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1 or low frequency (LFS; 3 h at 10 Hz to mimic endurance training).
2  instead, exhibits plasticity with long-term endurance training.
3 tal muscle and enhanced adaptive response to endurance training.
4 gnificantly with age, sex, body surface, and endurance training.
5 cise and facilitates molecular adaptation to endurance training.
6 strated physiology characteristic of extreme endurance training.
7 s sex, race, and age in response to 20 wk of endurance training.
8 events the adaptive intrinsic bradycardia of endurance training.
9        The latter 3 groups performed 5 wk of endurance training 5 x/wk.
10 plementation neither enhances nor attenuates endurance training adaptations in young healthy men.
11 pha signaling pathway which may explain some endurance training adaptations.
12                          We also report that endurance training and a well-balanced diet activate the
13        These results show that low intensity endurance training and well-balanced diet activate the N
14 training and better performance after forced endurance training compared smtnl1(+/+) mice.
15 ty): seven participants performed 2 weeks of endurance training (cycling) and were tested pre-post in
16 he first 6 to 9 months after commencement of endurance training depending on the duration and intensi
17 rint interval training (SIT) and traditional endurance training elicit similar physiological adaptati
18 flavanone bioavailability.A 7-d cessation of endurance training enhanced, rather than reduced, the bi
19                  The novel methods show that endurance training (ET) and high intensity interval trai
20 iglyceride (IMTG) utilization is enhanced by endurance training (ET) and is linked to improved insuli
21  in peak oxygen uptake (V(O2peak)) following endurance training (ET) are primarily determined by cent
22  proposed as a time efficient alternative to endurance training (ET) for increasing skeletal muscle o
23  oxidation during exercise and the effect of endurance training, exercise intensity, and lipid supple
24 years; 6 male, 3 female) completed 1 year of endurance training followed by repeat measurements.
25     All group B patients underwent intensive endurance training for a median of 15 h/week (interquart
26                                   High-level endurance training has been associated with right ventri
27                        Vigorous exertion and endurance training have been reported to increase atrial
28 solute intensity, persons who have undergone endurance training have greater fat oxidation during exe
29  type 2 diabetes and are decreased following endurance training in healthy young men and in rats.
30 physiological cardiac remodelling induced by endurance training in mice.
31 We hypothesized that prolonged and intensive endurance training in previously sedentary healthy young
32 inal study to assess the effects of one-year endurance training in the elderly.
33 e-type specific in the posterior muscles and endurance training increased its content in type I muscl
34                                              Endurance training increased the basal mRNA level of hex
35 idative and performance ability derived from endurance training-induced increases in muscle mitochond
36                                              Endurance training induces a partial fast-to-slow muscle
37          One year of prolonged and intensive endurance training leads to cardiac morphological adapta
38                                              Endurance training neither affected skeletal muscle FNDC
39 hanges are consistent with those elicited by endurance training of the limb muscles in normal subject
40 se, similar in extent to that achieved after endurance training of wild-type littermates.
41 ated in both LRT and HRT rats that underwent endurance training on a treadmill compared with those th
42 y was, therefore, to determine the effect of endurance training on O2 saturation of Mb (SmbO2) and Pm
43 rs) were randomized to 4 weeks of supervised endurance training or to a control group.
44                                     Although endurance training preferentially induces PGC-1alpha1 ex
45 cardiovascular stiffening, whereas life-long endurance training preserves left ventricular (LV) compl
46                         Prolonged, sustained endurance training preserves ventricular compliance with
47 sessed the age-dependent effects of a 4-week endurance training program on the catabolic-anabolic bal
48  evaluated in mice after completing a 6-week endurance training program.
49 ring over 30 years was reversed by a 6-month endurance training program.
50 ise were assessed before and after a 6-month endurance training program.
51  women) aged 17-63 y participated in a 20-wk endurance training program.
52 6J mice were compared with or without a 5 wk endurance training protocol at rest or after an acute ex
53 consisted primarily of four 30- to 40-minute endurance training sessions (low-impact aerobics; brisk
54  tissue (AT) lipolysis becomes elevated upon endurance training to cope with enhanced energy demands.
55 f a combination of a human PKP2 mutation and endurance training to trigger an ARVC-like phenotype.
56  Age, sex, body surface area, and high-level endurance training were determinants of RA area.
57  biochemical responses to acute exercise and endurance training were investigated in female Fischer 3
58 In contrast, the right ventricle responds to endurance training with eccentric remodeling at all leve

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