ライフサイエンスコーパス: enter the cell cycle

1 osis in transgenic mice whose enterocytes re-enter the cell cycle.

2 ate in apoptosis in neurons attempting to re-enter the cell cycle.

3 an induce postmitotic lens fiber cells to re-enter the cell cycle.

4 ells outside the wound area to synchronously enter the cell cycle.

5 lus for neurons in Alzheimer's disease to re-enter the cell cycle.

6 lin for stimulation of quiescent cells to re-enter the cell cycle.

7 of E2F activity caused post-mitotic cells to enter the cell cycle.

8 A component as leukocytes were stimulated to enter the cell cycle.

9 s can be activated or remaining cells can re-enter the cell cycle.

10 r cells, which are normally post-mitotic, to enter the cell cycle.

11 egulated in G1 phase as normal human T cells enter the cell cycle.

12 caused terminally differentiated cells to re-enter the cell cycle.

13 e, re-express neuroepithelial markers and re-enter the cell cycle.

14 alpha recruited additional Muller glia to re-enter the cell cycle.

15 NA pathway spontaneously exit quiescence and enter the cell cycle.

16 ells that are able to dedifferentiate and re-enter the cell cycle.

17 H-thymidine, increase as proliferating cells enter the cell cycle.

18 d and neonatal ventricular cardiomyocytes to enter the cell cycle.

19 rnal signals into an all-or-none decision to enter the cell cycle.

20 xperience acute nucleosome shifting as cells enter the cell cycle.

21 agents readily respond to growth factors and enter the cell cycle.

22 itical size in G1 in order to pass start and enter the cell cycle.

23 require a lower threshold of stimulation to enter the cell cycle.

24 uiescent state, from which they can later re-enter the cell cycle.

25 on is sufficient for G0-arrested cells to re-enter the cell cycle.

26 show increased apoptosis when stimulated to enter the cell cycle.

27 ing S-phase re-entry when quiescent cells re-enter the cell cycle.

28 to be a function of spheroids failing to re-enter the cell cycle.

29 otein increase in rodent hepatocytes as they enter the cell cycle.

30 cent p21/p27-null MEFs were stimulated to re-enter the cell cycle.

31 ted downstream of thrombin activation and re-enters the cell cycle.

32 n declined progressively after the cells had entered the cell cycle.

33 eased > 10-fold as naturally quiescent cells entered the cell cycle.

34 ease and identifies cells that have recently entered the cell cycle.

35 to Muller glia-like cells, some of which re-entered the cell cycle.

36 on and also prevented activated T cells from entering the cell cycle.

37 by developing a resistance to apoptosis and entering the cell cycle.

38 ycle entry, and apoptosis is induced in HSCs entering the cell cycle.

39 cells from responding to TCR stimulation and entering the cell cycle.

40 ithout significant numbers of quiescent HSCs entering the cell cycle.

41 erminally differentiated and incapable of re-entering the cell cycle.

42 the absence of mitogenic factors and without entering the cell cycle.

43 e cells migrated to the upper papilla before entering the cell cycle.

44 administration caused kidney cells to start entering the cell-cycle.

45 s that prevented cells with damaged DNA from entering the cell-cycle.

46 e subjects were impaired in their ability to enter the cell cycle after stimulation and this impairme

47 found that satellite cells, once activated, enter the cell cycle and a subset undergoes asymmetric d

48 Strains carrying the cdc14-1(ts) allele enter the cell cycle and arrest at restrictive temperatu

49 tic, terminally differentiated neuron can re-enter the cell cycle and be reprogrammed to a state of t

50 stnatal period, anterior lobe progenitors re-enter the cell cycle and expand the populations of speci

51 nt CSCs with intact telomeres that cannot re-enter the cell cycle and form a differentiated progeny.

52 but only rarely do mammalian glial cells re-enter the cell cycle and generate new neurons.

53 ls causes surrounding supporting cells to re-enter the cell cycle and give rise to both new hair cell

54 In response to the Wnt ligand, VECs enter the cell cycle and in the absence of survival sign

55 utamate directly stimulate Muller glia to re-enter the cell cycle and induce neurogenesis in vivo and

56 -Myc provides a stronger signal for cells to enter the cell cycle and is a more potent inducer of apo

57 However, senescent cancer cells may re-enter the cell cycle and lead to tumor relapse.

58 is the ability of differentiated cells to re-enter the cell cycle and lose their differentiated chara

59 echanisms that trigger quiescent cells to re-enter the cell cycle and proliferate in response to tiss

60 he cell cycle shortly after birth but can re-enter the cell cycle and proliferate when subjected to i

61 ns, that together cause the infected cell to enter the cell cycle and so provide a suitable cellular

62 n cytomegalovirus induces quiescent cells to enter the cell cycle and then arrests them in late G(1),

63 ippo-deficient adult mouse cardiomyocytes re-enter the cell cycle and undergo cytokinesis.

64 in response to injury, quiescent hepatocytes enter the cell cycle and undergo DNA replication to prom

65 loss, which induces quiescent hepatocytes to enter the cell cycle and undergo limited replication und

66 her TCC also stimulate differentiated OLG to enter the cell cycle and whether the cell cycle inductio

67 ase of transient renal ischemia, these cells entered the cell cycle and the BrdU signal quickly disap

68 factors (MRFs.) Most mutant satellite cells entered the cell cycle and upregulated expression of myf

69 rimary human T cells that respond to IL-2 by entering the cell cycle and avoiding apoptosis.

70 dblocks that limit adult cardiomyocytes from entering the cell cycle and completing division.

71 dition, we show that the percentage of cells entering the cell cycle and the percentage of cells in e

72 nonfibrillar (denatured) collagen, the cells enter the cell cycle, and p27(KIP1) is down-regulated.

73 conditions were able to synthesize protein, enter the cell cycle, and progress normally through the

74 We also observed that T cells do not enter the cell cycle, and stimulation via the TCR in TCR

75 1 activity decreases when quiescent cells re-enter the cell cycle, and this effect is MCM dependent.

76 cell fate, so that gut endodermal stem cells enter the cell cycle, and undergo cell division that ult

77 nse to acute damage, numerous Muller glia re-entered the cell cycle, and shortly thereafter, expresse

78 large percentage of CD21(int) T2 B cells has entered the cell cycle, and the cycling subpopulation ex

79 rresponding to previously quiescent cells re-entering the cell cycle, and later becomes hypophosphory

80 h the percentage of cells (approximately 2%) entering the cell cycle as defined by Ki-67 expression i

81 SHP-1+ reactive astrocytes do not appear to enter the cell cycle (as defined by PCNA immunoreactivit

82 tionally, 60% of productively infected cells entered the cell cycle, as evaluated by Ki67 staining, b

83 into DNA and a significant increase of cells entering the cell cycle, as indicated by flow cytometry

84 ergo p53-dependent cell cycle arrest, and re-enter the cell cycle at 72 h.

85 tal rat, corneal endothelial cells are still entering the cell cycle at birth, but cell cycle entry g

86 report here that this phosphorylation signal enters the cell cycle at mid S phase.

87 le checkpoint exit, such that tumor cells re-enter the cell cycle before DNA repair is complete.

88 tic neurons are unusual in that they then re-enter the cell cycle before later permanently exiting.

89 e cerebellum and retina of Hq mutant mice re-enter the cell cycle before undergoing apoptosis.

90 partially synchronised Arabidopsis cells re-entering the cell cycle, before induction of CycD3 and C

91 ase from p21-induced growth arrest, cells re-entered the cell cycle but displayed growth retardation,

92 lls require the actions of growth factors to enter the cell cycle, but how individual members of a po

93 ved M phi exited the resting G0 state and re-entered the cell cycle, but experienced a sustained arre

94 alovirus (HCMV) stimulates arrested cells to enter the cell cycle by activating cyclin-dependent kina

95 nt normal human fibroblasts stimulated to re-enter the cell cycle by addition of serum begin to expre

96 evidence that sublytic TCC stimulate OLG to enter the cell cycle by induction of c-jun through activ

97 The lymphocyte is stimulated to enter the cell cycle by the parasite and the multinuclea

98 More cells from old donors entered the cell cycle by 36 hours after wounding, the n

99 predominate form, but as the cells began to enter the cell cycle Cdk9(42) became the major form.

100 07(+/-);p130(-/-) horizontal interneurons re-entered the cell cycle, clonally expanded, and formed me

101 ases provides cancer cells with the power to enter the cell cycle continuously by triggering G1-S-pha

102 ferentiation does not prohibit cells from re-entering the cell cycle, de-differentiating, and acquiri

103 Cells that failed to re-enter the cell cycle exhibited similar responses to IGF-

104 e medium stimulates cells from old donors to enter the cell cycle faster, increases the relative numb

105 es is blocked due to the failure of cells to enter the cell cycle following beta-selection, the proce

106 of reactive astrocytes that do not appear to enter the cell cycle following deafferentation of the ch

107 ession of KSR1 allowed KSR1(-/-) cells to re-enter the cell cycle following MMC treatment.

108 on and induction of E2F activity, as B-cells enter the cell cycle following stimulation via surface I

109 evels were observed over 48 h after cells re-entered the cell cycle following the chemically-induced

110 This new mechanism could explain how cells enter the cell cycle from a quiescent state.

111 temporal activation of NIMX(cdc2) as spores enter the cell cycle from quiescence and suggest that th

112 h is required for Mcm2 loading when cells re-enter the cell cycle from quiescence.

113 cation complex (Pre-RC) assembly in cells re-entering the cell cycle from quiescence.

114 g to a decrease in the number of LNCaP cells entering the cell cycle (i.e., enhanced number of LNCaP

115 Nonproliferating A549 cells did not enter the cell cycle if they were irradiated before bein

116 tes rapidly lose differentiation markers and enter the cell cycle in adult mice in which the telomera

117 l of trophic support due to an attempt to re-enter the cell cycle in an uncoordinated and inappropria

118 c receptor, hematopoietic stem cells did not enter the cell cycle in increased numbers after tMCAO (n

119 ted in G(0)/G(1) phase of the cell cycle, to enter the cell cycle in response to antigen and cytokine

120 xhibit increased quiescence, an inability to enter the cell cycle in response to hematopoietic stress

121 ematopoietic stem cells (HSCs) were shown to enter the cell cycle in response to systemically distrib

122 omain receptor 2 activation allowed cells to enter the cell cycle in the presence of fibrillar collag

123 by serum deprivation and then allowed to re-enter the cell cycle in the presence or absence of the R

124 Myocytes that had entered the cell cycle in preparation for cell division

125 onally upregulated at this point in cells re-entering the cell cycle in response to plant hormones an

126 t, in the absence of p57, nascent neurons re-enter the cell cycle inappropriately but later exit to b

127 In cells capable of entering the cell cycle, including cancer cells, beta-ca

128 ion marker, CD69, and caused many B cells to enter the cell cycle, indicative of polyclonal activatio

129 ctivation and contributes to the decision to enter the cell cycle is largely unknown.

130 ns, suggesting that the number of cells that enter the cell cycle is specifically affected in sof(b12

131 Furthermore, the delay in entering the cell cycle is associated with decreased exp

132 ared with cells from young donors, old cells entered the cell cycle more slowly (48 versus 36 hours),

133 e arrest is transient, and GSCs appear to re-enter the cell cycle on correction of centrosome orienta

134 s that determine whether a precursor cell re-enters the cell cycle or exits and differentiates are cr

135 e the rate at which different cohorts of HSC entered the cell cycle over time.

136 at approximately 8% of LT-HSC asynchronously entered the cell cycle per day.

137 /+, 80.6% of renal epithelial cells that had entered the cell cycle (proliferating cell nuclear antig

138 otic neurons, this involves an attempt to re-enter the cell cycle resulting in apoptosis which is spe

139 They start resorbing when cells re-enter the cell cycle (S phase) and are practically invis

140 NaPP1-treated v-erbB-as1 cells failed to re-enter the cell cycle, showed decreased levels of D- and

141 CY/G-CSF treatment, virtually all BM LT-HSC enter the cell cycle; some of these HSC then migrate int

142 that is unable to induce quiescent cells to enter the cell cycle still retains the ability to accele

143 expected capacity of podocytes to reversibly enter the cell cycle, suggest that podocyte renewal may

144 rat et al. find that when quiescent yeast re-enter the cell cycle, the cell-cycle cyclin-dependent ki

145 ntiated cells with unrepaired DNA lesions re-entered the cell cycle, they manifested a spectrum of gr

146 As cells enter the cell cycle, this complex disappears, and there

147 de-differentiate into stem-like cells or re-enter the cell cycle to compensate for the tissue loss.

148 cells display decreased survival and fail to enter the cell cycle to proliferate.

149 ) glial cells that escaped depletion rapidly enter the cell cycle to repopulate the cortex with alter

150 ption 3 (Stat3) transcription factors and re-enter the cell cycle to yield undifferentiated neuronal

151 edly, dormant HSCs in the bone marrow do not enter the cell cycle upon Pten loss, they do not lose se

152 brain neurons (CaMKII-MYC) can be induced to enter the cell cycle using the physiologically relevant

153 d LDL and its lysoPC moiety stimulate SMC to enter the cell cycle via an oxidative mechanism that cau

154 ent endoreplicating cells, can be induced to enter the cell cycle when co-cultured with larval fat bo

155 le conditions and allows cells to rapidly re-enter the cell cycle when conditions are favorable.

156 splatin by preventing DNA-damaged cells from entering the cell-cycle, which would otherwise result in

157 found that cells released from senescence re-entered the cell cycle with strongly enhanced and Wnt-de

158 tivation or RAS expression, although they re-entered the cell cycle without growth after pRB inactiva

159 d the number of vascular smooth muscle cells entering the cell cycle without inducing apoptosis.