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1 or low frequency (LFS; 3 h at 10 Hz to mimic endurance training).
2 instead, exhibits plasticity with long-term endurance training.
3 tal muscle and enhanced adaptive response to endurance training.
4 gnificantly with age, sex, body surface, and endurance training.
5 cise and facilitates molecular adaptation to endurance training.
6 strated physiology characteristic of extreme endurance training.
7 s sex, race, and age in response to 20 wk of endurance training.
8 events the adaptive intrinsic bradycardia of endurance training.
10 plementation neither enhances nor attenuates endurance training adaptations in young healthy men.
15 ty): seven participants performed 2 weeks of endurance training (cycling) and were tested pre-post in
16 he first 6 to 9 months after commencement of endurance training depending on the duration and intensi
17 rint interval training (SIT) and traditional endurance training elicit similar physiological adaptati
18 flavanone bioavailability.A 7-d cessation of endurance training enhanced, rather than reduced, the bi
20 iglyceride (IMTG) utilization is enhanced by endurance training (ET) and is linked to improved insuli
21 in peak oxygen uptake (V(O2peak)) following endurance training (ET) are primarily determined by cent
22 proposed as a time efficient alternative to endurance training (ET) for increasing skeletal muscle o
23 oxidation during exercise and the effect of endurance training, exercise intensity, and lipid supple
25 All group B patients underwent intensive endurance training for a median of 15 h/week (interquart
28 solute intensity, persons who have undergone endurance training have greater fat oxidation during exe
31 We hypothesized that prolonged and intensive endurance training in previously sedentary healthy young
33 e-type specific in the posterior muscles and endurance training increased its content in type I muscl
35 idative and performance ability derived from endurance training-induced increases in muscle mitochond
39 hanges are consistent with those elicited by endurance training of the limb muscles in normal subject
41 ated in both LRT and HRT rats that underwent endurance training on a treadmill compared with those th
42 y was, therefore, to determine the effect of endurance training on O2 saturation of Mb (SmbO2) and Pm
45 cardiovascular stiffening, whereas life-long endurance training preserves left ventricular (LV) compl
47 sessed the age-dependent effects of a 4-week endurance training program on the catabolic-anabolic bal
52 6J mice were compared with or without a 5 wk endurance training protocol at rest or after an acute ex
53 consisted primarily of four 30- to 40-minute endurance training sessions (low-impact aerobics; brisk
54 tissue (AT) lipolysis becomes elevated upon endurance training to cope with enhanced energy demands.
55 f a combination of a human PKP2 mutation and endurance training to trigger an ARVC-like phenotype.
57 biochemical responses to acute exercise and endurance training were investigated in female Fischer 3
58 In contrast, the right ventricle responds to endurance training with eccentric remodeling at all leve
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