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1 ining the vasodilator/clearance functions of endothelin-B receptors.
5 to a cysteine sulfenic acid modification in endothelin B receptor and consequently decreased endothe
7 transcripts encoding fibronectin, integrins, endothelin B receptor, angiopoietin 2, and delta-like li
12 We have earlier shown that stimulation of endothelin B receptors by IRL-1620 provides significant
13 ult of autocrine ET-1 release and subsequent endothelin B receptor-dependent NO production, and sugge
14 s in this model, suggesting that blockade of endothelin-B receptors did not yield any additional prot
16 we have cloned three previously unrecognized endothelin B receptor (EDNRB) transcripts from a human m
17 ntified in the RET receptor tyrosine kinase, endothelin-B receptor (EDNRB) and its physiological liga
18 schsprung disease, carries a deletion in the endothelin-B receptor (EDNRB) gene that abrogates expres
19 ald locus on mouse chromosome 14 encodes the endothelin-B receptor (EDNRB), a G protein-coupled, seve
23 thelial nitric oxide synthase (eNOS) via the endothelin-B receptor (ET(B)), suggesting that this sign
24 reased expression of endothelin-1 (ET-1) and endothelin-B receptor (ET-BR) from human pulmonary micro
29 levels and immunoreactivity of ET-1 and the endothelin B receptor (ETBR) were increased in preeclamp
30 data suggest that the cytosolic tail of the endothelin B receptor is involved in calcium mobilizatio
33 ing IRL-1620 demonstrated an upregulation of endothelin B receptor only in the infarcted hemisphere 7
34 ng through the c-Ret tyrosine kinase and the endothelin B receptor pathways is known to be critical f
35 -1, and determined the vasomotor role of the endothelin-B receptors that are known to be located on r
36 e specific contributions of endothelin-A and endothelin-B receptors to the changes in renal function
38 In the present study, stimulation of the endothelin B receptor was found to activate three distin
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