ライフサイエンスコーパス: enteric bacteria

1 reside within a core regulon, shared by most enteric bacteria.

2 nrf operon promoter from various pathogenic enteric bacteria.

3 ersal regulator of virulence determinants in enteric bacteria.

4 A similar approach can be used for other enteric bacteria.

5 CusR/CusS regulatory system present in other enteric bacteria.

6 . thuringiensis-induced mortality depends on enteric bacteria.

7 in the host and an aberrant host response to enteric bacteria.

8 nt mucosal CD4+ T cell response to commensal enteric bacteria.

9 s a homodimeric DNA-binding protein found in enteric bacteria.

10 e extreme acid-resistance response common to enteric bacteria.

11 a crucial role in global gene regulation of enteric bacteria.

12 llow for spread of the pCoo plasmid to other enteric bacteria.

13 pid that is synthesized by all gram-negative enteric bacteria.

14 flagellar biosynthesis and swarming in many enteric bacteria.

15 t pathway that is typically used by LPS from enteric bacteria.

16 can be regulated by colonization with normal enteric bacteria.

17 hat resistance might be transferred to other enteric bacteria.

18 ntibodies, putatively in response to GalT(+) enteric bacteria.

19 s, the plague bacillus, from closely related enteric bacteria.

20 nd the generation of CD4 T cell responses to enteric bacteria.

21 ally by the adenylyltransferase (GlnE) as in enteric bacteria.

22 ation, and mechanisms of resistance to AP in enteric bacteria.

23 lls required the presence of HBGA-expressing enteric bacteria.

24 version of the genetic map relative to other enteric bacteria.

25 microorganisms, including some gram-negative enteric bacteria.

26 hism three orders of magnitude lower than in enteric bacteria.

27 and internalization of normally noninvasive enteric bacteria.

28 probably of other physiological processes in enteric bacteria.

29 s is significantly more complex than that of enteric bacteria.

30 erstanding of lipopolysaccharide function in enteric bacteria.

31 two-component Ntr regulatory system found in enteric bacteria.

32 ct from those that mediate oxygen control in enteric bacteria.

33 vation-induced cross-resistance to stress in enteric bacteria.

34 t committed step in heme biosynthesis in the enteric bacteria.

35 of the well-characterized Che system of the enteric bacteria.

36 sequence similarity to the type I enzymes of enteric bacteria.

37 bout 80% identical and 90% similar to Fis in enteric bacteria.

38 ex regulatory pathway than that found in the enteric bacteria.

39 or as a dissimilatory metal ion reductase in enteric bacteria.

40 ocyte infiltration, which may be mediated by enteric bacteria.

41 ins (22 genera) of non-E. coli gram-negative enteric bacteria.

42 s by epithelial cells infected with invasive enteric bacteria.

43 ary-phase and osmotically inducible genes in enteric bacteria.

44 and acquisition of pathogenicity islands in enteric bacteria.

45 ritoneal cavity with gram-negative and other enteric bacteria.

46 and thus the regulator is not restricted to enteric bacteria.

47 ral regulator of peroxide stress response in enteric bacteria.

48 r resistance to oxidative and heat stress in enteric bacteria.

49 ation in a manner analogous to that found in enteric bacteria.

50 c resistance genes among clinically relevant enteric bacteria.

51 ays sequence homology to porin proteins from enteric bacteria.

52 .01) internalization of the other strains of enteric bacteria.

53 ity and cobalamin-dependent metabolism among enteric bacteria.

54 ulence and/or regulatory stress responses in enteric bacteria.

55 inct from the Ntr regulatory system found in enteric bacteria.

56 ipenem, which has broad-spectrum coverage of enteric bacteria.

57 d to prevent systemic infection of mice with enteric bacteria.

58 the regulation of inflammatory responses to enteric bacteria.

59 effector that is widely conserved throughout enteric bacteria.

60 ed within dusB-fis operons of representative enteric bacteria.

61 les, along with discrimination against other enteric bacteria.

62 Escherichia coli, Salmonella spp., and other enteric bacteria.

63 Curli are functional amyloids produced by enteric bacteria.

64 roduce inflammatory cytokines in response to enteric bacteria.

65 romosomally encoded antibiotic resistance in enteric bacteria.

66 L-10, but not IL-12 p40, when activated with enteric bacteria.

67 er range of exogenous fatty acids than other enteric bacteria.

68 s different from that of flgJ mutants in the enteric bacteria.

69 he cell by sensing the level of glutamine in enteric bacteria.

70 gest that this function may be restricted to enteric bacteria.

71 which c-di-GMP initiates this transition in enteric bacteria.

72 -domain exoproteins found only in pathogenic enteric bacteria.

73 changes in the growth rate for fast adapting enteric bacteria.

74 role associated with E. coli Lrp limited to enteric bacteria?

75 Nitrogen regulatory protein C (NtrC) of enteric bacteria activates transcription of genes/operon

76 ing a global regulatory role evolved to help enteric bacteria adapt to their ecological niches and th

77 In enteric bacteria, adaptation to a number of different st

78 In enteric bacteria, adhesion to host cells is often promot

79 ndent acid resistance system, which protects enteric bacteria against the extreme acidity of the huma

80 Enteric bacteria also produce amyloids, termed curli, co

81 dga genes are largely restricted to certain enteric bacteria and a few species in the phylum Firmicu

82 dentify ZntB as a zinc efflux pathway in the enteric bacteria and assign a new function to the CorA f

83 ch helminths modulate the host's response to enteric bacteria and bacteria-mediated intestinal inflam

84 tosis but also to apoptosis induced by other enteric bacteria and by several toxic agents including r

85 p of genes involved in host-cell invasion by enteric bacteria and can be modeled to predict the amoun

86 able increases in levels of gastrointestinal enteric bacteria and Candida.

87 tory loops controlling expression of oxyR in enteric bacteria and characteristic of the LysR superfam

88 appendages are found on the surface of many enteric bacteria and enable the bacteria to bind to euka

89 The interactions between enteric bacteria and genetic susceptibilities are major

90 es stable increases in gastrointestinal (GI) enteric bacteria and GI Candida levels with no introduct

91 protein H-NS that is conserved in a range of enteric bacteria and had no known function in Salmonella

92 oup IV polysaccharide capsules are common in enteric bacteria and have more recently been described i

93 is homologous to both CheA and CheY from the enteric bacteria and is therefore a novel CheA-CheY fusi

94 step in the cysteine biosynthetic pathway in enteric bacteria and plants, substitution of the beta-ac

95 step in the cysteine biosynthetic pathway in enteric bacteria and plants, the replacement of the beta

96 step in the cysteine biosynthetic pathway in enteric bacteria and plants, the replacement of the beta

97 establish that gut epithelia actively sense enteric bacteria and play an essential role in maintaini

98 , an abundant nucleoid-associated protein in enteric bacteria and related species, can recognize and

99 eruginosa that is different from that in the enteric bacteria and seems to be most similar to that of

100 By blocking the interaction between enteric bacteria and the innate immune system, CRX-526 m

101 Given the enormous numbers of enteric bacteria and the persistent threat of opportunis

102 m/methylammonium transport (Amt) proteins of enteric bacteria and their homologues, the methylammoniu

103 nclude mutually beneficial signaling between enteric bacteria and their mammalian hosts.

104 Enteric bacteria and their products play an important ro

105 Flagellin is secreted by many enteric bacteria and, upon reaching the basolateral memb

106 This study is aimed at determining if enteric bacteria and/or immune signals regulate their ph

107 e of Escherichia coli, as well as many other enteric bacteria, and are involved in cell colonization

108 solfataricus, mutational data obtained from enteric bacteria, and the enzyme's mechanism of action.

109 r CD4+ T cells react to Ags of the commensal enteric bacteria, and the latter can mediate colitis whe

110 he YcgR-FliG interaction is conserved in the enteric bacteria, and the N-terminal YcgR/PilZN domain o

111 hat the structurally related GlnB protein of enteric bacteria-apparently a paralogue of GlnK-cannot s

112 Commensal enteric bacteria are a required pathogenic factor in inf

113 work in several laboratories has shown that enteric bacteria are common inhabitants of the interior

114 Enteric bacteria are frequently found in aquatic environ

115 We have tested the hypothesis that enteric bacteria are necessary for formation of intestin

116 We conclude therefore that resident enteric bacteria are necessary for the development of sp

117 This study tested the hypothesis that enteric bacteria are necessary for the development of sp

118 Multi-drug resistant (MDR) enteric bacteria are of increasing global concern.

119 the MgtA and MgtB Mg2+ transport systems of enteric bacteria are P-type ATPases by sequence homology

120 Virulence factors expressed by enteric bacteria are pivotal for pathogen colonization a

121 Mechanisms of pH homeostasis in other enteric bacteria are reviewed and provide insight into a

122 Several proteins secreted by enteric bacteria are thought to contribute to virulence

123 ells as a cellular target of noroviruses and enteric bacteria as a stimulatory factor for norovirus i

124 rogen metabolism and in the pathogenicity of enteric bacteria, available biochemical data are scarce.

125 t that glycogen storage may be widespread in enteric bacteria because it is necessary for maintaining

126 pression of outer membrane porin proteins in enteric bacteria, belongs to a large family of transcrip

127 other pathogenic E. coli strains and related enteric bacteria but differs in Salmonella enterica sero

128 ter crescentus or the peritrichous system of enteric bacteria but is pertinent to many Vibrio and Pse

129 CheS has no counterpart in enteric bacteria but revealed distinct similarities to p

130 n is associated with dramatic alterations in enteric bacteria, but little is known about other microb

131 age chi is known to infect motile strains of enteric bacteria by adsorbing randomly along the length

132 MarR and MarA confer multidrug resistance in enteric bacteria by modulating efflux pump and porin exp

133 ed in the regulation of rRNA biosynthesis in enteric bacteria by modulating the efficiency of transcr

134 intestinal homeostasis to commensal luminal enteric bacteria by regulating NF-kappaB signaling in IE

135 t that dysregulated host immune responses to enteric bacteria can influence the development of extrai

136 Certain enteric bacteria can inhibit the NF-kappaB pathway by bl

137 Populations of enteric bacteria can inhibit the NF-kappaB pathway by bl

138 cterial siderophore involved in virulence of enteric bacteria, can only be brought into the cell usin

139 ties to the major chemotaxis proteins of the enteric bacteria: CheA, CheY, CheW, CheR, CheB and Tar.

140 In contrast to enteric bacteria, chemotaxis in Rhodobacter sphaeroides

141 In contrast to the situation in enteric bacteria, chemotaxis in Rhodobacter sphaeroides

142 Enteric bacteria circumvent the gastric acid barrier by

143 hogenic Escherichia coli strain UTI89 and by enteric bacteria Citrobacter koseri and Salmonella enter

144 The central nitrogen metabolic circuit in enteric bacteria consists of three enzymes: glutamine sy

145 The PhoQ sensor domains of enteric bacteria contain an acidic cluster of residues (

146 the enteric pathogen salmonella, like other enteric bacteria, contains three putative membrane-assoc

147 Many enteric bacteria cope with phosphosugar accumulation and

148 Class I pauses, discovered in enteric bacteria, depend on interaction of a nascent RNA

149 Enhancer-dependent transcription in enteric bacteria depends upon an activator protein that

150 DNA restriction and modification systems of enteric bacteria display several enzymatic activities du

151 we report that mycobacteria, in contrast to enteric bacteria, do not form higher-order structures (e

152 ar functional amyloids that are assembled by enteric bacteria during biofilm formation and host colon

153 Type III secretion systems of enteric bacteria enable translocation of effector protei

154 In the enteric bacteria Escherichia coli and Salmonella enteric

155 by the PhoP/PhoQ two-component system in the enteric bacteria Escherichia coli and Salmonella enteric

156 Application of this method to the enteric bacteria Escherichia coli and Salmonella enteric

157 In the enteric bacteria Escherichia coli and Salmonella enteric

158 three steps of siroheme biosynthesis in the enteric bacteria Escherichia coli and Salmonella enteric

159 esses containing commensal organisms, mostly enteric bacteria, even when reared under specific pathog

160 Homologues of the amtB gene of enteric bacteria exist in all three domains of life.

161 binding sites at the nir promoter in related enteric bacteria fix the level of nir operon expression

162 In the enteric bacteria, flgJ null mutants fail to produce the

163 and is an essential enzyme in gram-negative enteric bacteria for maintenance of bacterial viability.

164 different larger systems of proteins used by enteric bacteria for molecular recognition and signaling

165 protects Escherichia coli and possibly other enteric bacteria from exposure to the strong acid enviro

166 ila, 20 vertebrate, 17 Staphylococcus and 20 enteric bacteria genomes, EvoPrinterHD employs a modifie

167 s were colonized with specific-pathogen-free enteric bacteria grown overnight either in anaerobic or

168 Germfree transgenic rats reconstituted with enteric bacteria grown under anaerobic conditions had mo

169 ction of crotonobetaine) from L-carnitine by enteric bacteria has been demonstrated in rats and human

170 Quorum sensing in enteric bacteria has been elusive for a long time.

171 The nitrogen regulatory protein (NtrC) from enteric bacteria has been the model for this family of a

172 Although the oligomerization of H-NS from enteric bacteria has been the subject of intense investi

173 We describe how genetic analysis of enteric bacteria has contributed to these issues.

174 nces reveal that chromosome heterogeneity in enteric bacteria has resulted from the acquisition and d

175 Dysregulated T cell responses to enteric bacteria have been implicated as a common mechan

176 pathways of infection utilized by pathogenic enteric bacteria have important implications for their c

177 rotaviruses, Sapporo-like caliciviruses, and enteric bacteria (i.e., Salmonella, Clostridium, and Shi

178 ses to a subset of commensal (nonpathogenic) enteric bacteria in genetically predisposed individuals.

179 with choledochocholedochostomy (CDC) and by enteric bacteria in patients with choledochojejunostomy

180 Chemotaxis of enteric bacteria in spatial gradients toward a source of

181 of undesirable immune responses to foods and enteric bacteria in the gastrointestinal tract.

182 This is in contrast to certain enteric bacteria in which the absence of a terminal deco

183 ral regulator of peroxide stress response in enteric bacteria) in Mycobacterium leprae, this gene is

184 (ECA), a surface glycolipid ubiquitous among enteric bacteria, in S. Typhimurium pathogenesis.

185 in many pathogenic strains of Gram-negative enteric bacteria, including E. coli, Salmonella spp., an

186 greater numbers of each of seven strains of enteric bacteria, including Listeria monocytogenes (two

187 for metabolizing 1,2-propanediol in certain enteric bacteria, including Salmonella.

188 n is also conserved in several Gram-negative enteric bacteria, indicating that this Mn(2+)-responsive

189 Normal, nonpathogenic enteric bacteria induce and perpetuate chronic intestina

190 This regulatory network in all enteric bacteria involves genetic, allosteric, and physi

191 Type III secretion (T3S) from enteric bacteria is a co-ordinated process with a hierar

192 An early process in the pathogenesis of enteric bacteria is colonization of the intestinal epith

193 The RpoS sigma factor of enteric bacteria is either required for or augments the

194 An aberrant T cell response to enteric bacteria is important in inflammatory bowel dise

195 xpression of histidine biosynthetic genes in enteric bacteria is regulated by an attenuation mechanis

196 The RpoS sigma factor of enteric bacteria is required for the increased expressio

197 The host immune response to enteric bacteria is tightly compartmentalized to the muc

198 ichia coli cytoplasm, but how nitrate enters enteric bacteria is unknown.

199 stimulation, from Escherichia coli and other enteric bacteria, is an interwined alpha-helical homodim

200 t of redox-cycling compounds, whereas in the enteric bacteria it defends the cell against the same ag

201 fis operon structures were identified in the enteric bacteria Klebsiella pneumoniae, Serratia marcesc

202 higella, and Yersinia, three other genera of enteric bacteria known to cause apoptosis.

203 The only regulatory protein in enteric bacteria known to serve exclusively as an NO-res

204 type; second, K. aerogenes and several other enteric bacteria lack a gene homologous to gltF; and thi

205 der region of the his biosynthetic operon of enteric bacteria like Escherichia coli.

206 The chemoreceptors of enteric bacteria mediate attractant responses by interru

207 Eradication of commensal enteric bacteria mitigates intestinal inflammation and I

208 To successfully colonize the human gut, enteric bacteria must activate acid resistance systems t

209 Invasive enteric bacteria must pass through the intestinal epithe

210 To reach the mammalian gut, enteric bacteria must pass through the stomach.

211 ed as a single organism (n=57), with another enteric bacteria (n=23), or with coagulase negative stap

212 y 1 hour of incubation with pure cultures of enteric bacteria, namely, Salmonella typhimurium (two st

213 ellar components, heterologous expression in enteric bacteria of the putative switch basal body genes

214 ntestinal and extraintestinal infections and enteric bacteria on digestive motor function continues t

215 tion in intestinal epithelia and the role of enteric bacteria on VDR expression.

216 Translocation of enteric bacteria or their components (or both) has been

217 mpetitive advantage this system provides for enteric bacteria, particularly those that inhabit an ana

218 ce of different exposure routes by measuring enteric bacteria (pathogenic Escherichia coli) and virus

219 e investigated the relative roles of various enteric bacteria populations in the induction and perpet

220 Enteric bacteria possess multiple fimbriae, many of whic

221 In the non-enteric bacteria Pseudomonas aeruginosa and Streptomyces

222 rminants may play a role in the evolution of enteric bacteria quite apart from, and perhaps with prec

223 The mutS-rpoS intergenic region of enteric bacteria ranges in size from 88 bp in Yersinia t

224 ic challenges in the gastrointestinal tract, enteric bacteria rapidly accumulate salts of glutamate a

225 Secretory IgA, which targets enteric bacteria, regulates the number, composition, and

226 In Escherichia coli and related enteric bacteria, repair of base-base mismatches is perf

227 lactamase-1 (NDM-1) has been found to confer enteric bacteria resistance to nearly all beta-lactams,

228 cuits that exist between the closely related enteric bacteria Salmonella and E. coli.

229 hanism responsible for the divergence of the enteric bacteria Salmonella enterica and Escherichia col

230 Small intestines exposed to enteric bacteria secreted zonulin.

231 there is increasing evidence to suggest that enteric bacteria sense and respond to the host NE stress

232 By contrast, Lrp-related proteins from enteric bacteria show more than 97% amino acid identity.

233 The sequence preceding ORF1 in the enteric bacteria showed a very strong similarity to the

234 The chemotaxis signal protein CheY of enteric bacteria shuttles between transmembrane methyl-a

235 r treatment, however, has limited effects on enteric bacteria so we tested the hypothesis that excret

236 i are functional amyloid fibers assembled by enteric bacteria such as Escherichia coli and Salmonella

237 The former is typically observed in enteric bacteria such as Escherichia coli and Salmonella

238 otein component of curli fibers assembled by enteric bacteria such as Escherichia coli and Salmonella

239 Enteric bacteria such as Escherichia coli must tolerate

240 Enteric bacteria such as Escherichia coli utilize variou

241 ntestinal tract is vital for the survival of enteric bacteria such as Escherichia coli.

242 hanism exhibited by many of the well-studied enteric bacteria, such as enteroinvasive Escherichia col

243 genomes in comparison with those of related enteric bacteria suggest that extensive changes includin

244 Enteric bacteria synthesize glutamate by the combined ac

245 Most species of enteric bacteria tested synthesize cobalamin under both

246 A and for outer membrane proteins from other enteric bacteria than either an isogenic DeltarfaH deriv

247 part because of an increasing prevalence of enteric bacteria that are resistant to 3(rd)-generation

248 epithelial cells infected with gram-negative enteric bacteria that can bypass TLR activation.

249 A binding regulator with orthologues in many enteric bacteria that exhibits classical regulator activ

250 nents of the outer membrane of gram-negative enteric bacteria that function as potent stimulators of

251 IS) is a 22 kDa homodimeric protein found in enteric bacteria that is involved in the stimulation of

252 nal epithelium may recruit E. coli and other enteric bacteria that possess a Tsr-like receptor to pre

253 RpoS, the sigma factor of enteric bacteria that responds to stress and stationary

254 h postwithdrawal, morphine-treated mice had enteric bacteria that were detected in the Peyer's patch

255 ented internalization of selected strains of enteric bacteria that were preferentially adherent on th

256 In contrast to the putA genes of enteric bacteria, the A. tumefaciens putA gene is not re

257 In enteric bacteria, the cellular response to oxidative str

258 hosphotransferase system of V. furnissii and enteric bacteria, the differences may be important for s

259 In contrast to the rfaF (waaF) gene of enteric bacteria, the H. ducreyi waaF gene was not locat

260 In enteric bacteria, the key player of carbon catabolite re

261 In enteric bacteria, the kinase LsrK catalyzes the phosphor

262 In enteric bacteria, the transcription factor sigma(E) main

263 ellular pH or synthesizing ATP in many other enteric bacteria; therefore, we used degenerate primers

264 Since cyanobacteria predate the enteric bacteria, this procapsid-mediated assembly pathw

265 We found that Paneth cells directly sense enteric bacteria through cell-autonomous MyD88-dependent

266 xtends the family affiliations identified in enteric bacteria to a wide range of other genera.

267 ity, flagellum expression, and resistance of enteric bacteria to acetic acid and bile salts.

268 ecting organisms away from staphylococci and enteric bacteria to Aspergillus species, although staphy

269 obactin (Ent) is produced by many species of enteric bacteria to mediate iron uptake.

270 iderophore enterobactin (Ent) is produced by enteric bacteria to mediate iron uptake.

271 The ability of species of enteric bacteria to recognize and colonize unique niches

272 Enteric bacteria tumble, swim slowly, and are then paral

273 Enteric bacteria use a limited array of macromolecular s

274 Most enteric bacteria use intestinal brushborder glycoconjuga

275 nces in our understanding of how pathogenic, enteric bacteria use quorum sensing to regulate several

276 Recent data indicate that enteric bacteria use several quorum-sensing mechanisms i

277 doubling time and the glutamine pool size in enteric bacteria was also seen in phylogenetically dista

278 V infection and the family-level taxonomy of enteric bacteria was detected.

279 een glutamine pool size and doubling time in enteric bacteria was far less obvious in B. subtilis.

280 The stimulatory material in the enteric bacteria was trypsin sensitive and restricted by

281 a nucleoid-associated DNA-binding protein of enteric bacteria, was discovered 35 years ago and subseq

282 termination at the intrinsic terminators of enteric bacteria, we observed, by using single-molecule

283 appears to be limited to motile, chemotactic enteric bacteria, we propose that CheAs may play an impo

284 leotide sequences of envZ genes from several enteric bacteria were determined.

285 Here, nucleotide substitutions in enteric bacteria were examined, and no difference in mut

286 Individual strains of enteric bacteria were incubated with HT-29 cells for 1 h

287 phore associated with increased virulence of enteric bacteria, were mapped within a pathogenicity isl

288 share homology with chemotaxis proteins from enteric bacteria, which are encoded in the frzA-F putati

289 this constraint is not shared by many other enteric bacteria, which can use either cysteine or methi

290 We show further that like H-NS in enteric bacteria, which functions as a transcriptional s

291 limit proliferation for ceftiofur resistant enteric bacteria while preserving the ability to use thi

292 Dairy herds with enteric bacteria with known low tetracycline susceptibil

293 Genome-wide comparisons between enteric bacteria yield large sets of conserved putative