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1 We previously identified CLEC14A as a tumour endothelial marker.
2 5+ and are thought to lose the expression of endothelial markers.
3 sing human specific hepatocyte, biliary, and endothelial markers.
4 lial dysfunction, as assessed by circulating endothelial markers.
5 of all dividing cells are immunoreactive for endothelial markers.
6 ting in formation of blood and activation of endothelial markers.
7 and retained expression of general vascular endothelial markers.
8 and graft outcomes to a greater extent than endothelial markers.
9 zed by immunoreactivity for cytokeratins and endothelial markers.
10 fferentiation and sustains the expression of endothelial markers.
11 ALK2, chondrocytes and osteoblasts expressed endothelial markers.
12 SECs have both hematopoietic and endothelial markers.
13 scular endothelial growth factor (VEGF), and endothelial markers.
14 in a two-step screen to predict novel tumour endothelial markers.
15 e subtraction was employed to predict tumour endothelial markers.
16 nesium intake is related to inflammatory and endothelial markers.
17 tric analysis and quantifying mRNA levels of endothelial markers.
18 and showed varying immunoreactivity for all endothelial markers.
19 er have focal regions that lack these common endothelial markers.
20 technology is useful for in vivo imaging of endothelial markers.
21 ally new pan-endothelial and tissue-specific endothelial markers.
27 (alternatively known as endosialin or tumour endothelial marker-1) is also a member of this family an
29 One binding partner was identified as tumor endothelial marker-4 (TEM4; ARHGEF17), a guanine nucleot
30 protein-coupled receptor (GPCR) GPR124/tumor endothelial marker 5 is highly expressed in central nerv
31 protein-coupled receptor 124 (GPR124) (tumor endothelial marker 5, TEM5), an orphan member of the adh
34 eceptors, anthrax toxin receptor (ATR)/tumor endothelial marker 8 (TEM8) and capillary morphogenesis
35 wo identified anthrax toxin receptors: tumor endothelial marker 8 (TEM8) and capillary morphogenesis
40 ins, protective antigen (PA), binds to tumor endothelial marker 8 (TEM8) or capillary morphogenesis p
41 sured in cells specifically expressing tumor endothelial marker 8 (TEM8) or capillary morphogenesis p
45 Surface expression of toxin receptors tumor endothelial marker 8 and capillary morphogenesis gene 2,
46 ular receptors: anthrax toxin receptor/tumor endothelial marker 8 and capillary morphogenesis protein
47 The soluble anthrax toxin receptor/tumor endothelial marker 8 and capillary morphogenesis protein
50 capillary morphogenesis gene 2 (CMG2), tumor endothelial marker 8, and beta1-integrin, and, with the
53 e differentiated cells co-expressed CD31, an endothelial marker, along with alpha-SMA, as seen by dou
55 ascular endothelial cells lost expression of endothelial markers and acquired expression of mesenchym
58 e change in the EYFP population with loss of endothelial markers and an increase in mononuclear marke
59 of circulation express the highest levels of endothelial markers and do not generate blood cells in v
60 -1 and podoplanin used as specific lymphatic endothelial markers and Ki-67 as a proliferation marker.
61 tures that were positive for three lymphatic endothelial markers and negative for three blood vessel
62 ead showed the stable expression of multiple endothelial markers and the capacity to form capillary n
63 the absence of RUNX1, the down-regulation of endothelial markers and the formation of round cells, a
64 identification of tissue-specific lymphatic endothelial markers and the study of congenital lymphede
66 s based on the expression of VE-cadherin and endothelial markers and with lack of CD45 and hematopoie
67 neuronal marker; vWF, VEGFA, VEGFC and IL-8, endothelial markers; and PPARgamma and FABP4, adipose ma
68 n of transductional targeting to a pulmonary endothelial marker (angiotensin-converting enzyme, ACE)
69 YFP reporter quail embryos combined with the endothelial marker antibody QH1 provides definitive evid
71 ated into ECs (miPSC-ECs), the expression of endothelial markers, arterial endothelial markers, pro-a
72 nments recruit progenitor cells that express endothelial markers, as determined by staining with isol
78 ely 30 to 50% of fibroblasts coexpressed the endothelial marker CD31 and markers of fibroblasts and m
79 report, the co-expression of HLA-DR and the endothelial marker CD31 are used to identify RMEC as a d
80 lonal lines showed reduced staining with the endothelial marker CD31 in Matrigel plug assay, indicati
81 1 was specifically colocalized with vascular endothelial marker CD31 surrounded by type IV collagen.
83 C1 expression closely paralleled that of the endothelial marker CD31, and the peak level of STC1 expr
84 stem cell marker SCA-1, also co-express the endothelial marker CD31, suggesting the presence of endo
87 lpha(1), alpha(2), beta(3), and beta(5); the endothelial marker CD31; and metalloproteinases MMP-2 an
88 wth and immunohistochemical staining for the endothelial markers CD31 and VEGFR-2 and terminal deoxyn
89 on-induced angiogenesis and a panel of known endothelial markers (CD31, VE-cadherin, BS-I lectin), we
90 negative for: hematopoietic (CD34, CD45) and endothelial markers (CD31, vWf, von Willebrand factor).
91 ith colocalization studies with TUNEL and an endothelial marker, CD31, in tumor sections of Ad-MMP-2-
93 ained prostatectomy tumor block sections for endothelial marker CD34 and assessed microvessel density
94 d the expression of HIF1alpha, VEGF, and the endothelial marker CD34 in a mouse xenograft model of br
96 l growth factor receptor 3) as well as blood endothelial markers (CD34, endomucin, platelet endotheli
98 hogenesis of PPH; the normalization of these endothelial markers concomitant with improvement in hemo
99 or inhibitor-1, protein C, antithrombin, and endothelial markers (E-selectin, intracellular adhesion
100 induced endothelium dysfunction, we measured endothelial markers, endothelium-dependent vasodilation,
101 injury models may induce lineage-independent endothelial marker expression in mesenchymal cells.
104 uently differentiate into cells that express endothelial markers, function in vitro as mature endothe
106 ith antiserum to human ecNOS, the functional endothelial marker GLUT1, and the anatomical endothelial
108 lly using podoplanin as a specific lymphatic endothelial marker in 21 globes that had been enucleated
110 th muscle actin, but negative for desmin and endothelial markers including Factor VIII, clonal design
111 , designated IEM, which expresses a range of endothelial markers, including Von Willibrand Factor VII
112 We have shown that podoplanin, a lymphatic endothelial marker, is highly expressed in oral cancer a
113 double immunostains for the novel lymphatic endothelial marker LYVE-1 and for the panvascular marker
114 munohistochemical staining for the lymphatic endothelial marker LYVE-1 was done to determine the cont
116 , more than 86% of these cells expressed the endothelial markers P1H12, CD34, and CD31 and leukocyte
117 smooth muscle (SM) alpha-actin; whereas the endothelial marker, PECAM-1, was lost from the invaded c
118 bryos based on the lack of expression of the endothelial marker platelet endothelial cell adhesion mo
119 nto immunoliposomes (Ab-MJ33/IL) targeted to endothelial marker platelet endothelial cell adhesion mo
120 a cobblestone cell morphology, expression of endothelial markers (platelet endothelial cell-adhesion
121 EM and immunohistochemical staining with the endothelial marker, platelet and endothelial cell adhesi
123 receptor (LYVE-1, a routinely used lymphatic endothelial marker), podoplanin, Flt4/VEGFR3, Sca-1, CD1
124 g/ml each), explants formed QH-1 (anti-quail endothelial marker)-positive mesenchymal cells, which in
125 -Bromo-2'-deoxyuridine incorporation and neo-endothelial markers present in the microvasculature of M
126 expression of endothelial markers, arterial endothelial markers, pro-angiogenic cytokines, and Notch
127 schaemic leukoaraiosis group had a different endothelial marker profile, with lower levels of TFPI (P
131 d to tissues undergoing inflammation, and an endothelial marker responsible for tumour homing to the
133 rived CD11b+ macrophages expressed lymphatic endothelial markers such as LYVE-1 and Prox-1 under infl
134 pulations uniformly co-expressed myeloid and endothelial markers, suggesting that peripheral blood pr
135 totrophoblasts fail to express most of these endothelial markers, suggesting that this adhesion pheno
136 pha1, Ialpha2, and IIIalpha1 are known tumor endothelial markers, suggesting that TSP1 coordinately r
140 We recently identified genes encoding tumor endothelial markers (TEMs) that displayed elevated expre
145 Based on protein expression levels of common endothelial markers using flow cytometry, 3 subpopulatio
147 r percentage of cells expressed the specific endothelial marker VE-cadherin (5.2+/-0.7%) or stem/prog
149 f mesenchymal markers and down-regulation of endothelial markers via transforming growth factor/Smad
151 , left ventricular protein expression of the endothelial markers von Willebrand factor and neuregulin
152 reproducible, and show characteristic brain endothelial markers (von Willebrand factor, glucose tran
153 ; in both places, it is co-localized with an endothelial marker, von Willebrand factor, and laminin-1
154 xpression occurred together with that of the endothelial marker, von Willebrand factor, in human and
157 A new leukocyte population expressing DC and endothelial markers was uncovered in mouse and human ova
163 pha, or IL-1beta decreased the expression of endothelial markers, whereas mesenchymal markers increas
164 the first step towards identifying selective endothelial markers, which may be useful in targeting ce
165 e G-protein-coupled receptor RDC1 as a tumor endothelial marker whose expression is distinctly induce
166 actin (a marker of arterioles) and CD34 (an endothelial marker), with separate analyses on grey and
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