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1 m endocardial endothelial cells via aberrant endothelial to mesenchymal transition.
2 , suggest that HYAL2 is important to inhibit endothelial-to-mesenchymal transition.
3 reases mitral valve area in association with endothelial-to-mesenchymal transition.
4 n-driven transcription program that leads to endothelial-to-mesenchymal transition.
5 gain of mesenchymal markers consistent with endothelial-to-mesenchymal transition.
6 genesis, enhanced vascular permeability, and endothelial-to-mesenchymal transition.
7 umor vasculature that displayed hallmarks of endothelial-to-mesenchymal transition, a process of prev
8 hogenetic proteins 7 effectively ameliorates endothelial to mesenchymal transition and experimental E
9 vely active ALK2 in endothelial cells caused endothelial-to-mesenchymal transition and acquisition of
10 vation of TGF-beta signaling causes aberrant endothelial-to-mesenchymal transition and altered extrac
11 hearts and establish a possible link between endothelial-to-mesenchymal transition and cardiac fibros
12 D3 pathways, protecting the endothelium from endothelial-to-mesenchymal transition and consequent liv
13 y inactive HDAC3 expression rescues aberrant endothelial-to-mesenchymal transition and epigenetic sil
14 olarization, M2 macrophage infiltration, and endothelial-to-mesenchymal transition and improves endot
15 Moreover, Frs2alpha mutants have defective endothelial-to-mesenchymal transition and neural crest c
17 mice causes increased HA, which may promote endothelial-to-mesenchymal transition and proliferation
19 sts is multifactorial, recent data implicate endothelial-to-mesenchymal transition as an important so
20 subset of cardiac fibroblasts by undergoing endothelial-to-mesenchymal transition, but whether cardi
22 ag1, similar to the loss of Notch1, disrupts endothelial-to-mesenchymal transition during endocardial
26 Here, we investigated the role played by endothelial-to-mesenchymal transition (EndMT) and its ke
27 ism by which M1-polarized macrophages induce endothelial-to-mesenchymal transition (EndMT) and promot
32 al cells into mesenchymal cells featuring an endothelial-to-mesenchymal transition (EndMT) phenotype.
33 ied the diagnostic value of three markers of endothelial-to-mesenchymal transition (EndMT), fascin1,
38 y2 transcription factor in radiation-induced endothelial-to-mesenchymal transition (EndoMT) and its i
39 ith COUP-TFII knockdown also readily undergo endothelial-to-mesenchymal transition (EndoMT) and subse
41 orming growth factor beta (TGF-beta) induces endothelial-to-mesenchymal transition (EndoMT) in murine
47 ooth muscle (SM)-like mesenchymal phenotype (endothelial-to-mesenchymal transition), explaining both
48 c endothelial cells were more susceptible to endothelial-to-mesenchymal transition in response to tra
49 We further demonstrate that such aberrant endothelial to mesenchymal transition involving endocard
50 ell adhesion molecule 1 (CD31) and increased endothelial-to-mesenchymal transition markers, alphaSM a
52 differentiation from bone marrow (35%), the endothelial-to-mesenchymal transition program (10%) and
55 ated that this signaling pathway promotes an endothelial-to-mesenchymal transition that supplies cell
56 olarization, M2 macrophage infiltration, and endothelial-to-mesenchymal transition were greatly suppr
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