ライフサイエンスコーパス: endothelium-dependent vasodilator

1 infusion of methacholine chloride (Mch), an endothelium-dependent vasodilator.

2 mulative administration of acetylcholine, an endothelium-dependent vasodilator.

3 ty and preserves renal vascular responses to endothelium-dependent vasodilators.

4 at rest, during exercise, and in response to endothelium-dependent vasodilators.

5 itric oxide bioavailability at rest and with endothelium-dependent vasodilators.

6 n produced by neocortical application of the endothelium-dependent vasodilator acetylcholine (10 micr

7 r changes were determined in response to the endothelium-dependent vasodilator acetylcholine (ACh, 10

8 nses to intra-arterial administration of the endothelium-dependent vasodilator acetylcholine (P=0.03)

9 n during intracoronary administration of the endothelium-dependent vasodilator acetylcholine and the

10 Coronary blood flow in response to the endothelium-dependent vasodilator acetylcholine as well

11 threshold dose of genistein potentiated the endothelium-dependent vasodilator acetylcholine but not

12 The endothelium-dependent vasodilator acetylcholine elicited

13 d by neural activity, hypercapnia, or by the endothelium-dependent vasodilator acetylcholine.

14 ndothelial cell function was assessed by the endothelium-dependent vasodilator acetylcholine.

15 cterized by vasoconstrictive response to the endothelium-dependent vasodilator acetylcholine.

16 coronary vasoconstriction in response to the endothelium-dependent vasodilator acetylcholine.

17 ) were calculated during (1) infusion of the endothelium-dependent vasodilators acetylcholine (ACh) a

18 008), Responses of resistance vessels to the endothelium-dependent vasodilators acetylcholine and ADP

19 Dose responses for an endothelium-dependent vasodilator, acetylcholine (ACh, v

20 Elevation of IP(3) by the endothelium-dependent vasodilator, acetylcholine, increa

21 absolute forearm blood-flow responses to the endothelium-dependent vasodilator, acetylcholine, increa

22 renergic receptor agonist, phenylephrine; b) endothelium-dependent vasodilator, acetylcholine; and c)

23 ents the impaired vascular relaxation to the endothelium-dependent vasodilator ACh, this agent may be

24 ion responses of precontracted arterioles to endothelium-dependent vasodilators adenosine 5'-diphosph

25 ter 2 hours of reperfusion, but those to the endothelium-dependent vasodilator ADP and the endotheliu

26 cetylcholine (7.5, 15 and 30 microg/min), an endothelium-dependent vasodilator, and sodium nitropruss

27 in) and bradykinin (BK, 2.5 micrograms/min), endothelium-dependent vasodilators, and sodium nitroprus

28 Although responses to acetylcholine, an endothelium-dependent vasodilator, are abnormal in patie

29 Responses to endothelium-dependent vasodilators, both in resistance v

30 /min), SLIGKV (160 to 800 nmol/min), and the endothelium-dependent vasodilator bradykinin (100 to 100

31 Relaxation to the endothelium-dependent vasodilator bradykinin was decreas

32 cid (LPA) has been recognized recently as an endothelium-dependent vasodilator, but several lines of

33 Similar to Ercc1(d/-) mice, age-related endothelium-dependent vasodilator dysfunction in Xpd(TTD

34 ate that blockade of ET-1 receptors improves endothelium-dependent vasodilator function in hypertensi

35 Since endothelium-dependent vasodilator function may be affect

36 n of increased ET-1 activity to the impaired endothelium-dependent vasodilator function of hypertensi

37 e and that this is associated with a reduced endothelium-dependent vasodilator function, endothelium-

38 s to acetylcholine and in vitro responses to endothelium-dependent vasodilators in angiotensin II-tre

39 sepiapterin or MH4 restores the response to endothelium-dependent vasodilators in pig coronary arter

40 ctors, indicating that the reduced effect of endothelium-dependent vasodilators in those with hyperte

41 blood flow elicited by neural activity or by endothelium-dependent vasodilators in WT mice but not in

42 Response of IP(DVP) to endothelium-dependent vasodilators may provide a measure

43 graded intrafemoral artery infusions of the endothelium-dependent vasodilator methacholine chloride

44 graded intrafemoral artery infusions of the endothelium-dependent vasodilator methacholine chloride

45 graded intrafemoral artery infusions of the endothelium-dependent vasodilator methacholine chloride

46 graded intrafemoral artery infusions of the endothelium-dependent vasodilator methacholine chloride

47 response to norepinephrine, and an impaired endothelium-dependent vasodilator response to acetylchol

48 erhomocyst(e)inemic mice restored the normal endothelium-dependent vasodilator response.

49 Endothelium-dependent vasodilator responses to acetychol

50 t with apo A-I(M)/PC prevented impairment of endothelium-dependent vasodilator responses to acetylcho

51 on and no impairments in vasoconstrictor and endothelium-dependent vasodilator responses, associated

52 amin E therapies similarly improved arterial endothelium-dependent vasodilator responsiveness consist

53 lipoprotein levels, specific improvement in endothelium-dependent vasodilator responsiveness is simi

54 ange in augmentation index in response to an endothelium-dependent vasodilator (salbutamol).

55 nd dilated in a dose-dependent manner to the endothelium-dependent vasodilators serotonin, ATP, and i

56 coronary artery constriction in response to endothelium-dependent vasodilator stimuli such as the co

57 w responses to intrabrachial infusion of the endothelium-dependent vasodilators substance P and acety

58 IK(Ca)/SK(Ca)), NS309 (10(-5) M), and to the endothelium-dependent vasodilators, substance P (10(-8)

59 Endothelium-dependent vasodilators, such as acetylcholin

60 roxide production and the response to either endothelium-dependent vasodilator used.

61 systemic vascular reactivity in response to endothelium-dependent vasodilators, which may be mediate

62 , we hypothesized that the responsiveness to endothelium-dependent vasodilators would be greater in t