ライフサイエンスコーパス: endurance

1 this inhibition is important for contractile endurance.

2 ed as a second redox strategy for increasing endurance.

3 maintained after folding tests, showing high endurance.

4 et the commercial demands of scalability and endurance.

5 bilitation to build strength, dexterity, and endurance.

6 these mice have marked improvement in motor endurance.

7 tochondrial biogenesis, and improved running endurance.

8 excellent chemical, thermal, and mechanical endurance.

9 thickness remained relatively stable during ENDURANCE.

10 h test and a test of lower-back trunk muscle endurance.

11 lism, C3KO mice demonstrate reduced exercise endurance.

12 biphasic stimulation cycles, confirming high endurance.

13 cardiac contractility and prolongs exercise endurance.

14 isms to regulate transcription and boost LTP endurance.

15 duced rate of force development and muscular endurance 1 yr after ICU discharge.

16 .0 Newton-meters [Nm]/kg, 60 degrees /s) and endurance (-11.4 +/- 4.6 Nm/kg, 300 degrees /s), exercis

17 istor with fast switching speed, record high endurance (120 billion cycles) and reliable retention.

18 577X, have been consistently associated with endurance (ACE I/I) and power-related (ACTN3 R/R) perfor

19 on loss after 10(5) s, and no degradation in endurance after 10(4) switching cycles, with stable oper

20 stic effects of graphene are revealed by its endurance after CNT growth and low contact resistances b

21 cking lysoNa(ATP) have much reduced exercise endurance after fasting.

22 nt therapies because of limited strength and endurance after stroke.

23 and peak; (b) prolonged (60 min) submaximal endurance and (c) high-intensity intervals.

24 BCL2 AAA mice show decreased endurance and altered glucose metabolism during acute ex

25 older people are participating in organized endurance and competitive sporting events, the incidence

26 The low endurance and compound muscle action potential amplitude

27 lications that require low variability, high endurance and fast speed.

28 oimaging variate represented higher physical endurance and fluid intelligence as well as better funct

29 LPE results in greater improvements in cycle endurance and health-related quality of life in patients

30 PE) training used in athletes to traditional endurance and progressive resistance (EPR) training in p

31 . while 'mixed' MPS increases similarly with endurance and RE, increases in myofibrillar MPS are spec

32 Endurance and resistance exercise training induces speci

33 ain an incompatibility in adaptation between endurance and resistance exercise, it now seems likely t

34 lin resistance in mice but markedly enhances endurance and resistance to muscle fatigue, despite redu

35 uin activator in red wine, improves exercise endurance and skeletal-muscle oxidative metabolism in an

36 ical activity, and patient-perceived walking endurance and speed in PAD participants with and without

37 ion of polyQ AR SUMOylation rescued exercise endurance and type I muscle fiber atrophy; it also prolo

38 dexterity, sensation, gait, balance, power, endurance), and a psychometrically robust 11-item scale

39 des the context for the organism's survival, endurance, and adaptation.

40 e switching with high retention time, cyclic endurance, and low set/reset voltages.

41 romuscular junction and for muscle strength, endurance, and motor coordination in mice in vivo.

42 adult life span (18-84 years of age) and if endurance- and/or strength-oriented exercise training co

43 vices), fast switching (</=30 ns), excellent endurance ( approximately 10(12) cycles), stability (>10

44 The endurance aquatic disciplines have a higher prevalence o

45 Asthma/AHR in the endurance aquatic disciplines is common at the elite lev

46 Muscular strength and endurance are other important elements of functional cap

47 ural programs controlling muscle fitness and endurance are unknown.

48 demonstrate attenuated muscular strength and endurance as a consequence of their disease process.

49 x-knockout mice exhibited lower muscle force/endurance as well as increased muscle damage when compar

50 healthy white competitive elite male master endurance athletes (age range, 30-60 years) with a train

51 fter a strenuous exercise session.Eight male endurance athletes (mean +/- SEM age: 29 +/- 2 y; peak o

52 Symptoms developed in endurance athletes (N = 56) at a younger age (30.1 +/- 1

53 mean follow-up of 8.4 +/- 6.7 years were all endurance athletes (p = 0.002).

54 locomotor-ventilatory interactions for elite endurance athletes and individuals who are overweight or

55 their surreptitious use as doping agents by endurance athletes and poorly understood efficacy in Duc

56 Airway dysfunction is prevalent in elite endurance athletes and when left untreated may impact up

57 king cardiac morphological manifestations of endurance athletes are a result of exercise training or

58 the 880 subjects was significantly larger in endurance athletes compared with the strength athletes a

59 to an LCHF diet impairs performance in elite endurance athletes despite a significant improvement in

60 y explain the increased longevity typical of endurance athletes despite the presence of more coronary

61 Endurance athletes exhibit sinus bradycardia, that is a

62 Subsequent experiments on elite endurance athletes performing the same HIIT exercise sho

63 ing as a substrate for fast VT in high-level endurance athletes that can be successfully treated by a

64 lete's artery' as previously shown for elite endurance athletes to elite power athletes, and presents

65 Elite endurance athletes typically have larger arteries contri

66 Most lifelong masters endurance athletes with a low atherosclerotic risk profi

67 cture and function in long-term elite master endurance athletes with special focus on the right ventr

68 age, 28+/-6 years; 38% women; 395 top-level endurance athletes, 255 strength athletes, and 230 nonat

69 In a small sample of competitive endurance athletes, exercise-induced cardiac remodeling

70 F diet negated performance benefits in elite endurance athletes, in part due to reduced exercise econ

71 cause RyR1 fragmentation in muscles of elite endurance athletes, which may explain why HIIT is less e

72 ommon chronic diseases, but less so in elite endurance athletes.

73 is and echocardiography were performed in 25 endurance athletes.

74 to that observed cross-sectionally in elite endurance athletes.

75 ed levels similar to those observed in elite endurance athletes.

76 l/energy bars, and high-energy foods used by endurance athletes.

77 on metabolism and performance of world-class endurance athletes.

78 subjects similar to those observed in elite endurance athletes; however, it is not sufficient to ach

79 antly develop resistance to fatigue (through endurance-based exercise) and increased muscle mass (thr

80 resulted in a significant decrease of muscle endurance both in vivo and in vitro, which could largely

81 Orai1 enhances muscle endurance by promoting fatigue-resistant type I fiber co

82 data indicate that musclin enhances physical endurance by promoting mitochondrial biogenesis.

83 Overall, the available data suggest exercise endurance can be augmented by redox-active supplements,

84 The development of endurance capabilities in Homo appears to parallel the e

85 bic fitness, as a physiological indicator of endurance capacity associated with physical activity, is

86 re, there were no significant differences in endurance capacity between the trained and DT-14-(-)-Epi

87 th protection against age-induced decline of endurance capacity compared with wild-type mice.

88 ce generation, HET mice have an intermediate endurance capacity compared with WT and KO.

89 Endurance capacity during exercise exhaustion test was i

90 group showed improvements in both speed and endurance capacity in acute treadmill running tests (P <

91 s associated with reduced power and enhanced endurance capacity in elite athletes and nonathletes due

92 ith preserved whole body muscle strength and endurance capacity in the absence of reduced HF.

93 or mitochondrial energy production, and thus endurance capacity, is set by the mitochondria volume.

94 or mitochondrial energy production, and thus endurance capacity, is set by the mitochondria volume.

95 orrelates with improvements in parameters of endurance capacity.

96 l muscle, along with impaired improvement of endurance capacity.

97 derwent: 6-minute walk test (cardiopulmonary endurance), chair stands in 30 seconds (muscle endurance

98 rexpressing MusaSAP1 displayed better stress endurance characteristics as compared to controls in bot

99 nomy, but were likely still limited in their endurance compared to modern humans.

100 3%; group x time interaction, P < 0.05) and endurance (CON compared with LEU: -14% +/- 3% and -2% +/

101 chanical tests, including maximum, fast, and endurance contractions, was administered during isometri

102 ls that show low muscle PDH activity, severe endurance defects, and chronic lactic acidemia, recapitu

103 t progress has led to fast, low-energy, high-endurance devices that can be scaled down to less than 1

104 osomal function and are crucial for physical endurance during food restriction.

105 The appearance of skeletal traits related to endurance (e.g., larger limb joints, spring-like plantar

106 economy (e.g., limb length and posture) and endurance (e.g., muscle volume and fiber type) and inves

107 Extended breath-hold endurance enables the exploitation of the aquatic niche

108 d to increase women's participation in elite endurance events are also discussed as is the role of in

109 ccurred as bans on participation by women in endurance events were lifted.

110 global warming will affect winning times in endurance events, and counterbalance improvements in rac

111 Endurance exercise and frequent exercise increase the ri

112 ondrial content and function are enhanced by endurance exercise and impaired in states or diseases wh

113 that both the swimming pool environment and endurance exercise are etiologic factors in the developm

114 raining studies provide robust evidence that endurance exercise can attenuate muscle hypertrophy and

115 nctional features and increased strength and endurance exercise capacity.

116 ls, exaggerated muscle fatigue, and impaired endurance exercise capacity.

117 Athletes had participated in endurance exercise for an average of 31+/-12.6 years.

118 ed regulator, muscle 1) as a gene induced by endurance exercise in skeletal muscle, and regulating mi

119 Chronic endurance exercise increased AF susceptibility in rats,

120 whether the cumulative effects of intensive endurance exercise induce chronic cardiac damage, mainly

121 However, a high level of endurance exercise is associated with an increased AF pr

122 Endurance exercise is effective to attenuate muscle atro

123 cern that athletes who perform extraordinary endurance exercise may injure the right ventricle.

124 Improved endurance exercise performance in adult humans after spr

125 Collectively, these data suggest that endurance exercise stimulates MyoPS and MitoPS in skelet

126 sceptible to oxidative stress following high endurance exercise stress (EES), but demonstrated increa

127 ction by muscle, limiting performance during endurance exercise tasks.

128 hanisms for improved exercise capacity after endurance exercise training (ET) in elderly patients wit

129 The study sought to evaluate the effects of endurance exercise training (ET) on endothelial-dependen

130 ht loss from calorie restriction (CR) and/or endurance exercise training (EX) is cardioprotective.

131 In contrast, endurance exercise training (initiated 2 weeks after AAV

132 diabetes mellitus, thyroid dysfunction, and endurance exercise training all cause structural remodel

133 LV) structural and functional adaptations to endurance exercise training among competitive male rower

134 l to atrial arrhythmogenesis associated with endurance exercise training and occlusive coronary arter

135 Endurance exercise training can increase the ability to

136 tion would affect the response to 3 weeks of endurance exercise training in CACs, muscle mitochondria

137 Six weeks of endurance exercise training increased the transcriptiona

138 We show that endurance exercise training increases multiple CAC types

139 In conclusion, endurance exercise training induced increases in multipl

140 Endurance exercise training induces substantial adaptive

141 blot data of other laboratories suggest that endurance exercise training leads to similar changes in

142 tion would affect the response to 3 weeks of endurance exercise training on CD3(+) , CD3(+) /CD31(+)

143 Endurance exercise training partially reversed these abn

144 Long-term endurance exercise training results in a reduction in th

145 defined physiological system, regardless of endurance exercise training status.

146 These findings reveal that endurance exercise training-induced increases in basal a

147 at increased basal autophagy is required for endurance exercise training-induced skeletal muscle adap

148 is not required for metabolic adaptation to endurance exercise training.

149 ical) form of cardiac hypertrophy because of endurance exercise training.

150 drial biogenesis in sedentary mice and after endurance exercise would be impaired in mKO mice.

151 During endurance exercise, Acsl1(M-/-) mice ran only 48% as far

152 lar metabolic perturbation during whole body endurance exercise, eight subjects performed 5 km cyclin

153 associated with alterations in capacity for endurance exercise, rates of muscle atrophy, and cardiac

154 roup III/IV muscle afferents in limiting the endurance exercise-induced metabolic perturbation assaye

155 dult skeletal muscle, nor is it required for endurance exercise-mediated mitochondrial adaptations.

156 (Body VO2 max , indirect calorimetry) in 10 endurance exercise-trained and 10 untrained young males.

157 e adaptations that also occur in response to endurance exercise.

158 muscle and brain in times of stress, such as endurance exercise.

159 hanges in the expression of genes related to endurance exercise.

160 ondrial (MitoPS) protein synthesis following endurance exercise.

161 t, for unknown reasons, is linked to intense endurance exercise.

162 , PPARalpha, results in reduced capacity for endurance exercise.

163 oxylic acid cycle, which is generated during endurance exercise.

164 to eliminate participation in competitive or endurance exercise.

165 om wild type (WT) and C3KO mice subjected to endurance exercise.

166 Blood glucose is an important fuel for endurance exercise.

167 DME trial were maintained through M12 of the ENDURANCE extension study with a reduced treatment frequ

168 ts completing VISTA DME elected to enter the ENDURANCE extension study.

169 and stable with individualized dosing during ENDURANCE, fluctuating by <1.5 mean letters from the bas

170 eet the demands of hemiparetic gait improves endurance for activities of daily living while promoting

171 yields a large nonlinearity (>10(4) ), high endurance (>10(8) ), low variability, and low temperatur

172 represent low power consumption, high cycle endurance, high thermal stability and suitable retention

173 NIOV+O(2) yielded substantial exercise endurance improvements accompanied by respiratory muscle

174 observed trade-off between maximum speed and endurance in athletes and some animals that has been exp

175 and in vivo exercise assays revealed reduced endurance in constitutive muscle-specific Orai-KO mice.

176 gher aerobic fitness and better trunk muscle endurance in models including adjustment for age, sex, b

177 important for the maintenance of contractile endurance in rat muscle it is unknown whether a similar

178 Reduced skeletal muscle contractile endurance in smokers may result from impaired oxygen del

179 and propulsion would have reduced locomotor endurance in the earliest hominins and likely restricted

180 s aerobic capacity and muscular strength and endurance in those with heart failure.

181 adaptation responsible for this increase in endurance is an increase in muscle mitochondria.

182 durance), chair stands in 30 seconds (muscle endurance), isometric knee extension (lower extremity st

183 ementation tensile surface or at the fatigue endurance limit (1 million cycles).

184 egradation, with nearly 40% reduction in the endurance limit and even more substantial decrease in th

185 s, chronic right ventricular damage in elite endurance master athletes with lifelong high training vo

186 ore importantly, interventions that increase endurance may benefit individuals whose physical activit

187 ion of longer than 10(6) seconds and cycling endurance of 300 cycles.

188 The endurance of charge collection by rubbing the CGM tip on

189 This is consistent with exceptional endurance of naked mole rat tissues to various genotoxic

190 ctively, we observed a dramatically improved endurance of the TaO(x) memristor.

191 Pb/Pt nanoplates appear to underlie the high endurance of these catalysts, which can undergo 50,000 v

192 Thus, the fact that the endurance of this improved performance was significantly

193 The robustness and endurance of this new coating were investigated by direc

194 at result after undertaking either prolonged endurance or resistance training.

195 Twelve weeks of endurance- or strength-oriented exercise training increa

196 tion, histopathology, swimming behaviour and endurance, parasite infestation, and reproduction.

197 deas about the physiological determinants of endurance performance but also touches on some historica

198 eries resistor (R(S)) on the variability and endurance performance of memristor was studied in the Ta

199 iew summarizes factors associated with elite endurance performance, trends in distance running traini

200 artificial and illicit enhancement of their endurance performance.

201 and end-exercise muscle fatigue and reduced endurance performance.

202 rcise program significantly improved walking endurance, physical activity, and patient-perceived walk

203 PPARbeta/delta was shown to activate muscle endurance programs in transgenic mice.

204 area, excellent scalability, retention, and endurance properties.

205 ictus) are described as highly collaborative endurance pursuit hunters based on observations derived

206 Two important causes of sudden death during endurance races are arrhythmic death and heat stroke.

207 percentage of life-threatening events during endurance races are due to heat stroke or cardiac causes

208 on or prolonged changes in the expression of endurance-related genes.

209 Subjects ran at self-selected endurance running and sprinting speeds.

210 igins of humans' economical walking gait and endurance running capabilities remain unclear.

211 Whole-body VO2, fatty acid oxidation, and endurance running capacity were markedly increased in HS

212 Endurance running may have a long evolutionary history i

213 f subjects instead used a rearfoot strike at endurance running speeds.

214 IL-37 resulted in a further 326% increase in endurance running time compared with the performance lev

215 mental shuttle walking test, 45-85 s for the endurance shuttle walking test, and 46-105 s for constan

216 In our cohort of athletes participating in endurance sports, for every serious cardiac adverse even

217 inct from liver dysfunction and incorporates endurance, strength, and balance.

218 ng PGC-1alpha and SIRT1, to improve physical endurance, strongly suggesting myricetin as a mitochondr

219 ) in Homo was somewhat mosaic, with the full endurance suite apparent only approximately 1 million ye

220 e efficiency, and enhance performance during endurance tasks.

221 scle function as evidenced from the physical endurance test of mutant mice on a treadmill.

222 ion to the resistance ratio over a 100-cycle endurance test.

223 test, and 46-105 s for constant-load cycling endurance tests), but there is currently no validated MC

224 speed, lower operating voltage, and superior endurance than other existing flexible counterparts.

225 um voluntary contraction/sec, p < 0.01), and endurance time (40%, 136 +/- 84 sec vs. 226 +/- 111 sec,

226 The primary outcomes were cycling endurance time and health-related quality of life using

227 Endurance time at 60% Vo2max was 574 (178) seconds for p

228 ignificantly greater improvements in cycling endurance time compared with EPR.

229 When compared with placebo, endurance time was significantly prolonged after spinal

230 The model accurately estimated endurance times for sustained isometric contractions acr

231 g and old men had similar maximal forces and endurance times for the fatiguing tasks.

232 cell survival that contributes to tumor cell endurance to stress favoring prometastatic activity of l

233 nd TBC1D1(-/-) mice showed impaired exercise endurance together with impaired exercise-mediated 2-deo

234 However, endurance trained athletes and type 2 diabetes mellitus

235 endurance trained/rested, untrained/EB, and endurance trained/EB).

236 exercise condition (i.e., untrained/rested, endurance trained/rested, untrained/EB, and endurance tr

237 Nine endurance-trained (age = 23.4 +/- 0.9 years; BMI = 21.2

238 ) hampers insulin sensitivity, albeit not in endurance-trained athletes (Trained).

239 ochondrial inner membranes in leg muscles of endurance-trained athletes have an increased ratio of su

240 ty), whereas comparable IMTG accumulation in endurance-trained athletes is associated with insulin se

241 Endurance-trained athletes, characterized by a high oxid

242 long-TE 1H-MRS to measure acetylcarnitine in endurance-trained athletes, lean and obese sedentary sub

243 Nine endurance-trained cyclists completed three constant-load

244 bility of orange juice (OJ) (poly)phenols in endurance-trained males before and after cessation of tr

245 after cessation of training for 7 d.Ten fit, endurance-trained males, with a mean +/- SD maximal oxyg

246 raction did not affect the increased mVO2 in endurance-trained muscle, the O2 release rate from Mb in

247 ntributing factors to faster VO2 kinetics in endurance-trained muscle.

248 her the O2 release rate from Mb increases in endurance-trained muscles remain unclear.

249 At the histological level, RV samples from endurance-trained R735X-infected mice displayed connexin

250 ole-body energy expenditure in sedentary and endurance-trained rats fed ad libitum either low fat or

251 and raised whole-body energy expenditure in endurance-trained rats.

252 ty): seven participants performed 2 weeks of endurance training (cycling) and were tested pre-post in

253 The novel methods show that endurance training (ET) and high intensity interval trai

254 iglyceride (IMTG) utilization is enhanced by endurance training (ET) and is linked to improved insuli

255 in peak oxygen uptake (V(O2peak)) following endurance training (ET) are primarily determined by cent

256 proposed as a time efficient alternative to endurance training (ET) for increasing skeletal muscle o

257 The latter 3 groups performed 5 wk of endurance training 5 x/wk.

258 plementation neither enhances nor attenuates endurance training adaptations in young healthy men.

259 We also report that endurance training and a well-balanced diet activate the

260 These results show that low intensity endurance training and well-balanced diet activate the N

261 he first 6 to 9 months after commencement of endurance training depending on the duration and intensi

262 flavanone bioavailability.A 7-d cessation of endurance training enhanced, rather than reduced, the bi

263 All group B patients underwent intensive endurance training for a median of 15 h/week (interquart

264 High-level endurance training has been associated with right ventri

265 type 2 diabetes and are decreased following endurance training in healthy young men and in rats.

266 physiological cardiac remodelling induced by endurance training in mice.

267 We hypothesized that prolonged and intensive endurance training in previously sedentary healthy young

268 e-type specific in the posterior muscles and endurance training increased its content in type I muscl

269 Endurance training increased the basal mRNA level of hex

270 One year of prolonged and intensive endurance training leads to cardiac morphological adapta

271 Endurance training neither affected skeletal muscle FNDC

272 ated in both LRT and HRT rats that underwent endurance training on a treadmill compared with those th

273 y was, therefore, to determine the effect of endurance training on O2 saturation of Mb (SmbO2) and Pm

274 rs) were randomized to 4 weeks of supervised endurance training or to a control group.

275 Although endurance training preferentially induces PGC-1alpha1 ex

276 sessed the age-dependent effects of a 4-week endurance training program on the catabolic-anabolic bal

277 evaluated in mice after completing a 6-week endurance training program.

278 6J mice were compared with or without a 5 wk endurance training protocol at rest or after an acute ex

279 tissue (AT) lipolysis becomes elevated upon endurance training to cope with enhanced energy demands.

280 f a combination of a human PKP2 mutation and endurance training to trigger an ARVC-like phenotype.

281 Age, sex, body surface area, and high-level endurance training were determinants of RA area.

282 In contrast, the right ventricle responds to endurance training with eccentric remodeling at all leve

283 instead, exhibits plasticity with long-term endurance training.

284 cise and facilitates molecular adaptation to endurance training.

285 tal muscle and enhanced adaptive response to endurance training.

286 gnificantly with age, sex, body surface, and endurance training.

287 events the adaptive intrinsic bradycardia of endurance training.

288 A similar trend was observed for oxidative endurance, tumorigenic potential, cellular proliferation

289 muscle many of the best known adaptations to endurance-type exercise but has no effects on muscle str

290 have been shown to delay fatigue or increase endurance under a variety of experimental conditions.

291 sterol trafficking and degradation, physical endurance under fasting conditions, autophagy regulation

292 ch exhibited long shelf and high-temperature endurance (up to 1 week at 100 degrees C).

293 polymeric matrix and its effect on probiotic endurance upon spray drying.

294 aerobic fitness, muscle strength, and muscle endurance, using data on 31-year-old participants of the

295 as an anti-fatigue protein to enhance muscle endurance via the CCR5 pathway.

296 Exercise endurance was 17.6 +/- 5.7 minutes using NIOV+O(2), grea

297 ncreased muscle glycogen and enhanced muscle endurance, whereas ORM1 deficiency resulted in a signifi

298 ch as low operation speed, and limited cycle endurance, which prevents it from becoming the "universa

299 These devices should offer good mechanical endurance with high power-conversion efficiency for viab

300 s, slow muscle fatigue, and increase running endurance without negatively impacting cardiac function.