ライフサイエンスコーパス: enflurane

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1 of which were insensitive to ethanol and/or enflurane.

2 glycine receptors by the volatile anesthetic enflurane.

3 eased the response to isoflurane (0.2-2 mM), enflurane (0.2-2 mM), and 1-chloro-1,2, 2-trifluorocyclo

4 er effects on the response to halothane than enflurane [1, 2].

5 Halothane (2%) and enflurane (4%) prolonged the decay phase of the slow com

6 annel recordings revealed that halothane and enflurane activated a high conductance anion channel, wh

7 At higher concentrations, effects of enflurane and halothane, but not of isoflurane, were sta

8 of low-threshold Ca2+ spikes (LTS), and both enflurane and nickel also suppressed LTS and neuronal bu

9 two different T currents in nRT neurones by enflurane and other volatile anaesthetics occurs within

10 th the extent of potentiation by isoflurane, enflurane, and 1-chloro-1,2,2-trifluorocyclobutane, wher

11 t concentrations of isoflurane, sevoflurane, enflurane, and desflurane sensitize TRPV1 to capsaicin a

12 s (10(-4), 10(-3), and 10(-2) M), halothane, enflurane, and isoflurane each caused a concentration-de

13 These results suggest that (1) halothane, enflurane, and isoflurane interfere with Ca(2+) electrod

14 volatile halogenated anesthetics halothane, enflurane, and isoflurane on Ca(2+) electrode measuremen

15 pyl disulfide, the usual ability of octanol, enflurane, and isoflurane to potentiate the function of

16 esthetic haloethers (desflurane, isoflurane, enflurane, and sevoflurane), a haloalkane (halothane), a

17 cal manipulation) performed under 7-8 min of enflurane anesthesia induced Fos staining in neurons of

18 her anesthetics isoflurane, sevoflurane, and enflurane are also bound to the hydrophobic core of the

19 Volatile anesthetics like halothane and enflurane are of interest to clinicians and neuroscienti

20 associated with isoflurane, sevoflurane, and enflurane binding were -7.7 +/- 0.1 kcal/mol, -8.2 +/- 0

21 es accompanying isoflurane, sevoflurane, and enflurane binding were -8.5 cal/mol K, -10.4 cal/mol K,

22 The halogenated volatile anaesthetic enflurane blocked both currents, but only the slowly ina

23 h K(d) values of 140 +/- 10 micro M, whereas enflurane bound with a K(d) value of 240 +/- 10 micro M.

24 n-response curves of the volatile anesthetic enflurane constructed in the presence and absence of eth

25 Thus, isoflurane, but not halothane or enflurane, enhanced nitric oxide signaling stimulated by

26 lues): desflurane > isoflurane approximately enflurane > halothane >or= sevoflurane, with the differe

27 Isoflurane and enflurane had larger effects than halothane.

28 o each of these agonists, while halothane or enflurane had no effect.

29 peculated that the proconvulsant property of enflurane is related to its depression of the fast somat

30 brane potentials recorded in the presence of enflurane, isoflurane and halothane did not differ signi

31 However, enflurane markedly reduced the amplitude of the fast com

32 hydrate, halothane, isoflurane, sevoflurane, enflurane, nitrous oxide, and xenon, have been demonstra

33 further studied the effects of halothane and enflurane on single ion channels in excised membrane pat

34 zed the effects of halothane, isoflurane and enflurane on the membrane conductances and ion channels

35 The inhalation anesthetic enflurane produced profound, and at times, opposite effe

36 e anesthetics tested (isoflurane, halothane, enflurane, sevoflurane, and methoxyflurane).

37 n two, a pattern more akin to that seen with enflurane than ethanol.

38 to determine how halothane, isoflurane, and enflurane, three commonly used volatile anesthetic agent

39 o inhibit reversibly the enhancing effect of enflurane, toluene, and chloroform in a concentration-de

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