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1 dicating that the virus may use endosomes to enter host cells.
2 which receptor is used by a viral strain to enter host cells.
3 d viruses exploit the endomembrane system to enter host cells.
4 ay utilize clathrin-dependent endocytosis to enter host cells.
5 that use different cell surface receptors to enter host cells.
6 unclear whether TrkC is used by T. cruzi to enter host cells.
7 ifferent pathways that animal viruses use to enter host cells.
8 often hijacked by intracellular microbes to enter host cells.
9 three proteins--sigma1, mu1, and sigma3--to enter host cells.
10 pilus and the products of tolQRA in order to enter host cells.
11 involved in the ability of L. pneumophila to enter host cells.
12 ium significantly impaired in its ability to enter host cells.
13 drug was removed the parasites were able to enter host cells.
14 gliding, to move on solid substrates and to enter host cells.
15 t binds up to five GM1 glycosphingolipids to enter host cells.
16 gene ssaH, are transcribed after Salmonella enters host cells.
20 antly increases the ability of Salmonella to enter host cells and required interaction with both acti
25 ine encephalomyelitis virus (TMEV) binds and enters host cells and the molecules involved are not com
26 ytic pathway, to NPC1(+) LE/Lys, in order to enter host cells, and that they do so to access high lev
27 vide new information about how the BoNTs can enter host cells as well as a basis for understanding th
29 mutant could produce normal appressoria and enter host cells but failed to develop, indicating the a
30 8-kDa mutant protein retains the capacity to enter host cells but lacks detectable toxic activity.
36 d pathogen human parainfluenza virus type 3, enter host cells by fusion of the viral and target cell
38 s organism not only is able to attach to and enter host cells by using its cell surface-associated fa
39 prevented BDV infection, indicating that BDV enters host cells by endocytosis and requires an acidic
41 al RNA-containing viral particles, and (iii) enter host cells, complete reverse transcription, enter
42 evidence that the ability of streptococci to enter host cells contributes to the frequent failure of
44 fs were required for the fungal effectors to enter host cells in the absence of any pathogen-encoded
45 lectively, these data suggest that C. jejuni enters host cells in a targeted and tightly controlled p
51 While the spherical virions are known to enter host cells through exploitation of clathrin-mediat
52 e prelimary results suggest that filoviruses enter host cells through receptor-mediated endocytosis v
53 The single-stranded DNA (ssDNA) parvoviruses enter host cells through receptor-mediated endocytosis,
59 e and fungal pathogens have been inferred to enter host cells, where they interact with host resistan
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