ライフサイエンスコーパス: enteral feeding

1 r treatment of diarrhea that develops during enteral feeding.

2 g, preterm infants during the first 12 wk of enteral feeding.

3 All patients were on enteral feeding.

4 s the delivery, digestion, and absorption of enteral feeding.

5 also decreased in humans who are deprived of enteral feeding.

6 ght junction protein losses due to a lack of enteral feeding.

7 cluding medications, underlying illness, and enteral feeding.

8 eral feed tolerance thereby permitting early enteral feeding.

9 = 0.063), respectively, even at the start of enteral feeding.

10 n are lost with starvation and maintained by enteral feeding.

11 were trauma patients (83%), and 90% received enteral feeding.

12 mass and mucosal immunity when compared with enteral feeding.

13 Group Gf received nasogastric enteral feeding.

14 e to acute amino acid supplementation during enteral feeding.

15 for renal protection, vasopressors, TPN, and enteral feeding.

16 ions were associated with the utilization of enteral feedings.

17 more cost-effective and timely initiation of enteral feedings.

18 ession, traumatic tissue injury, and lack of enteral feedings.

19 cal, as was the use of antibiotics, TPN, and enteral feedings.

20 "nothing by mouth" status (28%), started on enteral feeding (23%), or discharged from the intensive

21 cifications, 2) clinical/practical issues in enteral feeding, 3) gastrointestinal and surgical issues

22 calculated caloric requirements) or standard enteral feeding (70 to 100%) for up to 14 days while mai

23 failure-induced liver disease include early enteral feeding, a multidisciplinary approach to the man

24 s with acute lung injury, compared with full enteral feeding, a strategy of initial trophic enteral f

25 Enteral feeding affects MAdCAM-1 expression.

26 for the observed beneficial effects of early enteral feeding after major rectal surgery.

27 An IED is the preferred diet for early enteral feeding after severe blunt and penetrating traum

28 t of TPN support, can maintain themselves on enteral feedings after this intestinal rehabilitation pr

29 l period (C) of continuous high-carbohydrate enteral feeding alone, and (b) on the seventh day of ent

30 vival after transplant and both grew well on enteral feeding alone.

31 Lack of enteral feeding also induces mucosal effects.

32 and P = 0.002).A slow rate of progression of enteral feeding and a less favorable direct-breastfeedin

33 langiopancreatography acute pancreatitis and enteral feeding and antibiotics in severe acute pancreat

34 curs in patients who develop diarrhea during enteral feeding and may be involved in its pathogenesis.

35 We showed directly a relation between enteral feeding and small-intestinal mucosal growth.

36 of oligopeptides as a source of nitrogen for enteral feeding and the use of oral route for delivery o

37 y acids (SCFAs) in patients starting 14-d of enteral feeding and to compare these changes between pat

38 Early enteral feeding and, in particular, breast milk were cor

39 the major risk factors for NEC: prematurity, enteral feeding, and bacterial colonization.

40 ment of acute rejection, early postoperative enteral feeding, and ganciclovir prophylaxis.

41 Nursing care procedures, antacids, enteral feeding, and prophylactic antibiotics were admin

42 arenteral nutrition (PN), 41% were receiving enteral feeding, and the remaining 18% had already achie

43 y in the medical intensive care unit, use of enteral feeding, and use of sucralfate.

44 icant improvement in glucose control, use of enteral feeding, antibiotic use, adult respiratory distr

45 r upper GI surgery for malignancy with early enteral feeding are limited.

46 gth of hospital stay and early initiation of enteral feedings as compared with bedside self-migrating

47 espiratory tract immunity was preserved with enteral feeding but not with intravenous feeding.

48 These data indicate that enteral feeding can prolong survival and decrease renal

49 scopists play a key role in the placement of enteral feeding catheters.

50 Thus, we tested the hypothesis that enteral feeding could prevent renal ischemic injury usin

51 NALD who were unable to wean from PN to full enteral feeding developed cirrhosis and end-stage liver

52 s encountered during endoscopic placement of enteral feeding devices.

53 problems encountered during the placement of enteral feeding devices.

54 These observations underscore the value of enteral feeding during health and disease.

55 The most recent data suggest that early enteral feeding, even when patients are receiving vasopr

56 Initial trophic versus full enteral feeding for the first 6 days after randomization

57 randomized to receive either trophic or full enteral feeding for the first 6 days.

58 teral feeding, a strategy of initial trophic enteral feeding for up to 6 days did not improve ventila

59 The patients then received enteral feedings for 2 hrs, after which the gastric intr

60 e median net protein balance improved during enteral feeding from -8.6 to -5.8 mumol . kg body weight

61 ts, best practice guidelines for withholding enteral feeding from intubated patients before scheduled

62 the length of time necessary for withholding enteral feeding from intubated patients before scheduled

63 rograms for the length of time they withhold enteral feeding from intubated patients before seven sch

64 Enteral feeding has been strongly recommended in severe

65 Enteral feeding has largely replaced the parenteral rout

66 ermiT (Permissive Underfeeding versus Target Enteral Feeding in Adult Critically Ill Patients) trial.

67 ceiving cimetidine) were mixed with 60 mL of enteral feeding in an airtight container; the PCO2 of th

68 05), whereas PEG-CCK9 mimicked the impact of enteral feeding in fasted animals (p < 0.05).

69 Enteral feeding in malnourished patients may result in r

70 Time to achieve full enteral feeding in the open pyloromyotomy group was (med

71 thogenesis of diarrhea in patients receiving enteral feeding includes colonic water secretion, antibi

72 d with rats infused with diet A after 3 d of enteral feeding irrespective of endotoxin co-infusion.

73 In critical illness, delaying enteral feeding is associated with a reduction in small

74 hy and impaired mucosal transport occur when enteral feeding is not provided, residual transport can

75 ting a higher limit for glucose control when enteral feeding is principally used.

76 olerance (ie, achieving and maintaining full enteral feedings) is a significant problem in preterm in

77 Enteral feeding may ameliorate protein loss, but its eff

78 distribution, and clearance, and concomitant enteral feeding may decrease fluoroquinolone bioavailabi

79 dback, the rate of intestinal transit during enteral feeding may depend on a balance between the acce

80 a direct reaction between gastric fluid and enteral feedings may generate CO2, 30-mL aliquots of gas

81 In preterm infants, both parenteral and enteral feeding methods are modeled on term breast milk.

82 The continuous intragastric in vivo enteral feeding model in the rat developed by Tsukamoto

83 alues were similar to levels achieved in the enteral feeding model.

84 to 4 weeks via intragastric feeding using an enteral feeding model.

85 In such instances, enteral feeding must be temporarily discontinued or seve

86 ndard parenteral nutrition (n = 7) or normal enteral feeding (n = 8).

87 he consequences of frequent interruptions of enteral feeding need to be weighed against the possible

88 We compared the median hours of withholding enteral feeding of intubated patients according to train

89 infants, 2) clinical and practical issues in enteral feeding of preterm infants, 3) gastrointestinal

90 further information defining the effects of enteral feeding on mucosal immunity.

91 here are clinical trials showing benefits of enteral feeding on outcome of acute pancreatitis as well

92 ive underfeeding), as compared with standard enteral feeding, on 90-day mortality among critically il

93 e directives should be addressed long before enteral feeding or assistive ventilatory support might b

94 s no evidence to support specific methods of enteral feeding or increased frequency of ventilator cir

95 rtant clinical outcomes of parenteral versus enteral feeding or intravenous fluids in patients with t

96 ain-matched TLR4KO mice were provided either enteral feeding or TPN.

97 Compared with enteral feeding, parenteral nutrition was associated wit

98 feeding alone, and (b) on the seventh day of enteral feeding plus exogenous insulin (200 pmol/h = 28

99 DD], acidification of gastric content, early enteral feeding, prevention of microinhalation); circuit

100 ethanol diet or high-fat control diet via an enteral-feeding protocol for 3 weeks.

101 Enteral feeding provides nutrients for patients who requ

102 teworthy studies on endoscopic approaches to enteral feeding published from January 2005 to the prese

103 Enteral feeding reduced the death rate and organ permeab

104 on across neonatal networks, and investigate enteral feeding-related antecedents of severe necrotisin

105 The increased intraluminal CO2 following enteral feeding results in a spuriously low gastric intr

106 In experiment 1, enteral feeding significantly reduced the death rate com

107 Enteral feeding significantly reduces the incidence of p

108 cal microbiota or SCFAs were observed during enteral feeding, stark alterations occurred within indiv

109 Enteral feeding stimulates the secretion of hydrogen ion

110 wer and intermediate rates of progression of enteral feeding strategies were associated with a higher

111 is, 87% of surviving patients do not require enteral feeding support.

112 tegies concerning the rate of progression of enteral feeding, the direct-breastfeeding policy, and th

113 hed preterm infants commonly receive minimal enteral feedings, the aim being to enhance intestinal fu

114 Early enteral feeding through a nasoenteric feeding tube is of

115 h protein supplements are routinely added to enteral feeding to correct protein malnutrition, little

116 Enteral feeding to deliver a moderate amount of nonprote

117 and center) within 3 days after their first enteral feeding to receive either an enteral emulsion pr

118 This report describes a novel technique of enteral feeding tube placement, using external magnetic

119 The enteral feeding tube was withdrawn prematurely from 48.5

120 onic Health Evaluation II score, presence of enteral feeding tube, mechanical ventilation, and recent

121 Enteral feeding tubes are often used in this situation,

122 e enabled endoscopists to successfully place enteral feeding tubes in patients who previously require

123 ch as radiographs, fluoroscopic placement of enteral feeding tubes, and insertion of vena cava filter

124 determined in those patients receiving total enteral feeding (two-thirds polymeric/one-third elementa

125 uch as cholestasis might be reduced by early enteral feedings, ursodeoxycholic acid, and cholecystoki

126 r, as was the proportion of cumulative total enteral feeding volume provided as breast milk: median (

127 ith a nasogastric tonometer in situ, in whom enteral feeding was initiated.

128 of the gastric fluid before and after adding enteral feeding was measured tonometrically.

129 enty patients starting exclusive nasogastric enteral feeding were monitored for 14 d.

130 ormula, which was fed from the age when full enteral feedings were tolerated through expected term, o

131 Other important mechanisms include lack of enteral feeding, which leads to reduced gut hormone secr

132 Short-term enteral feeding with an eicosapentaenoic acid-enriched o

133 Early enteral feeding with an IEF has been associated with imp

134 Early enteral feeding with an IEF was not beneficial and shoul

135 was to determine whether early postoperative enteral feeding with an immune-enhancing formula (IEF) d

136 Additionally, patients received enteral feeding with an immunonutrition formula (experim

137 present study determined whether short-term enteral feeding with diets enriched with either eicosape

138 retrospectively testing the hypothesis that enteral feeding with EPA+GLA could reduce alveolar-capil

139 be rapidly modified by continuous short-term enteral feeding with EPA- and GLA-enriched diets irrespe

140 Early enteral feeding with FOSL-HN was safe and well tolerated

141 and hepatic 125I albumin leak compared with enteral feeding without increasing pulmonary myeloperoxi

142 ith functioning grafts are currently on full enteral feeding without need for any intravenous supplem