ライフサイエンスコーパス: enhancer RNA

1 a super-enhancer element and its associated enhancer RNA.

2 re not multiexonic, thus are more similar to enhancer RNAs.

3 s transcription units, generating non-coding enhancer RNAs.

4 tergenic lncRNAs, antisense transcripts, and enhancer RNAs.

5 and are required to produce their associated enhancer RNAs.

6 nhancer transcription process and non-coding enhancer RNAs.

7 er-enhancer interactions and the presence of enhancer RNAs.

8 re, we expand on current concepts to discuss enhancer RNAs and enhancer transcription, and how enhanc

9 Structured (CRS overlapping) enhancer RNAs and extended 3' ends have significantly in

10 s to be mediated by long, rather than short, enhancer RNAs and to be more prominent in intragenic, ra

11 atin remodeling, transcribes a bidirectional enhancer RNA, and loops to physically interact with the

12 haracterized by PU.1 binding, an increase of enhancer RNA, and P300 recruitment.

13 egulate RNAPII recruitment, the synthesis of enhancer RNAs, and the activation of tumor-promoting gen

14 cipitation of hnRNP H cross-linked to the N1 enhancer RNA, as well as gel mobility shift analysis of

15 of lncRNA biology, such as the functions of enhancer RNAs, circular RNAs and chemical modifications

16 The enhancer RNAs did not appear to contain a normal cap str

17 uggest that MUNC is not a classic cis-acting enhancer RNA (e-RNA) acting exclusively by stimulating t

18 We have identified new enhancer RNA elements and an extended stem-loop in the N

19 Genetic modification of enhancer RNA elements can be utilized for designing atte

20 thus, unstable transcripts such as putative enhancer RNA (eRNA) are dramatically increased.

21 An enhancer RNA (eRNA) is transcribed in thyroid tissue fro

22 f divergent gene promoters and bidirectional enhancer RNA (eRNA) production at enhancers.

23 mapped p53-regulated enhancers by looking at enhancer RNA (eRNA) production.

24 upancy correlates with enhancer activity and enhancer RNA (eRNA) transcription.

25 uridine-rich small nuclear RNA (UsnRNA) and enhancer RNA (eRNA), and in the transcription of coding

26 diminishes the signal-dependent induction of enhancer RNAs (eRNAs) and abrogates stimulus-induced enh

27 Active enhancers produce bi-directional enhancer RNAs (eRNAs) and display CBP/p300-dependent his

28 re located in transcribed sequences encoding enhancer RNAs (eRNAs) and were shown to impair enhancer

29 Enhancer RNAs (eRNAs) are a class of long noncoding RNAs

30 ription is pervasive at active enhancers and enhancer RNAs (eRNAs) are tightly coupled to regulated t

31 -on sequencing (Gro-seq) data found abundant enhancer RNAs (eRNAs) being transcribed at ESEs.

32 Enhancer RNAs (eRNAs) have emerged as an important compo

33 Here we report that enhancer RNAs (eRNAs) identified by global nuclear run-o

34 We discuss the potential roles of enhancer RNAs (eRNAs) in early elongation and highlight

35 Furthermore, the discovery of enhancer RNAs (eRNAs) provides an alternative approach t

36 understanding of enhancer transcription and enhancer RNAs (eRNAs) remains incomplete.

37 Unbiased examination of enhancer RNAs (eRNAs) that cluster in specific circadian

38 number of long non-coding RNAs (lncRNAs) and enhancer RNAs (eRNAs) with emergent functionality.

39 ranscribes bi-directionally a novel class of enhancer RNAs (eRNAs) within enhancer domains defined by

40 We identify 76 enhancer RNAs (eRNAs), 40 canonical lncRNAs, 65 antisens

41 , integrating transcription of coding genes, enhancer RNAs (eRNAs), and various other noncoding trans

42 miRNAs), long non-coding RNAs (lncRNAs), and enhancer RNAs (eRNAs), as well as yet undiscovered class

43 its upstream enhancers produce bidirectional enhancer RNAs (eRNAs), termed KLK3e.

44 ains (TADs) express noncoding RNAs, known as enhancer RNAs (eRNAs).

45 ed on enhancers, and are thus referred to as enhancer RNAs (eRNAs).

46 transcribed into long noncoding RNAs termed "enhancer RNAs" (eRNAs), their putative role in enhancer

47 primary micro-RNAs, long noncoding RNAs, and enhancer RNAs in a large animal model of acute infarctio

48 Here, we report that CTCF binding to enhancer RNAs is enriched when breast cancer cells are s

49 histone-, promoter upstream transcript- and enhancer RNA-loci.

50 hat enhancer transcription and the resulting enhancer RNAs may, in some cases, have functional roles,

51 ancers exhibiting differential expression of enhancer RNAs pointed a central role for Kruppel-like fa

52 Consequently, Brd4 deletion prevents enhancer RNA production, cell identity gene induction an

53 of engaged RNA polymerases showed a lack of enhancer RNAs, promoter-proximal pausing, and divergent

54 The enhancer RNAs remained in discrete spots within the nucl

55 rs with regulatory sites termed silencers or enhancers, RNA-RNA base-pairing interactions, or chromat

56 Competing endogenous RNAs and enhancer RNAs show interesting functional and regulatory

57 enhancers and is required for activation of enhancer RNA transcription and recruitment of coactivato

58 ctivators, exemplified by p300, causing both enhancer RNA transcription and target gene activation.

59 st cancer cells, and loss of FOXA1 increases enhancer RNA transcription for a representative basal ge

60 invokes changes in histone modifications and enhancer RNA transcription that correspond to altered ex

61 H3K27 acetylation and methylation and super-enhancer RNA transcription.

62 The enhancer RNAs were synthesized not from a defined site b

63 istically define svRNA as a small regulatory enhancer RNA, which functions to promote genome replicat

64 ence of purifying selection on the resulting enhancer RNAs within the human population.