14 gene was induced by either AA deprivation or endoplasmic reticulum stress.

15 inhibition, increased cytosolic calcium and endoplasmic reticulum stress.

16 as they tend to aggregate and induce robust endoplasmic reticulum stress.

17 of toll-like receptor-4 (TLR4) signaling and endoplasmic reticulum stress.

18 oreactive T-cells and apoptosis triggered by endoplasmic reticulum stress.

19 ons but no activation of XBP-1, a marker for endoplasmic reticulum stress.

20 oimmune attack and/or irreversible damage by endoplasmic reticulum stress.

21 phy mice, which was accompanied by increased endoplasmic reticulum stress.

22 11 promoter activity induced by oxidative or endoplasmic reticulum stress.

23 e inhibitor, and thapsigargin, an inducer of endoplasmic reticulum stress.

24 ells subjected to PERK-activation by chronic endoplasmic reticulum stress.

25 d calpain-10 but not caspase-3 activation or endoplasmic reticulum stress.

26 but is induced by cellular stress, including endoplasmic reticulum stress.

27 ibbon unlinking, unconventional secretion or endoplasmic reticulum stress.

28 tal to the adaptation to AKI associated with endoplasmic reticulum stress.

29 rols and had decreased lung inflammation and endoplasmic reticulum stress.

30 on, decreased lipid clearance, and increased endoplasmic reticulum stress.

31 xpression, beta cell functional failure, and endoplasmic reticulum stress.

32 tion, nephrin phosphorylation and attenuated endoplasmic reticulum stress.

33 type I interferon response via induction of endoplasmic reticulum stress.

34 s regulating dynein-associated molecules and endoplasmic reticulum stress.

35 cts, e.g. lipotoxicity, oxidative stress and endoplasmic reticulum stress.

36 s through inhibition of oxidative stress and endoplasmic reticulum stress.

37 tion, disordered autophagy, and pathological endoplasmic reticulum stress.

38 and an increase of hepatocellular injury and endoplasmic reticulum stress.

39 atic CES2 stimulates lipogenesis by inducing endoplasmic reticulum stress.

40 bolism, proliferation, and the resolution of endoplasmic reticulum stress.

41 eceptors, does protect these neurons against endoplasmic reticulum stress.

42 Chronic hypoxia also triggered mild endoplasmic reticulum stress, a conserved homeostatic re

43 e genes impaired protein translation, caused endoplasmic reticulum stress, activated DNA-damage-respo

44 Expression of CEL MODY increased endoplasmic reticulum stress, activated the unfolded pro

45 We induced OIR in C57BL/6, ATF4(+/-), and endoplasmic reticulum stress-activated indicator (ERAI)

46 XNIP post-transcriptionally via induction of endoplasmic reticulum stress, activation of inositol-req

47 on of cultured mammalian beta-cells and that endoplasmic reticulum stress acts upstream of FGF1 relea

48 ed ultrastructural changes characteristic of endoplasmic reticulum stress after 8 h and were no longe

49 erapeutic exploration of treatments inducing endoplasmic reticulum stress against mutant ERBB2-expres

50 Endoplasmic reticulum stress alone, however, did not ind

51 We detected prolonged, severe endoplasmic reticulum stress already at 20 weeks of age

52 reast epithelial cells to agents that induce endoplasmic reticulum stress, altering the unfolded prot

53 nstrate that the depletion of eIF5A leads to endoplasmic reticulum stress, an unfolded protein respon

54 54Arg) mutation showed evidence of increased endoplasmic reticulum stress and a reduction in Golgi vo

55 High-fat diet in sedentary mice led to endoplasmic reticulum stress and aberrant mitochondrial

56 dividuals with AATD by a mechanism involving endoplasmic reticulum stress and aberrant TNF-alpha sign

57 Induction of endoplasmic reticulum stress and activation of the intri

58 Endoplasmic reticulum stress and activation of the unfol

59 were dispensable for house dust mite-induced endoplasmic reticulum stress and airways fibrosis.

60 This resulted in endoplasmic reticulum stress and an unfolded protein res

61 feration of pancreatic cancer cells, induced endoplasmic reticulum stress and apoptosis, and reduced

62 y in a vast array of human cancers, inducing endoplasmic reticulum stress and apoptosis, toxic autoph

63 s monoclonal protein trafficking, leading to endoplasmic reticulum stress and apoptosis.

64 ," misfolding during assembly and leading to endoplasmic reticulum stress and autophagy responses.

65 ol level, which was associated with elevated endoplasmic reticulum stress and caused apoptosis.

66 response in islet beta cells with subsequent endoplasmic reticulum stress and cellular death.

67 p1), a transcription factor activated during endoplasmic reticulum stress and critically involved in

68 egenerating livers, XBP1 deficiency leads to endoplasmic reticulum stress and DNA damage.

69 from nearly 100 normal individuals following endoplasmic reticulum stress and exposure to ionizing ra

70 through UACA elevation, which by attenuating endoplasmic reticulum stress and GRP78 translocation to

71 asion, stress responses (e.g. DNA damage and endoplasmic reticulum stress and hypoxia), and cell-cell

72 light-chain dimers, significantly increasing endoplasmic reticulum stress and inducing cytotoxicity b

73 Endoplasmic reticulum stress and induction of autophagy

74 nesis and cholesterol efflux, but suppresses endoplasmic reticulum stress and inflammation.

75 nduce polymer formation, prime the cells for endoplasmic reticulum stress and initiate nuclear factor

76 mulation of Y437H myocilin in the TM induced endoplasmic reticulum stress and led to a 45% loss of sm

77 y, increased levels of p62, and increases in endoplasmic reticulum stress and mitochondrial damage, c

78 of reactive oxygen species and induction of endoplasmic reticulum stress and mitochondrial dysfuncti

79 (apoptosis or toxic autophagy) by promoting endoplasmic reticulum stress and modulating multiple sig

80 Although palmitate-induced endoplasmic reticulum stress and nuclear factor kappaB p

81 Similar effects on diabetes-induced endoplasmic reticulum stress and peripheral nerve dysfun

82 Dysregulated endoplasmic reticulum stress and phosphorylation of euka

83 ted FFA-derived toxic metabolites that drive endoplasmic reticulum stress and podocyte death.

84 Therefore, PP-IX leads to endoplasmic reticulum stress and proteasome inhibition i

85 NF-kappaB or UACA expression, which enhanced endoplasmic reticulum stress and restored GRP78 traffick

86 ition of mitochondrial Ca(+2) entry leads to endoplasmic reticulum stress and sensitizes cancer cells

87 In vivo, endoplasmic reticulum stress and SREBP-1-dependent effec

88 one loss reflects reversible calorie-induced endoplasmic reticulum stress and the associated unfolded

89 plasmic reticulum of neutrophils, leading to endoplasmic reticulum stress and the expression of proap

90 tifies L. pneumophila infection as a form of endoplasmic reticulum stress and the sensor pATF6 is pro

91 These cytokines induce endoplasmic reticulum stress and the unfolded protein re

92 We hypothesized that IFN-gamma induces endoplasmic reticulum stress and the unfolded protein re

93 ssion through calorie-dependent induction of endoplasmic reticulum stress and the unfolded protein re

94 egulated the expression of genes involved in endoplasmic reticulum stress and Toll-like receptor (TLR

95 Concomitantly, a significant increase of endoplasmic reticulum stress and unfolded protein respon

96 of misfolded or unfolded proteins - known as endoplasmic reticulum stress - and the activation of the

97 of the rough endoplasmic reticulum (without endoplasmic reticulum stress) and Golgi apparatus, incre

98 tial UPR response, elevating the response to endoplasmic reticulum stress, and apoptotic cell death.

99 icked gammaTE in upregulating A20, enhancing endoplasmic reticulum stress, and attenuating TNF-trigge

100 except for genes associated with apoptosis, endoplasmic reticulum stress, and autophagy (P < 0.05).

101 ion of islet amyloid, signs of oxidative and endoplasmic reticulum stress, and beta-cell death.

102 nuclear factor-kappaB inflammatory response, endoplasmic reticulum stress, and cell death.

103 s associated with cytoplasmic vacuolization, endoplasmic reticulum stress, and dysregulated autophagy

104 -kappaB activation, proinflammatory markers, endoplasmic reticulum stress, and insoluble phosphorylat

105 nounced potentiation of cellular heat shock, endoplasmic reticulum stress, and oxidative stress respo

106 ducible nitric oxide synthase; activation of endoplasmic reticulum stress, and the initiation of the

107 ilencing, Wnt signaling, neuronatin-mediated endoplasmic reticulum stress, and the laforin glycogen p

108 ted receptor-alpha, and Nrf-1; inhibition of endoplasmic reticulum stress; and inhibition of enhanced

109 ir functions are diverse, including roles in endoplasmic reticulum stress, apoptosis and retinal dege

110 as markedly down-regulated, genes related to endoplasmic reticulum stress, apoptosis, and inflammatio

111 proliferation and hypertrophy, angiogenesis, endoplasmic reticulum stress, apoptosis, and senescence.

112 and colleagues identify chemotherapy-induced endoplasmic reticulum stress as a novel strategy to targ

113 21 expression attenuated tunicamycin-induced endoplasmic reticulum stress, as demonstrated by using a

114 free fatty acid-induced oxidative stress and endoplasmic reticulum stress, as denoted by a reduction

115 ion of reactive oxygen species and increased endoplasmic reticulum stress, as revealed by biochemical

116 rease in post-ischemic proton production and endoplasmic reticulum stress, as well as an activation o

117 tein response at 8 weeks and overt beta cell endoplasmic reticulum stress at 12-16 weeks.

118 PERK activation in response to endoplasmic reticulum stress attenuates eIF2B activity b

119 ta-cell homicide) and beta-cell apoptosis by endoplasmic reticulum stress (beta-cell suicide).

120 To rescue beta-cell from the endoplasmic reticulum stress, beta-cells activate the un

121 orylation site mutants survive low levels of endoplasmic reticulum stress better than IRE1 deletions

122 Skin fibroblasts showed signs of increased endoplasmic reticulum stress, but despite the reported i

123 autoimmune attack against beta-cells causes endoplasmic reticulum stress by forcing the remaining be

124 Elevated markers of oxidative stress, endoplasmic reticulum stress, caspase activation, and ne

125 Endoplasmic reticulum stress, caused by the presence of

126 Endoplasmic reticulum stress causes unfolded proteins to

127 IGF2 led to beta-cell dedifferentiation and endoplasmic reticulum stress causing islet dysfunction i

128 bodies in the endoplasmic reticulum triggers endoplasmic reticulum stress, causing a disturbance of t

129 ptosis and necrosis and was not dependent on endoplasmic reticulum stress, ceramide generation, or re

130 Glucose-regulated protein 78 (GRP78) is an endoplasmic reticulum stress chaperone that regulates th

131 entify cell type-specific pathways involving endoplasmic reticulum-stress, circadian signaling, ion c

132 mTORC1 activation caused endoplasmic reticulum stress, columnar cell lesions, and

133 Under endoplasmic reticulum stress conditions, induction of CS

134 he Ire1 kinase-endonuclease, activated under endoplasmic reticulum stress conditions, relieves the in

135 revealed elevated expression of markers for endoplasmic reticulum stress, confirming an increase in

136 Furthermore, endoplasmic reticulum stress contributes to the aetiolog

137 dverse effects on mitochondrial function and endoplasmic reticulum stress, could have contributed to

138 XBP1, we observed of liver tissue persistent endoplasmic reticulum stress, defects in acute-phase res

139 t-TUCB consistently reduced endoplasmic reticulum stress demonstrated by the attenua

140 ECs induce CB1 R-mediated, endoplasmic reticulum stress-dependent synthesis of spec

141 pression may contribute to the oxidative and endoplasmic reticulum stress described in PE, as well as

142 3R activity, apoptosis, phosphorylation, and endoplasmic reticulum stress did not trigger the dissoci

143 anti-apoptotic, fatty acid biosynthesis, and endoplasmic reticulum stress effectors.

144 e, luciferase reporter assays showed that an endoplasmic reticulum stress element (ERSE)-like element

145 Cytokine-induced endoplasmic reticulum stress enhanced exosome secretion

146 Moreover, subthreshold endoplasmic reticulum stress (ER stress) drove insulin d

147 Activation of endoplasmic reticulum stress (ERS) with thapsigargin (TG

148 tic proteins BIM and BAX, JNK signaling, and endoplasmic reticulum stress, explaining why SRp55 deple

149 e R992C mutant collagen II molecules endured endoplasmic reticulum stress, had atypical polarization,

150 Endoplasmic reticulum stress has been linked to insulin

151 Endoplasmic reticulum stress has been reported to increa

152 binding of ABA to GRP78, a key regulator of endoplasmic reticulum stress, has also been proposed.

153 o pulmonary fibrosis and are associated with endoplasmic reticulum stress in alveolar type II epithel

154 mechanism for activation of autophagy during endoplasmic reticulum stress in Arabidopsis thaliana.

155 reticulum homeostasis and prevent cytotoxic endoplasmic reticulum stress in ccRCC.

156 vestigated whether mutant OA protein induces endoplasmic reticulum stress in D2J osteoblasts.

157 hondrial biogenesis, collagen deposition and endoplasmic reticulum stress in db/db mice.

158 ory stress induced both oxidative stress and endoplasmic reticulum stress in HFD-fed mice livers.

159 rdiometabolic disorders, including lipotoxic endoplasmic reticulum stress in macrophages.

160 ective actions that reduces inflammation and endoplasmic reticulum stress in other tissues.

161 Enhanced endoplasmic reticulum stress in response to S. pneumonia

162 sistance in multiple tissues but the role of endoplasmic reticulum stress in skeletal muscle has not

163 c role, possibly by mediating the effects of endoplasmic reticulum stress in the activation of pro-at

164 There is evidence implicating endoplasmic reticulum stress in the development and prog

165 Our findings reveal an important role of endoplasmic reticulum stress in the development of diabe

166 ndrogen receptor, induces AR aggregation and endoplasmic reticulum stress in the prostate glands of E

167 damage, apoptosis, and DNAJC3 (a marker for endoplasmic reticulum stress) in pancreatic islets.

168 conditions such as heat or agents that cause endoplasmic reticulum stress, including tunicamycin and

169 Transforming growth factor-beta1, but not endoplasmic reticulum stress, induced FKBP10 expression

170 binding and inhibiting p53, but its role in endoplasmic reticulum stress-induced apoptosis remains u

171 SF10 expression were far more susceptible to endoplasmic reticulum stress-induced apoptosis than cont

172 plasmic reticulum stress signaling and block endoplasmic reticulum stress-induced apoptosis, cardiac

173 hanism capable of protecting beta cells from endoplasmic reticulum stress-induced apoptosis.

174 criptionally downregulates CHOP and inhibits endoplasmic reticulum stress-induced apoptosis.

175 nocyte-adhesive hyaluronan matrix through an endoplasmic reticulum stress-induced autophagic mechanis

176 Furthermore, P12 inhibited endoplasmic reticulum stress-induced c-Jun N-terminal ki

177 pUL38 also plays a role in blocking endoplasmic reticulum stress-induced cell death during H

178 A twofold to threefold enhancement in endoplasmic reticulum stress-induced IL-10 expression wa

179 se data demonstrate that tribbles 3 mediates endoplasmic reticulum stress-induced insulin resistance

180 Genetic inactivation of the endoplasmic reticulum stress-induced JNK/TRAF1 axis as w

181 rotein metabolism and adaptive activation of endoplasmic-reticulum-stress-induced survival pathways.

182 Treatment with tunicamycin, an endoplasmic reticulum stress inducer, increased cleaved

183 To test the role of the endoplasmic reticulum stress inducer, PERK we compared t

184 actors such as Hrd1; homocysteine-inducible, endoplasmic reticulum stress-inducible, ubiquitin-like d

185 es consistent with facets of T2DM, including endoplasmic reticulum stress, inflammation, and hyperpro

186 tions include histone deacetylase inhibitor, endoplasmic reticulum stress inhibitor, ammonia sink, an

187 We examined whether endoplasmic reticulum stress is also involved in the pat

188 Endoplasmic reticulum stress is an evolutionarily conser

189 Endoplasmic reticulum stress is defined as the accumulat

190 In mouse OIR, adipose endoplasmic reticulum stress is increased, and APN produ

191 COMP in growth plate chondrocytes activates endoplasmic reticulum stress, leading to inflammation an

192 ounts of conjugation with polyubiquitin, and endoplasmic reticulum stress levels.

193 anscript levels of selected genes related to endoplasmic reticulum stress, lipid synthesis, and fatty

194 Endoplasmic reticulum stress manifest in upregulation of

195 two transcription factors NF-kappaB and the endoplasmic reticulum stress marker, the growth arrest a

196 ion was associated with higher expression of endoplasmic reticulum stress markers (CHOP and GRP78).

197 (AECs), which correlated with expression of endoplasmic reticulum stress markers in AECs.

198 umans significantly increases tribbles 3 and endoplasmic reticulum stress markers in skeletal muscle.

199 Neither endoplasmic reticulum stress markers nor endoplasmic ret

200 increases APN production by reducing adipose endoplasmic reticulum stress markers.

201 es, plaque ulcerations, and phosphoactivated endoplasmic reticulum stress markers.

202 In vitro, fumarate stimulated endoplasmic reticulum stress, matrix gene expression, an

203 detected presymptomatically, suggesting that endoplasmic reticulum stress may play an early and possi

204 tations in the WFS1 gene predispose cells to endoplasmic reticulum stress-mediated apoptosis and may

205 In addition, acrolein induced endoplasmic reticulum stress-mediated death of epithelia

206 edistribution of tight junction proteins and endoplasmic reticulum stress-mediated epithelial cell de

207 onatin-induced aberrant Ca(2+) signaling and endoplasmic reticulum stress might underlie LD pathogene

208 on of inflammatory response, oxidative load, endoplasmic reticulum stress, mitochondrial damage, and

209 rther validated by demonstrating increase in endoplasmic reticulum stress of MDA-MB-468 cells with ti

210 of Akt-SREBP-1 activation, and inhibition of endoplasmic reticulum stress or SREBP-1 prevented angiot

211 tion of FKBP10, as well as its regulation by endoplasmic reticulum stress or transforming growth fact

212 e (UPR), dictate whether cells will adapt to endoplasmic reticulum stress or undergo apoptosis.

213 lysis in mice without evidence of autophagy, endoplasmic reticulum stress, or necroptosis.

214 d hepatic metabolic disorders, which trigger endoplasmic reticulum stress, oxidative stress, and fina

215 emonstrated that heat shock, osmotic stress, endoplasmic reticulum stress, oxidative stress, DNA dama

216 Our results therefore indicate that the endoplasmic reticulum stress pathway could potentially b

217 The unfolded protein response is an endoplasmic reticulum stress pathway mediated by the pro

218 previously predicted a role for CHAC1 in the endoplasmic reticulum stress pathway, linked functionall

219 We found that endoplasmic reticulum stress pathways were activated and

220 ).d(-)(1), a prevention paradigm) attenuated endoplasmic reticulum stress, peripheral nerve dysfuncti

221 Endocrine disrupting chemicals induce endoplasmic reticulum stress, perturb NF-kappaB, and p53

222 Endoplasmic reticulum stress plays an important role in

223 Instead, we found that endoplasmic reticulum stress potentiated HIF-1 activity

224 s results in increased intracellular Ca(2+), endoplasmic reticulum stress, proteasomal dysfunction, a

225 Furthermore, pharmacological inhibition of endoplasmic reticulum stress reduced the endoplasmic ret

226 g accumulation of ubiquitinated proteins and endoplasmic reticulum stress-related apoptosis in variou

227 t to bortezomib, were sensitive to RA190 via endoplasmic reticulum stress-related apoptosis.

228 Endoplasmic reticulum stress-related dysfunction in Wolf

229 sitivity of mutant ERBB2-expressing cells to endoplasmic reticulum stress relied upon a UPR effector

230 ne CASP3 is likely governed by an integrated endoplasmic reticulum stress response (ERSR) and is cons

231 lpha/ATF4 signaling branch of the integrated endoplasmic reticulum stress response (IERSR) is activat

232 Liraglutide improved the cardiac endoplasmic reticulum stress response and also improved

233 signs of immunogenic cell death including an endoplasmic reticulum stress response and exposure of ca

234 l, we confirmed that podocyte injury induces endoplasmic reticulum stress response and upregulated un

235 T1 blockage does not alter mTOR signaling or endoplasmic reticulum stress response but can be modulat

236 cal functions in triglycerides transport and endoplasmic reticulum stress response due to its unique

237 increased GRP78 expression is indicative of endoplasmic reticulum stress response during HPS, which

238 isplayed aberrant expression of several host endoplasmic reticulum stress response genes and chaperon

239 ous GWAS findings in the HLA region; and (3) endoplasmic reticulum stress response genes.

240 n species levels, auranofin induced a lethal endoplasmic reticulum stress response in cultured and pr

241 The integrated endoplasmic reticulum stress response inLeishmania amazo

242 vidence suggests that after podocyte injury, endoplasmic reticulum stress response is activated, but

243 The endoplasmic reticulum stress response is postulated to p

244 stimulate protein synthesis, resulting in an endoplasmic reticulum stress response mediated by Perk.

245 rtners are associated with diseases in which endoplasmic reticulum stress response or sensitivity to

246 have illustrated detrimental changes in the endoplasmic reticulum stress response or unfolded protei

247 d activation of the proapoptotic arms of the endoplasmic reticulum stress response that is probably s

248 ologous protein 10 (CHOP), a mediator of the endoplasmic reticulum stress response, was elevated in s

249 testing of small-molecule modulators of the endoplasmic reticulum stress response, which improved bo

250 ggesting that specific TG6 mutants elicit an endoplasmic reticulum stress response.

251 uroplasticity and deployment of a successful endoplasmic reticulum stress response.

252 ls was TNFalpha independent but involved the endoplasmic reticulum stress response.

253 is believed to be a signal transducer in the endoplasmic reticulum stress response.

254 tro and in vivo, and subsequent induction of endoplasmic reticulum stress response.

255 critical tumor suppressor by augmenting the endoplasmic reticulum stress response.

256 ced proliferation, increased metabolism, and endoplasmic reticulum stress-response activation in trop

257 machinery and the IRE1-alpha-MKK4 arm of the endoplasmic-reticulum-stress-response pathway.

258 ATM repressed IL-23, we examined a role for endoplasmic reticulum stress responses by measuring gene

259 Recent findings have linked endoplasmic reticulum stress responses mediated by inosi

260 Endoplasmic reticulum stress resulting from abnormal fol

261 T protein aggregates in hepatocytes leads to endoplasmic reticulum stress, resulting in impairment of

262 enzyme-1 alpha (IRE1alpha) protein caused by endoplasmic reticulum stress results in the homodimeriza

263 pathway causing cardiac hypertrophy involves endoplasmic reticulum stress sensor PERK (protein kinase

264 w that during Brucella abortus infection, an endoplasmic reticulum stress sensor, IRE1alpha, initiate

265 master stress regulator-detaches from three endoplasmic reticulum stress sensors (IRE1alpha, PERK, a

266 GGF1 protein therapy and miR-183-5p regulate endoplasmic reticulum stress signaling and block endopla

267 34 phosphorylation on tyrosine 262 modulates endoplasmic reticulum stress signaling and cell fate.

268 onical, AGGF1-mediated regulatory system for endoplasmic reticulum stress signaling associated with i

269 Mechanistically, AGGF1 regulates endoplasmic reticulum stress signaling by inhibiting ERK

270 ence of alpha-synuclein expression, specific endoplasmic reticulum stress signaling events were signi

271 el consistently associated with induction of endoplasmic reticulum stress signaling in mouse models a

272 This protein complex is also essential for endoplasmic reticulum stress signaling induction, possib

273 The major endoplasmic reticulum stress signaling pathway causing c

274 CM-P), phosphorylation of stress kinases and endoplasmic reticulum stress signaling was increased, in

275 omote lesion stability through modulation of endoplasmic reticulum stress signaling, and attenuation

276 adipokines and macrophage infiltration), the endoplasmic reticulum stress signaling, and decrease of

277 tes to antiapoptotic-like cell death via the endoplasmic reticulum stress-signaling pathway rather th

278 f glioma proliferation rate, malignancy, and endoplasmic reticulum stress statuses.

279 100 only) exhibited only subtle increases in endoplasmic reticulum stress, suggesting that an altered

280 RCN1 mutant cell lines were revealed to have endoplasmic reticulum stress, suggesting the involvement

281 in 1 (Pdx1) regulates beta-cell survival and endoplasmic reticulum stress susceptibility, in part thr

282 Accumulation of misfolded proteins triggers endoplasmic reticulum stress that leads to unfolded prot

283 intracellularly, its accumulation triggering endoplasmic reticulum stress that results in abnormal SL

284 veolar epithelial metaplasia, and epithelial endoplasmic reticulum stress that were evident after the

285 resulting in the induction of oxidative and endoplasmic reticulum stress, the formation of the infla

286 breast cancer cells 1 induces DNA damage and endoplasmic reticulum stress, the latter being responsib

287 tokine and microbial stimulation to suppress endoplasmic reticulum stress, thereby assuring antiinfla

288 SREBP-1 and endoplasmic reticulum stress thus provide potential nove

289 at a functional RNase domain is required for endoplasmic reticulum stress tolerance, and that both th

290 f the spliced form of X-box protein-1, a key endoplasmic reticulum stress transcription factor.

291 ession of Creb3l2, whose gene product is the endoplasmic reticulum stress transducer crucial for chon

292 Oxidative and endoplasmic reticulum stresses up-regulate Slc7a11 expre

293 ty via MTT assay, neurosphere formation, and endoplasmic reticulum stress/UPR-responsive proteins.

294 Notably, angiotensin II-induced endoplasmic reticulum stress was identified as a key med

295 iptional and translational analyses revealed endoplasmic reticulum stress was not the etiology of our

296 rate under pathogenic, oxidative, heat, and endoplasmic reticulum stress was observed.

297 8), and C/EBP-homologous protein, markers of endoplasmic reticulum stress, was more prominent in the

298 udies demonstrate a role for this variant in endoplasmic reticulum stress, which is consistent with t

299 Endoplasmic reticulum stress, which results from protein

300 myeloid cells, polyploidization can trigger endoplasmic reticulum stress with consequent exposure of