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1 tes that pHi may modulate the release of the endothelium-derived relaxing factor.
2 act on the endothelium to cause synthesis of endothelium-derived relaxing factor.
3 have also been shown to influence release of endothelium-derived relaxing factor.
4 ienoic acids as new members of the family of endothelium-derived relaxing factors.
7 he production and the degradation of NO, the endothelium-derived relaxing factor and a primary mediat
8 nitric oxide (NO) is considered an important endothelium-derived relaxing factor and may function to
10 e (Hcy) decreases the biological activity of endothelium-derived relaxing factor and that this decrea
13 taglandin I2, staurosporine, wortmannin, the endothelium-derived relaxing factor congener S-nitroso-N
16 by signals arising in the endothelium (e.g., endothelium-derived relaxing factor, endothelium-derived
17 e (NO), the biologically active component of endothelium-derived relaxing factor, has critical roles
19 this study was to discover and identify new endothelium-derived relaxing factors involved in the reg
21 an impaired nitric oxide synthase-dependent endothelium-derived relaxing factor response and paradox
23 wing the discovery that NO is the prototypic endothelium-derived relaxing factor, this signaling mole
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