ライフサイエンスコーパス: endothelial

1 ystemic inflammation (interleukin 6 [IL-6]), endothelial activation (angiopoietin-2), and microvascul

2 -2 is a previously unrecognized component of endothelial adherens junctions.

3 myoferlin was shown to be also expressed in endothelial and cancer cells where it was shown to modul

4 ristics by expressing phenotypic markers for endothelial and mesenchymal cells.

5 way, resulting in time- and isoform-specific endothelial and neuronal nitric oxide synthase activatio

6 ns, a MSC subpopulation carrying markers for endothelial and pericytic cells was lower in the presenc

7 Pan-endothelial- and lymphatic-specific Hk2 knockouts phenoc

8 Endothelial LKB1 may regulate endothelial angiogenesis and tumor growth by modulating

9 Deficiency in endothelial autophagy also increased TNF-alpha-induced i

10 was unchanged in atheroprone areas, in which endothelial autophagy flux is already blocked.

11 In hypercholesterolemic mice, deficiency in endothelial autophagy increased plaque burden only in th

12 and adherens junction disassembly leading to endothelial barrier breakdown.

13 ress syndrome (ARDS) is caused by widespread endothelial barrier disruption and uncontrolled cytokine

14 1P effectively could reverse alcohol-induced endothelial barrier dysfunction using both cultured endo

15 le-positive areas further exhibited impaired endothelial barrier function as illuminated by Evans blu

16 hat initiates pericyte loss and breakdown of endothelial barrier function by generating the diol 19,2

17 The endothelial binding parameter Kb varied highly across br

18 ized and co-immunoprecipitated with vascular endothelial cadherin-based complexes, including beta- an

19 S by enhancing its interaction with vascular endothelial-cadherin.

20 in (Plvap) that is specifically localized to endothelial caveolae in vivo and compared its effects to

21 erosis and plaque composition by inducing an endothelial cell (BmxCreER(T2)-driven)-specific or smoot

22 giogenesis was not associated with classical endothelial cell (EC) activation signs, such as Vegfa/VE

23 To examine its role in endothelial cell (EC) biology, we generated mice with ca

24 In view of the fact that the spontaneous endothelial cell (EC) regeneration is a slow and insuffi

25 of cardiomyocytes and functionally distinct endothelial cell (EC) subtypes from cardiogenic versus h

26 All the effects are fully rescued by endothelial cell (EC)-specific overexpression of Kir2.1.

27 NPs (50, 100, and 150 nm) in a human corneal endothelial cell (HCEC) line, B4G12.

28 o-angiogenic cytokine that potently promotes endothelial cell activation and pathological angiogenesi

29 agulation and anticoagulation, fibrinolysis, endothelial cell activation, matricellular protein relea

30 acute flow perturbation promoted downstream endothelial cell activation, neutrophil accumulation, en

31 VEGF antagonism also increased markers of endothelial cell activation, which was partially reduced

32 n of Amigo2 in QRsP-11 cells increased liver endothelial cell adhesion and liver metastasis.

33 endothelial cell injury linked to increased endothelial cell AGEs and RAGE levels.

34 Endothelial cell angiogenic responses in vitro and in vi

35 type 1 receptor antibody (AT1R-Ab) and anti-endothelial cell antibody (AECA).

36 Endothelial cell apoptosis induced by oxidative stress i

37 Ablation of endothelial cell class II major histocompatibility compl

38 pids and carbohydrates over time, induced by endothelial cell contact.

39 es of tumor-associated signals in regulating endothelial cell contractility and adherens junction dis

40 ars; 8516 men and 6016 women), the mean (SD) endothelial cell count was 2732 (437) cells/mm2.

41 the vicious cycle of complement activation, endothelial cell damage, platelet activation, and thromb

42 al cell activation, neutrophil accumulation, endothelial cell death and desquamation, and mural throm

43 iferations (GMP), accompanied by only modest endothelial cell death.

44 Specular microscope examination revealed an endothelial cell density (ECD) of 1532/mm(2) in patient

45 sm (TA), central corneal thickness (CCT) and endothelial cell density 12 months postoperatively; and

46 Endothelial cell density was measureable in 6 cooperativ

47 ble transcription factor-1 (HIF-1)-dependent endothelial cell glycolysis, which is crucial for pathol

48 ry disease may affect tissue oxygenation and endothelial cell health.

49 c antibody (DSA) causes complement-dependent endothelial cell injury in kidney transplants, as assess

50 /or CS-exposed mice have pulmonary and renal endothelial cell injury linked to increased endothelial

51 Endothelial cell intercellular junction formation was ch

52 rix degradability switches three-dimensional endothelial cell invasion between two distinct modes: si

53 ing and quantifying extracellular changes in endothelial cell layer integrity following the activatio

54 helial cell types (the human brain capillary endothelial cell line hCMEC/D3 and human umbilical vein

55 biogenesis, Cavin-2 plays a critical role in endothelial cell maintenance and function by regulating

56 eature of cellular adhesion, locomotion, and endothelial cell mechanobiology.

57 istically, this occurs through alteration of endothelial cell metabolism.

58 lial barrier dysfunction using both cultured endothelial cell monolayer and in vivo models.

59 in decreased glycolysis, leading to impaired endothelial cell proliferation and migration.

60 ng mechanisms involved in regulating uterine endothelial cell proliferation during pregnancy.

61 ich laminins modulate vascular branching and endothelial cell proliferation during retinal angiogenes

62 17beta-oestradiol- and catecholamine-induced endothelial cell proliferation may be indicative of unap

63 fy the galectin-3-binding molecule(s) on the endothelial cell surface responsible for the galectin-3-

64 Intraportal delivery of endothelial cell therapy or saline was technically succe

65 ts of nitro-oleic acid (OA-NO2) on the human endothelial cell transcriptome.

66 nated) by two phenotypically different human endothelial cell types (the human brain capillary endoth

67 for lymphatic TEM for various migrating and endothelial cell types possesses the capacity to be high

68 ey transplants, as assessed by expression of endothelial cell-associated transcripts (ENDATs), that m

69 table connections are spatially regulated by endothelial cell-intrinsic modulation of mFlt1, suggesti

70 neoplastic cells most closely resemble: the endothelial cell.

71 Primary SJL mouse brain endothelial cells (a target of MAV-1 in vivo) infected e

72 (CRCs), including CTCs and circulating tumor endothelial cells (CECs).

73 Cytokine activation of endothelial cells (EC) upregulates VCAM-1 receptors that

74 igment epithelium (RPE), fenestrated choroid endothelial cells (ECs) and Bruch's membrane, a highly o

75 mune cells, its expression level and role in endothelial cells (ECs) are still unclear.

76 Human endothelial cells (ECs) are widely used to study mechani

77 ally been viewed from the perspective of how endothelial cells (ECs) coordinate migration and prolife

78 al polyethylene glycol (PEG) hydrogel NPs by endothelial cells (ECs) cultured in a microchannel compa

79 Endothelial cells (ECs) express O-glycoproteins that are

80 sintegration, it sensitizes retinal vascular endothelial cells (ECs) to VEGF-A, leading to upregulati

81 Here, we determined that aging of endothelial cells (ECs), a critical component of the BM

82 Estrogens protect against apoptosis of endothelial cells (ECs), one of the hallmarks of endothe

83 d PE STBEVs by primary human coronary artery endothelial cells (HCAEC) and the effects of free HbF on

84 We treated human umbilical vein endothelial cells (HUVECs) with E2, TNFalpha, or both an

85 ved cardiomyocytes, smooth muscle cells, and endothelial cells (in a 2:1:1 ratio) to generate the hCM

86 a1 gene in adult mice resulted in loss of LV endothelial cells (LECs) specifically from the leaflets

87 support the notion that, in pulmonary artery endothelial cells (PAECs), expression of transcription f

88 guanine) exclusively localized to glomerular endothelial cells after 3 weeks of diabetes, and these a

89 eased distance between Gli1(+) pericytes and endothelial cells after AKI (mean+/-SEM: 3.3+/-0.1 micro

90 f prolonged TNFalpha exposure on the fate of endothelial cells and found that such treatment induced

91 h reduced ADAMTS7 expression in human aortic endothelial cells and lymphoblastoid cell lines.

92 Only TLN-treated human endothelial cells and neonatal porcine islets prolonged

93 comprised mainly of astrocytes, while brain endothelial cells and pericytes encase the surface, acti

94 ion could alter the glycosylation pattern of endothelial cells and thereby impact adhesion of circula

95 tissues and blood revealed Vwf expression in endothelial cells and thrombocytes.

96 rate that the interactions between FCSCs and endothelial cells are essential for FCSC-derived vascula

97 helial cells enhanced neutrophil adhesion to endothelial cells but inhibited neutrophil transmigratio

98 TRIN affects the functional transcriptome of endothelial cells by down-regulating several genes impor

99 Y POINTS: A reduction in Kindlin-2 levels in endothelial cells compromises vascular barrier function.

100 Clarification of the origins of coronary endothelial cells during cardiac repair is essential for

101 on via silencing of small interfering RNA in endothelial cells enhanced neutrophil adhesion to endoth

102 In the retina, endothelial cells form a blood-retina barrier by virtue

103 Transcriptomic analysis in endothelial cells identified nitric oxide (NO) as major

104 nt (bMTM) comprising co-culture of tumor and endothelial cells in a 3D environment.

105 cross-talk between BM osteolineage cells and endothelial cells in regulating hematopoietic reconstitu

106 Nitric oxide (NO) produced by endothelial cells in response to cytokines displays anti

107 linked to degradation of tight junctions in endothelial cells in vitro, which is blocked by pharmaco

108 was found to promote the migration of human endothelial cells in vitro.

109 ion to P-selectin on activated platelets and endothelial cells induces shedding of the P-selectin ect

110 acquisition of tissue-specific properties in endothelial cells is essential for vascular function.

111 Lymphatic endothelial cells lack the Ca(2+) -activated K(+) channe

112 n of the cross-talk between granulocytes and endothelial cells may lead to new therapeutic approaches

113 produced from mammalian cells, infects fetal endothelial cells much more efficiently than other patho

114 ggest that itraconazole selectively inhibits endothelial cells rather than cancer cells by targeting

115 ells and CD34 expression by liver sinusoidal endothelial cells remained stable, consistent with the a

116 duced migration and proliferation of retinal endothelial cells stimulated with VEGF.

117 We demonstrated that these endothelial cells supply the hepatocyte growth factor (H

118 miR-204, whereas overexpression of Sirt1 in endothelial cells suppresses miR-204-induced ER stress.

119 Collectrin is also expressed in endothelial cells throughout the vasculature, where it r

120 nsactivator can reduce the capacity of human endothelial cells to recruit and activate alloreactive T

121 alone can activate human brain microvascular endothelial cells to stimulate adhesion molecules, CCL2,

122 rette smoke extract on human coronary artery endothelial cells under oscillatory, normal laminar and

123 Endocan is a proteoglycan secreted by endothelial cells under the control of inflammatory cyto

124 mal care and use committee, autologous liver endothelial cells were grown from core hepatic specimens

125 Instead, cardiac endothelial cells were likely to proliferate and generat

126 Arterial endothelial cells were robustly generated from multiple

127 Results Liver endothelial cells were successfully isolated, cultured,

128 Coculturing endothelial cells with astrocytes yielded the greatest r

129 We treated human aortic endothelial cells with exogenous amphiphiles, shown prev

130 n and leukocyte adhesion in quiescent tumour endothelial cells with intact insulin receptors and part

131 cell line hCMEC/D3 and human umbilical vein endothelial cells), and without interference of the fluo

132 y combining in vivo fluorescence labeling of endothelial cells, a novel tissue-clearing technique, li

133 Knockdown of Sirt1 in endothelial cells, and conditional deletion of endotheli

134 ls, committed progenitor cells, fibroblasts, endothelial cells, and immune cells.

135 ell types, such as tubular epithelial cells, endothelial cells, and podocytes, working in concert.

136 In human pulmonary microvascular endothelial cells, G was 20.4 +/- 12 Pa and decreased by

137 dels using a HCC cell line, HepG2, and human endothelial cells, HUVECs, as well as ex vivo and in viv

138 and secondary HUS are simultaneous damage to endothelial cells, intravascular hemolysis, and activati

139 through the endothelium by opening holes in endothelial cells, known as transcellular tunnels, which

140 which prevented VWF multimer accumulation on endothelial cells, or by an anti-VWF Ab.

141 In endothelial cells, this phenomenon might contribute to v

142 Moreover, they were internalized by endothelial cells, thus supporting their involvement in

143 al coupling-from the potential source of NO, endothelial cells, to the potential beneficiary from the

144 n of the Notch ligand delta-like 4 (DLL4) in endothelial cells, we find that activation of the MAPK/E

145 hese embryos exhibited aberrant alignment of endothelial cells, which disturbed the feto-maternal cir

146 dependent expression of the ET-B receptor in endothelial cells, which in turn mediates the decrease i

147 a shift toward inhibition of proteolysis in endothelial cells, with decreased expression of extracel

148 evaluated in vitro with human microvascular endothelial cells-1 and in vivo with the Matrigel plug a

149 activation of the mTOR pathway in lymphatic endothelial cells.

150 in vitro tube formation and proliferation of endothelial cells.

151 ed when VEGF binds to its receptors on tumor endothelial cells.

152 reduces ER-mitochondrial contact in retinal endothelial cells.

153 miR-204 mimic (miR-204 M) decreased Cav1 in endothelial cells.

154 of Slco2a1 in the tumour-associated vascular endothelial cells.

155 edium, promoted EC-to-OSB conversion of 2H11 endothelial cells.

156 ufficient to induce phenotypic conversion of endothelial cells.

157 akaryocytes, myeloid cells, fibroblasts, and endothelial cells.

158 N1/CYR61 is highly regulated by stiffness in endothelial cells.

159 latory roles in a number of keratinocyte and endothelial cellular traits associated with the wound he

160 al exercise in the endothelium that triggers endothelial communication to mesenteric vessel muscle ce

161 hrombin-induced increase in the amplitude of endothelial cytosolic Ca(2+) oscillations.

162 rombomodulin (sCD141) and ICAM-1, reflecting endothelial damage.

163 ession were significantly up-regulated after endothelial denudation at 24 h and 21 d compared with co

164 Using the mouse endothelial-derived EOMA cell line as a model of HE, we

165 Depletion of endothelial-derived MPs from ACS patients reduced the in

166 rmore, to mimic the antagonistic efficacy of endothelial-derived prostacyclin, we determined how Ilop

167 cal junction disassembly associated with the endothelial-derived tumor Kaposi sarcoma.

168 gest that optimal combinatorial ECMs enhance endothelial differentiation, compared to many single-fac

169 er, impaired AJ activity can account for the endothelial dilator dysfunction in old arteries.

170 evident after the occurrence of age-related endothelial dysfunction and diminished distensibility.

171 oscillatory shear stress further exacerbates endothelial dysfunction in patients with moderate-severe

172 ive against oscillatory shear stress-induced endothelial dysfunction in patients with moderate-severe

173 thelial cells (ECs), one of the hallmarks of endothelial dysfunction leading to cardiovascular disord

174 Endothelial dysfunction may underlie this; however, the

175 netically predisposed and is associated with endothelial dysfunction that is induced by oxidative str

176 injury, inflammation, oxidative stress, and endothelial dysfunction, all of which may perpetuate a n

177 apy can also cause myocardial damage, induce endothelial dysfunction, and alter cardiac conduction.

178 tic milieu characterized by blood stasis and endothelial dysfunction.

179 inducing oxidative stress, inflammation, and endothelial dysfunction.

180 ythematosus (SLE) is a known risk factor for endothelial dysfunction.

181 ERK5 signalling may be useful to counteract endothelial dysfunction.

182 g was performed after DMEK surgery for Fuchs endothelial dystrophy.

183 ndrial DNA damage associated with glomerular endothelial EDNRA expression.

184 s NO stems from neuronal NOS (nNOS), but not endothelial (eNOS).

185 Endothelial-enriched PCs (CD34(+)/VEGF(+)) were independ

186 rotects against tunicamycin-induced vascular/endothelial ER stress, associated impairment of endothel

187 Endothelial exocytosis of Weibel-Palade body (WPB) is on

188 Therapeutic targeting of endothelial FABP4 by siRNA in vivo has antiangiogenic an

189 mplicated IgG as the pathogenetic ligand for endothelial FcgammaRIIB in obesity-induced insulin resis

190 ther low flow oxygen administration improves endothelial function and is protective against oscillato

191 There was no effect on endothelial function as measured by reactive hyperaemia

192 d associations of PTSD with inflammatory and endothelial function biomarkers, but most work has been

193 erapy was not associated with improvement of endothelial function compared with the sham device.

194 el demonstrated a significant improvement in endothelial function following anti-TNF-alpha treatment

195 ontinuous positive airway pressure, improves endothelial function in patients with severe OSA.

196 ts that anti-TNF-alpha treatment may improve endothelial function in RA patients.

197 ymptomatic hyperuricemia but did not improve endothelial function in this sample of patients.

198 1 (Cav1) is also an important determinant of endothelial function.

199 These findings reveal a novel link between endothelial glutamine and asparagine metabolism in vesse

200 label provided the most reliable measure of endothelial glycocalyx anatomy, correlating with paired,

201 -1 inhibition, however, increased glomerular endothelial glycocalyx coverage, with preservation of he

202 g with paired, numerically smaller values of endothelial glycocalyx depth (0.078 +/- 0.016 mum) from

203 nd analysis techniques yield measurements of endothelial glycocalyx depth that vary by over an order

204 to determine the adjusted association of the endothelial glycocalyx layer (EGL) and tight and adheren

205 We thus identify Gpr124 as an endothelial GPCR specifically required for endothelial W

206 gulatory compliant tissue-engineered corneal endothelial graft substitute can alleviate this reliance

207 rating keratoplasty, while sparing a healthy endothelial graft.

208 or microvascular density (MVD), and vascular endothelial growth factor (VEGF) expression) from 9 pati

209 Vascular endothelial growth factor (VEGF) is implicated in the pe

210 as a negative feedback regulator of vascular endothelial growth factor (VEGF) receptor activation.

211 We also show how vascular endothelial growth factor (VEGF) regulates PRKCB promote

212 Because deficiency of vascular endothelial growth factor (VEGF) results in thrombotic m

213 rther demonstrate the importance of Vascular Endothelial Growth Factor (VEGF) secretion for this path

214 ling transducers and isoforms along vascular endothelial growth factor (VEGF) signaling pathways at c

215 Anti-vascular endothelial growth factor (VEGF) therapy has demonstrate

216 cell-derived factor 1 (SDF-1alpha), vascular endothelial growth factor (VEGF), hypoxia-inducible fact

217 otein 10, interleukin (IL)-6, IL-8, vascular endothelial growth factor (VEGF), monocyte chemoattracti

218 Vascular endothelial growth factor (VEGF)-A has been implicated i

219 Alternate splicing in the exon-8 of vascular endothelial growth factor (VEGF)-A results in production

220 ell proliferation by regulating the vascular endothelial growth factor (VEGF)-A, VEGF-C, FGFR3, and p

221 ls, von Willebrand factor (vWF) and vascular endothelial growth factor (VEGF)-C expression were measu

222 Vascular endothelial growth factor (VEGF)-D is capable of inducin

223 eriodontal diseases, supported with vascular endothelial growth factor (VEGF-A) and tumor necrosis fa

224 ells where it was shown to modulate vascular endothelial growth factor (VEGFR)-2 and epidermal growth

225 Inadequate tumor uptake of the vascular endothelial growth factor antibody bevacizumab could exp

226 Up-regulation of vascular endothelial growth factor enhances the therapeutic effec

227 Vascular endothelial growth factor has emerged as a significant c

228 vels and mesenchymal cells, but not vascular endothelial growth factor in Hyal2(-/-) embryonic hearts

229 t experiences specific to receiving vascular endothelial growth factor inhibitors (anti-VEGF) for wet

230 raised that intravitreal dosing of vascular endothelial growth factor inhibitors in DME could be ass

231 intervention requiring 1 or 2 anti-vascular endothelial growth factor injections only.

232 ing intravitreal injections of anti-vascular endothelial growth factor or verteporfin photodynamic th

233 rmation (evidenced by a decrease in vascular endothelial growth factor receptor 1 positive (VEGFR1(+)

234 he combination of anti-TLR2 and antivascular endothelial growth factor receptor 2 yielded an additive

235 To evaluate if the up-regulation of vascular endothelial growth factor strengthens the protective eff

236 Participants who received antivascular endothelial growth factor therapies for neovascular AMD

237 factors (fibroblast growth factor, vascular endothelial growth factor, and platelet-derived growth f

238 sels of the microcirculation is critical for endothelial homeostasis and inflammation.

239 SW620Exos) exhibited higher ability to cause endothelial hyperpermeability than exosomes from the non

240 We show that endothelial hypoxia-inducible factor 1alpha (HIF-1alpha)

241 These in vitro effects require endothelial IL-1 receptors, shown by immunofluorescence

242 -producing monocytes, which stimulated brain endothelial IL-1R1.

243 e complement activation, likely triggered by endothelial injury.

244 show that the rapid S1P-induced increase in endothelial integrity is mediated by a S1PR1-Galphai-Cdc

245 icle-tracking methods for studying bacterial-endothelial interaction biomechanics.

246 ere translated by assessing human neutrophil-endothelial interactions under flow: PD1n-3 DPA and RvD5

247 is review provides insight into the platelet-endothelial interface, based on in vitro flow chamber st

248 KEY POINTS: Endothelial inwardly rectifying K(+) (Kir2.1) channels r

249 ocular lenses (IOLs) after Descemet membrane endothelial keratoplasty (DMEK).

250 dynamics during Descemet stripping automated endothelial keratoplasty (DSAEK) using intraoperative op

251 Disruption of endothelial KLF2 results in dysregulation of lung microv

252 ther differentiation of the progenitors into endothelial lineage.

253 Endothelial LKB1 may regulate endothelial angiogenesis a

254 A search for epigenetically controlled endothelial lncRNAs yielded lncRNA n342419, here termed

255 to identify novel and functionally important endothelial lncRNAs.

256 Thus, our findings suggest that targeting endothelial LPP enhances the efficacy of chemotherapy in

257 uble molecules to pass through the capillary endothelial membrane while limiting the passage of patho

258 This cross-talk between nerves and endothelial metabolism could potentially be targeted as

259 alyses indicated that Cavin-2 is secreted in endothelial microparticles (EMPs) and is required for EM

260 ted the presence of the kinin B1 receptor on endothelial microvesicles and its contribution to the in

261 Thus, B1 receptor-positive endothelial microvesicles may contribute to chronic infl

262 , deletion of which caused severe defects in endothelial migration.

263 Glomerular endothelial mitochondrial dysfunction was associated wit

264 cell adhesion onto TNF-alpha-activated human endothelial monolayers.

265 used to characterize the following MP types: endothelial MPs, epithelial MPs (epithelial cell adhesio

266 lating the expression of adhesion-associated endothelial mRNA targets.

267 y underlie this; however, the association of endothelial nitric oxide (NO) pathways with disease seve

268 Instead, expression of endothelial nitric oxide synthase (eNOS), which generate

269 evated mitochondrial O2(. -), and diminished endothelial nitric oxide.

270 ore, cardiac O2 consumption is controlled by endothelial NO in a paracrine, but not intracrine, fashi

271 d with enhanced oxidative stress and reduced endothelial NO production is a further indication for th

272 vascular oxidative stress and improvement of endothelial NO production represent reasonable therapeut

273 1B siRNA-transfected mice also had augmented endothelial outgrowth.

274 1B knockdown or an EPAC antagonist increases endothelial permeability and that VEGF has no additive e

275 echanisms of angiogenesis, inflammation, and endothelial permeability.

276 a proinflammatory, apoptotic, and senescent endothelial phenotype.

277 like properties of tumor cells, also inhibit endothelial phenotypes of breast cancer cells adopted in

278 further enriched by selection for a CD133(+) endothelial progenitor cell population.

279 ted receptors and alphaVbeta3-overexpressing endothelial progenitor EP cells) and the kinase inhibito

280 proposal suggests that neovessels form from endothelial progenitors able to assemble the intimal lay

281 impairments after hypoperfusion possibly via endothelial protection supporting its potential use in t

282 requires the cofactor thrombomodulin and the endothelial protein C receptor.

283 teins of the PfEMP1 family that bind various endothelial receptors.

284 , injection of wild-type platelets inhibited endothelial recovery in wire-injured carotid arteries, b

285 annels play a critical role in physiological endothelial responses to flow.

286 with TNFalpha to induce the up-regulation of endothelial selectins and adhesion molecules, ultimately

287 Targeting endothelial senescence could be a new therapeutic avenue

288 dothelial cells, and conditional deletion of endothelial Sirt1 in mice, promotes ER stress via upregu

289 lium-dependent vasorelaxation, and preserves endothelial Sirt1.

290 Finally, we demonstrate that mice with endothelial-specific deletion of miR-322 (miR-424 orthol

291 of Spns2, either globally or in a lymphatic endothelial-specific manner, creates a circulating lymph

292 ur findings establish that pericytes promote endothelial sprouting, which results in the loss of side

293 rected differentiation of cardiomyocytes and endothelial subtypes from hPSCs.

294 ata2 reporter expression in cells undergoing endothelial-to-hematopoietic transition.

295 mice causes increased HA, which may promote endothelial-to-mesenchymal transition and proliferation

296 n and not derived from bone marrow lineages, endothelial-to-mesenchymal transition, or blood.

297 , suggest that HYAL2 is important to inhibit endothelial-to-mesenchymal transition.

298 (DCs), and residential macrophages near high endothelial venules, the results highlight critical role

299 n endothelial GPCR specifically required for endothelial Wnt signaling and BBB integrity under pathol

300 We concluded that corneal endothelial wound healing in rabbits has different outco