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1 activation by the basement membrane protein, entactin.
2 r to the G1-domain present in the nidogen or entactin (an extracellular matrix protein), contributes
3 2, E, and G3, to assess interactions between entactin and neutrophil integrin receptors.
4 et critical BBB proteins, including laminin, entactin, and collagen type IV, are elevated in the cere
5 gest that alterations of laminins, nidogen-1/entactin, and epithelial integrin in DR corneas may occu
6 observed that the protein levels of laminin, entactin, and fibronectin in an extracellular matrix (EC
7 n the basement membrane including laminin-5, entactin, and perlecan.
8 axis, and respiratory burst; PMN adhesion to entactin; and PMN transendothelial and transepithelial m
9 that the presence of nidogens (also known as entactins) at the NMJ is the main determinant for TeNT b
10                                     Nidogen (entactin) can form a ternary complex with type IV collag
11     Incubation in chamber slides coated with entactin-collagen IV-laminin (ECL) attachment matrix or
12 of Abs to alpha-actinin, aggrecan, collagen, entactin, fibrinogen, hemocyanin, heparan sulphate, lami
13 tronectin/collagen/chondroitin sulphate, and entactin/fibrinogen/hyaluronic acid) did not.
14 se studies indicate that multiple domains of entactin have the ability to ligate individual integrins
15  alpha5, and beta1 laminin chains; nidogen-1/entactin; integrin alpha3 and beta1 chains in diabetic a
16                                              Entactin is structurally and functionally organized into
17 n, which contains the single RGD sequence of entactin, is sufficient for ligation of the beta3-like i
18 atrix (ECM) proteins, including fibronectin, entactin, laminin, and insoluble elastin, as potently as
19 cribed decreased immunostaining of nidogen-1/entactin; laminin chains alpha1, alpha5, beta1,gamma1; a
20 sement membrane-associated molecules nidogen/entactin (NID-1) and type XVIII collagen (CLE-1) are ass
21                                              Entactin (nidogen) was a specific target for stromelysin
22  IV, collagen, perlecan, bamacan, laminin-1, entactin-nidogen, fibronectin) in SEF areas and in PCL.
23 ning for laminin-1 (alpha 1 beta 1 gamma 1), entactin/nidogen and fibronectin; accumulation of fibron
24 d the basement membrane components, laminin, entactin/nidogen, and collagen IV, was similar in the di
25                                     Laminin, entactin/nidogen, and fibronectin immunoreactivities wer
26             It binds collagen IV, laminin-1, entactin/nidogen-1, fibronectin and vitronectin, but not
27 ot adhere to the common laminin contaminants entactin or collagen type IV, neither of these proteins
28 psinized human type IV collagen, recombinant entactin, or NC1 domain of type VII collagen by dot blot
29             Although members of the nidogen (entactin) protein family are structural components of ba
30 ing laminins, fibronectins, elastin, nidogen/entactin, proteoglycans, and matrix-bound cytokines and
31   The enhanced cleavage of basement membrane entactin to above-normal levels was directly related to
32 E efficiently degrades fibronectin, laminin, entactin, type IV collagen, chondroitan sulfate, and hep
33 heparan sulfate proteoglycan, laminin-1, and entactin was observed in the GBM of the affected animals
34                     Furthermore, cleavage of entactin with the matrix metalloproteinase, matrilysin,

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