ライフサイエンスコーパス: enteral

1 Patients were randomized 1:1 to enteral acetaminophen 1 g every 6 hours for 3 days (n =

2 e role of the mucus layer was tested via the enteral administration of a mucus surrogate.

3 Furthermore, enteral administration of either live or heat-killed pre

4 Enteral administration of immune-modulating nutrients (e

5 Therefore, continuous enteral administration of lipid- and protein-rich nutrit

6 Enteral administration of lipid-rich nutrition was previ

7 t Th17 polarization could be reversed by the enteral administration of retinoic acid, which induced T

8 Enteral administration of talactoferrin reduced 28-day a

9 ing, negative pressure therapy, debridement, enteral and parenteral feeding, vitamin and mineral supp

10 surgery and was significantly higher in the enteral and parenteral groups than in the control group

11 Supplementation of these amino acids with enteral and parenteral nutrition before, during, and aft

12 Both enteral and parenteral solutions were prepared with comm

13 Hepatic GS contributes to both enteral and systemic ammonia detoxification.

14 d symptoms, and nutritional intake via oral, enteral, and parenteral routes to accurately assess the

15 intestinal adaptation after transplant, but enteral autonomy is attainable.

16 xpands absorptive surface areas and promotes enteral autonomy.

17 that leaves the food matrix, and crosses the enteral barrier to reach their cellular target.

18 fter head injury, the rats received a single enteral bolus of luminescent Escherichia coli Xen 14.

19 The full-feeding group received more enteral calories for the first 6 days, about 1300 kcal/d

20 teral nutrition without supplementation, and enteral causes such as malabsorption.

21 dings in 90.0% of patients who received IV + enteral contrast and 90.4% of patients scanned with IV c

22 om a diverse group of hospitals suggest that enteral contrast can be eliminated in CT scans for suspe

23 Enteral contrast does not improve CT evaluation of appen

24 ed trials from tertiary centers suggest that enteral contrast does not improve diagnostic performance

25 s study of intravenous (IV)-only versus IV + enteral contrast in computed tomographic (CT) scans perf

26 Eliminating enteral contrast may improve efficiency, patient comfort

27 foration, odds ratio of concordance for IV + enteral contrast versus IV contrast alone was 0.95 (95%

28 ence in concordance between IV-only and IV + enteral contrast.

29 se with IV contrast only and 28.5% with IV + enteral contrast.

30 have focused on the need for intravenous or enteral contrast.

31 Enteral delivery of proteins altered the duodenal mucosa

32 We recently reported that enteral delivery of proteins markedly stimulated global

33 Enteral DHA supplementation at a dose of 60 mg per kilog

34 r first enteral feeding to receive either an enteral emulsion providing DHA at a dose of 60 mg per ki

35 The mean delivery of enteral energy and protein was 36 +/- 35% and 37 +/- 38%

36 er function and suggests the possible use of enteral Epo as a therapeutic agent for gut diseases.

37 pathogenesis is incompletely understood, but enteral feed exposures are believed to affect risk.

38 calculated caloric requirements) or standard enteral feeding (70 to 100%) for up to 14 days while mai

39 for the observed beneficial effects of early enteral feeding after major rectal surgery.

40 and P = 0.002).A slow rate of progression of enteral feeding and a less favorable direct-breastfeedin

41 Early enteral feeding and, in particular, breast milk were cor

42 scopists play a key role in the placement of enteral feeding catheters.

43 NALD who were unable to wean from PN to full enteral feeding developed cirrhosis and end-stage liver

44 s encountered during endoscopic placement of enteral feeding devices.

45 problems encountered during the placement of enteral feeding devices.

46 Initial trophic versus full enteral feeding for the first 6 days after randomization

47 randomized to receive either trophic or full enteral feeding for the first 6 days.

48 teral feeding, a strategy of initial trophic enteral feeding for up to 6 days did not improve ventila

49 e median net protein balance improved during enteral feeding from -8.6 to -5.8 mumol . kg body weight

50 ermiT (Permissive Underfeeding versus Target Enteral Feeding in Adult Critically Ill Patients) trial.

51 05), whereas PEG-CCK9 mimicked the impact of enteral feeding in fasted animals (p < 0.05).

52 In critical illness, delaying enteral feeding is associated with a reduction in small

53 ting a higher limit for glucose control when enteral feeding is principally used.

54 Enteral feeding may ameliorate protein loss, but its eff

55 infants, 2) clinical and practical issues in enteral feeding of preterm infants, 3) gastrointestinal

56 ain-matched TLR4KO mice were provided either enteral feeding or TPN.

57 wer and intermediate rates of progression of enteral feeding strategies were associated with a higher

58 Early enteral feeding through a nasoenteric feeding tube is of

59 Enteral feeding to deliver a moderate amount of nonprote

60 and center) within 3 days after their first enteral feeding to receive either an enteral emulsion pr

61 The enteral feeding tube was withdrawn prematurely from 48.5

62 onic Health Evaluation II score, presence of enteral feeding tube, mechanical ventilation, and recent

63 e enabled endoscopists to successfully place enteral feeding tubes in patients who previously require

64 r, as was the proportion of cumulative total enteral feeding volume provided as breast milk: median (

65 cifications, 2) clinical/practical issues in enteral feeding, 3) gastrointestinal and surgical issues

66 s with acute lung injury, compared with full enteral feeding, a strategy of initial trophic enteral f

67 ive underfeeding), as compared with standard enteral feeding, on 90-day mortality among critically il

68 DD], acidification of gastric content, early enteral feeding, prevention of microinhalation); circuit

69 tegies concerning the rate of progression of enteral feeding, the direct-breastfeeding policy, and th

70 on across neonatal networks, and investigate enteral feeding-related antecedents of severe necrotisin

71 s the delivery, digestion, and absorption of enteral feeding.

72 also decreased in humans who are deprived of enteral feeding.

73 ght junction protein losses due to a lack of enteral feeding.

74 cluding medications, underlying illness, and enteral feeding.

75 lity evidence); no reduction in tolerance of enteral feeds (risk ratio, 0.94 [95% CI, 0.62-1.42]; p =

76 Probiotics reduced the time to full enteral feeds in preterm neonates.

77 efits are seen in patients receiving minimal enteral feeds versus full caloric enteral nutrition.

78 s (n = 4527) estimated that the time to full enteral feeds was shorter in the probiotic group (mean d

79 ile, drugs (e.g. laxatives, antibiotics) and enteral feeds.

80 Enteral fish oil administration was associated with incr

81 We therefore sought to determine whether enteral fish oil alone would reduce pulmonary and system

82 Intravenous and enteral fluid resuscitation are frequently used therapie

83 n the mildly to moderately dehydrated child, enteral fluid resuscitation with the aid of an antiemeti

84 ute lung injury when studied in a commercial enteral formula.

85 Protein-energy-enriched enteral formulas (PE-formulas) can improve nutrient inta

86 enriched in sodium nitrate--a precursor for enteral generation of nitrite and nitric oxide--and repl

87 ccurred significantly less frequently in the enteral group (1 patient) compared with parenteral suppl

88 roup and 409 of 1195 patients (34.2%) in the enteral group had died (relative risk in parenteral grou

89 erences between the parenteral group and the enteral group in the mean number of treated infectious c

90 e higher in the parenteral group than in the enteral group on postoperative day 1 (p = 0.027) and day

91 Mean length of stay in the enteral group was 13.4 +/- 2.2 days versus 16.7 +/- 2.3

92 1191 in the parenteral group and 1197 in the enteral group).

93 n the parenteral group, as compared with the enteral group, in rates of hypoglycemia (44 patients [3.

94 arginine concentrations were measured in the enteral group, whereas a better clinical outcome was obs

95 Enteral IAP supplementation may represent a novel approa

96 nts with bleeding risk factors (1B); oral or enteral (if necessary) feedings, as tolerated, rather th

97 2011 comparing isocaloric and isonitrogenous enteral IMN combinations with standard diet in patients

98 Enteral IMN or CON was resumed postoperatively and conti

99 showed that lactoferrin given orally before enteral infection with pathogenic Escherichia coli reduc

100 om order and received, during 5 h, either an enteral infusion of maltodextrins alone (0.25 g . kg(-)(

101 ctional intestine to sustain themselves with enteral intake alone.

102 d-expiratory pressure (P < 0.01), and higher enteral intake in the first 7 days (P = 0.01).

103 n expression in vivo after acute and chronic enteral iron administration in mice.

104 Daily parenteral and enteral manganese intakes were calculated.

105 Our previous data showed that enteral nutrient deprivation or total parenteral nutriti

106 Likewise, enteral nutrient deprivation selectively affected the mo

107 Enteral nutrient deprivation via total parenteral nutrit

108 We used a model of enteral nutrient deprivation, or total parenteral nutrit

109 odel of total parenteral nutrition (TPN), or enteral nutrient deprivation, to study this interaction

110 ies, such as humans, undergo long periods of enteral nutrient deprivation.

111 tocols for the initiation and advancement of enteral nutrient intake had a lower prevalence of acquir

112 strointestinal tract, which is influenced by enteral nutrients and microbial activity.

113 trition (TPN), which deprives the animals of enteral nutrients, displayed a significant decrease of I

114 Both groups received similar durations of enteral nutrition (5.5 vs. 5.1 days; p = .51).

115 ation) preoperatively into two groups: early enteral nutrition (early enteral nutrition, intervention

116 CD in 2010 and 2011 who commenced exclusive enteral nutrition (EEN) for 8 weeks were followed up for

117 iology of Crohn's Disease (CD) and exclusive enteral nutrition (EEN) is the primary induction treatme

118 dy was designed to investigate whether early enteral nutrition (EEN), as a bridge to a normal diet, c

119 nutrition (PN) is still widely preferred to enteral nutrition (EN) in malnourished patients undergoi

120 Us) with relative contraindications to early enteral nutrition (EN) may benefit from parenteral nutri

121 Enteral nutrition (EN) was advanced and PN weaned as ind

122 lled trial (RCT) hypothesized that prolonged enteral nutrition (EN) with supplemental eicosapentanoic

123 Home enteral nutrition (HEN) has always been recognized as a

124 (1-5 g/kg/day) (GA group, n=7) or a standard enteral nutrition (HP group, n=10).

125 f research suggests that ongoing maintenance enteral nutrition (MEN) can be beneficial in maintaining

126 this study was to compare nasojejunal early enteral nutrition (NJEEN) with total parenteral nutritio

127 interleukin (IL)-6 levels were decreased by enteral nutrition (p < 0.05).

128 ncluded rats with head injury fed a standard enteral nutrition (Sondalis HP, n = 10) and group 2 incl

129 on has a deleterious effect in comparison to enteral nutrition alone.

130 izing feeding practices improves delivery of enteral nutrition and decreases feeding complications.

131 PURPOSE OF REVIEW: Adequate enteral nutrition and growth are vital to recovery and s

132 Controlled studies of patients receiving enteral nutrition and observations made from patients on

133 might benefit from early intensive therapy, enteral nutrition and timely transfer to specialized cen

134 defecation was significantly shorter in the enteral nutrition arm than in the control arm (P = 0.04)

135 We compared the impact of administering enteral nutrition as either gastric feeding or jejunal f

136 tudy expands the immunomodulating effects of enteral nutrition as previously observed in rodents to m

137 ntilation whose physicians intended to start enteral nutrition at 44 hospitals in the National Heart,

138 ntilation whose physicians intended to start enteral nutrition at 44 hospitals in the National Heart,

139 ailure similar to those of early full-energy enteral nutrition but with fewer episodes of gastrointes

140 average time from ICU admission to start of enteral nutrition compared to the control group (40.7-29

141 Early combined parenteral/enteral nutrition delayed recovery irrespective of sever

142 immunomodulatory potential of a custom-made enteral nutrition during systemic inflammation in man.

143 ales, 6 females, age 54.9 +/- 3.3 yrs) or no enteral nutrition during the first 4 days of admission (

144 High-protein enteral nutrition enriched with immune-modulating nutrie

145 ally ventilated patients expected to receive enteral nutrition for >/=2 d were randomly assigned to r

146 ilated for more than 72 hours and to require enteral nutrition for more than 72 hours were randomized

147 stomy (PEG) is an effective and safe mode of enteral nutrition for patients needing chronic enteric n

148 er initial trophic (10 mL/hr) or full-energy enteral nutrition for the initial 6 days of ventilation.

149 ity: 44.4% of patients died in the intensive enteral nutrition group (95% confidence interval [CI], 3

150 In the intensive enteral nutrition group, enteral nutrition was given via

151 Delay in initiating enteral nutrition has been reported to disrupt intestina

152 Also, low-dose or trophic enteral nutrition has similar benefits with less gastroi

153 l, the Early Parenteral Nutrition Completing Enteral Nutrition in Adult Critically Ill Patients (EPaN

154 trial (Early Parenteral Nutrition Completing Enteral Nutrition in Adult Critically Ill Patients [EPaN

155 Current guidelines recommend enteral nutrition in critically ill adults; however, poo

156 rvations support recommendations for "early" enteral nutrition in critically ill patients.

157 outcomes as compared to initial full-energy enteral nutrition in patients with acute respiratory fai

158 sed the evidence on effects of probiotics on enteral nutrition in preterm neonates.

159 the effects of probiotic supplementation on enteral nutrition in preterm neonates.

160 r strains used specifically for facilitating enteral nutrition in this population.

161 59% days, B: 69%, C: 71%, p < .001), use of enteral nutrition increased from A to B (stable in C), a

162 enteral nutrition to supplement insufficient enteral nutrition increases morbidity in the intensive c

163 The optimization of enteral nutrition is a priority in preterm neonates worl

164 Early enteral nutrition is associated with less anastomotic le

165 Providing effective enteral nutrition is important during critical illness.

166 It appears that enteral nutrition is more likely to produce hypoglycemia

167 Early enteral nutrition is safe and associated with significan

168 ovide preliminary evidence that hypercaloric enteral nutrition is safe and tolerable in patients with

169 Using a mouse TPN model, removing enteral nutrition leads to decreased crypt proliferation

170 s adverse events considered to be related to enteral nutrition occurred in 5 patients.

171 s study was to examine the impact of delayed enteral nutrition on small intestinal absorption of 3-O-

172 ohn's disease patients starting therapy with enteral nutrition or anti-TNFalpha antibodies and reveal

173 rwent intravenous cannulation and were given enteral nutrition or TPN for 7 days.

174 The amount of enteral nutrition patients with acute lung injury need i

175 luded rats with head injury fed the standard enteral nutrition plus arginine (4 g/kg/d, n = 11).

176 :1) to groups that received either intensive enteral nutrition plus methylprednisolone or conventiona

177 Early enteral nutrition reduced postoperative ileus, anastomot

178 Continuous administration of enteral nutrition resulted in a rapid anti-inflammatory

179 Initial trophic enteral nutrition resulted in clinical outcomes in mecha

180 higher percentage of goal energy intake via enteral nutrition route was significantly associated wit

181 xplores management strategies for delivering enteral nutrition safely and effectively to this high-ri

182 The groups received similar volumes of enteral nutrition solution [1221 mL/d (95% CI: 1120, 132

183 he substitution of a 1.0- with a 1.5-kcal/mL enteral nutrition solution administered at the same rate

184 receive 1.5 (n = 57) or 1.0 (n = 55) kcal/mL enteral nutrition solution at a rate of 1 mL/kg ideal bo

185 ne whether the substitution of a 1.5-kcal/mL enteral nutrition solution for a 1.0-kcal/mL solution re

186 PURPOSE OF REVIEW: Enteral nutrition support is often required in patients

187 combination of corticosteroid and intensive enteral nutrition therapy is more effective than cortico

188 Since enteral nutrition therapy is the preferred nutritional s

189 Management of enteral nutrition varies widely both pre and postoperati

190 nteral nutrition, 354 (36%) of whom received enteral nutrition via the postpyloric route.

191 proportion of targeted energy delivered from enteral nutrition was 72% for the early nasojejunal nutr

192 In both groups, enteral nutrition was attempted early and intravenous mi

193 Intolerance to enteral nutrition was based only on regurgitation and vo

194 nutrition in critically ill patients in whom enteral nutrition was contraindicated did not significan

195 gal anti-inflammatory reflex with lipid-rich enteral nutrition was demonstrated to prevent tissue dam

196 ith corticosteroids, we found that intensive enteral nutrition was difficult to implement and did not

197 Enteral nutrition was found to be an independent factor

198 In the intensive enteral nutrition group, enteral nutrition was given via feeding tube for 14 days

199 f prescribed protein and energy delivered by enteral nutrition was greater in the intervention sites

200 d to stay in ICU for at least 3 days in whom enteral nutrition was indicated.

201 arly initiation of parenteral nutrition when enteral nutrition was insufficient (early parenteral nut

202 Enteral nutrition was subsequently interrupted on averag

203 Enteral nutrition was systematically offered, although P

204 Enteral nutrition was the only factor that was protectiv

205 Enteral nutrition was used in 67% of the patients and wa

206 critically ill patients suitable to receive enteral nutrition were compared with 12 healthy subjects

207 Enteral nutrition with gastric or jejunal feeding in hea

208 However, enteral nutrition with PEG transfers treatment responsib

209 Patients were randomized to either enteral nutrition within 24 hrs of admission (14 "early

210 l ventilation for more than 2 days and given enteral nutrition within 36 hours after intubation at 9

211 than 24 hours, of which 74,771 were ordered enteral nutrition within the first 48 hours.

212 d for 7 days with either a diabetes-specific enteral nutrition without (G group, n=7) or with graded

213 esis that initial low-volume (i.e., trophic) enteral nutrition would decrease episodes of gastrointes

214 an (SD) percentage daily nutritional intake (enteral nutrition) compared to prescribed goals was 38%

215 A total of 985 subjects received enteral nutrition, 354 (36%) of whom received enteral nu

216 odulating nutrients vs standard high-protein enteral nutrition, initiated within 48 hours of ICU admi

217 o two groups: early enteral nutrition (early enteral nutrition, intervention) by nasojejunal tube (n

218 7 trials that compared parenteral nutrition, enteral nutrition, or nutritional supplements to no nutr

219 djunctive nutritional support (parenteral or enteral nutrition, or nutritional supplements) to patien

220 versy exists on the use of immune-modulating enteral nutrition, reflected by lack of consensus in gui

221 omponents were protective ventilation, early enteral nutrition, standardization of antibiotherapy for

222 Current evidence suggests that enteral nutrition, started as soon as possible after acu

223 g mechanical ventilation and receiving early enteral nutrition, the absence of gastric volume monitor

224 ASN, the American Society for Parenteral and Enteral Nutrition, the Academy of Nutrition and Dietetic

225 Among patients ordered early enteral nutrition, the risk of mortality in the body mas

226 However, among those ordered early enteral nutrition, the survival disadvantage for body ma

227 Given that birth marks the first exposure to enteral nutrition, we investigated how nutrient-regulate

228 s is a viable long-term treatment option for enteral nutrition, with complication rates similar to th

229 ng minimal enteral feeds versus full caloric enteral nutrition.

230 with amyotrophic lateral sclerosis receiving enteral nutrition.

231 t common myths and misconceptions related to enteral nutrition.

232 and who were already receiving percutaneous enteral nutrition.

233 Animals were fasted or received lipid-rich enteral nutrition.

234 pneumonia (VAP) in patients receiving early enteral nutrition.

235 d among a subgroup of patients ordered early enteral nutrition.

236 ted energy requirement that was delivered as enteral nutrition.

237 unal nutrition would improve the delivery of enteral nutrition.

238 cations that may impede adequate delivery of enteral nutrition.

239 , could facilitate earlier and more complete enteral nutrition.

240 itically ill patients anticipated to receive enteral nutrition.

241 died critically ill patients receiving early enteral nutrition.

242 initial hypocaloric-hyponitrogenous dose of enteral nutrition.

243 o differences in need for postoperative (par)enteral nutritional support, other complications, hospit

244 Ninety-day regimen of daily enteral or oral citicoline (2000 mg) or placebo.

245 Enteral or parenteral nutrition before, during, and afte

246 d controlled trials (RCTs) have investigated enteral or parenteral nutritional support, and evidence-

247 ve known roles in carbohydrate digestion and enteral or renal glucose transport, suggesting that gene

248 onia-detoxifying capacity through either the enteral or the intravenous route is approximately 160 mu

249 t anaphylaxis, were subjected to a high-dose enteral OVA desensitization protocol (OIT).

250 nutritional guidelines recommend the use of enteral over parenteral nutrition in patients undergoing

251 daily, day 7, and discharge), feeding route (enteral, parenteral, combined, none-oral), length of int

252 afting (CABG) were randomly assigned between enteral, parenteral, or no nutrition (control) from 2 d

253 therapy and a need for opioid analgesic and enteral/parenteral nutrition, with an effect on patient

254 Enteral progesterone administration increased plasma lev

255 Unexpectedly, the glucoregulatory actions of enteral progesterone did not require classical incretin

256 iable practices at the bedside might enhance enteral protein delivery in the PICU with a potential fo

257 tian in the PICU were associated with higher enteral protein delivery.

258 ved during early hypocaloric-hyponitrogenous enteral protein feeding in these patients.

259 lished a method of quantifying the effect of enteral protein feeding on whole-body protein turnover a

260 In relation to mean enteral protein intake <20%, intake >/=60% of the prescr

261 The adequacy of enteral protein intake was significantly associated with

262 This might point to a ceiling effect for enteral protein intake with respect to its influence on

263 ine effects on growth of different levels of enteral protein supplementation in predominantly human m

264 We aimed to assess the influence of an enteral protein supply on the duodenal mucosal proteome

265 In the carbohydrate fed state, enteral proteins but not glutamine increased duodenal pr

266 Rapid enteral rehydration may be used in the uncomplicated, mi

267 RDS were randomly assigned to receive either enteral rosuvastatin or placebo in a double-blind manner

268 uman DAF did not facilitate infection by the enteral route either in immunocompetent animals or in an

269 he enhanced protein-energy provision via the enteral route feeding protocol is safe and results in mo

270 he enhanced protein-energy provision via the enteral route feeding protocol was associated with a dec

271 acebo three times a day orally or by another enteral route for 28 days or until ICU discharge.

272 intraperitoneal injection, infection by the enteral route is very inefficient.

273 criptions received by study patients via the enteral route over the first 12 days in the ICU.

274 be fed through either the parenteral or the enteral route to a delivery route, with nutritional supp

275 entage of prescribed dietary energy goal via enteral route was associated with improved 60-day surviv

276 nteral route is superior to that through the enteral route.

277 ARDS within the previous 48 hours to receive enteral simvastatin at a dose of 80 mg or placebo once d

278 ppropriate parenteral nutrition was added to enteral sources.

279 s also being pioneered by studies looking at enteral sphincter augmentation and regeneration.

280 ically ill patients by using intravenous and enteral stable-isotope Phe tracers.

281 ematic reviews have demonstrated the role of enteral stents in both the upper and lower gastrointesti

282 ployment mechanisms has enabled placement of enteral stents in the mid-gut; hence this has been incre

283 Lack of enteral stimulation during PN decreases both canonical a

284 We set out to determine whether an enteral supply of arginine would modulate bacterial inva

285 Dietary intervention studies have shown that enteral therapy, with defined formula diets, helps child

286 n the LBD that was successfully treated with enteral treatment using a calcium chloride infusion.

287 nutritional supplements (ONS; 11 RCTs), and enteral tube feeding (1 RCT)] with a control comparison

288 March 2016 we randomly allocated 59 women to enteral tube feeding and 57 women to standard care.

289 dverse effects (34%).In women with HG, early enteral tube feeding does not improve birth weight or se

290 0 wk of gestation were randomly allocated to enteral tube feeding for >/=7 d in addition to standard

291 /- SD birth weight was 3160 +/- 770 g in the enteral tube feeding group compared with 3200 +/- 680 g

292 We hypothesized that in women with HG, early enteral tube feeding in addition to standard care improv

293 Enteral tube feeding was discontinued within 7 d of plac

294 Of the women allocated to enteral tube feeding, 28 (47%) were treated according to

295 equently, she was fed an elemental diet with enteral tube feeding, and her condition gradually improv

296 eaned from parenteral nutrition to exclusive enteral tube feeding.

297 utrition was slowly tapered while increasing enteral tube feeding.

298 oric) or 50% of that value (hypocaloric) via enteral tube feeds or parenteral nutrition, with an equa

299 critically ill patients should be started on enteral tube feeds within 48 h of intubation whenever po

300 es morbidity through dermal injury, impaired enteral uptake and subsequent fluid imbalance.