ライフサイエンスコーパス: endothelial cell

1 neoplastic cells most closely resemble: the endothelial cell.

2 es with minimal deformation of the sensitive endothelial cell.

3 of Slco2a1 in the tumour-associated vascular endothelial cells.

4 activation of the mTOR pathway in lymphatic endothelial cells.

5 edium, promoted EC-to-OSB conversion of 2H11 endothelial cells.

6 ufficient to induce phenotypic conversion of endothelial cells.

7 akaryocytes, myeloid cells, fibroblasts, and endothelial cells.

8 otes strong activation of brain blood vessel endothelial cells.

9 and corneal keratocytes as well as vascular endothelial cells.

10 cting as its transcriptional co-activator in endothelial cells.

11 ks favor interactions with Flt1 mutant mouse endothelial cells.

12 es sprouting, migration and proliferation of endothelial cells.

13 on, subsequently preventing the apoptosis of endothelial cells.

14 N1/CYR61 is highly regulated by stiffness in endothelial cells.

15 00 ligand (CD200L) expressed by newly formed endothelial cells.

16 origins of fibroblasts that give rise to new endothelial cells.

17 d phospholipase C beta-mediated signaling in endothelial cells.

18 the release of microvesicles from glomerular endothelial cells.

19 imary cultures of vascular smooth muscle and endothelial cells.

20 , TNF-alpha, and IL-10 secretion in vascular endothelial cells.

21 ced transcription of ET-B in human and mouse endothelial cells.

22 s were observed in human brain microvascular endothelial cells.

23 -cadherin, and CD106/Endoglin, from vascular endothelial cells.

24 in vitro tube formation and proliferation of endothelial cells.

25 ed when VEGF binds to its receptors on tumor endothelial cells.

26 reduces ER-mitochondrial contact in retinal endothelial cells.

27 miR-204 mimic (miR-204 M) decreased Cav1 in endothelial cells.

28 evaluated in vitro with human microvascular endothelial cells-1 and in vivo with the Matrigel plug a

29 Primary SJL mouse brain endothelial cells (a target of MAV-1 in vivo) infected e

30 y combining in vivo fluorescence labeling of endothelial cells, a novel tissue-clearing technique, li

31 ibition of monocyte exosome release reverses endothelial cell activation and monocyte chemotaxis.

32 o-angiogenic cytokine that potently promotes endothelial cell activation and pathological angiogenesi

33 ification of lipoproteins and phospholipids, endothelial cell activation, and macrophage infiltration

34 agulation and anticoagulation, fibrinolysis, endothelial cell activation, matricellular protein relea

35 acute flow perturbation promoted downstream endothelial cell activation, neutrophil accumulation, en

36 VEGF antagonism also increased markers of endothelial cell activation, which was partially reduced

37 n of Amigo2 in QRsP-11 cells increased liver endothelial cell adhesion and liver metastasis.

38 dothelial markers (CD34, endomucin, platelet endothelial cell adhesion molecule 1, and plasmalemmal v

39 F-A165 b restores the expression of platelet endothelial cell adhesion molecule in glomerular endothe

40 dhesion molecule (MCAM)/MUC18, CD31/platelet endothelial cell adhesion molecule-1 (PECAM-1), CD144/VE

41 igher cell migration, transmigration through endothelial cells, adhesion to stromal cells, and cell p

42 guanine) exclusively localized to glomerular endothelial cells after 3 weeks of diabetes, and these a

43 eased distance between Gli1(+) pericytes and endothelial cells after AKI (mean+/-SEM: 3.3+/-0.1 micro

44 ODS AND Based on a transcriptome analysis of endothelial cells after miR-100 overexpression, we ident

45 endothelial cell injury linked to increased endothelial cell AGEs and RAGE levels.

46 markers of the neurovascular unit, including endothelial cells and astrocytes, in mouse models of AD.

47 s signaling in several cell types, including endothelial cells and bone marrow (BM)-derived cells.

48 We then stimulated primary aortic endothelial cells and ex-vivo atherosclerotic tissue wit

49 f prolonged TNFalpha exposure on the fate of endothelial cells and found that such treatment induced

50 thelial cell adhesion molecule in glomerular endothelial cells and glomerular capillary circumference

51 rgeted transcriptomic approaches in vitro in endothelial cells and in vivo in a radiation enteropathy

52 h reduced ADAMTS7 expression in human aortic endothelial cells and lymphoblastoid cell lines.

53 red under flow conditions between irradiated endothelial cells and monocytes.

54 increased TSP1 expression in the vicinity of endothelial cells and near regions of endocardial fibros

55 Only TLN-treated human endothelial cells and neonatal porcine islets prolonged

56 comprised mainly of astrocytes, while brain endothelial cells and pericytes encase the surface, acti

57 sion of chemokines and adhesion molecules in endothelial cells and reduced NF-kappaB activation and m

58 e, adiponectin and T-cadherin colocalized on endothelial cells and synthetic smooth muscle cells in t

59 the point of contact between donor vascular endothelial cells and the recipient's immune cells, with

60 ion could alter the glycosylation pattern of endothelial cells and thereby impact adhesion of circula

61 tissues and blood revealed Vwf expression in endothelial cells and thrombocytes.

62 cted DAG peptide homes to neurovascular unit endothelial cells and to reactive astrocytes in mouse mo

63 d metastasis by facilitating the adhesion to endothelial cells and transendothelial extravasation.

64 cell line hCMEC/D3 and human umbilical vein endothelial cells), and without interference of the fluo

65 in circulating monocytes, pulmonary arterial endothelial cells, and also activated B cells.

66 Knockdown of Sirt1 in endothelial cells, and conditional deletion of endotheli

67 aining protein ACAP2 as an Rab35 effector in endothelial cells, and depletion as well as overexpressi

68 ls, committed progenitor cells, fibroblasts, endothelial cells, and immune cells.

69 ell types, such as tubular epithelial cells, endothelial cells, and podocytes, working in concert.

70 ppressed alpha3beta1-mediated stimulation of endothelial cells, and wounds of epidermis-specific alph

71 Endothelial cell angiogenic responses in vitro and in vi

72 type 1 receptor antibody (AT1R-Ab) and anti-endothelial cell antibody (AECA).

73 Endothelial cell apoptosis induced by oxidative stress i

74 rate that the interactions between FCSCs and endothelial cells are essential for FCSC-derived vascula

75 Human endothelial cells are initiators and targets of the reje

76 duced VWF synthesis, specifically in hepatic endothelial cells, as a critical factor that is regulate

77 Cells, such as cancer or endothelial cells, as well as tumor/tissue grafts, can b

78 ey transplants, as assessed by expression of endothelial cell-associated transcripts (ENDATs), that m

79 ce acute significant cytotoxicity in corneal endothelial cells at concentrations up to 100 microg/mL.

80 ort studies across blood-brain barrier (BBB) endothelial cells, at circulating concentrations, provid

81 In human aortic endothelial cells, BA increased beta1-integrin-Arg-Gly-A

82 mixed gel by coculturing brain microvascular endothelial cells (BMECs) and human mesenchymal stem cel

83 erosis and plaque composition by inducing an endothelial cell (BmxCreER(T2)-driven)-specific or smoot

84 helial cells enhanced neutrophil adhesion to endothelial cells but inhibited neutrophil transmigratio

85 y regulates MEKK3-KLF2/4 signalling in brain endothelial cells, but upstream activators of this disea

86 TRIN affects the functional transcriptome of endothelial cells by down-regulating several genes impor

87 reorientation ("organization") of Prox1(hi) endothelial cells by postnatal day 0.

88 vious changes, specific deletion in vascular endothelial cells caused CNV and a phenotype similar to

89 XCL8 as factors secreted by EVTs that induce endothelial cell CCL14 and CXCL6 expression.

90 (CRCs), including CTCs and circulating tumor endothelial cells (CECs).

91 VE-cadherin at endothelial cell-cell junctions links the contractile ac

92 th factor receptors (VEGFRs) that resides at endothelial cell-cell junctions transduces signals impor

93 Here, isolated primary human corneal endothelial cells (CEnCs) propagated using a dual media

94 Ablation of endothelial cell class II major histocompatibility compl

95 Y POINTS: A reduction in Kindlin-2 levels in endothelial cells compromises vascular barrier function.

96 pids and carbohydrates over time, induced by endothelial cell contact.

97 es of tumor-associated signals in regulating endothelial cell contractility and adherens junction dis

98 ars; 8516 men and 6016 women), the mean (SD) endothelial cell count was 2732 (437) cells/mm2.

99 AECA was evaluated using an endothelial cell crossmatch (ECXM) in patients whose HLA

100 group began to devise a method to establish endothelial cell cultures from human peripheral blood, w

101 the vicious cycle of complement activation, endothelial cell damage, platelet activation, and thromb

102 early on in the disease, causing glomerular endothelial cell damage.

103 al cell activation, neutrophil accumulation, endothelial cell death and desquamation, and mural throm

104 iferations (GMP), accompanied by only modest endothelial cell death.

105 ty (BSCVA), central corneal thickness (CCT), endothelial cell density (ECD) and complication rates.

106 Specular microscope examination revealed an endothelial cell density (ECD) of 1532/mm(2) in patient

107 erative best-corrected visual acuity (BCVA), endothelial cell density (ECD), and complications.

108 ral corneal thickness (CCT, micrometers) and endothelial cell density (ECD).

109 sm (TA), central corneal thickness (CCT) and endothelial cell density 12 months postoperatively; and

110 mean CCT was 492+/-62.10 mum; postoperative endothelial cell density averaged 2026+/-397cells/mm(2)

111 spectively (P = 0.84), and the postoperative endothelial cell density changes were -3+/-10% (P = 0.07

112 Endothelial cell density was measureable in 6 cooperativ

113 ncy-specific proliferation of uterine artery endothelial cells derived from pregnant (P-UAECs), but n

114 a-catenin signaling, is expressed in retinal endothelial cells during angiogenesis and barrier format

115 Clarification of the origins of coronary endothelial cells during cardiac repair is essential for

116 giogenesis was not associated with classical endothelial cell (EC) activation signs, such as Vegfa/VE

117 describe mechanisms by which molecules cross endothelial cell (EC) barriers in normal tissues and in

118 To examine its role in endothelial cell (EC) biology, we generated mice with ca

119 r smooth muscle cell (SMC) proliferation and endothelial cell (EC) dysfunction are critical in the pa

120 We investigated effects of PGA2 on pulmonary endothelial cell (EC) permeability and inflammatory acti

121 In view of the fact that the spontaneous endothelial cell (EC) regeneration is a slow and insuffi

122 of cardiomyocytes and functionally distinct endothelial cell (EC) subtypes from cardiogenic versus h

123 All the effects are fully rescued by endothelial cell (EC)-specific overexpression of Kir2.1.

124 wer this question, we generated a mouse with endothelial cell (EC)-specific overexpression of the hum

125 Cytokine activation of endothelial cells (EC) upregulates VCAM-1 receptors that

126 igment epithelium (RPE), fenestrated choroid endothelial cells (ECs) and Bruch's membrane, a highly o

127 Endothelial cells (ECs) are critical determinants of vas

128 mune cells, its expression level and role in endothelial cells (ECs) are still unclear.

129 Human endothelial cells (ECs) are widely used to study mechani

130 ally been viewed from the perspective of how endothelial cells (ECs) coordinate migration and prolife

131 al polyethylene glycol (PEG) hydrogel NPs by endothelial cells (ECs) cultured in a microchannel compa

132 Endothelial cells (ECs) express O-glycoproteins that are

133 We demonstrate that microvascular endothelial cells (ECs) from Anxa2(-/-) mice display red

134 Glutamine metabolism in endothelial cells (ECs) has been poorly studied.

135 ese results suggest an important role for BM endothelial cells (ECs) in regulating hematopoietic agin

136 Endothelial cells (ECs) in the tumor microenvironment ha

137 eptor 2 (VEGFR2) localized on the surface of endothelial cells (ECs) is a key determinant of the magn

138 We cultured endothelial cells (ECs) on collagen-coupled stiff or com

139 examined the potential of MPs from senescent endothelial cells (ECs) or from patients with acute coro

140 s, we performed proximity ligation assays in endothelial cells (ECs) to demonstrate compound-induced

141 Transplanting vascular endothelial cells (ECs) to support metabolism and expres

142 sintegration, it sensitizes retinal vascular endothelial cells (ECs) to VEGF-A, leading to upregulati

143 Here, we determined that aging of endothelial cells (ECs), a critical component of the BM

144 Estrogens protect against apoptosis of endothelial cells (ECs), one of the hallmarks of endothe

145 mbers are expressed in retinal and choroidal endothelial cells (ECs).

146 on via silencing of small interfering RNA in endothelial cells enhanced neutrophil adhesion to endoth

147 Induced endothelial cells expressed specific endothelial cell su

148 compatibility complex class II expression on endothelial cells for at least 4 to 6 weeks after transp

149 In the retina, endothelial cells form a blood-retina barrier by virtue

150 Here, we report the derivation of arterial endothelial cells from human pluripotent stem cells that

151 Pancreatic endothelial cells from NOD-Idd22 animals expressed lower

152 his study investigates the effect of EMPs on endothelial cell function and dysfunction in a model of

153 ry (SCI), and diabetes mellitus (DM) impairs endothelial cell function and integrity of BSCS.

154 ive product, nitric oxide (NO), mediate many endothelial cell functions, including angiogenesis and v

155 In human pulmonary microvascular endothelial cells, G was 20.4 +/- 12 Pa and decreased by

156 ed primary human podocytes, renal glomerular endothelial cells (GECs), and mesangial cells with ZIKV.

157 ble transcription factor-1 (HIF-1)-dependent endothelial cell glycolysis, which is crucial for pathol

158 Thus, by delivering TNFalpha to endothelial cells, granulocytes promote blood vessel gro

159 ective concentrations; protects human aortic endothelial cells (HAEC) from cold hypoxia/reoxygenation

160 d PE STBEVs by primary human coronary artery endothelial cells (HCAEC) and the effects of free HbF on

161 NPs (50, 100, and 150 nm) in a human corneal endothelial cell (HCEC) line, B4G12.

162 d PD-L1 expression in human dermal lymphatic endothelial cells (HDLECs), which play key roles in immu

163 ry disease may affect tissue oxygenation and endothelial cell health.

164 ro studies using human retinal microvascular endothelial cells (HRMECs) showed increased RUNX1 RNA an

165 F, member 1 (SCARF-1) on hepatic sinusoidal endothelial cells (HSEC).

166 BBB models were constructed with human brain endothelial cells, human astrocytes, and human brain per

167 l stem cells (MSCs) and human umbilical vein endothelial cells (HUVECs) as previously reported.

168 ty test of the Mg extract via human vascular endothelial cells (HUVECs) indicates that the corrosion

169 We treated human umbilical vein endothelial cells (HUVECs) with E2, TNFalpha, or both an

170 dels using a HCC cell line, HepG2, and human endothelial cells, HUVECs, as well as ex vivo and in viv

171 Transcriptomic analysis in endothelial cells identified nitric oxide (NO) as major

172 nt (bMTM) comprising co-culture of tumor and endothelial cells in a 3D environment.

173 sed (18)F-FDG uptake by human umbilical vein endothelial cells in a concentration-dependent manner.

174 This first report on brain lymphatic endothelial cells in a vertebrate embryo identifies cell

175 cross-talk between BM osteolineage cells and endothelial cells in regulating hematopoietic reconstitu

176 Nitric oxide (NO) produced by endothelial cells in response to cytokines displays anti

177 ibited angiogenic sprouting and alignment of endothelial cells in response to shear stress.

178 linked to degradation of tight junctions in endothelial cells in vitro, which is blocked by pharmaco

179 was found to promote the migration of human endothelial cells in vitro.

180 t with VEGFA reduced expression of CX3CL1 in endothelial cells in vitro.

181 ved cardiomyocytes, smooth muscle cells, and endothelial cells (in a 2:1:1 ratio) to generate the hCM

182 uced ROS productions in human umbilical vein endothelial cells incubated with H2O2 for 2 hours, accom

183 ion to P-selectin on activated platelets and endothelial cells induces shedding of the P-selectin ect

184 c antibody (DSA) causes complement-dependent endothelial cell injury in kidney transplants, as assess

185 /or CS-exposed mice have pulmonary and renal endothelial cell injury linked to increased endothelial

186 uently have albuminuria (indicative of renal endothelial cell injury) associated with hypoxemia.

187 d VE-cadherin, thereby compromising pericyte-endothelial cell interactions and inter-endothelial cell

188 Endothelial cell intercellular junction formation was ch

189 Pluripotency-independent conversion of endothelial cells into autologous authentic engraftable

190 that orchestrate the fleeting transition of endothelial cells into haematopoietic stem cells remain

191 and secondary HUS are simultaneous damage to endothelial cells, intravascular hemolysis, and activati

192 table connections are spatially regulated by endothelial cell-intrinsic modulation of mFlt1, suggesti

193 rix degradability switches three-dimensional endothelial cell invasion between two distinct modes: si

194 Adhesion of cancer cells to endothelial cells is a key step in cancer metastasis; th

195 acquisition of tissue-specific properties in endothelial cells is essential for vascular function.

196 cyte-endothelial cell interactions and inter-endothelial cell junctions.

197 through the endothelium by opening holes in endothelial cells, known as transcellular tunnels, which

198 Lymphatic endothelial cells lack the Ca(2+) -activated K(+) channe

199 ndered by barriers presented by the vascular endothelial cell layer and by the aberrant nature of tum

200 ntly reduced leukocyte migration through the endothelial cell layer by 93%.

201 ing and quantifying extracellular changes in endothelial cell layer integrity following the activatio

202 , stabilizes the microtubule cytoskeleton in endothelial cells leading to endothelial lumenogenesis.

203 pes across primary mouse and human lymphatic endothelial cells (LEC), and validated the model for the

204 In vitro cultured mouse lymphatic endothelial cells (LECs) expressed VitD Receptor (VDR),

205 a1 gene in adult mice resulted in loss of LV endothelial cells (LECs) specifically from the leaflets

206 In vitro, siRNA-mediated FABP4 knockdown in endothelial cells led to a marked increase of endothelia

207 helial cell types (the human brain capillary endothelial cell line hCMEC/D3 and human umbilical vein

208 ABSTRACT: The human cerebral microvascular endothelial cell line hCMEC/D3 was used to characterize

209 Endothelial cells lining SC elaborate tight junctions (T

210 Endothelial cells lining the vessel wall control importa

211 The monolayer of endothelial cells lining the vessel wall forms a semiper

212 To determine whether endothelial cell loss 3 years after successful DSAEK is

213 Liver sinusoidal endothelial cells (LSECs) are uniquely differentiated to

214 s of Kindlin-2 in Kindlin-2(+/-) mice aortic endothelial cells (MAECs) from these mice, and human umb

215 biogenesis, Cavin-2 plays a critical role in endothelial cell maintenance and function by regulating

216 n of the cross-talk between granulocytes and endothelial cells may lead to new therapeutic approaches

217 eature of cellular adhesion, locomotion, and endothelial cell mechanobiology.

218 ilised isogenic keratinocytes, microvascular endothelial cells, melanocytes and fibroblasts isolated

219 istically, this occurs through alteration of endothelial cell metabolism.

220 In high glucose-exposed endothelial cells, metformin treatment and adenoviral ov

221 y growth by activating Cxcl12 expression and endothelial cell migration against blood flow into devel

222 pathway to promote actin polymerization and endothelial cell migration.

223 igestion and the inhibitory activity towards endothelial cell migration.

224 lial barrier dysfunction using both cultured endothelial cell monolayer and in vivo models.

225 produced from mammalian cells, infects fetal endothelial cells much more efficiently than other patho

226 respond to greater stiffness of PGPC-treated endothelial cells observed by measuring cellular elastic

227 1) as a gene upregulated in CD31(+) vascular endothelial cells obtained from human PDR fibrovascular

228 nnal glycoprotein receptors, particularly on endothelial cells of arterioles in the brain and immune

229 ported exclusively within the neovasculature endothelial cells of nonprostatic cancers; however, ther

230 e receptors and the gap junction coupling in endothelial cells of the blood-brain barrier.

231 ivate/inhibit all cell types (neurons, glia, endothelial cells, oligodendrocytes) in the stimulated a

232 erdin to the surface of human umbilical vein endothelial cells or Neisseria meningitidis after incuba

233 which prevented VWF multimer accumulation on endothelial cells, or by an anti-VWF Ab.

234 shed in Nature, Wong et al. (2016) show that endothelial cells oxidize fatty acids to produce acetyl-

235 Vulnerability of pulmonary arterial endothelial cells (PAEC) to apoptosis was increased by H

236 support the notion that, in pulmonary artery endothelial cells (PAECs), expression of transcription f

237 in culture, Dach1 overexpression stimulated endothelial cell polarization and migration against flow

238 Dach1 had small coronary arteries, decreased endothelial cell polarization, and reduced expression of

239 in decreased glycolysis, leading to impaired endothelial cell proliferation and migration.

240 ng mechanisms involved in regulating uterine endothelial cell proliferation during pregnancy.

241 ich laminins modulate vascular branching and endothelial cell proliferation during retinal angiogenes

242 17beta-oestradiol- and catecholamine-induced endothelial cell proliferation may be indicative of unap

243 econd, IL-6 trans-signaling directly induces endothelial cell proliferation to promote tumor angiogen

244 ytic enzyme expression, glycolytic flux, and endothelial cell proliferation, sprouting and tubule for

245 to influence vascular branching density and endothelial cell proliferation.

246 a type of angiomatous lesions that features endothelial cell proliferation.

247 uired for NOSTRIN-induced down-regulation of endothelial cell proteins.

248 r study shows that the actin cytoskeleton of endothelial cells provides both passive and active resis

249 ggest that itraconazole selectively inhibits endothelial cells rather than cancer cells by targeting

250 dental pulp cells, and human umbilical vein endothelial cells (recell-dTBs); 3) dTBs seeded with bon

251 Descemet's membrane supports corneal endothelial cell regeneration in rabbits after endotheli

252 patterning, as did supplementation with the endothelial cell-regulated mesenchymal factors IGFBP2 an

253 ells and CD34 expression by liver sinusoidal endothelial cells remained stable, consistent with the a

254 mediated largely by Edn1-Ednra in glomerular endothelial cells representing an early event in DKD pro

255 apeutically targeting tumor stiffness or the endothelial cell response to tumor stiffening may help r

256 In vitro (macrophages, endothelial cells, skeletal muscle cells under normal an

257 Endothelial cell-specific, but not macrophage-specific A

258 duced migration and proliferation of retinal endothelial cells stimulated with VEGF.

259 We demonstrated that these endothelial cells supply the hepatocyte growth factor (H

260 miR-204, whereas overexpression of Sirt1 in endothelial cells suppresses miR-204-induced ER stress.

261 Induced endothelial cells expressed specific endothelial cell surface markers and also exhibited the

262 fy the galectin-3-binding molecule(s) on the endothelial cell surface responsible for the galectin-3-

263 nts, expression of adhesion molecules on the endothelial cell surface, and decreased endothelial barr

264 gered tube-like morphogenesis in neighboring endothelial cells that could restore blood perfusion in

265 Intraportal delivery of endothelial cell therapy or saline was technically succe

266 In endothelial cells, this phenomenon might contribute to v

267 Collectrin is also expressed in endothelial cells throughout the vasculature, where it r

268 hat induces oxidative stress on the vascular endothelial cells, thus mediating progression of diabeti

269 Moreover, they were internalized by endothelial cells, thus supporting their involvement in

270 ecreted CLIC3 promotes invasive behaviour of endothelial cells to drive angiogenesis and increases in

271 approach for fully reprogramming adult mouse endothelial cells to haematopoietic stem cells (rEC-HSCs

272 how MFSD2A regulates lipid metabolism of gut endothelial cells to promote resolution of intestinal in

273 Addition of exogenous endothelial cells to reconstituted glands restored epith

274 nsactivator can reduce the capacity of human endothelial cells to recruit and activate alloreactive T

275 alone can activate human brain microvascular endothelial cells to stimulate adhesion molecules, CCL2,

276 al coupling-from the potential source of NO, endothelial cells, to the potential beneficiary from the

277 ts of nitro-oleic acid (OA-NO2) on the human endothelial cell transcriptome.

278 nated) by two phenotypically different human endothelial cell types (the human brain capillary endoth

279 for lymphatic TEM for various migrating and endothelial cell types possesses the capacity to be high

280 Endothelial cells typically rely on aerobic glycolysis f

281 rette smoke extract on human coronary artery endothelial cells under oscillatory, normal laminar and

282 Endocan is a proteoglycan secreted by endothelial cells under the control of inflammatory cyto

283 eir activation and firm adhesion to vascular endothelial cells (VECs), leukocytes preferentially extr

284 pathy mouse model by promoting glycolysis in endothelial cells via the ERK/Akt/HIF-1alpha pathway, th

285 deletion of prostaglandin synthesis in brain endothelial cells, we demonstrate that local prostagland

286 n of the Notch ligand delta-like 4 (DLL4) in endothelial cells, we find that activation of the MAPK/E

287 mal care and use committee, autologous liver endothelial cells were grown from core hepatic specimens

288 Instead, cardiac endothelial cells were likely to proliferate and generat

289 Arterial endothelial cells were robustly generated from multiple

290 Results Liver endothelial cells were successfully isolated, cultured,

291 heart transplantation models to confirm that endothelial cells were the only accessible targets for D

292 hese embryos exhibited aberrant alignment of endothelial cells, which disturbed the feto-maternal cir

293 dependent expression of the ET-B receptor in endothelial cells, which in turn mediates the decrease i

294 Coculturing endothelial cells with astrocytes yielded the greatest r

295 and ELISA analyses showed that treatment of endothelial cells with EVT conditioned medium significan

296 We treated human aortic endothelial cells with exogenous amphiphiles, shown prev

297 dothelial progenitor cells or spleen-derived endothelial cells with inflammatory cytokines.

298 n and leukocyte adhesion in quiescent tumour endothelial cells with intact insulin receptors and part

299 receptor tyrosine kinases found primarily in endothelial cells with key roles in development and main

300 a shift toward inhibition of proteolysis in endothelial cells, with decreased expression of extracel