ライフサイエンスコーパス: energy expenditure

1 d with individual daily activity budgets and energy expenditure).

2 ared with 3 meals/d, meal skipping increased energy expenditure.

3 s from increased energy intake or defects in energy expenditure.

4 resistance due to a compensatory increase in energy expenditure.

5 he periphery to balance food consumption and energy expenditure.

6 OCN, impaired glucose tolerance, and reduced energy expenditure.

7 duce diminishingly small increments in daily energy expenditure.

8 esis is an important contributor to adaptive energy expenditure.

9 y homeostasis, whereby energy intake exceeds energy expenditure.

10 ibits UCP1-dependent increases in whole-body energy expenditure.

11 ty acids to produce heat, thereby increasing energy expenditure.

12 ystems, the net effect of which is to reduce energy expenditure.

13 ity is a primary factor in the regulation of energy expenditure.

14 eride, cholesterol, glucose homeostasis, and energy expenditure.

15 erations increase both aerobic and anaerobic energy expenditure.

16 e tissue, have increased BAT mass and higher energy expenditure.

17 , and triglyceride levels, as well as higher energy expenditure.

18 othalamic output to control both feeding and energy expenditure.

19 e improves glucose homeostasis and increases energy expenditure.

20 winds further along the route, thus reducing energy expenditure.

21 imize resource localization while minimizing energy expenditure.

22 tivity levels, and resting, active and total energy expenditure.

23 xidative genes and increase FA oxidation and energy expenditure.

24 we show that they are sufficient to inhibit energy expenditure.

25 mice is associated with a 20-30% increase in energy expenditure.

26 s facilitated weight gain while exacerbating energy expenditure.

27 ) could facilitate weight loss by increasing energy expenditure.

28 xergames) may provide short-term increase in energy expenditure.

29 pathetic premotor neurons, have no effect on energy expenditure.

30 oxidative capacity, and enhances whole-body energy expenditure.

31 yle factors, but also by inducing endogenous energy expenditure.

32 7 +/- 3 versus 48 +/- 2 gm) due to increased energy expenditure.

33 he result of an increased metabolic rate and energy expenditure.

34 fat absorption, triglyceride metabolism, or energy expenditure.

35 havioral control of food intake and systemic energy expenditure.

36 consumption, carbon dioxide production, and energy expenditure.

37 are being rearranged minimizing the overall energy expenditure.

38 posity than L(2.1)KOs, solely due to reduced energy expenditure.

39 reased hepatic beta-fatty acid oxidation and energy expenditure.

40 d mitochondrial beta-oxidation and decreased energy expenditure.

41 ity and the metabolic syndrome by increasing energy expenditure.

42 ptl6), neuromedin B, and nesfatin, linked to energy expenditure.

43 issue (BAT) activity, a major contributor of energy expenditure.

44 ortant role for SLN in muscle metabolism and energy expenditure.

45 critical roles for both basal and inducible energy expenditure.

46 tochondria and increased heat production and energy expenditure.

47 issue (WAT), leading to increased whole body energy expenditure.

48 in body fat with corresponding reductions in energy expenditure.

49 ertical displacement leads to an increase in energy expenditure.

50 of mass (CoM) with the objective to optimize energy expenditure.

51 mentia might also be associated with altered energy expenditure.

52 pregnant mice on offspring thermogenesis and energy expenditure.

53 mice reduced food intake and also increased energy expenditure.

54 dementia, and are associated with changes in energy expenditure.

55 (fibroblast growth factor 21) and increases energy expenditure.

56 rn led to differences in flight activity and energy expenditure.

57 ovides the brain with the ability to control energy expenditure.

58 ary exercise- wheel running- and total daily energy expenditure.

59 be associated with different levels of daily energy expenditure.

60 cid oxidation and thermogenesis, and overall energy expenditure.

61 seen as a possible mechanism for increasing energy expenditure.

62 erall host protein synthesis shutoff to meet energy expenditure.

63 ids (AAs) and energy at 1.3x estimated basal energy expenditure].

64 ounteracted by decreases in other aspects of energy expenditure [1, 5-10].

65 They also increased energy expenditure (+15%) as they increased their foragi

66 DIT ( approximately 30 kJ/2.5 h) and resting energy expenditure (243 kJ/d) and an anorexigenic appeti

67 tion of carbohydrate for fat found that both energy expenditure (26 kcal/d; P <.0001) and fat loss (1

68 elations between physical activity and total energy expenditure [4] but are challenged by ecological

69 active populations do not have higher total energy expenditure [5-8].

70 rown adipose tissue (BAT) is specialized for energy expenditure, a process called adaptive thermogene

71 tion of creatine levels decreases whole-body energy expenditure after administration of a beta3-agoni

72 groups, and decreased locomotor activity and energy expenditure after OVX can explain these metabolic

73 accounted for the lack of increase in daily energy expenditure after the first week.

74 ontributes to obesity through alterations in energy expenditure and activity.

75 t on a high fat diet and displayed increased energy expenditure and adipose tissue thermogenesis.

76 have maintained the ability to reduce their energy expenditure and adjust their Tb under adverse env

77 of derived traits suggests major changes in energy expenditure and allocation in the human lineage,

78 pathway accounted for 8.9 +/- 5.6% of total energy expenditure and although experienced players were

79 knockdown (KD) mouse line and assessed both energy expenditure and appetitive motivation under condi

80 ecific Kcnk3 knockout mice display increased energy expenditure and are resistant to hypothermia and

81 pha signaling is important for regulation of energy expenditure and BAT activation, but not the metab

82 GDF15 regulates food intake, energy expenditure and body weight in response to metabo

83 Pharmacological T3 treatment increases energy expenditure and causes weight loss, but is contra

84 potential therapeutic strategies to enhance energy expenditure and combat metabolic disease.

85 prevents obesity as the result of increased energy expenditure and decreased food intake.

86 tion of Ucp1 blocked LP-induced increases in energy expenditure and food intake, and exacerbated LP-i

87 adipose tissue uncoupling protein 1 (UCP1), energy expenditure and food intake, and these effects re

88 etic activation drives robust suppression of energy expenditure and food intake, which lowers body te

89 Cold exposure stimulates energy expenditure and glucose disposal.

90 MC4R effects on energy expenditure and glucose metabolism are primarily

91 reduction of feeding and body weight; their energy expenditure and glucose tolerance were unaffected

92 more exercise is better' by suggesting daily energy expenditure and health plateaus are reached beyon

93 ys promote relatively higher levels of daily energy expenditure and health?

94 cyte development, or WAT browning, increases energy expenditure and holds potential for combating met

95 ow birth weight, increased body weight gain, energy expenditure and hyperphagia.

96 dy weight gain in early adulthood, increased energy expenditure and hyperphagia.

97 gallate and resveratrol (EGCG+RES) increased energy expenditure and improved the capacity to switch f

98 ly expressed JNK1 and JNK2 isoforms regulate energy expenditure and insulin resistance.

99 ring of RES-treated mothers showed increased energy expenditure and insulin sensitivity when on an ob

100 s to summarize existing evidence on pubertal energy expenditure and intake in healthy nonobese adoles

101 lity evidence on the pubertal alterations of energy expenditure and intake, and this has limited our

102 rons (FoxO1 KO(DAT)) show markedly increased energy expenditure and interscapular brown adipose tissu

103 cal/hour/kg in WT mice) explaining increased energy expenditure and lean phenotype in these mice.

104 ffects of bariatric surgery, which encompass energy expenditure and macronutrient preference, the lum

105 l, subfamily A, member 1 and may thus induce energy expenditure and metabolic changes.The objective o

106 t diet-induced obesity and exhibit increased energy expenditure and oxygen consumption in beige and w

107 H melanocortin receptor activation increases energy expenditure and physical activity, switches fuel

108 spects of energy balance, including feeding, energy expenditure and physical activity.

109 ill lead to corresponding increases in total energy expenditure and prevent or reverse energy imbalan

110 temperature, increasing body temperature and energy expenditure and preventing diet-induced obesity.

111 tially subvert these processes by increasing energy expenditure and promoting fat loss, our meta-anal

112 creted proteins correlates with elevation of energy expenditure and promotion of beige and white fat

113 after fibre intake, increased fat oxidation, energy expenditure and PYY, and decreased lipolysis in o

114 t1 KO mice also displayed reduced whole-body energy expenditure and reduced mitochondrial oxygen cons

115 dusp6-deficient mice significantly increased energy expenditure and reduced weight gain in recipient

116 cal conditions, muscle TRIB3 also influences energy expenditure and substrate metabolism, indicating

117 crobiota composition, plasma and fecal SCFA, energy expenditure and substrate oxidation, body composi

118 tter reflect the constrained nature of total energy expenditure and the complex effects of physical a

119 e reduces UCP1 expression in BAT, whole-body energy expenditure and the number of brown-like/beige ce

120 While LP diet increased energy expenditure and Ucp1 expression in an FGF21-depen

121 in peripheral sensory neurons have increased energy expenditure and weight loss when fed a Western di

122 eptor beta increases mitochondrial function, energy expenditure, and brown adipose tissue.

123 olution of energy and macronutrient intakes, energy expenditure, and changes in body composition over

124 tion, incorporating energy intake and waste, energy expenditure, and daily activity.

125 sity, improved insulin sensitivity, enhanced energy expenditure, and fat depot-specific cellular remo

126 ndrial respiration and increased glycolysis, energy expenditure, and fat metabolism.

127 obesity, with increased food intake, reduced energy expenditure, and impaired glucose tolerance.

128 the brown adipose tissue, reduced whole-body energy expenditure, and increased fat deposition, which

129 ile acid, lipid and carbohydrate metabolism, energy expenditure, and inflammation by acting predomina

130 associated with with abdominal obesity, low energy expenditure, and muscle weakness.

131 n-angiotensin-aldosterone system, adiposity, energy expenditure, and pancreatic cell activity.

132 gain, body fat mass accumulation, increased energy expenditure, and reduced arcuate suppressor of cy

133 body composition, hip circumference, resting energy expenditure, and respiratory quotient.

134 ads to higher-firing neurons, with increased energy expenditure, and that sleep serves to return acti

135 tion or increased activity, but by increased energy expenditure, and was accompanied by a transient i

136 a crucial role in the control of feeding and energy expenditure, and within the hypothalamus, the arc

137 ant to diet-induced obesity due to increased energy expenditure ( approximately 10%) and physical act

138 sought to determine if body temperature and energy expenditure are influenced by a cholinergic input

139 are well characterized, but the limiters of energy expenditure are largely unknown.

140 nergy X-ray absorptiometry; activity-related energy expenditure (AREE) by doubly labeled water; and d

141 ion in vivo were not the result of increased energy expenditure, as measured by indirect calorimetric

142 nd fat loss, and attenuated the reduction in energy expenditure associated with calorie restriction.

143 s index support a role for increased resting energy expenditure before clinical onset of ALS.

144 s and provide new evidence for adipocyte and energy expenditure biology, widening the potential of ge

145 ma surprisingly failed to improve whole-body energy expenditure, block muscle accumulation of triglyc

146 oes not affect body weight, food intake, and energy expenditure but results in an exaggerated diabeti

147 ked effects of 17beta-estradiol to stimulate energy expenditure, but did not affect estrogen-induced

148 vity is associated with an increase in daily energy expenditure, but further increases in physical ac

149 ts are hypermetabolic with increased resting energy expenditure, but if and how hypermetabolism contr

150 This increased their calculated annual energy expenditure by 1.8%-3.6% (depending on region and

151 Cold exposure can enhance energy expenditure by activating BAT, and it has been sh

152 Enhancing energy expenditure by cold exposure will likely not impa

153 (LSD1) as a pivotal regulator of whole-body energy expenditure by controlling the oxidative and ther

154 approach for obesity is to optimize/maximize energy expenditure by increasing energy-utilizing thermo

155 er, the FGF21-dependent increase in UCP1 and energy expenditure by LP has no effect on the ability to

156 iding excessive sample handling and reducing energy expenditure by more than ten times.

157 , and its ability to control body weight and energy expenditure by targeting PGC-1b, a transcriptiona

158 othalamic control exerted on food intake and energy expenditure by the leptin-melanocortin pathway.

159 y weight, organ mass, fasting blood glucose, energy expenditure, cardiac geometry and function, cardi

160 of a test drink, measurements were taken of energy expenditure, circulating glucose, lipids (fasting

161 ITC administration induced a 32% increase in energy expenditure compared with vehicle (17.5 +/- 4.9 J

162 n mass, and enhances insulin sensitivity and energy expenditure compared with wild-type mice.

163 Whole-body daily energy expenditure (DEE) during rest and cold stress was

164 ed rates of glucose oxidation with increased energy expenditure, despite reduced overall food intake.

165 Spontaneous activity and energy expenditure did not differ significantly between

166 Energy expenditure did not increase after ingestion of c

167 ight loss results from smaller reductions in energy expenditure during caloric restriction is not kno

168 enhanced the reduction of temperature and of energy expenditure during calorie restriction.

169 Here we test whether the induction of energy expenditure during protein restriction requires U

170 tilation (VE [L min(-1)]) with the change in energy expenditure (EE [W]) at each speed, we were able

171 Glucagon (GCG) acutely stimulates energy expenditure (EE) and hepatic glucose production (

172 n system both play vital roles in increasing energy expenditure (EE) and physical activity, decreasin

173 Energy expenditure (EE) during walking includes energy c

174 Energy expenditure (EE) increases with overfeeding, but

175 Enhancing energy expenditure (EE) is an attractive strategy to com

176 ave previously shown that a relatively lower energy expenditure (EE) predicts weight and fat mass gai

177 f white adipose tissue (WAT), an increase in energy expenditure (EE), and enhancement of insulin sens

178 nemia and results in adaptive suppression of energy expenditure (EE).

179 seen between measured and predicted resting energy expenditure either within or between groups.

180 ry activity pattern was recorded (ambulatory energy expenditure estimation).

181 and less closely with their measured resting energy expenditure expressed as kcal/d (r = 0.69, P = 0.

182 low protein (LP) diets to assess changes in energy expenditure, food intake and other metabolic endp

183 Body weight, body composition, energy expenditure, food intake, and insulin/glucose tol

184 ults in the uncoupling of caloric intake and energy expenditure, fostering overeating and further wei

185 from synergistic glucagon action to increase energy expenditure, GLP-1 action to reduce caloric intak

186 derscore the regulation of feeding behavior, energy expenditure, glucose homeostasis and autonomic ou

187 n adipose tissue (BAT) as a key regulator of energy expenditure has sparked interest in identifying n

188 ulties in feeding during infancy and reduced energy expenditure, hyperphagia, and developmental delay

189 SP2, increased lean body mass and whole body energy expenditure, hyperplastic brown/white adipose tis

190 gate how well flipper strokes correlate with energy expenditure in 33 foraging northern and Antarctic

191 ody composition, psychological function, and energy expenditure in 39 nonobese [body mass index (in k

192 ment of epididymal fat depots and suppresses energy expenditure in a nutritional- and age-dependent m

193 ulin action and increased metabolic rate and energy expenditure in diet-induced obese mice.

194 vant role in mediating estrogenic actions on energy expenditure in females.

195 e of objectively estimated energy intake and energy expenditure in humans.

196 T)-long known to promote heat production and energy expenditure in infants and hibernating mammals-al

197 Metabolic profiling indicated increased energy expenditure in KCP-overexpressing mice and reduce

198 Uncoupling proteins (UCPs) regulate energy expenditure in living cells by inducing proton le

199 Energy expenditure in Maf1(-/-) mice is increased by sev

200 se tissue (BAT) is an important component of energy expenditure in mammals.

201 ters, and energy intake (secondary outcomes).Energy expenditure in mice was measured after subcutaneo

202 tolerance, and altered physical activity and energy expenditure in mice.

203 nt changes in the expression of mediators of energy expenditure in muscle.

204 d and could in theory be exploited to reduce energy expenditure in species that do not normally use t

205 ocytes could be a novel strategy to increase energy expenditure in the context of obesity, diabetes a

206 esting that BChE gene transfer did not alter energy expenditure in the long term.

207 lation with change in 24EE was the change in energy expenditure in tissue other than muscle or fat-fr

208 ronic treatment with IL-4 failed to increase energy expenditure in wild-type, Ucp1(-/-) and interleuk

209 describe increasing food intake (relative to energy expenditure) in response to food insecurity as a

210 ing fat oxidation (P < 0.01), whilst resting energy expenditure increased after HA and HP compared wi

211 4Rs) leads to reduced food intake, increased energy expenditure, increased insulin sensitivity, and r

212 tal energy expenditure model, in which total energy expenditure increases with physical activity at l

213 erge with latitude, with foraging effort and energy expenditure increasing when birds winter further

214 change in energy intake, and increased basal energy expenditure independent of physical activity.

215 an osteoblast-derived hormone that increases energy expenditure, insulin sensitivity, insulin secreti

216 Increased energy expenditure is a primary metabolic effect of diet

217 umption of palatable foods or a reduction in energy expenditure is highly variable between individual

218 of curtailed sleep on physical activity and energy expenditure is less clear, but changes are unlike

219 early hours of the dark cycle, during which energy expenditure is only slightly lower than in wild-t

220 erived hormone favoring glucose homeostasis, energy expenditure, male fertility, brain development, a

221 of obesity, results suggest that increasing energy expenditure may be more effective for reducing bo

222 s in the genes that regulate food intake and energy expenditure may contribute to obesity.

223 al energy x-ray absorptiometry scanning with energy expenditure measured over 10 days at home by doub

224 We compared total energy expenditure, measured using doubly labeled water,

225 ysical) activity types, (2) methods based on energy expenditure, METs (metabolic equivalents of task)

226 Resting metabolic rate, daily energy expenditure, milk energy output and suckling time

227 Here we tested a Constrained total energy expenditure model, in which total energy expendit

228 re plateaued, supporting a Constrained total energy expenditure model.

229 Such Additive total energy expenditure models are supported by exercise inte

230 glucose homeostasis and of both compounds on energy expenditure occur even in the absence of muscle P

231 Finally, LP-induced energy expenditure occurred even in the absence of hyper

232 observations on the survival, behaviour and energy expenditure of animals during such events.

233 ercise of any intensity at the equivalent of energy expenditure of approximately 10 hours/week of wal

234 We provide the first data on activity and energy expenditure of birds, Eurasian cranes Grus grus,

235 reater lipid fuel preference and non-resting energy expenditure, one-half the body fat, and better gl

236 mount of daily wheel running and total daily energy expenditure or energy intake across mice.

237 ic-pituitary-adrenal axis, without affecting energy expenditure or glucose metabolism.

238 cillations is dynamic regulation of cellular energy expenditure over the daily cycle.

239 ing energy expenditure (P = 0.045) and total energy expenditure (P = 0.035).

240 o controls (P = 0.002) and increased resting energy expenditure (P = 0.045) and total energy expendit

241 B4 levels, were leaner, and showed increased energy expenditure, partly due to browning of white adip

242 the upper range of physical activity, total energy expenditure plateaued, supporting a Constrained t

243 ion of the metabolic regulator POMC and less energy expenditure prior to the onset of obesity.

244 plitude x number of strokes) predicted total energy expenditure (R(2) = 0.63) better than flipper str

245 ity, suggesting that OH cells facilitate net energy expenditure rather than energy intake [2, 21-23].

246 red D2R signaling to obesity lies in altered energy expenditure rather than the induction of compulsi

247 se and fatty acids to heat, and may increase energy expenditure, reduce adiposity and lower blood glu

248 ass I histone deacetylases (HDACs) increases energy expenditure, reduces adiposity, and improves insu

249 The combined resting energy expenditure (REE) and handgrip strength provided

250 mass (FFM)-independent reduction of resting energy expenditure (REE) to caloric restriction (CR).

251 lear.We studied the relation between resting energy expenditure (REE), the estimated energy balance,

252 ved in the undernourished children to reduce energy expenditure, reflected by increased N-methylnicot

253 proximately 1 kcal/day; however, total daily energy expenditure remained stable after the first week

254 respectively, without altering body weight, energy expenditure, respiratory quotient, or adiposity.

255 unning wheel for 3 to 7 days increased daily energy expenditure, resulting in a caloric deficit of ap

256 tide (PP), are involved in the regulation of energy expenditure, satiety, and food intake.

257 EF deletion resulted in an acute increase in energy expenditure, selectively impaired early adipose t

258 ics: clinically underweight, exhaustion, low energy expenditure, slow walking speed, and muscle weakn

259 w muscle mass, self-reported exhaustion, low energy expenditure, slow walking speed, and weakness.

260 capsulated placebo mixture, measurements of energy expenditure, substrate oxidation, core temperatur

261 s seasonal adjustment mechanisms in terms of energy expenditure, Tb and locomotion.

262 Llamas adjusted their energy expenditure, Tb and locomotor activity according

263 esting metabolic rate (BMR/RMR), total daily energy expenditure (TDEE), and/or energy intake (EI) for

264 We quantified total energy expenditure (TEE) in patients with SBS by using t

265 e fed FF exhibited consistently higher total energy expenditure (TEE) than their corresponding WT.

266 doubly labelled water measurements of total energy expenditure (TEE; kcal day(-1)) in humans, chimpa

267 costs three to six times the resting rate of energy expenditure) that rapidly depleted onboard oxygen

268 adiposity by regulating feeding behavior and energy expenditure, the roles for individual brain regio

269 ight due to reduced food intake and elevated energy expenditure; they also manifested glucose intoler

270 y inert PIC intermediate, which necessitates energy expenditure to complete the process.

271 ls, improves glucose tolerance, and promotes energy expenditure to treat symptoms and underlying caus

272 n, initiating changes in eating behavior and energy expenditure, to maintain energy balance.

273 body weight, body composition, food intake, energy expenditure, total cage activity, and running whe

274 th shore and ice bears in summer, resembling energy expenditures typical of fasting, nonhibernating m

275 topoietic cells of adult mice neither alters energy expenditure upon cold exposure nor reduces browni

276 They also showed an increase in energy expenditure using indirect calorimetry, which was

277 d water method and derived activity-specific energy expenditures using fine-scale time-activity budge

278 e results reveal a role for Egr1 in blocking energy expenditure via direct Ucp1 transcription repress

279 .When compared with the 3-meal control, 24-h energy expenditure was higher on both skipping days (BSD

280 Energy expenditure was lower during ER in the morning (P

281 Daily energy expenditure was measured as field metabolic rate

282 Resting energy expenditure was measured by using indirect calori

283 ssed with the use of bio-impedancemetry, and energy expenditure was measured with the use of indirect

284 Energy expenditure was not altered (P-time x treatment =

285 justing for body size and composition, total energy expenditure was positively correlated with physic

286 Energy expenditure was significantly higher (~145%; P <

287 Metabolic risk factors and total energy expenditure were associated with PC6, PC9 (AA and

288 ions; however, no significant differences in energy expenditure were observed 24 h (~6%; P > 0.49) an

289 pose tissue (BAT) activity, WAT browning and energy expenditure were significantly higher in Ern1(f/f

290 On chow diet, Sucnr1(-/-) mice had increased energy expenditure, were lean with a smaller WAT compart

291 s (AVGs) have been shown to acutely increase energy expenditure when compared with seated video games

292 UCP1-dependent thermogenesis and whole-body energy expenditure, which opens the way to improved ther

293 t GPR45 is a regulator of POMC signaling and energy expenditure, which suggests that it may be a pote

294 sponse reduces cell surface area to minimize energy expenditure while conserving biomass, suggesting

295 opranolol decreases cardiac work and resting energy expenditure while increasing peripheral lean mass

296 re) mice exhibited increased food intake and energy expenditure with no net effect on body weight.

297 In the absence of energy expenditure, with regulatory DNA at thermodynamic

298 levels as the body adapts to maintain total energy expenditure within a narrow range.

299 thalamus (DMH) developed obesity and reduced energy expenditure without hyperphagia.

300 ce exhibited normal food intake but elevated energy expenditure, yielding reduced weight gain.