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1 ecting the gut epithelium, immune system and enteric nervous system.
2 eristalsis is significantly dependent on the enteric nervous system.
3 s show a significant increase in glia in the enteric nervous system.
4 (Hand2(+/-) mice) numbers of neurons in the enteric nervous system.
5 in contrast to the neurodegeneration in the enteric nervous system.
6 ural crest that colonize the gut to form the enteric nervous system.
7 role in the development and survival of the enteric nervous system.
8 eric neurons but also is latent in the human enteric nervous system.
9 es produced by enteroendocrine cells and the enteric nervous system.
10 gulating the development and function of the enteric nervous system.
11 neurite fasciculation necessary to form the enteric nervous system.
12 e most common congenital malformation of the enteric nervous system.
13 pin-releasing factor (CRF) in the guinea pig enteric nervous system.
14 nal development with a partial rescue of the enteric nervous system.
15 ory neurons (EP cells) within the developing enteric nervous system.
16 s of cells to the terminal bowel to form the enteric nervous system.
17 s a major inhibitory neurotransmitter in the enteric nervous system.
18 excitatory actions of CRF on neurons in the enteric nervous system.
19 n many inhibitory neurones of the vertebrate enteric nervous system.
20 ing factor (CRF) receptors in the guinea pig enteric nervous system.
21 pes and glial cells present in the mammalian enteric nervous system.
22 ole of erbB2 in postnatal development of the enteric nervous system.
23 ng neurogenesis in the fully developed adult enteric nervous system.
24 n myenteric and submucosal components of the enteric nervous system.
25 ing fast and slow synaptic excitation in the enteric nervous system.
26 ic and submucosal plexuses of the guinea pig enteric nervous system.
27 gically identified neurons in the guinea pig enteric nervous system.
28 ney organogenesis and the development of the enteric nervous system.
29 ng renal development and histogenesis of the enteric nervous system.
30 for neuronal replacement in disorders of the enteric nervous system.
31 iate into the neurons and glial cells of the enteric nervous system.
32 neurotransmitter roles for NO and CO in the enteric nervous system.
33 l role in the development of the kidneys and enteric nervous system.
34 l dysgenesis and abnormal development of the enteric nervous system.
35 known to be critical for development of the enteric nervous system.
36 cells and cranial ganglia as well as in the enteric nervous system.
37 sary for normal development of the mammalian enteric nervous system.
38 ganglia, and also contributes to the foregut enteric nervous system.
39 ntial for the development of kidneys and the enteric nervous system.
40 odulating cholinergic neural reflexes in the enteric nervous system.
41 inhibits cholinergic neural reflexes in the enteric nervous system.
42 nesyl-prelamin A are toxic to neurons of the enteric nervous system.
43 nd adaptive immunity, and dysfunction of the enteric nervous system.
44 HGF and MET are expressed in fetal and adult enteric nervous system.
45 n between the enteroendocrine system and the enteric nervous system.
46 e along with the drivers of their input, the enteric nervous system.
47 Little is known of how Tat affects the enteric nervous system.
48 ed during HIV infection are regulated by the enteric nervous system.
49 neuronal as well as glial progenitors in the enteric nervous system.
50 role of miRNAs in HFD-induced damage to the enteric nervous system.
51 motility disorders caused by diseases of the enteric nervous system.
52 axonal number in the peripheral, central and enteric nervous systems.
53 hat which transports 5-HT in the central and enteric nervous systems.
54 differentiated neurons of the peripheral and enteric nervous systems.
55 Mas AT is expressed in both the central and enteric nervous systems.
56 rs are localized to central, peripheral, and enteric nervous systems.
59 t accumulation of 5-HITCA in the central and enteric nervous systems, along with data showing the deg
60 appear to be an intrinsic capability of the enteric nervous system and are not related to slow waves
61 inergic receptor subtype is expressed in the enteric nervous system and at intestinal neuromuscular j
62 the autonomic nervous systems, including the enteric nervous system and central autonomic network.
63 ion between these genes is restricted to the enteric nervous system and does not affect renal, coat c
64 ofound alterations in the development of the enteric nervous system and extensive vacuolar changes in
65 pathway of adrenergic differentiation in the enteric nervous system and have defined a transient requ
66 ides a selective mechanism through which the enteric nervous system and innate immune system integrat
69 e involves an enterotoxin, activation of the enteric nervous system and malabsorption, suggesting tha
70 d carbon monoxide - that are produced by the enteric nervous system and share common molecular target
73 singly evident that interactions between the enteric nervous system and the immune system play an imp
74 hindbrain normally gives rise to most of the enteric nervous system and the superior cervical ganglio
75 relationships among cell populations in the enteric nervous system and the sympathetic nervous syste
76 des convey nutrient-regulated signals to the enteric nervous system and to distal organs, acting as c
77 he possible common origin of sympathetic and enteric nervous systems and provides new hope that we ma
79 e sensory, sympathetic, parasympathetic, and enteric nervous systems and the kidneys, as well as for
80 inhibitory nitrergic neural inputs from the enteric nervous system, and (iv) stretch receptors that
81 the immaturity of their intestinal barrier, enteric nervous system, and immune response to pathogens
82 Ralpha) display deficits in the kidneys, the enteric nervous system, and spinal motor and sensory neu
83 ions between the central nervous system, the enteric nervous system, and the gastrointestinal tract.
84 the interactions among the vagus nerve, the enteric nervous system, and the intestinal epithelium ma
85 he genetic control of the development of the enteric nervous system, and the potential role of the in
87 iation of progenitor cells in the developing enteric nervous system are controlled by molecules such
88 tent of the sacral crest contribution to the enteric nervous system are not well established in roden
89 neuronal subtypes during development of the enteric nervous system are poorly understood despite its
90 es a viral enterotoxin and activation of the enteric nervous system, as well as malabsorption, sugges
91 nding and isolating early progenitors of the enteric nervous system based on their ability to form di
92 tatin subtype 2A (sst2A) receptor in the rat enteric nervous system by using a C-terminus-specific, a
93 ardenburg-Shah syndrome combines the reduced enteric nervous system characteristic of Hirschsprung's
94 Vagal neural crest-derived precursors of the enteric nervous system colonize the bowel by descending
95 is sequence also modulates expression in the enteric nervous system consistent with its proposed role
97 ition to ionotropic glutamate receptors, the enteric nervous system contains functional group I metab
103 y recently to discuss the latest research on enteric nervous system development at a meeting organise
104 ng the critical time for EDNRB expression in enteric nervous system development begins after separati
105 sts between GFR alpha1 and GDNF in renal and enteric nervous system development, significant cross-ta
106 To clarify the role of these receptors in enteric nervous system development, the effect of ligand
113 losion of interest in the development of the enteric nervous system driven by the need to understand
114 ation of immunity, changes that occur in the enteric nervous system during inflammation, the fundamen
115 nism by measuring colonization in hosts with enteric nervous system dysfunction due to a mutation in
116 cells (NCC) that ultimately give rise to the enteric nervous system (ENS) along the entire length of
117 Enteric glia are important components of the enteric nervous system (ENS) and also form an extensive
119 uding the gastrointestinal tract to form the enteric nervous system (ENS) and hematopoietic organs (b
120 the hypotheses that OT is endogenous to the enteric nervous system (ENS) and that OTR signaling may
121 ous system (CNS), there is evidence that the enteric nervous system (ENS) and the peripheral nervous
122 The majority of neurones and glia of the enteric nervous system (ENS) are derived from the vagal
124 t signaling is critical for formation of the enteric nervous system (ENS) because Ret activation prom
125 derived progenitors (ENPs) that generate the enteric nervous system (ENS) can lead to aganglionosis i
126 (HSCR) is a severe congenital anomaly of the enteric nervous system (ENS) characterized by functional
130 irschsprung disease is a serious disorder of enteric nervous system (ENS) development caused by the f
134 rify the role of Ret signaling components in enteric nervous system (ENS) development, we evaluated E
141 and A30P lines show robust abnormalities in enteric nervous system (ENS) function and synuclein-immu
143 sodium channel (VGSC) alpha subunits in the enteric nervous system (ENS) has not been described.
147 stinal obstruction due to the absence of the enteric nervous system (ENS) in the distal bowel and is
150 iets, the consumers' gut microbiota, and the enteric nervous system (ENS) interact to regulate gut mo
153 tanding the neurochemical composition of the enteric nervous system (ENS) is critical for elucidating
156 h the idea that phenotypic expression in the enteric nervous system (ENS) is determined, in part, by
161 ronal development in vitro; nevertheless, an enteric nervous system (ENS) is present in mice lacking
164 S: Neural stem and progenitor cells from the enteric nervous system (ENS) might serve as a source of
165 en have aggregated alpha-synuclein (aSyn) in enteric nervous system (ENS) neurons, which may be assoc
166 es in resident and inflammatory cells in the enteric nervous system (ENS) of macaques during the acut
172 ocate calcium (Ca2+) channel proteins in the enteric nervous system (ENS) of the rat and guinea pig.
175 ant oligogenic birth defect that occurs when enteric nervous system (ENS) precursors fail to colonize
177 ral crest-derived epidermal melanoblasts and enteric nervous system (ENS) precursors to completely co
179 tion of the bowel, results from a failure of enteric nervous system (ENS) progenitors to migrate, pro
185 Crohn's disease (CD) and the ability of the enteric nervous system (ENS) to produce PGD2 in inflamma
188 a balanced microbial community and that the enteric nervous system (ENS), a chief regulator of physi
189 estinal obstruction, striking defects in the enteric nervous system (ENS), and abnormal intestinal mo
190 rons are the major excitatory neurons of the enteric nervous system (ENS), and include intrinsic sens
191 function is controlled by its own intrinsic enteric nervous system (ENS), but it is additionally reg
192 of axonal tract configuration in the mature enteric nervous system (ENS), but profound abnormalities
193 ointestinal tract, and more specifically the enteric nervous system (ENS), in search of an early biom
195 the neural crest, impacts development of the enteric nervous system (ENS), possibly by regulating the
197 ium signaling plays an essential role in the enteric nervous system (ENS), the role of CaMKII in neur
199 pathway in the development of the mammalian enteric nervous system (ENS), very little is known regar
200 ointestinal physiology are controlled by the enteric nervous system (ENS), which is composed of neuro
219 ortant in the functional neurobiology of the enteric nervous system (ENS); nevertheless, details for
220 natide reduces food intake and activates the enteric nervous system (ENS; myenteric and submucosal pl
221 ord primary motor neurons (PMN), kidney, and enteric nervous systems (ENS) and have identified areas
222 , have been identified and several mammalian enteric nervous systems express CB1 receptors and produc
224 aluminal virus is able to gain access to the enteric nervous system from the gastrointestinal lumen.
225 effect of this Gaucher mutation on motor and enteric nervous system function in these transgenic anim
226 otential of neural crest stem cells from the enteric nervous system (gut NCSCs) in vivo to evaluate t
229 (EGCs), the major cellular component of the enteric nervous system, have long been considered mere s
234 nteractions can influence development of the enteric nervous system in mouse models and suggests that
239 studies to describe the regeneration of the enteric nervous system in the sea cucumber Holothuria gl
241 e viable and fertile but have defects in the enteric nervous system, including reduced myenteric plex
242 isceral afferents (sensory) pathways and the enteric nervous system, including the interstitial cells
243 progenitor function in the dentate gyrus or enteric nervous system, indicating regional differences
245 ory detection in these processes, disordered enteric nervous system integration in diarrhea and const
246 es reveals that, in the absence of Pax3, the enteric nervous system is ablated from its inception.
247 flecting the current interest in the way the enteric nervous system is altered in disease and the sec
251 ibution of sacral crest-derived cells to the enteric nervous system is not affected by cocolonization
252 the requirement for erbB2 in maintaining the enteric nervous system is not cell autonomous, but rathe
258 eural crest cells fated to contribute to the enteric nervous system migrate ventrally away from the n
262 major changes in the smooth muscle layers or enteric nervous system occurred in the absence of these
264 processes of enteric glial cells within the enteric nervous system of CWD-infected Tg(CerPrP-E) mice
265 ade an important contribution to the ancient enteric nervous system of early jawless vertebrates, a r
267 ons of both brain stem emetic nuclei and the enteric nervous system of the gut; and 4) whether select
273 be selectively ablated by ganciclovir in the enteric nervous system, or in the injured forebrain or s
275 l neural crest to interrupt the migration of enteric nervous system precursor cells and thus create a
276 e expressed very early in the development of enteric nervous system precursors, and are already prese
277 und that control of differentiation of mouse enteric nervous system progenitor cells by EDN3 requires
280 holothurians provide a model system to study enteric nervous system regeneration in deuterostomes.
284 recent advances in our understanding of the enteric nervous system, sensory physiology underlying pa
286 We describe neurochemical coding of the enteric nervous system, specifically the myenteric plexu
287 hin-3 plays a role in the development of the enteric nervous system suggest that trkC and neurotrophi
288 ating GDNF and ET-3 in the patterning of the enteric nervous system, suggest that specific pairing of
289 rity of neurons and glia that constitute the enteric nervous system, the intrinsic innervation of the
291 tite and energy balance by engagement of the enteric nervous system through cannabinoid receptors.
293 terface by demonstrating the capacity of the enteric nervous system to influence the microbiota.
296 year's issue on developmental aspects of the enteric nervous system, we have focused on several key t
298 fore alter innervation and morphology of the enteric nervous system, which may contribute to colonic
299 hesized that Lewy pathology initiates in the enteric nervous system years prior to debut of clinical
300 during the development of the kidney and the enteric nervous system, yet no ligand has been identifie
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