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1 c signals are not well regulated and produce endothelial cell dysfunction.
2 tein CIII links hyperlipidemia with vascular endothelial cell dysfunction.
3 4, thrombomodulin, and eNOS mRNA, suggesting endothelial cell dysfunction.
4 a possible mechanism of HHcy induced retinal endothelial cell dysfunction.
5 itonavir does not appear to directly promote endothelial cell dysfunction.
6 ps because higher albumin excretion reflects endothelial cell dysfunction.
7 g inflammatory responses and contributing to endothelial cell dysfunction.
8 critical early events in fatty acid-mediated endothelial cell dysfunction.
9 ant irradiation induces a sustained vascular endothelial cell dysfunction.
10 tly attenuated APAP-induced liver sinusoidal endothelial cell dysfunction and ameliorated hepatic mic
11                                              Endothelial cell dysfunction and apoptosis are critical
12            We hypothesized that IGF-1 causes endothelial cell dysfunction and expression of neutrophi
13 le of eliciting cell contact-dependent brain endothelial cell dysfunction and increased barrier perme
14 n demonstrated to promote pulmonary arterial endothelial cell dysfunction and induce pulmonary arteri
15 ty, migration, and proliferation, as well as endothelial cell dysfunction and inflammation.
16 lmonary arterial hypertension (PAH) includes endothelial cell dysfunction and proliferation and migra
17 ed soluble VEGF receptor 1 protein (sFlt-1), endothelial cell dysfunction and proteinuria.
18  a progressive disease characterized by lung endothelial cell dysfunction and vascular remodeling.
19 aling will be useful targets for controlling endothelial cells dysfunction and consequently atheroscl
20 irculating IL-6 levels that can promote both endothelial cell dysfunction (and subsequent vascular dy
21 ent approaches to the clinical assessment of endothelial cell dysfunction; and outline some promising
22                             Nitric oxide and endothelial cell dysfunction are also implicated in the
23 zeta has emerged as a pathologic mediator of endothelial cell dysfunction, based on its essential rol
24                      The likely mechanism is endothelial cell dysfunction due to increased MEK1 activ
25 elp preservation of hepatocyte function, but endothelial cell dysfunction during the cold preservatio
26                                      Diffuse endothelial cell dysfunction (ECD) documented by several
27 and obliterative vasculopathy resulting from endothelial cell dysfunction, extensive fibrosis seconda
28 ew, we trace the evolution of the concept of endothelial cell dysfunction, focusing on recent insight
29                                              Endothelial cell dysfunction has been described in the c
30                                      Retinal endothelial cell dysfunction has been implicated in the
31  aspartate aminotransferase), and sinusoidal endothelial cell dysfunction (hyaluronic acid).
32 ion for understanding the pathophysiology of endothelial cell dysfunction in the multiple organ dysfu
33 is pressure transmission could contribute to endothelial cell dysfunction in various pathologies.
34                                              Endothelial cell dysfunction, in its broadest sense, enc
35 ion are hallmarks of early and late vascular endothelial cell dysfunction induced by diabetes.
36                                              Endothelial cell dysfunction is associated with bioavail
37                                              Endothelial cell dysfunction is pivotal to the pathophys
38                                     Vascular endothelial cell dysfunction mediated by antiphospholipi
39 d circulating interleukin-6 levels), reduced endothelial cell dysfunction (reduced endothelial activa
40                           On the other hand, endothelial cell dysfunction suggested by poor hyaluroni
41                 Dasatinib treatment mediated endothelial cell dysfunction via increased production of

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