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1 t requires protease activated receptor-1 and endothelial cell protein C receptor.
2 can inhibit neutrophil binding to selectins: Endothelial cell protein C receptor, a protein C/APC bin
3 ur findings support the hypothesis that both endothelial cell protein C receptor and thrombomodulin a
4 APC's cytoprotection requires its receptor, endothelial cell protein C receptor, and protease-activa
5 lantation were immunostained for CD31, CD34, endothelial cell protein C receptor, and thrombomodulin
6 It is evident that thrombomodulin and the endothelial cell protein C receptor are critical players
7 tudy examined whether thrombomodulin and the endothelial cell protein C receptor are down-regulated o
8 ed APC's proteolytic activity and binding to endothelial cell protein C receptor but not protease act
9 icating that APC up-regulates TF activity by endothelial cell protein C receptor-dependent shedding o
10 common genetic and acquired abnormalities of endothelial cell protein C receptor do contribute to pat
11 cell signaling that requires two receptors, endothelial cell protein C receptor (EPCR) and protease-
12 or APC's signaling emphasizes its binding to endothelial cell protein C receptor (EPCR) and subsequen
21 served that there is about 100 ng/ml soluble endothelial cell protein C receptor (EPCR) in human plas
22 Our recent studies showed that expression of endothelial cell protein C receptor (EPCR) in MPM cells
26 issue of the JCI, Kerschen et al. show that endothelial cell protein C receptor (EPCR) is specifical
27 ng of protein C/activated protein C (APC) to endothelial cell protein C receptor (EPCR) on endothelia
30 tibodies that block protein C binding to the endothelial cell protein C receptor (EPCR) reduce protei
32 ed receptor-1 (PAR1), as part of a novel APC-endothelial cell protein C receptor (EPCR) signaling pat
36 shown that factor VIIa (FVIIa) binds to the endothelial cell protein C receptor (EPCR), a cellular r
37 gulant proteins, thrombomodulin (TM) and the endothelial cell protein C receptor (EPCR), impair endot
40 investigated the influence of tissue factor, endothelial cell protein C receptor (EPCR, PROCR), and p
42 c disease suggests that the understanding of endothelial cell protein C receptor genotype and the kno
46 eads to the prediction that abnormalities in endothelial cell protein C receptor might be associated
47 way, activated protein C, interacts with the endothelial cell protein C receptor, protease-activated
48 matory signals along the activated protein C-endothelial cell protein C receptor-protease activated r
49 C pathway by converting protein C (bound to endothelial cell protein C receptor) to activated protei
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