ライフサイエンスコーパス: endothelium-dependent

1 eading dilatation and hyperpolarization were endothelium dependent.

2 in the radial artery, suggesting that it is endothelium-dependent.

3 nduced vasodilation in mesenteric vessels is endothelium-dependent.

4 rest and during intra-arterial infusions of endothelium-dependent (acetylcholine and substance P) an

5 To test this, we measured endothelium-dependent (acetylcholine) and -independent v

6 serotonin; both P = .03); relaxation to the endothelium-dependent agonist acetylcholine was attenuat

7 Under baseline conditions, responses to the endothelium-dependent agonist acetylcholine were not aff

8 Deletion of PTP1B improved both endothelium dependent and independent NO-mediated dilati

9 tionally, obese mice demonstrate an impaired endothelium dependent and independent vasodilation to ac

10 Endothelium-dependent and -independent arterial vasodila

11 Intravital microscopy was used to assess endothelium-dependent and -independent arteriolar vasodi

12 Resveratrol elicits endothelium-dependent and -independent dilation of retin

13 Endothelium-dependent and -independent flow-mediated vas

14 ears that the dilatation is mediated by both endothelium-dependent and -independent signalling pathwa

15 and SV graft vasomotor and flow responses to endothelium-dependent and -independent stimuli 5 years a

16 e, distal segments failed to respond to both endothelium-dependent and -independent stimuli.

17 al coronary segments that fail to respond to endothelium-dependent and -independent stimuli.

18 We examined whether endothelium-dependent and -independent vascular reactivi

19 for (1) NADPH-dependent O2- production, (2) endothelium-dependent and -independent vascular relaxati

20 hy, higher white blood cell count, and lower endothelium-dependent and -independent vasodilation in t

21 disease have a peripheral vascular defect in endothelium-dependent and -independent vasodilation that

22 ronary calcification; and 4) brachial artery endothelium-dependent and -independent vasodilation.

23 ignificantly improved vascular reactivity to endothelium-dependent and -independent vasodilators as w

24 lar responses to intra-arterial infusions of endothelium-dependent and -independent vasodilators have

25 peroxynitrite (ONOO(-)) inhibitors improved endothelium-dependent and EDHF-mediated relaxations and

26 Forearm endothelium-dependent and endothelium-independent vasodi

27 gen species, endothelial cell apoptosis, and endothelium-dependent and endothelium-independent vasore

28 Endothelium-dependent and independent vasodilation was t

29 Nebivolol but not metoprolol induced endothelium-dependent and nitric oxide-dependent relaxat

30 Flow-mediated, endothelium-dependent and nitroglycerin-induced, endothe

31 to measure the flow-mediated dilation (FMD; endothelium dependent) and nitroglycerin-induced dilatio

32 y at rest, during cold pressor test (largely endothelium-dependent), and after dipyridamole administr

33 to determine brachial artery, flow-mediated, endothelium-dependent, and nitroglycerin-mediated, endot

34 The activation of PAR-1 causes endothelium-dependent arterial vasodilation and the rele

35 y during transfusion, followed by testing of endothelium-dependent blood flow with increasing doses o

36 erol supplementation significantly increased endothelium-dependent brachial artery flow-mediated dila

37 The primary outcome was change in endothelium-dependent brachial artery flow-mediated dila

38 of isolated arteries with palmitate impaired endothelium-dependent but not vascular smooth muscle fun

39 vasodilator responses to acetylcholine (ACh; endothelium dependent) but not sodium nitroprusside (SNP

40 stal segments: incremental atrial pacing for endothelium-dependent cases; and intracoronary nitroglyc

41 kidney function and hemodynamic responses to endothelium-dependent challenge were assessed in pigs af

42 en evaluated for suspected ischemia, and the endothelium-dependent component is linked with adverse o

43 d releases endothelin-1 (ET-1) and initiates endothelium-dependent constriction.

44 lial stimulation, and initiate ET-1-mediated endothelium-dependent constriction.

45 Affs from Ang II 200 rabbits had an endothelium-dependent contraction factor (EDCF) response

46 Thrombin did not cause endothelium-dependent contraction of young arteries.

47 Our findings show that endothelium-dependent control of swine coronary artery t

48 tment is effective in partial restoration of endothelium-dependent coronary flow.

49 Compared with IS, endothelium-dependent coronary vasomotion was significan

50 n the collateral dependent region, preserves endothelium-dependent coronary vessel function, and upre

51 This allows the combined assessment of endothelium-dependent (CPT) and endothelium-independent

52 osine hydroxylase-positive axon density) and endothelium-dependent dilatation (carbachol) of the MCA

53 Endothelium-dependent dilatation (ED) is abnormal in pat

54 aerobic exercise is associated with enhanced endothelium-dependent dilatation (EDD) in older humans,

55 Endothelium-dependent dilatation (EDD) is impaired with

56 Acetylcholine (ACh)-induced peak endothelium-dependent dilatation (EDD) was lower in isol

57 Endothelium-dependent dilatation (EDD), assessed by the

58 ximately 30% (P <0.05) reduction in arterial endothelium-dependent dilatation (EDD).

59 anism involved in the age-related decline in endothelium-dependent dilatation (EDD).

60 Acetylcholine caused endothelium-dependent dilatation that was decreased in o

61 Heat therapy improved endothelium-dependent dilatation, arterial stiffness, in

62 cular endothelial dysfunction (e.g. impaired endothelium-dependent dilatation, EDD).

63 protective endothelial functions, including endothelium-dependent dilatation.

64 term high-fat diet feeding had no effect on endothelium dependent dilation.

65 Ex vivo carotid artery endothelium-dependent dilation (EDD) to increasing doses

66 al function, as indicated in part by reduced endothelium-dependent dilation (EDD).

67 othesis in humans, we measured flow-mediated endothelium-dependent dilation (FMD) in a population sub

68 s bolus injections of apo A-I(M)/PC restored endothelium-dependent dilation in a dose-dependent manne

69 e inhibitor (apocynin), or anti-TNF restored endothelium-dependent dilation in Lepr(db) mice.

70 bitor (apocynin), or anti-TNF-alpha restored endothelium-dependent dilation in the ZOF rats.

71 Endothelium-dependent dilation is mediated by the releas

72 the control diet, the HM/LF diet diminished endothelium-dependent dilation to 10 micromol/L acetylch

73 Small mesenteric artery endothelium-dependent dilation to acetylcholine was impa

74 ioglitazone treatment significantly improved endothelium-dependent dilation to bradykinin (P=0.01) wi

75 Finally, endothelium-dependent dilation was lower (P < 0.01) in i

76 sion of these NAD(P)H subunits and abrogated endothelium-dependent dilation.

77 Vascular endothelial function (endothelium-dependent dilation; EDD), nitric oxide (NO)/

78 t sympathetic vasoconstriction competes with endothelium-dependent dilator activity to determine post

79 Dilatation of cerebral arterioles to the endothelium-dependent dilator, acetylcholine (10 micromo

80 ure to high glucose has a concentration- and endothelium-dependent effect on the myogenic tone of rat

81 ear-activated Kir currents and inhibition of endothelium-dependent flow-induced vasodilatation (FIV)

82 and 30 normotensive control subjects during endothelium-dependent flow-mediated dilatation induced b

83 After 4 weeks of therapy, endothelium-dependent flow-mediated vasodilation of the

84 o measure changes in segmental lumen volume (endothelium-dependent function).

85 ) venous occlusion plethysmography to assess endothelium-dependent (% Hyper) and endothelium-independ

86 re sites of dynamic Ca(2+) events leading to endothelium dependent hyperpolarization (EDH)-mediated r

87 esenteric arteries the K(Ca)2.3 component of endothelium-dependent hyperpolarization (EDH) is lost fo

88 To further study the role of endothelium-dependent hyperpolarization (EDH), ACh trial

89 ions and limits the impact of the disease on endothelium-dependent hyperpolarization (EDH)-mediated v

90 We propose that initiation of endothelium-dependent hyperpolarization is the underlyin

91 xation of vascular smooth muscle through the endothelium-dependent hyperpolarizing factor (EDHF) path

92 helium independent) and acetylcholine (ACh) (endothelium dependent) iontophoresis, flicker-light-indu

93 ascular superoxide and H2O2 production in an endothelium-dependent manner and elicited a concentratio

94 ferent physiologic or pharmacologic stimuli, endothelium-dependent (micro)vascular reactivity can be

95 In men, the HF F&V diet increased endothelium-dependent microvascular reactivity (P = 0.01

96 Endothelium-dependent microvascular relaxation response

97 Endothelium-dependent microvessel relaxation was moderat

98 d stable basal tone and the vasodilations to endothelium-dependent nitric oxide (NO)-mediated agonist

99 The authors examined whether CRP can affect endothelium-dependent nitric oxide (NO)-mediated dilatio

100 of CRP (7 microg/mL; 60 minutes) attenuated endothelium-dependent nitric oxide-mediated and prostacy

101 on the effects of aging and hypertension on endothelium-dependent nitric oxide-mediated vasodilation

102 We sought to determine whether endothelium-dependent NO regulates TG2 activity by S-nit

103 nificantly attenuated arteriolar dilation to endothelium-dependent NO-mediated agonists bradykinin an

104 CRP inhibits endothelium-dependent NO-mediated dilation in retinal ar

105 direct evidence for selective impairment of endothelium-dependent NO-mediated dilation of retinal ar

106 Endothelium-dependent NO-mediated dilation of retinal ar

107 The endothelium-dependent NO-mediated vasodilations to brady

108 By impairing endothelium-dependent NO-mediated vasoreactivity, CRP ca

109 Simvastatin elicits mainly an endothelium-dependent, NO-mediated dilation of retinal a

110 ial fluid, with emphasis on whether they are endothelium dependent or O(2) dependent.

111 CRP elicits endothelium-dependent oxidative stress and compromises n

112 his is the first study in humans to identify endothelium-dependent regulation of sympathetic vasocons

113 cellular signaling mechanism responsible for endothelium-dependent regulation of vascular smooth musc

114 ability of vascular rings to respond to the endothelium-dependent relaxant acetylcholine, both durin

115 ic tone was significantly potentiated, while endothelium-dependent relaxation (EDR) was impaired in s

116 38 prevented NADP(H) depletion and preserved endothelium-dependent relaxation and NO generation with

117 t to diabetes mellitus-induced impairment in endothelium-dependent relaxation and reendothelializatio

118 cids and GLP-1 were associated with improved endothelium-dependent relaxation compared with sham-oper

119 2-CreNox2KO mice, along with preservation of endothelium-dependent relaxation during angiotensin II s

120 significantly improved acetylcholine-induced endothelium-dependent relaxation in AMPKalpha2(-/-) mice

121 ion of l-sepiapterin normalized the impaired endothelium-dependent relaxation in aortas isolated from

122 ease in STIM1 protein expression, attenuates endothelium-dependent relaxation in diabetic coronary ar

123 stress and SERCA oxidation and improved the endothelium-dependent relaxation in isolated mouse aorta

124 lose inspection reveals a specific effect on endothelium-dependent relaxation in mesenteric resistanc

125 In conclusion, reduced endothelium-dependent relaxation in OHF mesenteric arter

126 We also observed impaired endothelium-dependent relaxation in resistant vessels fr

127 e control, exhibited striking improvement in endothelium-dependent relaxation in response to acetylch

128 Endothelium-dependent relaxation in response to acetylch

129 Endothelium-dependent relaxation in response to shear st

130 Functionally, acetylcholine-induced, endothelium-dependent relaxation is impaired in T1DM mes

131 reduced ejection fraction, mitral E/A ratio, endothelium-dependent relaxation of coronary arteries, t

132 with preserved left ventricular function and endothelium-dependent relaxation of coronary microvessel

133 emporal profile of Ca(2+) dynamics underlies endothelium-dependent relaxation of swine coronary arter

134 re no acute changes in BP or the NO-mediated endothelium-dependent relaxation of the brachial artery

135 the Ca(2+) concentration in the ER, and (3) endothelium-dependent relaxation that was attenuated in

136 Endothelium-dependent relaxation was enhanced in male CF

137 Vasodilator prostanoid contribution to endothelium-dependent relaxation was reduced in lobar ar

138 ared to the wild type, acetylcholine-induced endothelium-dependent relaxation was significantly impai

139 heterozygout mice (db/m) mice and effects on endothelium-dependent relaxation, insulin sensitivity, a

140 ha release, and improved coronary arteriolar endothelium-dependent relaxation.

141 howed increased aortic stiffness and reduced endothelium-dependent relaxation.

142 ich restored NO bioavailability and improved endothelium-dependent relaxations and HDL endothelium-pr

143 Impaired endothelium-dependent relaxations in renal arteries, car

144 mice exhibited an accelerated impairment of endothelium-dependent relaxations in response to in vitr

145 Hcy impaired endothelium-dependent relaxations of rat aortae and led

146 ived hyperpolarizing factor (EDHF)-mediated, endothelium-dependent relaxations of small mesenteric ar

147 helium-independent and prostacyclin-mediated endothelium-dependent relaxations were not changed.

148 gh glucose in vitro induced an impairment of endothelium-dependent relaxations, which was prevented b

149 ction, which is characterized by an impaired endothelium-dependent response to vasodilators and hyper

150 the alteration in neurovascular coupling and endothelium-dependent responses in somatosensory cortex

151 Endothelium-dependent responses to acetylcholine in pres

152 In this study, increasing endothelium-dependent signalling during exercise signifi

153 ver, H(2)O(2) has never been shown to be the endothelium-dependent transferrable hyperpolarization fa

154 studies suggest that aldosterone may impair endothelium-dependent vascular function through suppress

155 subjects with resistant hypertension impairs endothelium-dependent vascular reactivity as indexed by

156 peraldosteronism is associated with impaired endothelium-dependent vascular reactivity owing to incre

157 contributes, at least partially, to impaired endothelium-dependent vascular relaxation and hypertensi

158 oxLDL-induced impairment of endothelium-dependent vascular relaxation of vascular ri

159 riments from mice showed that L5 compromised endothelium-dependent vascular relaxation through a nitr

160 ild and moderate HHcy aggravated HG-impaired endothelium-dependent vascular relaxation to acetylcholi

161 xposure of WKY rat aortas to IL-17F impaired endothelium-dependent vascular relaxation, whereas IL-17

162 hat this mechanism is important in impairing endothelium-dependent vascular relaxation.

163 mental role in regulating endothelial NO and endothelium-dependent vascular tone by deacetylating eNO

164 lationship between the two in the context of endothelium-dependent vascular tone is unknown.

165 Nor-NOHA markedly increased endothelium-dependent vasodilatation (up to 2-fold) in p

166 Higher endothelium-dependent vasodilatation by ACh or leptin wa

167 Further, the altered endothelium-dependent vasodilatation caused by hypertens

168 ilure Assessment score, leukocyte count, and endothelium-dependent vasodilatation conferred an increa

169 ctional hyperaemia are reduced with age, and endothelium-dependent vasodilatation declines with age i

170 ance (HOMA-IR), was associated with impaired endothelium-dependent vasodilatation in FH- (p < 0.03, a

171 Ageing reduces endothelium-dependent vasodilatation in humans and anima

172 related increases in oxidative stress impair endothelium-dependent vasodilatation in humans, leading

173 Obesity also results in impairment of endothelium-dependent vasodilatation in response to insu

174 termine whether arginase activity diminishes endothelium-dependent vasodilatation in skeletal muscle

175 ameliorated the ageing-induced reduction in endothelium-dependent vasodilatation in soleus muscle ar

176 rginine completely inhibited the increase in endothelium-dependent vasodilatation induced by nor-NOHA

177 e results demonstrate that the impairment of endothelium-dependent vasodilatation induced by old age

178 We hypothesized that impaired endothelium-dependent vasodilatation is a predictor of m

179 In vivo bedside assessment of endothelium-dependent vasodilatation is an independent p

180 potassium (Kir) channels, but their role in endothelium-dependent vasodilatation is not clear.

181 Endothelium-dependent vasodilatation is reduced with adv

182 sine triphosphate release and stimulation of endothelium-dependent vasodilatation may explain impairm

183 Ageing reduces endothelium-dependent vasodilatation through an endothel

184 An endothelium-dependent vasodilatation value of 0.5% or le

185 Endothelium-dependent vasodilatation was calculated as t

186 Endothelium-dependent vasodilatation was impaired in cor

187 Endothelium-dependent vasodilatation was impaired in hig

188 annel function was not altered, although the endothelium-dependent vasodilatation was severely impair

189 In logistic regression analysis, endothelium-dependent vasodilatation was the only predic

190 adrenergic-induced vasoconstriction, reduced endothelium-dependent vasodilatation, and enhanced hypox

191 ostatic conditions demonstrated a decline in endothelium-dependent vasodilatation, but restored the f

192 probably due (in part) to an improvement in endothelium-dependent vasodilatation.

193 nnels, thereby causing hyperpolarization and endothelium-dependent vasodilatation.

194 reverses the ageing-associated reduction in endothelium-dependent vasodilatation.

195 airment of coronary blood flow responses and endothelium-dependent vasodilatation.

196 Endothelium-dependent vasodilatations in response to mus

197 It induces endothelium dependent vasodilation, but the precise rece

198 tolerance and brachial artery flow-mediated, endothelium-dependent vasodilation (EDV) were assessed i

199 Brachial artery flow-mediated endothelium-dependent vasodilation (FMD) and endothelium

200 The change in endothelium-dependent vasodilation achieved with fasudil

201 IRT1 in the endothelium of arteries inhibits endothelium-dependent vasodilation and decreases bioavai

202 active hyperemic index (RHI), which measures endothelium-dependent vasodilation and is a surrogate ma

203 Here we show that SIRT1 promotes endothelium-dependent vasodilation by targeting endothel

204 of the MS, black subjects exhibited reduced endothelium-dependent vasodilation compared with white s

205 MA levels and reduce nitric oxide levels and endothelium-dependent vasodilation in a murine model and

206 Fasudil increased endothelium-dependent vasodilation in CAD subjects from

207 tabolite, 5-methyltetrahydrofolate, increase endothelium-dependent vasodilation in human peripheral c

208 pressure in healthy individuals and improves endothelium-dependent vasodilation in obese and overweig

209 Indeed, both flow-mediated dilation and endothelium-dependent vasodilation in response to acetyl

210 determine whether putative modifications in endothelium-dependent vasodilation of the principal nutr

211 glutathionylation is increased with impaired endothelium-dependent vasodilation that is restored by t

212 histocompatibility complex class I inhibited endothelium-dependent vasodilation to acetylcholine.

213 de for a possible coupling mechanism linking endothelium-dependent vasodilation to bone remodeling.

214 mCMV-ND mice exhibited impaired endothelium-dependent vasodilation versus mock-ND at 9 a

215 Endothelium-dependent vasodilation was assessed as the i

216 PNA endothelium-dependent vasodilation was assessed in vitro

217 Coronary endothelium-dependent vasodilation was examined by infus

218 Endothelium-dependent vasodilation was less in smokers t

219 (estimated using the homeostasis model) with endothelium-dependent vasodilation were examined in 42 w

220 on was associated with larger improvement in endothelium-dependent vasodilation) (r=-0.48; P=0.01).

221 diabetic mice exhibited a marked decrease in endothelium-dependent vasodilation, a modest decrease in

222 s an important determinant of eNOS coupling, endothelium-dependent vasodilation, and superoxide produ

223 ysfunction, we measured endothelial markers, endothelium-dependent vasodilation, arteriolar glycocaly

224 placebo, allopurinol significantly improved endothelium-dependent vasodilation, by both forearm veno

225 LRs on endothelial cells leading to impaired endothelium-dependent vasodilation, increased vascular t

226 (*-) generation, is associated with impaired endothelium-dependent vasodilation.

227 pothesis that Ca(2+) influx via TRPA1 causes endothelium-dependent vasodilation.

228 n, including Ca(2+) signaling, integrity and endothelium-dependent vasodilation.

229 pid levels or the change in lipid levels and endothelium-dependent vasodilation.

230 rate for nitric oxide synthesis, and impairs endothelium-dependent vasodilation.

231 ange in augmentation index in response to an endothelium-dependent vasodilator (salbutamol).

232 nses to intra-arterial administration of the endothelium-dependent vasodilator acetylcholine (P=0.03)

233 Coronary blood flow in response to the endothelium-dependent vasodilator acetylcholine as well

234 The endothelium-dependent vasodilator acetylcholine elicited

235 d by neural activity, hypercapnia, or by the endothelium-dependent vasodilator acetylcholine.

236 /min), SLIGKV (160 to 800 nmol/min), and the endothelium-dependent vasodilator bradykinin (100 to 100

237 Similar to Ercc1(d/-) mice, age-related endothelium-dependent vasodilator dysfunction in Xpd(TTD

238 response to norepinephrine, and an impaired endothelium-dependent vasodilator response to acetylchol

239 Endothelium-dependent vasodilator responses to acetychol

240 t with apo A-I(M)/PC prevented impairment of endothelium-dependent vasodilator responses to acetylcho

241 on and no impairments in vasoconstrictor and endothelium-dependent vasodilator responses, associated

242 Elevation of IP(3) by the endothelium-dependent vasodilator, acetylcholine, increa

243 absolute forearm blood-flow responses to the endothelium-dependent vasodilator, acetylcholine, increa

244 cid (LPA) has been recognized recently as an endothelium-dependent vasodilator, but several lines of

245 ) were calculated during (1) infusion of the endothelium-dependent vasodilators acetylcholine (ACh) a

246 ion responses of precontracted arterioles to endothelium-dependent vasodilators adenosine 5'-diphosph

247 blood flow elicited by neural activity or by endothelium-dependent vasodilators in WT mice but not in

248 , we hypothesized that the responsiveness to endothelium-dependent vasodilators would be greater in t

249 IK(Ca)/SK(Ca)), NS309 (10(-5) M), and to the endothelium-dependent vasodilators, substance P (10(-8)

250 Endothelium-dependent vasodilators, such as acetylcholin

251 itric oxide bioavailability at rest and with endothelium-dependent vasodilators.

252 endothelial intracellular [Ca(2+)] regulate endothelium-dependent vasodilatory pathways, the molecul

253 We tested the hypothesis that stimulating endothelium-dependent vasodilatory signalling will enhan

254 1.0 [0.9-1.0], P<0.001), and displayed less endothelium-dependent vasomotion (% change segmental lum

255 vasodilation and the nitric oxide-mediated, endothelium-dependent vasomotion in response to cold pre

256 tory abnormalities of nitric oxide-mediated, endothelium-dependent vasomotion occur with increasing s

257 Per2, a circadian gene, is known to regulate endothelium-dependent vasomotion.

258 X-1 contributes to adverse effects of CRP on endothelium-dependent vasomotor function in resistance a

259 Endothelium-dependent vasomotor function, as measured by

260 identify p53 as an important determinant of endothelium-dependent vasomotor function.

261 Thus, the relationship between coronary endothelium-dependent vasomotor reactivity and atheroma

262 icate that endothelial Rac1 is essential for endothelium-dependent vasomotor response and ischemia-in

263 5-MTHF improved NO-mediated endothelium-dependent vasomotor responses and reduced va

264 However, the role of SIRT1 in regulating endothelium-dependent vasomotor tone is not known.

265 can partially restore the normal response to endothelium-dependent vasorelaxants and myocardial perfu

266 ditions, Sirt1(endo-/-) mice showed impaired endothelium-dependent vasorelaxation and angiogenesis, a

267 ary arteries, sCD40L significantly decreased endothelium-dependent vasorelaxation and eNOS mRNA level

268 temic antagonism of miR-204 rescues impaired endothelium-dependent vasorelaxation and vascular Sirt1,

269 Improvement of endothelium-dependent vasorelaxation by antibiotics is l

270 s in vitro, confirming that ceramide impairs endothelium-dependent vasorelaxation in a tissue-autonom

271 nificantly improved myocardial perfusion and endothelium-dependent vasorelaxation in chronically isch

272 her also improved vascular H(4)B content and endothelium-dependent vasorelaxation in diabetes.

273 vailable vascular nitric oxide, and improves endothelium-dependent vasorelaxation in mouse aortas.

274 o reduce systemic blood pressure and improve endothelium-dependent vasorelaxation in patients with at

275 ) and the 3-plex significantly also impaired endothelium-dependent vasorelaxation in response to brad

276 Endothelium-dependent vasorelaxation in response to brad

277 RAAS inhibition normalized MAP and improved endothelium-dependent vasorelaxation in sGCalpha1-defici

278 release, at least partly contributes to the endothelium-dependent vasorelaxation induced by the KATP

279 Collectrin knockout mice display impaired endothelium-dependent vasorelaxation that is associated

280 downregulated endothelial Cav1 and impaired endothelium-dependent vasorelaxation that was rescued by

281 endothelial dysfunction measured by blunted endothelium-dependent vasorelaxation to acetylcholine, w

282 endothelial dysfunction measured by blunted endothelium-dependent vasorelaxation to acetylcholine, w

283 Endothelium-dependent vasorelaxation was impaired in you

284 malization, blunted ROS production, restored endothelium-dependent vasorelaxation, and attenuated apo

285 arrow endothelial progenitor cells, improved endothelium-dependent vasorelaxation, and markedly delay

286 elial nitric oxide synthase (eNOS), impaired endothelium-dependent vasorelaxation, and mild hypertens

287 othelial ER stress, associated impairment of endothelium-dependent vasorelaxation, and preserves endo

288 tigates angiotensin II-induced impairment of endothelium-dependent vasorelaxation, decrease in bioava

289 dothelial dysfunction, signalled by impaired endothelium-dependent vasorelaxation, is an early marker

290 ss of type I IFN receptor signaling improved endothelium-dependent vasorelaxation, lipoprotein parame

291 APE1/ref-1+/- mice have impaired endothelium-dependent vasorelaxation, reduced vascular N

292 administration markedly attenuated impaired endothelium-dependent vasorelaxation, SERCA oxidation, E

293 tion, and rescue decline of aortic Sirt1 and endothelium-dependent vasorelaxation, triggered by high-

294 cg1(-/-) mice exhibited a marked decrease in endothelium-dependent vasorelaxation, while Abca1(-/-) m

295 R-204 expression, reduced Cav1, and impaired endothelium-dependent vasorelaxation.

296 e, insulin-mediated glucose utilization, and endothelium-dependent vasorelaxation.

297 al membrane potential and play a key role in endothelium-dependent vasorelaxation.

298 es in the regulation of vasoconstriction and endothelium-dependent vasorelaxation.

299 chanical stress, and its knock-down inhibits endothelium-dependent vasorelaxation.

300 ent increase in blood pressure, and impaired endothelium-dependent vasorelaxation.