ライフサイエンスコーパス: enolase

1 sminogen activators (cytokeratin 8 and alpha-enolase).

2 evoked potentials, and serum neuron-specific enolase.

3 it the oxidation of actin and UNP similar to enolase.

4 ation of microRNA levels and neuron-specific enolase.

5 hey did not compete for the interaction with enolase.

6 nts: RNase E, PNPase, RhlB RNA helicase, and enolase.

7 issue that did not stain for neuron-specific enolase.

8 he mouse model of BA was identified as alpha-enolase.

9 four were identified by mass spectrometry as enolase.

10 regions of sequence homology between RRV and enolase.

11 caspase-11 and the caspase-1 substrate alpha-enolase.

12 with citrullinated recombinant P gingivalis enolase.

13 NA is degraded via RNaseE, RhlB, PNPase, and enolase.

14 ), polynucleotide phosphorylase (PNPase) and enolase.

15 change in MCAv, S100beta and neuron-specific enolase.

16 carbonyls; in both, lower glycolytic enzyme enolase.

17 l as the binding mode of BV to P. falciparum enolase.

18 in serum S100beta, GFAP, and neuron specific enolase.

19 The glycolytic gene enolase 1 (ENO1) in the 1p36 locus is deleted in gliobla

20 o 3 proteins identified in previous studies: enolase 1 (ENO1), NK-tumor recognition protein (NKTR), a

21 utoantigens identified including arginase 1, enolase 1, and keratin 10.

22 hydrogenase, triose phosphate isomerase, and enolase 1, are targeted using RNAi in Ras-transformed NI

23 es, including enzymes sponsoring glycolysis (enolase 1, triosephosphate isomerase 1, and hexokinase 2

24 Cell surface-associated enolase-1 (ENO-1) enhances plasmin formation and thus pa

25 between glycolysis and Foxp3-E2 variants via enolase-1 shows a previously unknown mechanism for contr

26 n 2 (Foxp3-E2) through the glycolytic enzyme enolase-1.

27 pathway, leading to ERK phosphorylation and enolase 2 (ENO2) expression.

28 d in glycolysis (lactate dehydrogenase A and enolase 2), oxidant stress (FOXO3a), angiogenesis (VEGF)

29 ent selection (isocitrate dehydrogenase 1/2, Enolase 2).

30 e (9 of 12 mice), citrullinated P gingivalis enolase (6 of 6 mice), and uncitrullinated P gingivalis

31 of 6 mice), and uncitrullinated P gingivalis enolase (6 of 6 mice).

32 sitive for antibody (Ab) reactivity to gamma-enolase (8%); alpha-enolase (9%); heat-shock protein 90

33 (9 of 12 mice), uncitrullinated human alpha-enolase (9 of 12 mice), citrullinated P gingivalis enola

34 ice immunized with citrullinated human alpha-enolase (9 of 12 mice), uncitrullinated human alpha-enol

35 (Ab) reactivity to gamma-enolase (8%); alpha-enolase (9%); heat-shock protein 90 (13%); osteopontin (

36 membrane vesicles of B. burgdorferi contain enolase, a glycolytic-cycle enzyme that catalyzes 2-phos

37 dentified the pneumococcal glycolytic enzyme enolase, a nonclassical cell surface and plasminogen-bin

38 diate AMD, and 46% of those with GA had anti-enolase AAbs, compared with 29% of individuals with NV a

39 Enolase activity was also assessed in the presence of ar

40 s truncated form of LOS2 has little, if any, enolase activity, indicating that an intact N-terminal r

41 for quantification was conducted with yeast enolase added to serum as an internal standard.

42 ehyde-3-phosphate dehydrogenase (GAPDH), and enolase, all of which are responsible for energy metabol

43 ermore, in the presence of exogenously added enolase, an increased C4BP binding to and subsequently d

44 ObcA catalyzes its reaction by combining the enolase and acetyltransferase superfamilies, but the pre

45 was evaluated using tryptic digests of yeast enolase and alcohol dehydrogenase.

46 an unexpected role for the metabolic enzymes enolase and aldo-keto reductase as positive and negative

47 In cooked or roasted foods, enolase and aldolase were detectable in chicken breast w

48 LN, where IgG2 autoantibodies against alpha-enolase and annexin AI predominate in the glomerulus and

49 Notably, IgG2 autoantibodies against alpha-enolase and annexin AI were detected in 11 and 10 of the

50 utoantibodies detected, including anti-alpha-enolase and antiannexin AI, identified LN versus SLE and

51 of neuronal markers such as neuron specific enolase and beta-III tubulin.

52 Serum IgG reactivity with alpha-enolase and citrullinated alpha-enolase was assayed by W

53 e IgG2 recognized specific epitopes of alpha-enolase and did not cross-react with dsDNA.

54 peaked earliest, followed by neuron-specific enolase and finally myelin basic protein.

55 s (aldolase, phosphoglycerate mutase 2, beta enolase and glycogen phosphorylase), transport proteins

56 e, glycerol-3-phosphate-dehydrogenase, alpha enolase and L-lactate dehydrogenase B-chain) and in oxid

57 Recombinant human alpha-enolase and P gingivalis enolase, either citrullinated o

58 enes involved in glycolysis (muscle-specific enolase and phosphofructokinase).

59 nally we identified two TMs (neuron-specific enolase and pro-gastrin-releasing peptide) that differen

60 d anti-enolase antibodies cross-reacted with enolase and RRV proteins; we identified regions of seque

61 Neuron-specific enolase and S-100 levels increased in the ensuing 4 hrs

62 Serum neuron-specific enolase and S100b concentrations were increased in the u

63 erized biomarkers, including neuron-specific enolase and S100B protein.

64 terized as a mimotope of an ookinete surface enolase and SM1 presumably competes with enolase, the pr

65 s has been identified as citrullinated alpha-enolase and the importance of genetic factors in anticit

66 ere noted in organs from mice immunized with enolase and then challenged with WT bacteria compared to

67 e form, pyruvate kinase muscle isozyme, beta-enolase and triosephosphate isomerase and phosphoglucomu

68 eling standards (human carbonic anhydrase I, enolase, and alpha-lactalbumin) is achieved at 50- to 10

69 from acidic residues (DNA topoisomerase II, enolase, and C-Raf) show that the relevant acidic residu

70 in complex ions (e.g., superoxide dismutase, enolase, and hemoglobin) desorbed from solution by liqui

71 erized by the presence of flotillin-1, alpha-enolase, and Hsp70, the same proteins that associate wit

72 Enrollment leptin, neuron-specific enolase, and intracellular cell adhesion molecule-1 leve

73 ne levels of chromogranin A, neuron-specific enolase, and multiple soluble angiogenic biomarkers were

74 data show that serum S100b, neuron-specific enolase, and myelin basic protein may aid in outcome cla

75 noblotting for citrullinated proteins, alpha-enolase, and the deiminating enzymes peptidylarginine de

76 (matrix metallopeptidase-9, neuron-specific enolase, and vascular cellular adhesion molecule-1) and

77 ctive allergens involved being parvalbumins, enolases, and aldolases.

78 Plg-Rs include histone 2B, alpha-enolase, annexin 2, and p11, all proteins which lack sig

79 bodies against podocyte antigens (anti-alpha-enolase/antiannexin AI) were also investigated.

80 Anti-RRV and anti-enolase antibodies cross-reacted with enolase and RRV pr

81 The cross-reactivity between an anti-enolase antibody and RRV proteins indicates that molecul

82 e glycolytic enzymes phosphofructokinase and enolase are presented and discussed in relation to their

83 elicase B, polynucleotide phosphorylase, and enolase) are organized as helical filamentous structures

84 rted biomarkers of acute IS (neuron-specific enolase: area under the curve=0.69; interleukin 6: area

85 nd plasminogen bound to different domains of enolase as they did not compete for the interaction with

86 rasites identified P. falciparum enolase (Pf enolase) as the strongest candidate.

87 y measure injury to neurons (neuron-specific enolase), astrocytes (S100b), and axons (myelin basic pr

88 alpha-Enolase autoantibody specificity was confirmed by ELISA

89 th SM1 and ookinete surface enolase, termed "enolase-binding protein" (EBP).

90 The enolase binds plasminogen in a lysine-dependent manner b

91 Antibodies to human alpha-enolase, both citrullinated and unmodified, and to CEP-1

92 Recombinant enolase bound in a dose-dependent manner C4BP purified f

93 Under anaerobic conditions, enolase bound to the RNase E/degradosome stabilizes the

94 We provide a mechanism by which Ecoli uses enolase-bound degradosomes to switch from rod-shaped to

95 Citrullination of human fibrinogen and alpha-enolase by P gingivalis was studied by incubating live w

96 Importantly, we demonstrate that surface enolase captures plasminogen from the mammalian blood me

97 Because the RuBisCO- and "enolase"-catalyzed reactions differ in the regiochemistr

98 d-type P gingivalis with fibrinogen or alpha-enolase caused degradation of the proteins and citrullin

99 tibodies to peptide 1 of citrullinated alpha-enolase (CEP-1) and its arginine-bearing control peptide

100 As compared to neuron-specific enolase, circulating microRNAs are modest but significan

101 Taken together, the CvfA-enolase complex in S. pyogenes is involved in the regula

102 lcium-binding protein B, and neuron-specific enolase concentrations in plasma and serum were measured

103 Serum S100 beta (S100B) and neuron-specific enolase concentrations rise after brain injury.

104 es to interfere with the function of surface enolase could contribute to the development of novel pre

105 the geometric area under the neuron-specific enolase curve from 24 to 72 hours after admission.

106 rences in the area under the neuron-specific enolase curve, or a composite end point of death and poo

107 ssed spots were found and identified, namely enolase, cyclophilin-A, ribosomal protein L13 and actin-

108 nctionalized sol-gels was performed using an enolase digested peptide mixture, a beta-casein digested

109 As expected, enolase displayed consistent expression in vivo, however

110 RNase E/enolase distribution changes from membrane-associated pa

111 scopy and proteinase K treatment showed that enolase does not appear to be exposed on the surface.

112 mbinant human alpha-enolase and P gingivalis enolase, either citrullinated or uncitrullinated, were u

113 B. burgdorferi enolase, either in a recombinant form or as a membrane-b

114 Confounders of neuron-specific enolase elevation should be actively considered: neuron-

115 gory 1-2 had confounders for neuron-specific enolase elevation.

116 of lactate dehydrogenase (LDH1 and LDH2) and enolase (ENO1 and ENO2) that are expressed in a stage-sp

117 In mammalian cells, the alpha-enolase (ENO1) gene encodes both a 48 kDa glycolytic enz

118 Antibodies against alpha-enolase (ENO1), a glycolytic enzyme, are detected in mor

119 tamate (Met6); hyphal wall protein-1 (Hwp1); enolase (Enol); glyceraldehyde-3-phosphate dehydrogenase

120 synaptophysin, chromogranin, neuron specific enolase, epidermal growth factor receptor, HER2, CD5, CD

121 entiation, neuron-specific beta3-tubulin and enolase expression was reduced together with an increase

122 active immunization of mice with recombinant enolase failed to evoke protective immunity against subs

123 I, 2.56-12.16), and elevated neuron-specific enolase (false-positive rate, 0.12; 95% CI, 0.06-0.23; p

124 ceraldehyde-3-phosphate dehydrogenase, alpha-enolase, filamin-A, and heat shock protein 90, were iden

125 , we demonstrated that the surface-expressed enolase from diarrheal isolate SSU of Aeromonas hydrophi

126 unodominant peptide showed 82% homology with enolase from Porphyromonas gingivalis, and the levels of

127 re detected in the levels of neuron-specific enolase from preseason values (median, 6.5 mug/L; range,

128 o determined the structure of the activated "enolase" from G. kaustophilus (carboxylated on Lys 173)

129 cal course of the reaction catalyzed by the "enolases" from Bacillus subtilis and Geobacillus kaustop

130 asting serum chromogranin A, neuron-specific enolase, gastrin, glucagon, vasoactive intestinal peptid

131 he insertion-deletion (indel) process in the enolase gene across the Tree of Life using the phylogene

132 We also showed that the enolase gene could potentially be important for the viab

133 we could delete the chromosomal copy of the enolase gene only when another copy of the targeted gene

134 Since these alpha-enolase gene products have important functions in glucos

135 nally diverse superfamilies (amidohydrolase, enolase, glutathione transferase, haloalkanoic acid deha

136 , presence of myoclonus, and neuron-specific enolase greater than 75 microg/L; accuracy was highest f

137 All three patients with neuron-specific enolase greater than 90 mug/L and Cerebral Performance C

138 f good outcome patients with neuron-specific enolase greater than 90 mug/L and poor outcome patients

139 We measured levels of neuronal-specific enolase, growth-associated protein 43, nerve growth fact

140 romogranin A (>/=600 mug/L), neuron-specific enolase (>/=25 mug/L), and classic grading (Ki-67-based)

141 High levels of serum anti-alpha-enolase (>15 mg/L) IgG2 and/or anti-annexin AI (>2.7 mg/

142 specificity, serum S100b and neuron-specific enolase had optimal positive and negative predictive val

143 xin AI IgG2 and patients with low anti-alpha-enolase/high anti-annexin AI IgG2.

144 ormothermia (p = 0.013), and neuron-specific enolase higher than 33 mug/L (p = 0.029), but not somato

145 Anti-H3 and anti-alpha-enolase IgG2 levels had the most remarkable increase in

146 Anti-alpha-enolase IgG2 recognized specific epitopes of alpha-enola

147 atients with BA had increased levels of anti-enolase IgM and IgG.

148 In contrast, enolase immunization of murine hosts significantly reduc

149 of WT bacteria in the livers and spleens of enolase-immunized mice than that found in the nonimmuniz

150 Instead, CvfA interacted with enolase, implying that CvfA, a putative RNase, controls

151 have identified autoantibodies against alpha-enolase in a mouse model of BA (infected with RRV) and i

152 at positions 343, 394, 420, 427, and 430 of enolase in A. hydrophila SSU; the mutated forms of enola

153 Lyme disease patients exhibit recognition of enolase in serologic assays.

154 cytoadherence of a plasminogen-binding alpha-enolase in T. vaginalis.

155 defined by antibodies to citrullinated alpha-enolase in the context of DR4.

156 However, enolase in the outer membrane vesicles is accessible to

157 the direct involvement of surface-expressed enolase in the pathogenesis of A. hydrophila SSU infecti

158 However, the role of enolase in the RNase E/degradosome is not understood.

159 Here, we report that presence of enolase in the RNase E/degradosome under anaerobic condi

160 rillum rubrum is also able to function as an enolase in vivo as part of an MSP, but only under anaero

161 M. aeruginosa RLPs function as tautomerases/enolases in a methionine salvage pathway (MSP).

162 tial of ENO1-deleted GBM cells, and that the enolase inhibitor phosphonoacetohydroxamate is selective

163 252)FYDAEKKEY(260)) in the A. hydrophila SSU enolase involved in plasminogen binding.

164 Alpha-enolase is a bifunctional gene encoding both a glycolyti

165 Neuron-specific enolase is an easily available, observer-independent pro

166 eferentially localized in nuclei while alpha-enolase is found in the cytoplasm.

167 lly blocked, while the wild-type chromosomal enolase is secreted normally in the same cultures during

168 the secretion of plasmid gene-encoded mutant enolase is totally blocked, while the wild-type chromoso

169 evoked potentials, and serum neuron-specific enolase, is recommended; however, no study examined the

170 One such difference was the number of enolase isoforms and their sum abundance; castor had app

171 d poor outcome patients with neuron-specific enolase less than or equal to 17 mug/L (upper limit of n

172 majority of 14 patients with neuron-specific enolase less than or equal to 17 mug/L who died had a ca

173 mor burden, and the baseline neuron-specific enolase level.

174 Exenatide did not reduce neuron-specific enolase levels and did not significantly improve a compo

175 at all time points and lower neuron-specific enolase levels on days 1 and 3 compared with those with

176 lysosomal thiol-reductase GILT, and a 47-kDa enolase-like protein.

177 Here, we show that enolase lines the ookinete surface.

178 Enolase localization in photoreceptors was assessed by i

179 d two cohorts: patients with high anti-alpha-enolase/low anti-annexin AI IgG2 and patients with low a

180 ted protein 2), and energy metabolism (alpha-enolase, malate dehydrogenase, triosephosphate isomerase

181 i and Plasmodium falciparum, suggesting that enolase may act as an invasion ligand.

182 n suggests that the unmodified form of alpha-enolase may be important in initiating the corresponding

183 milarity and cross-reactivity with bacterial enolase may indicate a role for bacterial infection, par

184 Levels of alpha-enolase, MBP-1, and c-myc expression were compared to le

185 otein, CD11b), and neuronal (neuron-specific enolase, neuronal nitric oxide synthase) markers in IR(A

186 Serum levels of neuron-specific enolase (NSE) and neuron-enriched S100 beta (S100beta) w

187 Neuron-specific enolase (NSE) is a biomarker for neuronal stress.

188 Neuron-specific enolase (NSE) is a widely-used biomarker for prognostica

189 leasing peptide (ProGRP) and neuron specific enolase (NSE) is presented, which involves coextraction

190 Serum neuron specific enolase (NSE) measurements, brain imaging findings, soma

191 or BMP4 under control of the neuron specific enolase (NSE) promoter.

192 d electrochemical sensor for neuron specific enolase (NSE) was developed by electrochemical polymeriz

193 Chromogranin A (CgA) and neuron-specific enolase (NSE) were assessed monthly if elevated at basel

194 sfully used for detection of neuron-specific enolase (NSE), a traumatic brain injury (TBI) protein bi

195 for the neuromarkers S100B, neuron-specific enolase (NSE), and glial fibrillary acidic protein (GFAP

196 y of neurologic examination, neuron-specific enolase (NSE), and median nerve somatosensory-evoked pot

197 ium bromine ionic liquid and neuron specific enolase (NSE).

198 d higher accuracy than serum neuron-specific enolase (NSE; the area under the receiver operating char

199 lactate dehydrogenase and 77 neuron-specific enolase observations), the statistical framework prospec

200 ylase, the cytoskeletal-like organization of enolase occurred only in the presence of the RNaseE coil

201 d a baseline plasma level of neuron-specific enolase of greater than 15 ng/mL independently predicted

202 graphy reactivity, and serum neuron-specific enolase offers the best outcome predictive performance f

203 ported cases of plasminogen binding to alpha-enolase on mammalian cells, as well as mechanisms by whi

204 The results support the hypothesis that enolase on the surface of Plasmodium ookinetes plays a d

205 tochemistry revealed colocalization of alpha-enolase or annexin AI with IgG2 in glomeruli.

206 ubsequently characterized by neuron-specific enolase or glial fibrillary acidic protein expression, a

207 hromosomal gene product BB0337, annotated as enolase or phosphopyruvate dehydratase, is associated wi

208 motif) ligand 2 (p = 0.030), neuron-specific enolase (p = 0.006), and S100b (p = 0.015) and in patien

209 is, markedly elevated plasma neuron-specific enolase (P = 0.016; hazard ratio, 2.9; 95% CI, 1.2-7.0)

210 ase activity of GD3- cell-derived Yes toward enolase, p125, and Yes itself.

211 Antibodies to citrullinated alpha-enolase peptide 1 (CEP-1) and cyclic citrullinated pepti

212 levels of antibodies to citrullinated alpha-enolase peptide 1 correlated with the levels of antibodi

213 immunodominant peptide, citrullinated alpha-enolase peptide 1, was identified.

214 c phosphorylated and un-phosphorylated alpha-enolase peptides with sera of healthy and PDAC patients.

215 ISA) for reactivity with citrullinated alpha-enolase peptides.

216 ithin the parasites identified P. falciparum enolase (Pf enolase) as the strongest candidate.

217 -PHOSPHATE-ISOMERASE1 (MTI1) and DEHYDRATASE-ENOLASE-PHOSPHATASE-COMPLEX1 (DEP1) under different S co

218 se Y, polynucleotide phosphorylase (PNPase), enolase, phosphofructokinase, and a DEAD box RNA helicas

219 ted baseline chromogranin A, neuron-specific enolase, placental growth factor, and soluble vascular e

220 ide and (EPIP)(4), four copies of Plasmodium enolase-plasminogen interaction peptide that prevents pl

221 lamina propria occupied by neuronal-specific enolase-positive (57.7% increase) and growth-associated

222 ould be actively considered: neuron-specific enolase-producing tumors, acute brain diseases, and hemo

223 ig4 under the control of the neuron-specific enolase promoter and the astrocyte-specific glial fibril

224 gin under the control of the neuron-specific enolase promoter) or fewer than normal (Hand2(+/-) mice)

225 forced expression, via the neuronal specific enolase promoter, showed protection against the learned

226 tured from brain cortices of neuron-specific enolase promoter-driven apoE3 (NSE-apoE3) or apoE4 (NSE-

227 CatB under the control of a neuron-specific enolase promoter.

228 d1) under the control of the neuron-specific enolase promoter.

229 cytoplasmic functions, include GroEL, DnaK, enolase, pyruvate dehydrogenase subunits PdhB and PdhD,

230 increases key glycolytic proteins, including enolase, pyruvate kinase M2 (PKM2), lactate dehydrogenas

231 se, unnamed protein product (UNP) similar to enolase, pyruvate kinase, isoforms of creatine kinase, a

232 - and 4-hr postresuscitation neuron-specific enolase (r = -.86, p < .001 and r = -.87, p < .001, resp

233 that this RuBisCO catalyzes the DK-MTP 1-P "enolase" reaction either in vitro or in vivo.

234 -methylthiopentane 1-phosphate (DK-MTP 1-P) "enolase" reaction in the well-known "methionine salvage"

235 sh structure-function relationships for the "enolase" reaction so that the structural basis for the f

236 Enolase recruited C4BP and plasminogen, but not factor H

237 C4BP bound to the enolase retained its cofactor activity as determined by

238 When the enolase-RNase E/degradosome interaction is disrupted, th

239 and biochemical parameters (neuron-specific enolase, S-100).

240 We measured serum neuron-specific enolase, S100b, and myelin basic protein on days 1-4 and

241 mokine (C-C motif) ligand 2, neuron-specific enolase, S100b, intercellular adhesion molecule-5, and b

242 A neuron-specific enolase serum concentration greater than 90 mug/L predic

243 An neuron-specific enolase serum concentration less than or equal to 17 mug

244 We analyzed neuron-specific enolase serum concentrations 3 days after nontraumatic i

245 Neuron-specific enolase serum concentrations less than or equal to 17 mu

246 High neuron-specific enolase serum concentrations reliably predicted poor out

247 munization of mice with purified recombinant enolase significantly protected the animals against a le

248 correlated with higher serum neuron-specific enolase (Spearman r = -0.52, p < 0.0001).

249 try using recombinant full-length Plasmodium enolase suggested one binding site for BV.

250 rone MRL-lpr/lpr mice recognized human alpha-enolase, suggesting homology between animal models and h

251 amidohydrolase, metallo-beta-lactamase, and enolase superfamilies.

252 ate lactonizing enzyme (MLE) subgroup of the enolase superfamily (UniProt ID A0NXQ8 ).

253 gous proteins in the mechanistically diverse enolase superfamily by screening a library of acid sugar

254 tructure of an uncharacterized member of the enolase superfamily from Oceanobacillus iheyensis (GI 23

255 The mechanistically diverse enolase superfamily is a paradigm for elucidating Nature

256 The mechanistically diverse enolase superfamily is a paradigm for understanding the

257 d investigation of a group of enzymes in the enolase superfamily that are involved in epimerizing dip

258 erized family in the mechanistically diverse enolase superfamily that is encoded by the genome of Esc

259 es for assigning functions to members of the enolase superfamily that should be applicable to other s

260 In the enolase superfamily, a set of conserved active site resi

261 Mg2+ are the only conserved residues in the enolase superfamily, establishing the primary functional

262 different homologous progenitors within the enolase superfamily, in which different spatial arrangem

263 ed in the structures of other members of the enolase superfamily, ManD contains two domains, an N-ter

264 The OSBS family belongs to the enolase superfamily, members of which use a set of conse

265 Like other members of the enolase superfamily, RhamD contains an N-terminal alpha

266 In the mechanistically diverse enolase superfamily, we discovered that a monofunctional

267 gs to the mandelate racemase subgroup in the enolase superfamily.

268 d structurally characterized subgroup in the enolase superfamily.

269 d by other members of the MR subgroup of the enolase superfamily.

270 tor that binds both SM1 and ookinete surface enolase, termed "enolase-binding protein" (EBP).

271 toglobulin), dimeric (beta-lactoglobulin and enolase), tetrameric (streptavidin, concanavalin A, and

272 ed a hydrophobic alpha-helical domain within enolase that contributes to its secretion.

273 ace enolase and SM1 presumably competes with enolase, the presumed ligand, for binding to a putative

274 hat links an immune response to P gingivalis enolase to an important subset of RA, defined by antibod

275 r curves for serum S100b and neuron-specific enolase to classify favorable versus unfavorable outcome

276 he serum biomarkers S100 and neuron-specific enolase to clinical characteristics for predicting outco

277 We propose that BV targets enolase to reduce parasite glycolysis rates and changes

278 mmunodominant epitope in citrullinated alpha-enolase, to which antibodies are specific for RA.

279 amino acids with sequence identity to alpha-enolase (tv-eno1).

280 2.89 (95% CI, 1.09-7.73) for neuron-specific enolase, using a cutoff of 62.0 ng/mL, and 2.15 (95% CI,

281 Enolase, UTP-glucose-1-phosphate uridylyltransferase and

282 The LOQ with extracted serum samples for enolase was 1 muM, linear from 1 to 40 muM, the highest

283 Neuron-specific enolase was an accurate predictor of neurological outcom

284 y with alpha-enolase and citrullinated alpha-enolase was assayed by Western blotting and enzyme-linke

285 trophoresis provided evidence that the alpha-enolase was citrullinated in RA synovial fluid.

286 The enolase was cloned, expressed, purified, and used to gen

287 Alpha-enolase was detected in all of the samples, with mean le

288 This increased enolase was enzymatically inactive and associated with t

289 ELISAs, and cross-reactivity with bacterial enolase was investigated by immunoblotting.

290 xhibits significant similarity to the enzyme enolase was isolated.

291 its classic role in carbohydrate metabolism, enolase was recently found to localize to membranes, whe

292 lysine residues at positions 420 and 427 of enolase were crucial in plasminogen-binding activity.

293 e in A. hydrophila SSU; the mutated forms of enolase were hyperexpressed in Escherichia coli, and the

294 ng individual domains, two binding sites for enolase were identified on the complement control protei

295 potentials (SSEP), and serum neuron-specific enolase were performed in parallel, as part of standard

296 valbumin and two new allergens, aldolase and enolase, were identified at 12, 40, and 50 kDa, respecti

297 cluding ATPase, clathrin, peroxiredoxins and enolase, which may provide clues to the molecular mechan

298 ci specifically interact with human C4BP via enolase, which represents an additional mechanism of hum

299 The LOS2 gene in Arabidopsis encodes an enolase with 72% amino acid sequence identity with human

300 ephalography reactivity, and neuron-specific enolase yielded the best predictive performance (receivi