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1 I, respectively, but infection with wt virus enhanced transcription of 5S rRNA by RNAPIII.
2  influences E2F-1 protein stability, and the enhanced transcription of a variety of downstream target
3 ddition to the C1/P1 DNA-binding sites for R-enhanced transcription of a1.
4 ed Ang-2 protein synthesis and secretion via enhanced transcription of Ang-2.
5 (NRF2) is orchestrated and amplified through enhanced transcription of antioxidant and antiinflammato
6 licited by activation of TRPM3 channels with enhanced transcription of AP-1-regulated genes.
7 the unfolded protein response, manifested by enhanced transcription of ATF4 and C/EBP homologous prot
8 egions (-8.2 to -7.5 kb and -7.5 to -7.0 kb) enhanced transcription of both A-chain and heterologous
9 ely dependent on YvrI and is correlated with enhanced transcription of both the yvrI-yvrHa and the ox
10 nc finger transcription factor GKLF-mediated enhanced transcription of c-myc.
11               That the G488A mutation indeed enhanced transcription of ccl1-2 was demonstrated by the
12  G1 cyclins and CDKs and the other involving enhanced transcription of CDIs by the activated receptor
13                RBP2-/- mouse cells displayed enhanced transcription of certain cytokine genes, which,
14 epithelial cells with PMA is associated with enhanced transcription of Cox-2 and increased production
15 t cellular transformation is associated with enhanced transcription of Cox-2 and increased production
16 of transcription factor, NF-kappaB; and (iv) enhanced transcription of cytochrome oxidase Vb (COX Vb)
17  the mouse dor promoter was required for the enhanced transcription of dor gene in phytohemagglutinin
18 lation induced by DNA damage correlates with enhanced transcription of downstream p53 target genes.
19 res despite the fact that neither polyphenol enhanced transcription of elastin mRNA or cellular proli
20 tion of AI-2 increased cell motility through enhanced transcription of five motility genes, we propos
21 f lamprey blood cells with rIL-17D.1 protein enhanced transcription of genes expressed by the B-like
22 e acetylation, chromatin reorganization, and enhanced transcription of genes, such as ET-1, enhancing
23  active Smo and does not necessarily involve enhanced transcription of Gli1.
24 ives HIF-1alpha protein accumulation through enhanced transcription of HIF-1alpha mRNA, a process tha
25 ylated form of nuclear MLC(20), resulting in enhanced transcription of ICAM-1.
26 ich causes nuclear accumulation of TRX-1 and enhanced transcription of inflammatory mediators through
27 ed the inflammatory response, as measured by enhanced transcription of interleukin-6 and tumor necros
28 ng to an altered chromatin landscape and the enhanced transcription of low-expressed genes.
29 increased C-Jun transcriptional activity and enhanced transcription of matrix metalloproteinase 10 (M
30         Stimulation of memory cells revealed enhanced transcription of "memory-primed" genes compared
31                It was found that OSM induced/enhanced transcription of MMP-1 (interstitial collagenas
32 d through an induction process that requires enhanced transcription of mtrCDE when gonococci are grow
33 of NOXA at the protein level was preceded by enhanced transcription of NOXA mRNA.
34 lycosaminoglycan binding was associated with enhanced transcription of peptidoglycan recognition prot
35 that recognizes the inducers and signals the enhanced transcription of phase 2 genes does so by virtu
36 ERH1 results in salicylic acid accumulation, enhanced transcription of RPW8 and RPW8-dependent sponta
37         We discovered that csrR mutants have enhanced transcription of sagA, a gene associated with s
38 pendent interactions are required to achieve enhanced transcription of specific estrogen-receptor tar
39 ere with beta-catenin turnover, resulting in enhanced transcription of target genes through the incre
40 Jun, resulting in diminished degradation and enhanced transcription of the Bcl-2 homology 3 domain-on
41 tigen and beta-catenin in colon cancer cells enhanced transcription of the c-myc promoter, the downst
42                                       NtSLT1 enhanced transcription of the CaN-dependent ENA1 gene pr
43 ncrease in Egr-1 protein and mRNA levels and enhanced transcription of the Egr-1 gene via serum respo
44               The mechanisms contributing to enhanced transcription of the gene coding for PKCbetaII,
45 stasis by insulin is mediated in part by the enhanced transcription of the gene encoding SREBP-1c (st
46 stasis by insulin is mediated in part by the enhanced transcription of the gene encoding sterol regul
47 and the overexpression of COX-2 is caused by enhanced transcription of the gene.
48 hese, is amplification of the SERCA gene and enhanced transcription of the gene.
49 This increase in enzyme activity reflects E6-enhanced transcription of the human telomerase reverse t
50 dence that these mutations in SRC-3 promoted enhanced transcription of the IGFBP3 gene and globally i
51  we present evidence that anguibactin itself enhanced transcription of the iron-transport genes fatA
52  the activation of nuclear factor kappaB and enhanced transcription of the KC gene with equal potency
53  the leukotoxin gene operon which results in enhanced transcription of the leukotoxin.
54 the short half-lives of the P2Y(1) mRNAs, it enhanced transcription of the P2Y(1) gene.
55           This latter property resulted from enhanced transcription of the streptolysin S biogenesis
56            Moreover, Myd88(MYEL) mice showed enhanced transcription of the Tnfa gene and an excessive
57 ases in RPE cells occurred primarily through enhanced transcription of the VEGF gene and via the IGF-
58 secreted protein levels in RPE cells through enhanced transcription of the VEGF gene.
59 rd4 with growth factor-responsive genes, and enhanced transcription of these genes that could be atte
60                          PR55alpha-PP2A also enhanced transcription of these genes, without affecting
61 ription factors, and that is associated with enhanced transcription of this gene and increased enzyme
62 ha (HIF-1alpha) and HIF-2alpha, resulting in enhanced transcription of transforming growth factor-alp
63            Reduced expression of NME2 led to enhanced transcription of vinculin.

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