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1 patients, was a complex mass or masses in an enlarged spleen.
2  CT as multiple hypoattenuating masses in an enlarged spleen.
3 fe; 60% of children younger than 5 years had enlarged spleens.
4 rved that MIM-deficient mice often developed enlarged spleens.
5 tive risk 0.65 [14-50], p=0.0023), 26% fewer enlarged spleens (46/79 [58%] vs 67/90 [74%], p=0.0045),
6  these animals reveal that they have greatly enlarged spleens, altered thymic histology, and lymphocy
7 oly(Y,F,A,K)n have been established from the enlarged spleen and lymph nodes that result from copolym
8 ing mice show increased liver apoptosis, and enlarged spleen and lymph nodes, respectively.
9                   The mice developed grossly enlarged spleens and a leukemia involving ATL cells that
10  At 2 y of age the mice showed significantly enlarged spleens and an increase in the CD5(+) B-cell po
11                            Moribund mice had enlarged spleens and lungs, while surviving mice had eve
12 e (37.7 degrees C or above), nailbed pallor, enlarged spleen, and being seen at one of the clinics ra
13 terations of hemopoietic tissues, such as an enlarged spleen due to lymphoid hyperplasia, extramedull
14 pes include shortened long bones, a markedly enlarged spleen, elevated neutrophil counts, an enlarged
15                  Leukemic mice typically had enlarged spleens, invasion of parenchymal organs with ma
16                             Furthermore, the enlarged spleens of FMLV-IL-1beta-infected mice correlat
17 review found no evidence of infection and an enlarged spleen that showed active germinal centers.
18 cKO (conditional knockout) mice exhibited an enlarged spleen with disrupted spleen architecture and l
19                        PC1/3 KO mice have an enlarged spleen with marked disorganization of the margi
20     At this stage, mice showed significantly enlarged spleens with abnormal B cell-derived white pulp
21 acking AMPKalpha2, and the mice had markedly enlarged spleens with dramatically increased proportions
22                       Accompanying this were enlarged spleens with prominent white-pulp macrophage in
23 % of the Tat-transgenic population developed enlarged spleens within 1 year after birth.

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